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50 Cards in this Set

  • Front
  • Back
Supraoptic nucleus
Paraventricular nucleus
Lateral
Suprachiasmastic
Ventromedial
Septal
Anterior
Posterior
All parts of the hypothalamus
Supraoptic - ADH
Paraventricular - oxytocin
Lateral - Hunger, inhibited by leptin
Ventromedial - satiety
Suprachiasmatic - circadian rhythm
Septal - sexuality
Anterior - Cooling, parasympathetic
Posterior - Heating, sympathetic
MPTP linked to what diseaese?
Parkinsons
Myoclonus
Sudden, brief muscle contraction
Dystonia
Sustained, involuntary muscle contraction
Damage to the reticular activating system
Coma
Reduced level of arousal and wakefulness
Damage to subthalamic nucleus
Contralateral hemiballismus
Damage to hippocampus
Cannot form new memories
Damage to PPRF
Lose ability to make ipsilateral horizontal eye movements
Look away from lesion
Damage to FEF
Look towards lesion
muscle that is spared in recurrent laryngeal damage
Cricothyroid
Connects Brocas to Wernickes
Arcuate fasiculus
Poor repetition
Follows commands
Fluid speech
Conduction aphasia
Poor repetition
Nonfluent speech
Follows commands
Broca
"Worst headache of my life"
SAH
Blood supply to the internal capsule
Lenticulostriate arteris from MCA
Common sites in the brain for HTN injury
Basal ganglia
Thalmaus
Pons
Cerebellum
How strokes will appear on imaging
MRI - 3-30 min to appear, and last for 10 days. appear white on diffusion weighted

CT - appear dark, take 24 hrs to appear
Sx of anteior spinal artery (medial medulary syndrome)
Hypoglossal paralysis (toung deviate to ipsilateral side)
Contralateral hemiparesis
Contralatearl proprioception
Wallenberg syndrome
Lateral medullary
PICA

Nystagmus
Contralateral loss of pain/temp body
Ipsilateral loss pain/temp face
Nystagmus, diplopia, vomiting, nausea
Ipsilateral Horners
Ipsilateral ataxia
Hoarsness
Vertigo
Diploplia
Artery infarcted leading to Homonomous hemianopa w/ macular sparing
PCA infarct
What is the cause of Charcot-Bouchard microaneurysms
Chronic HTN
Foramen of Magendie
Connects 4th ventrical (medially located)
Foramen of monro
connects lateral to 3rd vetnricles
Sx of Normal pressure hydrocephalus
Urinary incontinence
Wide gait ataxia
Dementia
Cells that are destroyed in MS
Oligodendrocytes
Use IFN-beta to treat
MS
Romberg test
What does it test and what do results mean
Used for ataxia
If posivite - then a dorsal column (sensory) problem
If negative - then a cerebellar defect
Respond to pressure and vibration
pacinian corpuscles
Clinical reflex roots
Biceps - C5,C6
Brachioradialis - C5-C6
Triceps, - C7, C8
Achilies - S1, S2
Patellar - L3, L4
Rooting reflex
Stroke side of cheek and head moves lookin for nipple
Superior colliculi
Vision
Conjugate vertical gaze center
Inferior colliculi
Hearing
Parinaud syndrome
Paralysis of conjugate vertical gaze b/c of superior colliculi lesion
Taste to anterior 2/3 of tongue
VII
Taste to posterior 1/3 of tongue
Glossopharyngeal
Innervates parotid gland
Glossopharyngeal
Innervates sublinguial gland
Facial
Corneal reflex
Afferant and Efferent
Afferent V1 (nasociliary branch)
Efferent VII - temporal branch
Vagal nuclei
Nucleus ambiguious (motor)
Nucleus solitarius (sensory)
Dorsal motor nucleus --< autonomics to heart, gut, lungs
CN XI lesion symptoms
Cannot move head to contralateral side + shoulder drop
How do opiods work?
How is tolerance formed?
What drug can you use to prevent this?
Inc K+ out of cell and Dec Ca in; this decreases the release of neurotramsitters: 5-HT, NE, Substance P, ACh, Glu
Tolerance is formed by:
Inc NO, phosphorylation of opioid receptors, and increasing adenylate cyclase action

Things like glutamate increase NO and phosphorylation by activating NMDA receptors. So give an NMDA receptor blocker like Ketamine
Gram positive microbe with tumbling motility
Listeria
Allosteric activator of carbamoyl phosphate synthetase I
N-acetylglutamate
Conduction deafness in left ear; what would W & R say
BC>AC on left
Weber lateralize to left
Sensineural deafness in left ear; what woudl W & R say
AC>BC on left
Weber lateralize to the right
How ciliary muscles work?
What are they innervated by?
When they contract, they cause relaxation of the pupil and it gets wider. this helps w/ near sight
They are stimulated by M3 receptors for accomodation
Where is aqueous humor produced from?
Stimualted by what?
Ciliary proccess --> B receptors
What controls the size of the iris?
PS --> M3 --> miosis --> Constrictor/circular/sphincter

Symp --> alpha 1 --> mydriasis --> radial muscle
Innervates stapedius
Facial
Nerve for lacrimation
Facial