The structure most likely injured with the direct blow of the tibial tuberosity resulting in presentation of the lower limb (figure 1) would be the posterior cruciate ligament (PCL). The presented picture shows the tibia being pushed back posteriorly. The PCL, within the knee joint, prevents posterior translation of the tibia bone related to the femur bone (Moore et., al, 2014). If the PCL is torn, then the tibia can move backward too much leading to instability of the knee. The knee joint serves as a hinge joint that connects the femur to the tibia allowing for flexion, extension, and minimal internal/external rotation. The tibial tuberosity also serves as the attachment for the patellar ligament, which attaches to the patella from
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Anterior compartment syndrome results from an initial injury that was compromising vascular components leading to cell death due to hypoxia increasing intra-compartmental pressure (Moore et. al, 2014). Increased intra-compartmental pressure will be evident due to the vasculature being compromised with will further lead to cell damage, muscle necrosis, and maybe lead to permanent disability, amputation of the limb, or even death. Some signs this individual might suspect if this occurs are edema, pain, loss of motor function, or loss of sensation in the affected lower …show more content…
Palpating for this artery in the foot and finding a pulse will re-assure that the PCL injury did not affect the vasculature structure. The dorsalis pedis artery is a continuation of the anterior tibial artery. A common sign that there is a vasculature injury due to the PCL injury would be palpating the dorsal pedal pulse and not finding a pulse and/or cyanosis of the foot of the affected leg. Another area to palpate for normal blood flow would be within the popliteal fossa for the popliteal artery because of the tibia being displaced posteriorly. The posterior tibial artery could be palpated posterior to the medial malleolus for a sign of whether or not the injury affected this
Anterior compartment syndrome results from an initial injury that was compromising vascular components leading to cell death due to hypoxia increasing intra-compartmental pressure (Moore et. al, 2014). Increased intra-compartmental pressure will be evident due to the vasculature being compromised with will further lead to cell damage, muscle necrosis, and maybe lead to permanent disability, amputation of the limb, or even death. Some signs this individual might suspect if this occurs are edema, pain, loss of motor function, or loss of sensation in the affected lower …show more content…
Palpating for this artery in the foot and finding a pulse will re-assure that the PCL injury did not affect the vasculature structure. The dorsalis pedis artery is a continuation of the anterior tibial artery. A common sign that there is a vasculature injury due to the PCL injury would be palpating the dorsal pedal pulse and not finding a pulse and/or cyanosis of the foot of the affected leg. Another area to palpate for normal blood flow would be within the popliteal fossa for the popliteal artery because of the tibia being displaced posteriorly. The posterior tibial artery could be palpated posterior to the medial malleolus for a sign of whether or not the injury affected this