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157 Cards in this Set

  • Front
  • Back
Treatment for Mental Retardation in 1850 with facilities focused on what?
skills training
fostered caring
warm environment
What's AAMR?
helping to promote research, intervention, and social policy
Eugenics movements
(Eugenics movement) Eugenics is "the study of, or belief in, the possibility of improving the qualities of the human species or a human population by such means as discouraging reproduction by persons having genetic defects or presumed to have inheritable undesirable traits (negative eugenics)

The plan of the eugenics movement was that since the poor had these genes for feeblemindedness, which led them to misery, vice, and crime, the obvious solution to American social problems was to sterilize them, and restrict the immigration of more poor.

killing children with severe disabilities
sterilization laws
prohibit the unfit to have children by sterilizing them
the three main criteria of MR
limitations in

intellectual functioning
adaptive behavior
present before 18 years old
significantly sub-average intellectual functioning of IQ
70 or below
concurrent deficits in MR for adaptive functioning, at least 2 of the following areas
communication -- can they talk
self-care -- can they talk care of themselves?
home living -- do they live by themselves?
social/interpersonal skills -- can they socialize?
use of community resources -- can they use the post office?
self-direction -- can they make decisions?
functional academic skills -- can they attend school?
work -- can they hold a job?
leisure -- can they enjoy life?
health -- do they know where to visit the doctor?
safety -- self-injurious?
mild MR
50-70
moderate MR
30-50
severe
20-40
profound
under 20
for normal people, IQ high stability at
age 4
for MR
IQs are highly stable from early in life to adulthood
least stable at mild MR
but most stable with IQs under 50
Down Syndrome
act like 7-11 years old
early intervention programs boost IQs by
10-15 points
Fragile X MR
have steady IQs until puberty
prevalence
1-2%, below 3%
20-40% more males
MR, due to sex links
MR more common among
low SES and minorities,

especially African Americans
no differences in levels of
severe or profound MR across SES
similar-sequence approach
said that retarded and non-retarded chldren pass through same sequence, but slower

strong evidence
similar-structures approach
said that retarded children have the same "organization of intelligence as non-retarded children

support for familial retardation but organic MR do show numerous deficits
faimilial retardation
we don't know the exact cause

maybe due to polygenic, environmentally deprived, or undetected organic conditions

most prevalence in mild MR
organic retardation
we can point to the exact biological cause

maybe due to prenatal, perinatal, postnatal cause

such as down syndrome, fragile x, williams syndrome, prader-willi syndrome

most prevalence in moderate, severe, & profound MR
double ABCX model
X is the crisis of raising child with MR

A is the function of child's characteristics

B is the family's resources

C is the family's perceptions
Down syndrome
moderate MR

slowing rate of development

have social strengths

but weaknesses in grammar and speech
fragile x
long narrow face

prominent ears, jaws, and forehead

high arched palate, flat feet, hyper-extensible joints, enlarged testicles

moderate MR

more males than females

strength in Gestalt reasoning

weakness in sequential processing

austistic and ADHD like behaviors
williams syndrome
small upturned nose

long philtrum

wide mouth

full lips

small chin

puffiness around the eyes

weak health:

heart and blood vessel problems
feeding problems

usually have good verbal and language skills
prader-willi syndrome
low muscle tone

short stature

incomplete sexual development

chronic feeling of hunger

life-threatening obesity

V-shaped uppper lip

small hands

and fat
hyperphagia
An abnormal appetite for food.
fragile x show high rates of
hyperactive

attention

speech

and autistic-type problems
bleuler "autism"
schizophrenics who had lost touch with reality
kanner saw
language deficits

echolalia

pronounce reversal

social interaction problems
asperger saw
many of the same problems but without language deficits
looking at faces
birth
following person's gaze &

turning when name called
6-9 months
showing objects to others &

pointing at interesting objects

pointing to request
9-12 months
symbolic play
14 months
for autistics
looking at faces
following person's gaze &

turning when name called
following person's gaze &

turning when name called
following person's gaze &

turning when name called

12 months and up
autistics delays in
social interaction

communication

symbolic/imaginative play
Rett disorder

childhood disintegrative disorder
very rare
social interaction
impairments in:

eye-to-eye gaze

facial expression

body postures

failure to develop peer relationships

lack of seeking to share enjoyment, interests, or achievements
communications
impairments in:

delay in spoken language

impairment in initiate or sustain a conversation with others

repetitive use of language

lack of make-believe play or social imitative play

pronoun reversal
repetitive patterns of behaviors, interests, and actitivies
intensely talk about one subject

stick to specific routines or rituals, like lining up shoes

repetitive motor mannerisms, like rocking, finger flapping,

persistently preoccupy with parts of objects, like looking at wheels or shinny objects
deficits in social abilitites because
failure to understand and respond to social information
show very
secure attachment to family members
don't know how to
imitative others
impairment in
joint attention
don't focus on upcoming
stimuli

don't care about those

act robotlike
facial perception impairment
don't focus on the eyes
what's the earliest sign of autism?
symbolic play
how many % do not develop language?
50% do not speak
what's deviant in language?
if develop

echolalia

abnormal prosody

pronoun reversal
mind-blindness
can't put yourself in others' shoes
semantic language impairment
grammar
syntax
language comprehension is very concrete and literal -- no abstract thinking
express frustration through what?
self-injury

like hand banging, slapping, hair pulling
sleep disturbances
little to 4 hours of sleep per day
eating disturbances
very picky with foods
abnormal fears of everyday objects
mostly related to sensory response, sounds, etc.

clown, etc.
high pain tolerances
don't feel pain, don't get to live very long
symptoms can be seen at
12 months (1 year)

but at least 24 months for diagnosis

average diagnosis is at 4 years
high co-morbidity with
MR (40-69%)
asperger's disorder
intact intellectual and language functioning compared to autism


appropriate but unsual intense interests

motor clumsiness

more object-focused than people-focused
what can resemble autism?
developmental language disorders

childhood-onset schizophrenia
very poor prognosis
75% will not live independently

even with early interventions
signs for better prognosis
high IQs

communication skills
male to female ratio
4:1
females tend to have
lower intellectual functioning and more severe symptoms
what's crucial for better prognosis?
early detection & intervention
effective programs should focus on
attention

motor imitation

communication

appropriate toy use

social skills


high structured environments with low student to staff ratio

high levels of family involvememt
ASB in children and adolescents can fall into two categories, such as
conduct disorder

oppositional defiant disorder
delinquency in
children
criminal act in
adults
externalizing behaviors
acting out
internalizing behaviors
acting in
ASB are in the
externalizing, disruptive, acting out arena
ODD & CD in children
ASPD for adults
developmental perspective examines what?
examines development of callous/unemotional traits in childhood, and how it relates to traits of psychopathy in adults
verbal vs. physical
physical -- emerges early, peak during preschool years,

verbal shows later onset
instrumental
goal-directed

bully someone for money, target the money not the person
hostile
inflicting pain is the goal

target the person but the objects
proactive
bullying
reactive
retaliatory
direct vs. indirect
indirect seen more in females
overt vs. covert
covert = lying, stealing, destroying property
began in
DSM III

revise in DSM III-R, oppositional disorder was renamed ODD
what's ODD?
recurrent pattern of negative, hostile, and disobedient behavior towards authority fitures
leads to impairment in functioining
don't follow direction, don't do hw
A. four of the following for at least 6 months
often loses temper

often argues with adults

actively defies or refuses to comply with adults' requests or rules

annoy others

blames others for their mitakes

touchy

angery

spiteful and vindictive
B. cause impairment in


C. not part of psychotic or mood disorder

D. not met for CD or ASPD
social, academic, or occupational functioning
What's CD?
the basic rights of others or major age-appropriate societal norms or rules are violated
four main categories of symptoms
threaten physical harms

property damage

deceitfulness or theft

serious violations of rules
A. have to have 3 or more in the past 12 months, with at least one in last 6 months

B. cause significant in social, academic, and occupational setting

C. criteria not met for ASPD
.
1. aggression to people and animals
bullies, threatens, intimates others

initiate physical fights

use weapon

physically cruel to people

animals

steal something and confront that person

force someone into sexual activity
2. destruction of property
fire setting with the intention of causing serious damge

destroyed others' property
3. deceitfulness or theft
has broken into someone's else house

lies to obtain goods or favors

has stolen items
4. violations of rules
stays out at night despite parental prohibitions

run away from home for at least twice

truant from school
what are the CD subtypes?
child onset type

adolescent-onset type

unspecified onset

code severity
both ODD & CD co-morbit
with ADHD
most with CD have ODD
but most ODD do not turn into CD
prevalence
ODD 3%

CD 10%
sex differences
initially no sex different,

by elementary school, evident of sex differences

maybe males act more violent, physical

and females more internalized, gossip, etc.
snowball effect
as you go along, you are more falling behind
internalizing problems
depression

anxiety
risk factors
child factors

family factors

peer factors
1. child factors
difficult temperament

impulsivity

hyperactive

low intelligence
2. family factors
parental substance use

modeling of ASB from parents

parental history of metnal problems
3. peer factors
rejection by peers

association with delinquent peers/siblings

hang out with the wrong crowd
4. parenting practices
poor parenting skill

poor parent-child relationship

poor supervision

physical punishment

parental neglect

single parenthood

low SES

poorly educated parents

high turnover caretakes

carelessness in allowing access to weapons
5. neighborhood factors
neighborhood disadvantage or poverty

disorganized neighborhood

availability of weapons

media portrayal of violence
assessment & diagnosis
structured or semi-structured interview

family, teacher, and self-reports of behavior
should cover
developmental and family history
Treatment outcome
better for ODD

than CD
effective treatment based on what?
operant conditioning and

social-cognitive learning principles
treatment
contingency management programs

parent management training

CBT

stimulant medication

multisystemic therapy
contingency management programs
establish clear behavior goals

monitor the child's progress

reinforce appropriate behaviors

provide consequences for inappropriate behaviors
parent management training
teach parents to use the contingency management programs

also focus on:

improving parent-child interactions

changing antecedents to problem behaviors

improving parents' monitoring of child's behavior

using more effective disciplines strategies
CBT
role-play, modeling

to over come deficits in social cognitions
stimulant medications
if have ADHD
multsystemic therapy
focus on the family and adjust how the family responds and reacts to both the child and each other
what's developmental psychopathology?
devoted to studying the origins and course of individual maladaptation in the context of normal growth process
ADHD
explosive will

volitation inhibition

minimal brain dysfunction

hyperactive child syndrome

starts in DSM II -- hyperkinetic reaction of childhood

DSM III ADD

DSM IV ADHD

DSM V ADHD
What's ADHD?
persistent pattern of inattention and/or hyperactive-impulsive behaviors

severe and more frequent in same-aged peers

onset must be before 7 years old but diagnosis can occur much later
A. 6 or more symptomns of either inattention or hyperactive-impulsive for at least 6 months

B. symptoms present before age 7
.
1. Inattention symptoms
don't pay attention to close details

make careless mistakes

does not seem to listen when spoken to directly

does not follow instructions or finish tasks

have problems with organizations

avoid things that takes a lot of mental effort

often lose things

easily distracted

forgetful in daily tasks
ADHD
unable to sustain attention

have working memory problem

more off-task time
Hyperactivity symptoms
often fidgeting

get up and down, can't sit still

run and climbs when it is not appropriate

often loud and have trouble enjoying activities quietly

often on the go, seem like driven by a motor

talk excessively
impulsive symptoms
blurts out answers before questions have been finished

trouble waiting for one's turn

interrupt or intrudes on others (butts into conversations)
hyperactive-impulsive behaviors
is also called disinhibition

have problems with voluntary inhibition of response not by motivators
2. hyperactive-impulsive behaviors
more activity than other children

greater difficulties in stopping ongoing behavior

excessive talking

more frequent interruptions

less able to delay gratitificaiton

act too quickly and too often when they have to wait
B. some impairment from the symptoms is present in two or more setting (school/work/home)

C. there must be clear evidence of signiticant impairment in social, school, or work functioning

C. the symptoms bo not happen during the course of PDD, schizophrenia, or other psychotic disorders
.
ADHD subtypes
combined type -- both inattentive and hyper-impulsive symptoms

inattentive type -- inattentive symptoms no hyper-impulsive symptoms

hyperactive-impulsive type -- criteria for hyperactive-impulsive type met but not inattentive symptoms
what's sluggish cognitive style?
selective attention deficits

process information slower
problems of the inattentive type
sluggish cognitive style

more passive social relationships

memory retrieval problems

different developmental course
symptoms may not apply outside of
4-16 age range
inattention more geared toward
school-age or adolescents
hyper/impulsive seem more geared toward
younger children
DSM criticisms
onset before age 7 -- not research supported

no research support for symptom duration of 6 months; some supports for 12 months

situational specificity -- how they act differently in different environment
prevalence
5%
parents report lower figures than
teacher-reports
sex differences
3:1
females have greater deficits than
males
SES/Cultural differences
no support
hower
higher rate outside of US

maybe due to expectations
higher rates in US
non-white
co-morbit
ODD
CD
ASPD
learning disorder
anxiety disorders
mood disorders
course
disinhibition 3-4

inattention 5-7

slow cognitive tempo 8-10
developmental impairments
phsyical problmes

working memory impairments

poor planning

lack of verbal fluency

inefficient self-monitoring

poor regulation of emotion

impaired academic functioning

poor social skills
behavior inhibition executive functioning
all of these can fall under the domain of BI
caused by which part of the brain?
prefrontal cortex, the prefrontal cortical-striatal cortex
what cause ADHD?
environmental
genetic
neurological factors
Barkle's model includes?
poor working memory
delayed interalizatio speech
immature regulation of affect
impaired reconstitution
ADHD imapirs the BI,

then in turn impairs the EF,

then EF in turn impair
social self-sufficiency
this model does not apply to
the inattentive type
Diagnosis
structured or semi-structured interview


intelligence and achievement testing

parent, teacher, and self-reports of behaviors
Treatment
medication, highly effective -- such as Ritalin and Adderall

behavior therapy cannot reduce core symptoms but can help treat co-occurring problems