• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/168

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

168 Cards in this Set

  • Front
  • Back
Cardiac Diseases with NO murmur
DCM
HCM
HWD
Pericardial Dzs
Some Congenital Dzs
Chronic Cough DDX (2)
LHF
Pulmonary Disease (Collapsing Trachea, Bronchitis, HWD, Asthma)

PL
Acute Cough DDX (7)
LHF
Tonsillitis
Pharyngitis
Tracheobronchitis
Acute bronchitis
Pleuritis
Pneumonia

P3T2LA
Types of cough (8)
Loud, harsh, coarse dry - larynx, trachea, or bronchi

Cardiac - harsh and low

Chronic Non-productive - tumor

Rattling with whistling - Bronchiectasis

Honking - Collapsing trachea

After drinking - CT, Cardiac dzs, chronic tracheitis tracheobronchitis

With an inciting factor - Cardiac/pulmonary dzs extrapulmonary disorder, neoplasia

At eating - achalasia or vascular ring anomalies, esophageal diverticula
How to tell upper airway cough from lower
rattling, wheeze - lower
Difference between dyspnea and tachypnea
Tachypnea = increased rate
Dyspnea = rate and effort
Types of Dyspnea
Paroxysmal (comes and goes)
Exertional (early sign of Dyspnea)
Orthopneic (when lying down)
Inspiratory vs. Expiratory Dyspnea
Increased Inspiratory effort = upper airway
Increased Expiratory = lower
Causes of hemoptysis
Pulmonary contusions from trauma
Pulmonary edema
Lung/bronchi diseases
Pulmonary embolism
Clotting problems
DIC
Cause of head, neck, forelimb edema
mediastinal mass obstructing venous return
Causes of abnormal jugular pulses
RHF w/tricuspid regurg
Heart block or arrhythmia where right atrium contracts agains a closed tricuspid valve
PSten
Pulm hypertension
Jugular distension caused by
RHF (congestive)
Obstruction
Pulmonary Dzs
PMI displacement caused by
Cardiac enlargement
Mass pushing on heart
Collapsed lung lobe on R side
Just got up from right lat recumbency
Decreased intensity of heart sounds caused by
pleural/pericardial effusion
thoracic masses
Hernia with liver/intestines in thorax
Obesity
Arrhythmias
Causes of Hyperkinetic pulse (3)
PDA
Aortic regurg
Left ventricular hypertrophy
Causes of weak pulses (6)
myocardial disease
arrhythmia
Pericardial dzs
dehydration
ASten
Systemic Hypertension
Normal HR for Dog
Dog - 70-180
Valve areas for ascultation
Left side
Pulmonic 2-4 ICS, sternal
Aortic 4th ICS, costal-chondral junction
Mitral 5th ICS, costal-chondral junction

Right side
Tricuspid 3-5 costal-chondral junction
S1 Heart Sounds and causes
Blood surging to the AV valves to start AV closure; abrupt deceleration of blood as valves close
S2 Heart Sounds and causes
closure of semilunar valves
S1 splitting caused by
delayed closure of mitral or tricuspid valve from RBBB, VP beats
S2 splitting caused by
delayed closure of the pulmonic valve from

Pulmonic Hypertension (HWD)
RBBB
VP Beats from Left Ventricle
ASD
PSten
RHF
COPD (horses)
S3 heart sound caused by
Rapid Ventricular filling
S4 heart sound caused by
Atrial contraction of blood into ventricles
Types of murmurs
1. Functional - increased velocity
2. Pathologic - structural problem
Types of Pathologic murmurs
1. Stenosis (aortic, pulmonic)
2. Shunt (VSD, ASD)
3. Regurg (PAMT)
Most common type of murmur
Systolic
These valves should be open during systole
Aortic & Pulmonic
Causes of systolic murmurs
flow obstruction (aortic/pulmonic)
Leaky valves (mitral/tricuspid)
Holes (ASD/VSD)
Causes of continuous murmurs
PDA
Arteriovenous connections
Ateriopulmonary windows
flow disturbance (pulmonary emboli)
Significance of systolic clicks
Idiopathic. Leads to mitral valve problems.
99% of murmurs are ____.
Systolic
Endocardiosis is defined as _____ and is found two places (____ & _____ (with____ being the most common).
Chronic Valvular Disease; Mitral; Tricuspid; Mitral
Systolic murmur over the pulmonic region
psten
T of F
ASD
Systolic murmur over the aortic region
Subaortic Stenosis
Systolic murmur over tricuspid region
Tric. regurg
VSD
Tric. dysplasia
Continuous murmurs
PDA
AV Fistula
Diastolic Murmurs
Aortic Regurg
Pulm Regurg
Mitral Sten
Tric. Sten
4 most important things to do on a cardiac PE
Signalment
Complaint/Clinical History
PE/Ascult
Rads
Common breeds with PSten
Bassett
Beagle
Boxer
Boykin
Chihuahua
Chow
Cocker
English Bulldog
Lab
Mastiff
Newfie
Samoyed
Schnauzer
Terrier (All)
Westie
Common breeds with PDA
Bichon
Chihuahua
Cocker
Collie
Springer
GSD
Keeshond
Lab
Maltese
Pom
Poodle
Shetland
Corgi
Yorkie
Common breeds with SAS
Boxer
GSD
GSP
Golden
Great Dane
Newfie
Rottie
Samoyed
Common breeds with ASD
Boxer
Dobie
Samoyed
Common breeds with MVD (mitral valve dysplasia)
Bull Terrier
GSD
Golden
Dane
Mastiff
Newfie
Common breeds with CTD (Cor Toratatum Dexter)
Chow
Common breeds with AS
Bull Terrier
Common breeds with T of F
English Bulldog
Keeshond
Common breeds with VSD
English Bulldog
Springer Spaniel
Common breeds with TVD
GSD
Golden
Dane
Lab
Tx for Acute heart failure Cat
Lasix
O2
+/- vasodilation (nitroglycerine)
Tx for Chronic HF Cat
Lasix - NOTHING ELSE EFFECTIVE.
Enalapril MAY affect RAAS, but no evidence up to this point.
Normal Hr for Cat
Cat 145-220
Normal HR for cattle
Cattle 60 - 80
Normal HR for Horse (and foal)
Horse 30-40 (80 in foals)
Normal HR for sheep/goats
Sheep/goats 70-90
Normal HR for pig
Pig 60-100
S3 and S4 normal in what species?
Cow and horse at rest

Always abnormal in small animals
Respiratory abnormality found on both inspiration and expiration
Pleural Friction rubs
P wave reflects what?
Atrial depolarization
PR interval reflects what?
time for AV node conduction
P wave with NO QRS
AV node blockage
QRS reflects what?
Ventricular depolarization
T wave reflects what?
Ventricular repolarization
How many seconds should you count for HR?
3 sec. = 1 bic pen

50mm/sec = multiply by 20

25mm/sec = multiply by 10
Characteristics of a Sinus Rhythm
No sudden starts/stops
Positive P wave in 1, 2, 3, and aVF
Normal QRS
Can't sustain high rates for long
Places non-sinus rhythyms can originate
Atrium (has a P wave, but might look weird
AV Node (abnormal PR)
Ventricle (VPCs)
Describe the Atrial Premature Contraction
Funny P wave
Normal QRS
Describe the AV node/Junctional rhythm
+/- P wave
Weird PR interval
Describe the propagation of a normal heartbeat
Starts at the Upper right atrium at the sinus node

Upper RA>Lower LA>Cross AV node>Upper R AV node>Lower LV
Where is the normal Mean electrical axis (MEA)
LL quadrant
What happens with a Right Axis Shift?
MEA moves to Right side
What happens with a Left Axis Shift?
MEA moves to the Upper Left Quadrant
Which bundle branch feeds the upper L part of the LV?
Anterior. Posterior gets everything else.
What to expect in a RBBB?
Right axis shift
Wide and bizarre QRS
What to expect in a LBBB?
Normal MEA
Wide and bizarre QRS
RBBB makes you worry about?
Psten
tricuspid displasia
tricuspid regurg
RV CM
LBBB makes you worry about what?
SAS
PDA
Mitral sten
Mitral regurg
DCM
3rd heart sounds come from?
Big Atrium
Big Ventricle
Systolic Click (non-pathologic)
Gallop
Premature
Why don't you usually have a murmur with ASD?
Pressure gradient NSF
Holosystolic/Pansystolic murmur causes
Mitral regurg
Tricuspid regurg
VSD
Mid-systolic murmur causes
Asten
Psten
Tachycardia
pleural effusion
pericardial effusion
anemia
fever
Most common Diastolic murmurs
Aortic regurg
Mitral regurg
All causes of systolic murmurs
Obstruction to outflow (aortic and pulmonic)
Leaky valves (mitral and tricuspid regurg)
Holes (VSD and ASD)
Causes of diastolic murmurs
Leaky valves (aortic and pulmonic)
Stiff valves (mitral and tricuspid stenosis) - rare
Causes of continuous murmurs
PDA
Pulmonary emboli
True/False: APCs and VPCs are often associated with underlying structural heart disease
True
Tx for tachycardias
AADs
AADs targeting muscle (VeAt)
NCB
KCB
AADs targeting nodal cells (AV/Sinus)
B
C
D
Tx for Sinus tachycardia
Treat underlying problem

B-blockers
Tx for Atach
Target muscle
NBCs
KCBs
Bbs

Slow conduction
Bbs
CCBs
Dig
Names of NCBs
Procainamide
Lidocaine (No FX in atrium)
Mexiletine
Quinidine
Names of KCBs
Amiodarone
Sotalol
Names of Bbs
Esmolol
Propanolol
Altenolol
Tx for JTach
B
C
D
Tx for VTach
Go on a hunt for extra cardiac dzs

NCBs
KCBs
Bbs
MgSO4 (horses only)
Tx for Afib
Rhythm control - Cardiovert
NCBs
KCBs
Electrical

Rate control - slow conduction through AV
Bbs
CCBs
Digoxin
Names of CCBs
Diltiazem
About Quinidine
Class Ia NCB - most commonly used AAD for cardioversion

DO NOT USE IN SA! DO NOT GIVE ORALLY!

Toxic levels cause:
UR Stridor
Wide QRS
Ataxia

But NOT:
Tach
Hypotension
Diarrhea
Colic
About Digoxin
Direct Vagal FX - decreases Vent response in Afib

+ inotrope
Class I AADs
NCBs

1A - Quinidine, Procainamide

1B - Lidocaine, Mexilitine
Class 2 AADs
B-blockers

Selective - Altenolol, Esmolol

Non-selective - Propanolol
Class 3 AADs
KCBs

Sotalol, Amiodarone
Class 4 AADs
CCBs

Diltiazem, Digoxin
NCBs good for
Vent arrhythmias
Atach
Bbs good for
Stach
Jtach
Afib rate
KCBs good for
Vent arrythmias
Atach
CCBs good for
Afib rate
Atach
Jtach
About lidocaine
doesn't do anything in Atrial tissue

hypoK makes less effective

Have to bolus then CRI
About Mexiletine
Good for Atach, VtArr

Good for use with Altenolol or sotalol
About Amiodarone
Lots of Side FX (hepatic toxicity, erythema, pruritus, swelling, hives, pain at injxn site)

Pretreat w/Benadryl or steroids

Great at VtArr, Atach
About Diltiazem + Digoxin
great to slow Vt response to Afib
Tx for AV Block and SSS
Pacemaker or + chronotropes
Names of + Chronotropes
Theophylline
Terbutaline
Hycosamine
Define HF
When the heart can no longer maintain normal function resulting in:

low arterial BP
NSF perfusion of tissues at rest
Increased Venous BP
Increased Capillary pressures
Define CO
CO=HR*[(Contractility*Preload)/Afterload]
Name the six major principles of cardiac performance
1. Preload - amount of blood that goes back into the heart
2. Afterload - what the LV works against
3. Contractility - can the heart contract?
4. Compliance - can the heart fill?
5. HR
6. Synergy - how well does it all come together?
Types of Heart failure
1. Congestive
2. Low Output
L CHF cascade
LV Pressure>LA Pressure>Pulmonary pressure>increased oncotic pressure>pulmonary edema>decreased O2 exchange in lungs
Causes of increased L Vt Diastolic Pressure
1. increased preload (leaking mitral due to endocarditis)
2. decreased compliance (HCM)
3. Increased after load (ASten, hypertension)
4. Combination
Signs of L CHF
edema
dyspnea
R CHF cascade
RV Pressure>RA Pressure>Central Venous pressure (jugular distension)>Systemic VP>Systemic Capillary Pressure>Hepatic Sinusoid pressure>Ascites (dogs)/pleural effusion (cats)/ ventral (horses) or brisket (cows) edema
Causes of increased R Vt Diastolic pressure
Increased RV Preload
Increased Afterload (plum art hypertension)
Decreased compliance
Combination
Low output failure is a result of what?
Big, flabby heart. NSF blood pumped.

Symptoms: hypotension, cool extremities, depression, lethargy
Pressure overload is a systolic dysfunction and a cause of heart failure. Name some causes.
SAS, PSten, ToF, Hypertension, HWD, L to R Shunt, pheochromocytoma
Volume overload/excessive preload is a systolic dysfunction and a cause of heart failure Name some causes.
M, T, or A Regurg
Shunts (PDA, VSD, ASD)
AV Fistula
Pump/low output failure (NSF contractility) is a systolic dysfunction and a cause of heart failure. Name some causes.
Cardiomyopathy
Infarct
Cardiodepressive drugs (- Inotropes)
Myocardial restriction is a diastolic dysfunction and a cause of heart failure. Name some causes.
HCM
Restrictive CM
Pericardial Effusion
Pericarditis
Tumors
High output states can cause HF. Name the causes.
Chronic anemia
Chronic fever
Hyperthyroidism
Differentiate between the stages of HF
Mild - Coughing, dyspnea, and fatigue with exercise
Moderate - with mild activity
Severe - at rest
Draw the RAAS
How does the SNS attempt to combat HF?
1. HR - speeds up conduction and discharge - at a certain point become detrimental because decr filling.
2. Contractility - E! and NE! released by SNS - incr contractility, but sensitizes the heart to arrhythmias
3. Systemic Art BP - causes vasoconstriction via A-!, but incr afterload
Eccentric Hypertrophy caused by what and lays more sarcomeres down how?
Chronic volume overload; end-to-end

Internal dimensions increased
Concentric hypertrophy caused by what and lays more sarcomeres down how?
Chronic pressure overload; parallel

Internal dimensions decreased>less compliant ventricle
What's the deal with ANF
Comes from Atrial myocytes and is released when At is stretched

Increases GFR
Antagonizes Renin, Ald. release, AT2 vasoc.
Inhibits ADH
Vasodilates
What are the cardiac peptides?
ANF
Troponin
Vasopressin
What's the deal with vasopressin?
Increases with CHF
Retains water
Causes vasoc
What's up with Troponin?
Most commonly measured Cardiac peptide
Elevated in renal dz, PTE with dogs/cats
Somewhat high in boxers with ARVC
How can you detect CHF in dogs/cats?
1. Aldosterone ^ x 7
2. Renin ^ x 10
3. NE ^
4. ANF ^ if Class 3-4 CHF
5. NT-proBNP - hi Sn/Sp
What's up with NT-proBNP?
NOT a screening test.
Can tell you if it's cardiac and how bad.
Hi Sn/Sp
Poor prognosis if ^ AND ALT ^
Canine LHF CS
Rales
^ CRT
Weak femoral pulses
Pacing
+/- murmur
Canine RV failure CS
+/- murmur
Ascites
Hepatomegaly
Splenomegaly
Distended jugular
Cardiac cachexia
Feline Vent Failure
dyspnea
murmur
R - pleural effusion
L - pulm edema
Equine/Cow LHF
^ HR
^ RR
+/-Murmur/gallop
Equine RHF
^ HR
Murmur
Legs swell
Brisket edema
Cow RHF
Brisket edema
^ HR
+/- murmur
Goals of HF Tx?
1. V fluid accumulation
2. ^ CO
3. V workload
4. Control rhythm
5. Find cause
6. Neurohormonal (RAAS, SAS, ADH)
7. Cardiac Remodeling
ACEI do what to RAAS?
Prevent AT1>AT2

V preload
V afterload
Bb do what to RAAS?
V renin release from JG cells
Bbs do what to the SNS?
V contractility
Aas do what to the SNS?
Vasodilate
V afterload
How can you prevent Cardiac remodeling?
ACEI - V fibrosis, V afterload, V preload
Aldant - V fibrosis
Bbs - V remodel
Lasix - V preload
Name an Aldant.
Spironolactone
Why/How reduce preload?
v preload = v venous congestion

Lasix
ACEI
V sodium
Venodilators
Why/How reduce after load?
v afterload = v workload

ACEI
Art.dilators
Why/how alter contractility with systolic dysfunction?
Perfusion

+inotrope (pimo, dig, catecholamine)
Why/how alter Compliance with diastolic dysfunction?
Improve filling time by v HR

CCB
LHF vs. RHF TX?
LHF - Lasix, cage rest

RHF - tap it.
Name the arterial vasodilators.
Hydralazine
Nitro
ACEI
How to slow HR?
Bb, CCB, Dig
Why use + Inotropes and how do they work?
Big Flabby Heart - ^ systolic contractility

alter Ca+ at actin/myosin level
Name + inotropes
Dig
Pimo
Catecholamines (emergencies)
Name the catecholamines
Epi!
Isoproterenol
Dopamine
Dobutamine
Name the types of diuretic
Xanthines
Thiazides
Aldant
Lasix
Things that ^ blood digoxin levels
Quinidine
Aspirin
Amiodarone
Sotalol
Spironolactone
Cimetidine
Phenobarb
Hypothyroid
CRF/ARF
Hypokalemia
When to NOT use dig
Sinus node dysfunction
HCM
A/P Sten
RCM
Pericardial dzs
Vt Arr
A murmur
Pulm Hypertension
When to use Isoproterenol
SSS
3rd Degree AV Block

DO NOT USE unless you're getting ready to put in a pacemaker
When to use Dopamine
Sinus Tach