Decompression Sickness

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1) With the increase in aquatic depth, Po2 & Pn2 increase. Increasing Po2 in the atrial blood will provide the necessary oxygenation to other vital organs as well as the brain unless the diver depletes his/her oxygen supplies which results in a state of hypoxia (Caton-Richards. M, 2013). It is also said that Po2 along with increasing depth plays a role with inhibiting the urge respirate thus, allowing the diver to stay under water longer. Pn2 on the other hand activates the urge to respirate. With increasing depths Pn2 will begin to be absorbed into the blood and tissues keeping in mind that with depth, the chest is being compressed simultaneously. Consequences can be the immediate release of Pn2 and Po2 back into the body causing nitrogen narcosis (Encyclopedia, 2014).
2) Decompression Sickness is when the Nitrogen that was stored in the blood and tissues while descending in the water, forms bubbles within the tissues upon the ascending or pressure decrease (Encyclopedia, 2014).
3) Signs and symptoms of decompression sickness are itching of the skin, fatigue, dizziness, confusion, tingling/numbness, tremor, pain in the extremities and joints as well as shortness of breath and possible syncopal episodes (Caton-Richards. M, 2013).
4)
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Fluids will be given to ensure proper hydration. During the high-flow oxygen, the patient will receive 100% oxygen that allows the reversal of the pressure changes that allowed the gas bubbles to form in the tissues and blood stream (Lee, Kent, 2008). This process also aids nitrogen back into the liquid form so that it can be exerted from the body gradually over the period of several hours. Treatment for decompression sickness is not to be done by the patient themselves; this type of treatment will involve emergency help (Mcqueen, Kent, Murrsion,

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