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7 Cards in this Set
- Front
- Back
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Substrates for VT/VF
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Usually in someone who has underlying structural heart disease.
Most common= Acute ischemia, Dilated Cardiomyopathy Then you have= Metabolic (hyperkalemia, hypomagnesemia, hypoxia), medications (anti-arrhythmics) |
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Phases of VT During MI
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Early= first 48hrs, small percent have VT w/in 48hrs MI, not really a big deal if treated
Late= after 48hrs, probably because of reentry, a lot worse prognosis since you are already treated and it must be a bad problem if it didn't have to do with the MI itself |
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Torsade de Pointes
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Long QT results in polymorphic VT
Causes of Long QT= congenital (K or Na channel): Lange-Nielsen also get deafness Acqd= Erythromycin, hyperkalemia, meth, antihistamines, Class 1A/III anti-arrhythmics Treatment- Magnesium Sulfate***, increase HR to shorten the QT, use 1B (lidocaine, mefexitine, tocainamide) |
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R on T
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Depolarization happening at bad time of repolarization, so some cells will depolarize and some wont= not synchronized and you can get commotio cordis (R on T from trauma to the chest)
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ICD Indications
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Making tissue refractory to reset it and have the SA node take control again.
Use for: -cardiac arrest due to VT/VF -spontaneous, sustained VT -syncope of undetermined origin with significant sustained VT/VF -ischemic or dilated CM with EF<35% |
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Outflow Tract VT
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See in normal heart, no structural defect; triggered/automatic and results in monomorphic VT (vs. polymorphic= long QT).
-More common in younger, you can just ablate. Vs. in structural heart disease (CM ischemic and non-ischemic) you should have ICD |
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Arrhythmogenic RV Dysplasia
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Fibrofatty replacement of RV, common cause of SCD in rare population; young age
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