Adiadochokinesia Essay

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Our sensorimotor has many parts to function and manipulate what we want our body part to do. We simply must think and react to what are next move and in rapid millisecond either our hand or leg moves. As this occurs, our primary somatosensory cortex that’s located in our central sulcus has a full chart of information lined up for our function to travel all the way to our spinal cord and make a movement.
As Agnosia damages the path of any function of either of the temporal lobe or cerebellum, it creates a rare disorder that may damage the ability of the signals of movement to malfunction. A personage won’t know if we have a damage function. As doctor’s witness damage brain function with all kinds of structural technology then realized all
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(Bodranghien, F.,2016) As this agnosia occurs, a patient will have an abnormality of a certain way of how he/she will handle is feature. Is oblivion until you test the patient, you would see how they would have a movement of either the left hand and the right hand will try to mimic and would not have the same rhythm as what it should normally do. We find this function that starts on the primary motor cortex that travels to the cerebellum, that takes part of the motor control and takes it down on the brain stem that sends spinal activity to the spinal cord on to the muscle that results to the movement or abnormal movement. (Watson, N. V.,2012) Cerebellum is the most important motor learning. As this has said with damage part of the cerebellum, decreases the motor control on any part that cause to learn a movement or just simply affects the movement. With a damage cerebellum disease travels to the nerves to cause either slowing or improper movement to where the path of the damage cervical nerve. Parkinson’s and Huntington’s disease both have a similar but different affect to adiadochokinesia, as I observed and read (Watson, N.V.,2012) they both affect the muscular movement and mentally and

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