A1: Acute and Chronic inflammation differ in many ways. For example, acute inflammation as blatant as it is, acute inflammation is short, only lasting from a few minutes to days. On the other hand, chronic inflammation, is longer, lasting around days to even years. Furthermore, acute inflammations deal with fluid and plasma oozing. In addition, to leukocytes leaving to the extravascular tissues, which are mainly habited by neutrophils. Chronic inflammation, additionally, is associated with lymphocytes and macrophages, while having an increase in blood vessels, also, dealing with fibrosis and tissue necrosis. An example of each would be, a sore throat from a cold for acute inflammation and asthma for chronic inflammation.
A2: Platelets are …show more content…
They produce eicosanoid family inflammatory mediators. They then split up to either the lipoxygenase pathway or cyclooxygenase. The lipoxygenase produces leukotrienes while the cyclooxygenase produces prostaglandins and thromboxane. All help in inflammatory responses. Platelet-Activating Factor, or PAF, activates neutrophils and attracts eosinophils, additionally, starting platelet cluster formation. Furthermore, PAF can cause many different things if injected or inhaled, some examples are, wheal-and-flare reaction and bronchospasm.
Plasma Proteins, help in several inflammatory responses. For example, clotting, complement and kinin systems. Clotting triggers inflammation while complement systems destroy other proteins playing an important role in inflammation and immunity. The kinin system creates peptides from kininogens through actions of kallikreins. Thus, activating the release of bradykinin.
A4: Wound healing is split up into three steps. First, the body is protected by covering up the wound and sending your white-blood cells to keep it healthy. Second, the body then try to re-create the skin before the wound. Lastly, leading to the body after a while to produce a small scar or fully heal the
A2: Platelets are …show more content…
They produce eicosanoid family inflammatory mediators. They then split up to either the lipoxygenase pathway or cyclooxygenase. The lipoxygenase produces leukotrienes while the cyclooxygenase produces prostaglandins and thromboxane. All help in inflammatory responses. Platelet-Activating Factor, or PAF, activates neutrophils and attracts eosinophils, additionally, starting platelet cluster formation. Furthermore, PAF can cause many different things if injected or inhaled, some examples are, wheal-and-flare reaction and bronchospasm.
Plasma Proteins, help in several inflammatory responses. For example, clotting, complement and kinin systems. Clotting triggers inflammation while complement systems destroy other proteins playing an important role in inflammation and immunity. The kinin system creates peptides from kininogens through actions of kallikreins. Thus, activating the release of bradykinin.
A4: Wound healing is split up into three steps. First, the body is protected by covering up the wound and sending your white-blood cells to keep it healthy. Second, the body then try to re-create the skin before the wound. Lastly, leading to the body after a while to produce a small scar or fully heal the