Case Study Susie

Currently, Susie is not a physically fit individual. Even though she has not had any major cardiac events in her life, she does pose a risk for cardiac events due to her current health state. Her family history includes her father having a heart attack at the age of 62. Susie's resting heart rate is within the normal limit. Her systolic blood pressure is in the range of hypertension crisis since it is 182. However, her diastolic blood pressure is 94 which is below the range of hypertension crisis. Therefore, she is in stage two hypertension. Her BMI is in the overweight range, close to obesity. Her cholesterol is at a normal level. Susie's blood glucose level is also within the normal range, but closer to the end of the normal spectrum. Based on the stratification assessment, I deem that Susie is a low risk for exercise. She has not had any major cardiac events occur in the past. She has exercised in the past, but not frequently. Susie has not complained or mentioned any signs or symptoms during or after physical activity that …show more content…
High blood pressure increases mean arterial pressure, which could damage arteries and lead to further complications to organs if left untreated. The renin-angiotensin-aldosterone system (RAAS) regulates blood pressure and is stimulated in response to hypotension. The kidneys release renin into the bloodstream that combines with angiotensinogen, which is released from the liver. This process produces angiotensin I. Angiotensin I combines with Angiotensin-converting enzyme (ACE) to produce angiotensin II. Angiotensin II causes vasoconstriction, which increases systemic vascular resistance and reduces arterial compliance. The hormones vasopressin and aldosterone are also released due to angiotensin II. Vasopressin stimulates water retention and aldosterone stimulates sodium retention. This increases blood volume, along with vasoconstriction, which then increases blood