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44 Cards in this Set

  • Front
  • Back
P normally pos in
I,II, V4-6
P normally neg
AVR
p wave duration
.08-.11
` do PACs look different than normal p waves
yes
WAP and MAT must have at least how many different pacemakers?
3
do PACs change the rate?
yes
T waves should always appear the same in any give lead; if you have a T wave that looks different, it is probably because
a p wave is buried inside
are PACs usually compensatory?
no
What causes inverted p waves?
low atrial or AV nodal (retrograde conduction of the atria)
P mitrale
P> .12 and notched, left atrial enlargement
P pulmonale
P is peaked more than 2.5mm, right atrial enlargement
When the first half of the P wave in V1 is taller than the first half of the P wave in V6, think
right atrial enlargment
When the second half of the P wave is wider and deeper than 1 small block, think
left atrial enlargement
length of pr interval
.11 to .2
what does the pr interval include
atrial depolarization, atrial repolarization, AV node stim, his stim, BBB stim and purkinje stim
where should the pr interval be measured
widest p to the beginning of the widest qrs
if pr interval is long in all leads, think
meatabolic problem (maybe hyperkalemia)
causes of short pr intervals
1. junctional p waves
2. PACs
3. LGL or WGW
Wolff-Parkinson White syndrome
young individuals, atria to ventricles through the Kent bundle without AV node, could pass out, circus rhythm
Lown Ganong Levine Syndrome
short pr, no tachy, benign bypass around AV, no signs of WPW
signs of WPW**
know this slide
1. short ps interval
2. wide qrs
3. delta wave
4. St-t wave changes
5.associated with paroxysmal tachy (Syndrome)
first 4=pattern
delta wave
two impulses meet each other, WPW, initial slurring of the qrs, 3 types with A most common; negative delta wave is a pseudoinfarct
AV node that has a long refractory period until it drops a beat
Wenkebach
more dangerous mobitz II blocks
infranodal or infrahisian
Accrochage
p waves and qrs go marching along together but independent; third degree heart block
significant Qs
.03 s or wider and depth equal to or greater than 1/3 of the R wave
A negative wave after an R wave must be a
S wave
ST-T help diagnose a
acute MI
Q waves are more indicative of
previous MI damage
MI Q signs
significant size and in a minimum of 2 continuous leads
length of normal qrs**
Know this
.06 to .11
axis -30 to 105, downward and to the left
qrs deflection transition zone
between V3 and V4
increase in muscle mass does what to the size of qrs
larger
qrs size after MI and decrease in ventricular muscle mass and effusions
smaller
why dose qrs voltage look smaller if you have COPD or are over weight
leads further away from the heart
pericardial effusions
limb leads less than 5 mm (QRS)
precordial leads less than 10 mm
left ventricular hypertrophy
increased muscle mass causes increased action potential, increased vector with resultant increased EKG amplitude
EKG criteria for LVH
S in V1 or V2 added to R of V5 or V6> 34mm *
Any precordial lead > 45mm
R wave in aVL is >10mm
R wave in lead I is > 11mm
R in AVF > than 19mm
cant call LVH if what is present?
LBBB present
RVH
look at V1 and V2: R waves >S wave; cant make diagnosis with RBBB; could be due to WPW or posterior wall myocardial infarction
if tachy >100 with wide qrs, think
v tach until proven otherwise
3 possibilities for >.11 QRS
RBBB, LBBB or hyperkalemia
significant Q waves
dead muscle, must be in more than 1 lead, >1/3 of height and >.03 sec
osborn wave
aka J wave, hypothermia, elevated notch after qrs, often associated with bradycardia