This conditioning causes increase of systolic blood pressure, which increase the systolic workload of the left ventricle and reduce diastolic compliance. As a result, there is increase in oxygen consumption, left ventricular hypotrophy and imbalance in myocardial oxygen supply and demand and a widening in PP (Steppan, Barodka, Berkowitz, & Nyhan, 2011). Vascular stiffness can be measured by pulse wave velocity (PWV), the speed at which pressure waves move along the artery, which is increased with
This conditioning causes increase of systolic blood pressure, which increase the systolic workload of the left ventricle and reduce diastolic compliance. As a result, there is increase in oxygen consumption, left ventricular hypotrophy and imbalance in myocardial oxygen supply and demand and a widening in PP (Steppan, Barodka, Berkowitz, & Nyhan, 2011). Vascular stiffness can be measured by pulse wave velocity (PWV), the speed at which pressure waves move along the artery, which is increased with