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24 Cards in this Set

  • Front
  • Back
trauma and burns
renal disease
acute tubular necrosis
crush injury with rhabdomyolosis: myoglobin release
renal vascular disease
ultimate result is decreased renal blood flow
renal hypertension (renin-angiotensin system)
renal hypertension
renin and angiotensinogen
angiotensin I
angiotensin II
1.peripheral vasoconstriction
2. aldosterone secretion
Na+, H2O increased retension
plasma volume increase
**increase BP
reflux from urinary bladder
-cystic (urinary bladder infection)
-obstruction (urinary calculus, enlarged prostate)
-neurogenic bladder
-hematogenous spread
females>males (shorter urethra)
high fever and chills
urine contains WBC, bacteria
dialysis dementia
-speech impaired
acute glomerulonephritis (bright's disease)
inflammation of the glomeruli
-approximately 70% follows beta hemolytic streptococcus pharyngitis (APSGN)
acute glomerulonephritis
type III immue reaction
antigen antibody complexes
circulate to glomerulus and become mechanically trapped
injury/ inflammation result
acute glomerulonephritis
inflammation of the glomerulis
-disease occurs mostly in children (>50%)
-affects boys twice as much as girls
acute glomerulonephrtisi
50% of adults with glomerulonephritis completely recover
those who don't recover may proceed to a progressive of chronic stage
acute glomerulonephritis S&S
HTN (50%)
proteinuria (albumen)
coke colored urine (RBCs and Casts)
HA, N, & V (electrolyte imbalance)
oliguria, polyuria
tam-horsfall protein
24 hour trace protein
normal finding
rapid progressive glomerulonephritis
small percentage follow APSGN
reapidly progressive- fatal in about 2 years
glomerular subendothelial immune complexes
fibrin accumulation in the glomeruli
r,p,g : good pastures syndrome
diffuse glomerular loss
pulmonary alveolar lesions (hemorrhage, hemoptysis)
chronic glomerulonephritis
small percentage follow APSGN
slowlsy progressive- renal failure and uremic syndrome "end stage renal disease"
Diabetes Nephropathy
25-30% of renal failure
progressive disease
thickening of glomerular basement membrane (glomerulosclerosis)
diffuse diabetic (most common)
nodular diabetic (kimmelstiel- wilson)
end stage renal disease
metabolic acidosis-confusion, lethary
electrolyte imbalance (heart dysrhythmia, nausea & vomiting, diarrhea, muscle cramps)
-Na+ vs K+ inverse renal relationship (K+ is removed, Na+ is kept, visaversa)
end stage renal disease
renal osteodystrophy - decreased vit D (made by kidney)
azotemia retnetion of nitrogenous metabolic waste products (urea, creatine, uric acid; elevated BUN (bllod urea nitrogen), gout)
end staeg renal disease
peritoneal dialysis
renal transplant
physical rehabilitation
acute renal failure
an abrupt, frequently reversible decline in renal fxn, with recovery expected in about 6=8 weeks
acute renal failure common causes
acute tubular necrosis
acute obstruction
nephrotoxin consumption
acute interstitial nephritis
pigments (hemoglobin 2ndary to disseminated intravascular coagulation (DIC), myoglobin 2ndary to trauma and crush injuries
chronic renal failure common causes
chronic pyelonephritis
chronic hypertensive vascular disease
collagen vascular disease
polycystic kidney disease
renal tubular acidosis
diabetes mellitus
obstructions (prostate enlargment, neurogenic bladder)
renal diagnosis procedures
blood urea nitrogen (BUN) 15-25 mg%
blood creatine (<1.5)
urine sticks