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43 Cards in this Set
- Front
- Back
this links cognition with sex drive, sexual awareness, mood-emotion, and memory
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interactive limbic pathways
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what are the four primary sites of sexual differentiation
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gonads
internal ducts external genitalia brain |
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gonad differentiation
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occurs at 6-9 weeks
common primordia under genetic control male begins by 6 weeks in absence of Y chormosome ovaries develop at 7-9 weeks |
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function of the sex determining region on the Y chromosome (SRY gene)
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causes testis to develop
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internal genital ducts
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diff. occurs at 8-12 wks
separate sets of primordia which transiently coexist in embryos of both sexes |
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maleness requires positive influences of
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testosterone
mullerian inhibiting substance absence of these yields mullerian (female) ducts |
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external genitalia
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diff. at 8-12 wks
common primordia undergo continuous process maleness requires + effect of DHT alone when androgen effect is intermediate, newborn shows partial virilization diff. is virtually complete by 16th week |
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masculinization will never occur if T is not present in
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the first 12 weeks of gestation
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genetic females with excess androgens may have
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complete phallic urethra and prostatic tissue
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what enzyme is needed for this reaction to occur T --> DHT
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5alpha-reductase
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Bipotential hypothalamic neurons in absence of androgens
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GnRH pulsing
LH surge |
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bipotential hypothalamic neurons in presence of androgens
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no GnRH pulsing or LH surge
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LH surge can be elicited in
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women and homosexual men, but not in heterosexual men
surge in women is much larger than gay men |
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psychosexual diff.
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occurs at 8-12 wks and postnatal
it appears that androgen presence during sexual diff. favors but does not determine development of masculine sexuality and the androgen absence during sexual diff. favors but does not determine development of feminine sexuality |
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these factors influence how the prenatal input is expressed in adults
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genetic predisposition
postnatal experiences gender role and gender ID |
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adrenogenital women, despite being corrected in infancy show these tendencies
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increased bisexual and homosexual fantasies
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chornically elevated cortisol (stress) does what to the male fetus
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supresses testosterone
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sexual ambiguity is most commonly due to
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incomplete external genitalia development at birth
external genitalia complete but opposite of genetic sex examples are adrenogenital syndrome and androgen insensitivity syndrome |
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#1 rule for successful sexuality development in cases of ambiguity
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do not send ambiguous signals to child
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hormonal control of libido in males
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androgens from testes increase libido
E or P in large amts can inhibit libido |
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hormonal control of libido in females
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androgen from adrenal (&ovary) increase libido
hormones from ovary may alter libido across the menstrual cycle E or P in large amts can inhibit libido |
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androgen increase and libido
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increases intensity of sex drive but does not change direction of interest
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androgen deprivation and libido
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results in incomplete loss of libido
this suggests behavioral conditioning component of libido |
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pheromones
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airborne molecules which act via the olfactory system to influence drives and behaviors
express a physiological/behavioral state to the recipient that yields a response from the recipient |
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most likely reception pw for pheromones in mammals is via
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vomeronasal organ in nasal septa mucosa
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pheromones are involved in
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social recognition
territorial marking location mapping sexual attraction |
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pheromone chemistry and effects
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volatile steroids but no T, E, or P action
present in axillary sweat glands subtle emotion/mood effects can advance and retard ovulation short-chain aliphatic acids that are present in vaginal secretions |
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pheromones are copulins that
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stimulate male libido
have highest production around ovulation |
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sexuality from a physio perspective
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hormonally-determined process of sexual differentiation of the repro system and brain contributes to development of sexuality
hormonal environment in adolescence and adulthood contributes both to development and expression of sexuality pheromones can enhance sexual attraction and expression of behavior |
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sexuality and non-physiological factors
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there may be genetic predisposition for sexual orientation
social and cultural norms and values affect attitudes and expression of sexuality species-survival drives imbedded in social and cultural patterns |
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events of sexual response can be grouped into 3 physio responses
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vasocongestion
myotonia rapid release of A and B due to orgasm |
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4 stages of sexual response pattern
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excitement
plateau orgasm resolution |
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variation in human sexual response
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great variation in psychological contributions
little variation in physio events |
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excitement, plateau, and resolution stages show little sex differences
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homologous structures respond similarly
female response patterns may be more varied and are commonly more gradual |
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frequency of orgasm
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refractory period more common in men
multiple orgasms more common in women orgasm/intercourse ration greater in men |
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sexual stimuli
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senses
memory mood context |
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sexual response cycle operates via
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intergration of CNS and genito-pelvic events in a repetitive, summating positive feedback loop that leads to orgasm
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this occurs when orgasmic threshold is reached in CNS
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massive motor nerve output with focus in genito-pelvic (perineal) region
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orgasm
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repeated, rhythmic contractions of perineal musculature in response to motor nerve activity described above
CNS cortico-sensory experience of this |
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testosterone optimizes sensitivity of
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CNS substrate regulating libido
CNS components of the summing cycle leading to orgasm peripheral tissues involved in the sacral reflex |
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CNS and peripheral events during orgasm
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while normally integrated, are not mutually dependent
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disorders of sexual function
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physical basis- 10-15%
psychological basis- 85-90% |
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% of patients that have some sexual matter they would like to discuss with their doctor
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50%, but <10% actually do so
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