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95 Cards in this Set

  • Front
  • Back
What is the approach to aquired heart disease on a plain film
What is the first thing to do decide in aquired heart disease
if the heart is big or normal
What are the 2 sign post of aquired heart disease
left atrial enlargement
enlarged aortic arch
If a pt has a small/normal heart and has left atrial enlargement what is the differential
mitral stenosis
reduced LV compliance
What are 3 causes of left ventricular compliance
restrictive CM
hypertrophic CM
constrictive pericarditis
If a pt has restrictive CM, hypetrophic CM, or constrictive pericarditis they have a normal/small heart with LAE
yes
What is the patient has a small/normal heart and has a large aorta
aortic stenosis
What is the ddx for a normal sized heart with disease
Acute MI
restrictive CM
hypertrophic CM
contrstrictive CM
Can restrictive CM, hypertrophic CM and Constrictive pericarditis have either a enlarged left atrium or normal
yes
What are 2 classic plain film findings of mitral stenosis
LAE
enlarged PA
What do you expect to see in a pt with aortic stenosis
enlarged aorta and normal sized heart
What is the MC cause of acute pulmonary edema with normal heart size
acute MI
What are the two sign post for big heart disease
LAE
Aortic regurtitation
What is the only cause of LAE in a big heart disease
mitral regurgitation
What is the only cause of AoE in a big heart disease
aortic regurgitation
What should you suspect if there is big heart disease without LAE or AoE
idiopathic dilated CM
Ischemic CM
tricuspid regurgitation
RV failure
Pericardial effusion
What do you suspect if there is left atrial enlargement in a big heart
mitral regurgitation
What do you suspect if there is AoE in a big heart
aortic regurgitation
What is the differential of a markedly enlarged heart
tricuspid regurgitation (wall to wall heart)
large pericardial effusion
Why is differentiation of restrictive and constrictive pericarditis important
difference in managment (pericarditis you just take the pericardium off)
Where do you typically see pericardial calcifications in constrictive pericarditis
pericardial grooves
What are the 4 MC causes of constrictive pericarditis
cardiac surgery
radiation therapy
viral
TB
What are 5 diagnostic features of constrictive pericarditis
pericardial thickness greater than 4mm
septal bounce
RA enlargement
No RV enlargement
Dilated IVC
What is septal bounce
this is bounce of the septum towards the left ventricle upon opening of the tricuspid valve
What are the 2 most common causes of constrictive pericarditis
surgery
radiation
How do you determine constrictive Vs restrictive cardiac physiology
cardiac catheterization
Which way does the septum bounce in restrictive/constrictive heart diseasee
towards the left ventricle
What is the appearance of an acute hematoma on MRI
bright
What is the appearance of a subacute hematoma
heterogeneous
What is the appearance of a chronic hematoma on MRI T1
(fix last two its for T1)
Homogenous intermediate
What is the characteristic area you may see a pericardial cyst
right cardiophrenic angle
What does a pericardial cyst look like
homogenous low density lesion on T1 and bright on T2 (no enhancement)
What are two patterns of delayed enhancement of ischemic heart disease
subendocardial
transmural
What are the 2 patterns of non-ischemic cardiomyopathy
sub-epicardial
mid wall
What pattern of delayed enhancement would be expected in a patient with myocarditis
midwall
What would you expect to see 2 months later in a patient who had myocarditis
resolution of delayed enhancement
How do you differentiate between ischemic dilated cardiomyopathy and non ischemic dilated CM
look for delayed patterns of enhancement
What are some causes of non-ischemic dilated CM
6
HTN
alcohol
toxins
obesity
DM
idopathic
What is the MCC of dilated CM
ischemic
What are the findings of Non-ischemic dilated CM
LV enlargement
decreased systolic function
normal wall thickness
delayed enhancement (Not always but if they do it will most likely be a non-ischemic pattern but it can be ischemic)
What are the patterns of delayed enhancemet for non-ischemic dilated CM
59% None
28% midwall
13% ischemic pattern
When do you make diagnosis of HOCM
hypertrophy with no cause for it
What percent of HOCM have asymmetric septal hypertrophy
90%
Why is MR better than echo for looking at the potential HOCM pts
because you can measure complete LV mass
What ratio is diagnostic of asymmetric HOCM
septal/lateral: >1.5
Do you see delay enhancement in HOCM
yes (80% of cases) in the diseased portions
What do you see in HOCM
the anterior leaflet of the mitral valve will not close during systole and therefore you will never see those valves close together
What type of dysfunction occurs in restrictive CM; diastolic or systolic
diastolic
can pt with restrictive CM have systolic dysfunction too
yes
What chambers tend to be enlarged restrictive CM
RA and LA
Are the ventricles small in restrictive CM
yes with wall thickening
What are the findings of restrictive CM
RA and LAE
small ventricles
wall thickening
What type of CM is caused by amyloid
restrictive CM
What should you suspect if there is global subendocardial delayed enhancement
amyloid (will not respect coronary artery territories)
What percent of cases of amyloid will have global delayed hyperenhancement
70%
Are both amyloid and sarcoid types of restrictive CM
yes
What percent of pts with pulmonary sarcoid will have cardiac sarcoid
11%
Where is hyperenchancement typical seen in sarcoid
anterolateral and anteroseptal regions
What pattern of delayed enhancement is seen in sarcoid
midwall
What does ARVD stand for
arrhythomgenic right ventricular dysplasia
What ventricle is effected by ARVD
the right ventricle
What is the clinical SS of ARVD
syncope/sudden death during exercise
What do you see on EKG as a result of ARVD
recurrent VT or PVCs with a RV origin
What is the pathophysiology of ARVD
fatty or fibrous degeneration of RV
What are the MR finding of ARVD
5
increased signal on T1
wall thinning
aneurysm
RV enlargement
Regional or global contraction abnormalities
What is the T1 MR characteristic of ARVD
bright (fatty)
What is the role of MRI for cardiac mass evaluations
extent and location
tumor Vs thrombus
primary Vs secondary
benign Vs malignant
What are 4 descriptive terms for location of the tumor with regards to the heart
intracavitary
itnramural
pericardial
paracardial
What is the MC cardiac mass
thrombus
What is the appearance of a thrombus on Cine MR
dark
Does a thrombus enhance
no
What are 2 characteristics of a thrombus in the heart
dark on cine
no enhancment
What is the appearance of tumor on cine
intermediate
Does a tumor enhance
yes
what are two characterictis of a tumor on MR
intermediate on MR
contrast enhancement
What is the only exception of a intermediate tumor on MR
myxoma
Does a myxoma have a stalk
yes
What is the appearance of a mxyoma on cine
brighter than muscle
T or F; secondary tumors are 40 x more frequent than primary tumors of the heart
true
What are the 3 MC secondary tumors of the heart
breast
lymphoma
melanoma
What are the 2 most common primary benign tumors of the heart
myxoma
lipoma
What is a common primary malignant tumor of the heart
angiosarcoma
What are malignant tumor characteristics of the heart
6
wide point of attachment
necrosis or cavitation
involvment of > 1 chamber
pericardial effusion
pulmon mets
extension beyond the heart
Is angiosarcoma a primary malignancy of the heart
yes
Is a wide point of attachement to the heart a sign of a malignant tumor
yes
What is the primary technique for evaluation of valvular disease
echo
What are the qualitative ways to evaluate the heart
Cardiac CT and Cine MRI
What is the best way to quantitatively evaluate the heart
cine MRI (velocity encoded)
Note Cine MRI is good for qualtitative also
What is quantitative cine MR used to evaluate
the amount of regurgitation
What is the mercedes benz sign associated with
inabiltiy of the aortic leaflets to open....aortic stenosis
If there is alot of calcifications does it significantly limit echocardiography
yes
What is a fish mouth aortic valve characteristic of
a bicuspid aortic valve
Can you estimate the pressure gradient across a stenosis with velocity encoded cine
yes (by the bernoulli equation and the peak velocity)
What annular aortic ectasia (widening of the sinus of valsalva) associtated with
aortic regurgitation
What do you expect to see in the flow curve in aortic regurgitation on a flow diagram
postive flow during systole
negative flow during diastole