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35 Cards in this Set
- Front
- Back
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THE SOURCE OF EPO CAN BE FOUND IN WHAT ORGAN
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KIDNEY
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VITAMIN D IS ACTIVATED IN WHAT ORGAN
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KIDNEY
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3 COMPONENTS OF URINE FORMATION
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FILTRATION
REABSORPTION SECRETION |
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NORMAL URINE DOES NOT CONTAIN WHAT?
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RBC AND LARGE PROTEINS.
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% OF THE BLOOD AT ANY GIVEN TIME THAT IS BEING FILTERED BY THE KIDNEY
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2%
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SHOCK WILL GENERALLY INCREASE OR DECREASE THE GFR DUE TO POOLILNG AT THE EXTREMITIES
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DECREASE
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RENIN SECRETED WILL
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ACTIVATE NA+/K+ PUMP. NA+ AND WATER REABSORBED, K+ IS SECRETED
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ANP IS MADE WHEN WHAT AREA IS OVERSTRETCHED?
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ATRIA
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BNP IS CREATED WHEN WHAT PART OF THE HEART IS OVERWORKED
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VENTRICLES
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ANEMIA AND WEAK BONES ARE COMING FOR SOMEONE WITH COMPRIMISED KIDNEYS DUE TO
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INABILITY TO CREATE EPO AND ACTIVATE VITAMIN D
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AS LONG AS THIS PERCENT OF YOUR KIDNEY'S ARE FUNCTIONING, IT IS CONSIDERED NORMAL
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50%
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BUN IS SHORT FOR
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BLOOD UREA NITROGEN
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KIDNEY FUNCTION EQUALS
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NORMAL SERUM CREATININE/CURRENT SERUM CREATININE
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BUN CAN INCREASE DUE TO DIFFERENT FACTORS SUCH AS;
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HIGH PROTEIN DIET
PRERENAL FAILURE 1. DEHYDRATION 2. CHF POST RENAL FAILURE OBSTRUCTIVE UROPATHY |
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DIURETICS, LITHIUM, AND CORTICOSTEROIDS WILL INCREASE OR DECREASE BUN
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INCREASE
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LIVER DISEASE, POOR NUTRITION, OR OVERHYDRATION MAY INCREASE OR DECREASE BUN
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DECREASE
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RENAL DISEASE
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INCREASES BUN
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SINCE BUN CAN BE AFFECTED BY THINGS OTHER THAN JUST RENAL FAILURE, WHAT OTHER MEASUREMENT IS A GOOD INDICATION OF RENAL FUNCTION?
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SERUM CREATININE
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URINE IN THE BLOOD IS CALLED
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UREMIA
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DURING RENAL FAILURE YOU WILL SEE AN INCREASE IN WHAT LAB VALUES?
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SERUM CREATININE
BUN |
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tOO MUCH NITROGEN IN THE BLOOD
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AZOTEMIA
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WITH CHRONIC RENAL FAILURE YOU CAN EXPECT TO SEE WHAT OTHER DISORDERS?
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HTN
CHF BONEDISORDERS |
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EDEMA LEADS TO PULMONARY EDEMA WHICH LEADS TO
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DYSPNEA
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MICOALBUMINUREA
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ALBUMIN IN THE URINE
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HYPOALBUMINEMIA
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LOW ALBUMIN IN THE BLOOD
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PYELONEPHRITIS
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BACTERIAL INFECTION THAT COMES FROM A DOWNSTREAM AREA SUCH AS THE BLADDER
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LESS EPO WILL CREATE
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ANEMIA
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LOWER BLOOD VISCOSITY WILL ALLOW BLOOD TO FLOW MORE SWIFTLY WHICH WILL LEAD TO
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HEART RATE INCREASE WHICH INCREASES HEART LOAD AND LEFT HYPERTROPY
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CHRONIC RENAL FAILURE HAS INTERMITTENT OR PERMANENT LOSS OF NEPHRONS
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PERMANENT LOSS OF NEPHRONS
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DIMINISHED RESERVE
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~50% LOSS
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RENAL INSUFFICIENCY 20-50% OF NORMAL, WILL CAUSE
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AZOTEMIA, ANEMIA, HYPERTENSION, POLYURIA
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RENAL FAILURE <20% OF NORMAL
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EDEMA, ACIDOSIS, HYPERKALEMIA
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ANURIC
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NO URINE
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PHASES OF ACUTE RENAL FAILURE
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1. ANURIC
2. DIURETIC 3. RECOVERY |
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TUBULOINTERSTITIAL DISORDERS INCLUDE
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1. ACUTE TUBULAR NECROSIS - CELL DEATH
2. PYELONEPHRITIS -INFLAMMATION OF KIDNEY 3. DRUG-RELATED NEPHROPATHIES |
