Unfortunately there has not been a specific test, or even a trial period, for definitive diagnosis of ALS. The only way to diagnose a series of physical neurological examinations and tests are to test them in many ways, which are actually intended to reject the assumption of a person with similar illnesses. There are many other illnesses that have symptoms like Alzheimer's disease (Steinberg et al. 2012). To diagnose ALS, a neurologist performs an electromyogram test. This test is used to detect neural damage. It is also possible to test the patient in order to rule out the possibility of developing diseases such as muscular dystrophy, MS, tumors and other similar illnesses.There are tests that came be performed to diagnose ALS. These tests include; extensive
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The defining characteristic of ALS is the death of the upper and lower motor neurons in the brain, brain stem, and spinal cord of the brain. Prior to their destruction, motor neurons develop protein components in their cellular bodies and axons. These components are often combined with and combining one of the proteins associated with ALS: SOD1, TDP-43, or FUS. These components are non-amyloid aggregates. This disease has been found to contradict plaque and protein accumulation. The TDP-43 protein produced by this genetic anomaly is toxic. Patients with the disease have been found to have abnormal TDP proteins in dying neurons, but it is believed that this structure can be created by damaged cells that attempt to repair themselves. The oxygen uptake system consists of superoxide dismutase enzymes, catalase and glutathione peroxidase. In this collection, SOD is a key enzyme, because it is involved in the first phase of the removal of free radicals. Three ISO-enzymes have been identified, including SOD, cytosolic (Cu / Zn SOD), mitochondrial SOD (Mn-SOD) and extracellular SOD
The defining characteristic of ALS is the death of the upper and lower motor neurons in the brain, brain stem, and spinal cord of the brain. Prior to their destruction, motor neurons develop protein components in their cellular bodies and axons. These components are often combined with and combining one of the proteins associated with ALS: SOD1, TDP-43, or FUS. These components are non-amyloid aggregates. This disease has been found to contradict plaque and protein accumulation. The TDP-43 protein produced by this genetic anomaly is toxic. Patients with the disease have been found to have abnormal TDP proteins in dying neurons, but it is believed that this structure can be created by damaged cells that attempt to repair themselves. The oxygen uptake system consists of superoxide dismutase enzymes, catalase and glutathione peroxidase. In this collection, SOD is a key enzyme, because it is involved in the first phase of the removal of free radicals. Three ISO-enzymes have been identified, including SOD, cytosolic (Cu / Zn SOD), mitochondrial SOD (Mn-SOD) and extracellular SOD