Final Exam
Bacteria’s gene is only coding (makes a protein) meaning one gene equals one protein. Bacteria’s life history is a short lifespan, reproduces quickly in large number of individuals. Griffith finds that bacterial cells can be transformed. (Raven) There are two forms of bacteria, one that causes pneumonia and a nonvirulent that does not. The nonvirulent lacks enzymes to manufacture the coat of polysaccharide. Griffith performed a series of experiments with these types of bacteria on mice. Some mice died and some lived. In the end polysaccharide coat was necessary for virulence. If the virulent bacteria S formed in the first heat-killed then the infection doesn’t harm the experiment, because the coat itself was not sufficient that lead …show more content…
So there is definitely and qualitative and quantitative in size difference. Eukaryotes have a nucleus membrane bound that bacteria doesn’t. So the difference in general between bacteria and eukaryotes is that genetic information is formed in protein and is converted into organization, function and cellular structure. Eukaryotes also have a replication of machinery that is found in bacteria but are more complex and larger. The phase of replication has more factors for helicase and primase complexes, it then loads onto a sliding clamp unit where polymerase is there as …show more content…
Together these three are accomplished transcription in the nucleus of eukaryotic cells. Polymerase I showed no similarities in the coding region. Polymerase II have the most complex of the three type of polymerase, reflection is a huge diversity of genes and are transcribed by the polymerase. The first eukaryotic gene was isolated and had many sequences called (TATA box) from the start site. Prokaryotic was similar to ten sequences and the TATA was the primary promoter to the element. The sequencing of the genomes have many more genes that have been analyzed and assumptions have been provided too simple. Polymerase III was that the promoters were found to actually be internal to the gene itself. This has not been proven to be the case for all three polymerase, but it does appear to be for the most part.
Bacteria’s gene is only coding (makes a protein) meaning one gene equals one protein. Bacteria’s life history is a short lifespan, reproduces quickly in large number of individuals. Griffith finds that bacterial cells can be transformed. (Raven) There are two forms of bacteria, one that causes pneumonia and a nonvirulent that does not. The nonvirulent lacks enzymes to manufacture the coat of polysaccharide. Griffith performed a series of experiments with these types of bacteria on mice. Some mice died and some lived. In the end polysaccharide coat was necessary for virulence. If the virulent bacteria S formed in the first heat-killed then the infection doesn’t harm the experiment, because the coat itself was not sufficient that lead …show more content…
So there is definitely and qualitative and quantitative in size difference. Eukaryotes have a nucleus membrane bound that bacteria doesn’t. So the difference in general between bacteria and eukaryotes is that genetic information is formed in protein and is converted into organization, function and cellular structure. Eukaryotes also have a replication of machinery that is found in bacteria but are more complex and larger. The phase of replication has more factors for helicase and primase complexes, it then loads onto a sliding clamp unit where polymerase is there as …show more content…
Together these three are accomplished transcription in the nucleus of eukaryotic cells. Polymerase I showed no similarities in the coding region. Polymerase II have the most complex of the three type of polymerase, reflection is a huge diversity of genes and are transcribed by the polymerase. The first eukaryotic gene was isolated and had many sequences called (TATA box) from the start site. Prokaryotic was similar to ten sequences and the TATA was the primary promoter to the element. The sequencing of the genomes have many more genes that have been analyzed and assumptions have been provided too simple. Polymerase III was that the promoters were found to actually be internal to the gene itself. This has not been proven to be the case for all three polymerase, but it does appear to be for the most part.