Copd Exacerbation Essay

2972 Words Mar 1st, 2012 12 Pages
Case Study: COPD Exacerbation
Jeffrey Sherman
Due Date: January 16, 2012
Wayland Baptist University
Adult Health II

COPD Exacerbation The purpose of this paper is to discuss an exacerbation of Chronic Obstructive Pulmonary Disease (COPD) and its effect on my patient, Mr. HS, a 78 year old male. In this paper we will look at the various facets in the disease process including its incidence, pathophysiology, presenting complaints, analysis of his clinical presentation, and discuss treatment. We will analyze the effect the disease process has on Mr. HS and will examine his clinical manifestations and laboratory work, as well as provide an outcome analysis. Understanding these various facets will enable one to understand
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This causes the airways to become hyperactive, creating constriction and narrowing of the airways excessively. Infiltration of the airways with substances such as polynuclear, mononuclear phagocytes, and CD4 T cells increases with each stage of disease progression. The entire chronic inflammatory process, occurring in the lungs is associated with tissue repair and remodeling that ultimately results in COPD. COPD is seen commonly in patients who smoke. It appears that smoking may overcome the body's natural mechanisms for limiting this immune response. The process may also continue in susceptible individuals even after smoking cessation. In this case, even if the original noxious insults are removed, the continuation of COPD may develop and is characterized by progressive accumulation of cells of the immune system, fibrosis, and mucus hypersecretion. The molecular basis for the lung inflammation seen in COPD is still an area of great research and debate, with the potential roles of cytokines, complex autoimmune processes, and immune modulation from chronic infection all under investigation (Shapiro, 2003). The defining feature of COPD is irreversible airflow limitation during forced expiration. This may be a result of a loss of elastic recoil due to lung tissue destruction or an increase in the resistance of the conducting airways. The standard measure of COPD is the measure of forced expiratory volume in 1 second

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