Obesity remains the number one leading factor associated with Type 2 Diabetes. Type 2 Diabetes, previously identified as adult-onset diabetes, affects the way one metabolizes glucose; a crucial energy source for the body. An increase or decrease in insulin production also results from Type 2 Diabetes. Insulin resistance occurs when the body does not use insulin properly. According to the Center for Disease Control, Type 2 Diabetes accounts for ninety to ninety-five percent of diabetic cases; making it the greatest form of diabetes. Research from the Center for Disease Control, indicates Type 2 Diabetes results from a combination of genetic and environmental influences. (CDC, 2014). Obesity, a significant risk factor, enhances the effects of
…show more content…
Extra abdominal fat triggers and worsens insulin resistance. Signals inside the nervous system cause a breakdown of fat cells and a release of stored fat. Insulin promotes the opposite effect, signaling the fat cells to stop the release of fat. Paul Insel, a Stanford University student, states “An increase of body fat that reaches three to five times the normal amount of body fat or greater than thirty billion fat cells with a weight of thirty pounds, fat tissue is unable to be affect by insulin.” (Insel et. al, 2016). The fat cell’s response to the insulin decreases, leading to a high amount of fatty acids in the bloodstream. American Diabetes Association found, the higher levels of fatty acid lead to hyperglycemia, stimulating the production of insulin inside of the pancreas. Hyperglycemia injures the insulin manufacturing cells in the pancreas, making it progressively difficult for low blood glucose levels (ADA, 2014). The bodies, based off of research by Stephen Guyenet, MD, who produce insulin greater than the average insulin levels of 8.8 mcU/ml (microunits per milliliter) for men and 8.4 mcU/ml for women obtain a greater chance of developing insulin resistance (Guyenet, 2013). The decrease levels of glucose keep the cells from getting the proper amount of nutrients. The cells, despite the presence of insulin in the bloodstream, block the glucose from entering the cell. The remaining glucose stay in the bloodstream, resulting in high glucose
Extra abdominal fat triggers and worsens insulin resistance. Signals inside the nervous system cause a breakdown of fat cells and a release of stored fat. Insulin promotes the opposite effect, signaling the fat cells to stop the release of fat. Paul Insel, a Stanford University student, states “An increase of body fat that reaches three to five times the normal amount of body fat or greater than thirty billion fat cells with a weight of thirty pounds, fat tissue is unable to be affect by insulin.” (Insel et. al, 2016). The fat cell’s response to the insulin decreases, leading to a high amount of fatty acids in the bloodstream. American Diabetes Association found, the higher levels of fatty acid lead to hyperglycemia, stimulating the production of insulin inside of the pancreas. Hyperglycemia injures the insulin manufacturing cells in the pancreas, making it progressively difficult for low blood glucose levels (ADA, 2014). The bodies, based off of research by Stephen Guyenet, MD, who produce insulin greater than the average insulin levels of 8.8 mcU/ml (microunits per milliliter) for men and 8.4 mcU/ml for women obtain a greater chance of developing insulin resistance (Guyenet, 2013). The decrease levels of glucose keep the cells from getting the proper amount of nutrients. The cells, despite the presence of insulin in the bloodstream, block the glucose from entering the cell. The remaining glucose stay in the bloodstream, resulting in high glucose