Abstract Chronic inflammatory demyelinating polyneuropathy is considered an autoimmune disorder of the peripheral nervous system. It is the chronic form of Guillain-Barre syndrome, the difference between the two diseases is GBS is an acute inflammatory disease of peripheral nerves and most patients recover spontaneously while CIDP requires more attention. The cause of CIDP is unknown it is believed that it may be caused due to the immune response of the body attacking the nerve fibers causing demyelination to take place. Chronic inflammatory demyelination polyneuropathy is not curable but it is treatable, however due to variation of onset of symptoms per individual treatment methods contrast amongst on another producing a trial and error to take place. Due to the nature of the
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Austin went forth and proceeded to evaluate the patient and saw that the patient was reacting well with the steroids administered and was speculated to be CIDP (Albert and Knoefel 1984). In 1975 individuals who were reflecting the same assessments were said to have chronic inflammatory polyradiculoneuropathy, until demyelination was discovered to be distinguished as a primary factor the name was then changed to chronic inflammatory demyelination polyneuropathy (Albert and Knoefel 1984). CIDP can go by various other terms such as chronic relapsing polyneuropathy (CRP) or by chronic inflammatory demyelinating polyradiculoneuropathy due to the involvement of nerve fibers (Kissel 2003). Common onsets of symptoms are around 30’s to 60’s but symptoms may arise at any age (Dalakas 2011). The cause of the disease is still unknown but it is believe that it is the body’s immune response that plays a role in the disease thus causing the body to deteriorate the myelin sheath on neurons (Lewis 2013). The nature of the disease includes these symptoms but not limited to numbing
Austin went forth and proceeded to evaluate the patient and saw that the patient was reacting well with the steroids administered and was speculated to be CIDP (Albert and Knoefel 1984). In 1975 individuals who were reflecting the same assessments were said to have chronic inflammatory polyradiculoneuropathy, until demyelination was discovered to be distinguished as a primary factor the name was then changed to chronic inflammatory demyelination polyneuropathy (Albert and Knoefel 1984). CIDP can go by various other terms such as chronic relapsing polyneuropathy (CRP) or by chronic inflammatory demyelinating polyradiculoneuropathy due to the involvement of nerve fibers (Kissel 2003). Common onsets of symptoms are around 30’s to 60’s but symptoms may arise at any age (Dalakas 2011). The cause of the disease is still unknown but it is believe that it is the body’s immune response that plays a role in the disease thus causing the body to deteriorate the myelin sheath on neurons (Lewis 2013). The nature of the disease includes these symptoms but not limited to numbing