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56 Cards in this Set

  • Front
  • Back
CARDIAC DYSRHYMIA
ANY CARDIAC RHYTHM THAT DEVIATES FROM THE NORMAL SINUS RHYTHM
SINUS TACHYCARDIA
>100-150
SINUS BRADYCARDIA
<60
SUPRAVENTRICULAR TACHYCARDIA
150-250
ATRIAL FIBRILLATION
ELECTRICAL ACTIVITY IN THE ATRIA IS DISORGANIZED CAUSING ATRIA TO QUIVER
AFIBRILLATION RATE
350-600
ATRIOVENTRICULAR BLOCK
1ST, 2ND OR 3RD DEGREE, ELECTRICAL ALTERATION BETWEEN ARTIA & VENTRICLES
S/S OF PACEMAKER MALFUNCTION
WEAKNESS, DIZZINESS, & SYNCOPE
PVC'S
PREMATURE VENTRICULAR CONTRACTIONS - EARLY VENTRICULAR BEATS
VENTRICULAR TACHYCARDIA
WHEN 3 OR MORE SUCCESSIVE PVC'S OCCUR
DRUGS USED FOR VENT. TACHYCARDIA
PROCAINAMIDE & LIDOCAINE
VENTRICULAR FIBRILLATION
QUIVERING OF THE VENTRICLE RESULTING IN NO CARDIAC OUTPUT
TX FOR VFIBRILLATION
DEFIBRILLATION, CPR, LIDOCAINE, PROCAINAMIDE
DEFIBRILLATION
TERMINATION OF VENTRICULAR FIBRILLATION BY DELIVERING DIRECT ELECTRICAL SHOCK
TIMEFRAME FOR DEFIBRILLATION
SHOULD BE DELIVERED WITHIN 15-20 SECONDS OF THE ONSET OF DYSRHYTHMIA
IF DEFIBRILLATION IS NOT PERFORMED
BRAIN DAMAGE WILL OCCUR FROM LACK OF BLOOD FLOW/OXYGEN
3 DIAGNOSTIC TESTD FOR DETERMINING DYSRHYTHMIAS
ECG, TELEMETRY, HOLTER MONITORING
NURSING INTERVENTIONS FOR CARDIAC DYSRHYTHMIAS
ASSESS APICAL FOR 1 MINUET, ASSESS PT'S ANXIETY & DEGREE OF UNDERSTANDING
CARDIAC ARREST
SUDDEN CESSATION OF CARDIAC OUTPUT & CIRCULATION
CAUSE FOR CARDIAC ARREST
VENTRICULAR TACHYCARDIA, VENTRICULAR FIBRILLATION, VENTRICULAR ASYSTOLE
TX FOR CARDIAC ARREST
CPR, ABC & DEFIBRILLATION
CAD
A VARIETY OF CONDITIONS THAT OBSTRUCT BLOOD FLOW IN THE CORONARY ARTERIES
ATHEROSCLEROSIS
ARTERIAL DISORDER CHARACTERIZED BY YELLOWISH PLAQUES OF CHOLESTEROL ON ARTERY WALLS
ASHD
ATHEROSCLEROTIC HEART DISEASE (CAUSED BY ATHEROSCLEROSIS)
LUMEN
CAVITY OF CHANNEL WITHIN ANY ORGAN OF THE BODY
WHAT HAPPENS DURING ATHEROSCLEROSIS OF CORONARY ARTERIES
BLOOD FLOW TO THE HEART IS OBSTRUCTED
ANGINA PECTORIS
CARDIAC MUSCLE IS DEPRIVED OF OXYGEN
ANGINA
SPASMODIC, CRAMP LIKE, CHOKING FEELING
PECTORIS
BREAST OR CHEST AREA
ANGINA NOTES
OCCURS WITH INCREASED CARDIAC WORKLOAD, INTENSE COLD, EXERCISE, HEAVY MEALS, STRESS
MED. MANAGEMENT FOR ANGINA
GIVE O2, NITROGLYCERINE 1 TAB SUBLINGUAL EVERY 5 MINUTES X 3, BETA BLOCKERS, CALCIUM CHANNEL BLOCKERS MAY CAUSE HEADACHES
CABG
CORONARY ARTERY BYPASS GRAFT( TAKE VEIN FROM LEG & REDIRECT BLOOD FLOW TO THE HEART)
PTCA
PERCUTANEOUS TRANSLUMINAL CORONARY ANGIOPLASTY( INSERTING A BALLOON TO STRECH OUT ARTERY)
STENT PLACEMENT
EXPANDABLE MESH INSERTED INTO ARTERY TO HOLD IT OPEN (KEEP PT ON ANTICOAGULANTS)
MYOCARDIAL INFARCTION
BLOCK OF MAJOR CORONARY ARTERY CAUSING NECROSIS OF MYOCARDIUM
CLINCAL MANIFESTATIONS OF MI
CAN BE SILENT, PAIN NOT RELIEVED BY REST OR NITROGLYCERINE, SOB, SWEATING, PALLOR
MED. MGMT. FOR MI
O2, FIBRINOLYTIC AGENTS, PTCA, CABG, D.O.L.M.A.
4 COMMON LOCATIONS OF MI
ANTERIOR, LATERAL, SEPTAL, INFERIOR
HEART FAILURE
CIRCULATORY CONGESTION RESULTING FROM HEARTS INABILITY TO ACT AS AN EFFECTIVE PUMP
LEFT VENTRICULAR FAILURE
MOST COMMON, PULMONARY CONGESTION, DYSPNEA, PINK FROTHY SPUTEM, CRACKLES LEADS TO RVF
RIGHT VENTRICULAR FAILURE
DISTENDED JUGULAR VEIN, ANOREXIA, ABD DISTENTION, LIVER ENLARGEMENT, ASCITES, EDEMA LOWER 1/2 OF BODY
DIGOXIN
INCREASES PUMPING/CONTRACTIONS ALWAYS CHECK B/P NEEDS TO BE >60
ACE INHIBITORS
DECREASE BLOOD PRESSURE
MED MGMT FOR HF
DIGOXIN. VASODILATORS, ACE INHIBITORS, HOB HIGH FOWLERS, O2, MONITOR I & O
PULMONARY EDEMA
ACCUMULATION OF FLUID IN LUNGS & ALVEOLI, COMPLICATIONS OF CHF (LEFT)
D.O.L.M.A.
Digoxin, O2, Lasix, Morphine, Aminophylline (bronchodilator)
3 DIURETICS
THIAZIDES, SULFONAMIDES (LASIX), ALDOSTERONE **** MONITOR I & O
NITROGLYCERINE
DILATES ARTERIES, IMPROVES BLOOD FLOW, MONITOR FOR HEADACHES
3 ACE INHIBITORS
CAPTOPRIL, ENALAPRIL, VAMIPRIL - MONITOR FOR DROP DRASTIC DROP IN B/P
INOTROPIC AGENTS
DILATES RENAL BLOOD VESSELS TO EXCRET SODIUM WHICH HELPS CHF PT. MONITOR VERY CLOSELY!
VALVULAR HEART DISEASE
HEART VALVES ARE COMPROMISED & DO NOT OPEN & CLOSE PROPERLY
STENOSIS VHD
THICK VALVES CAUSING BLOCKAGE LESS BLOOD BEING CIRCULATED
INSUFFICIENCY
VALVES MAY NOT CLOSE PROPERLY CAUSING BACK FLOW & NOT ENOUGH BLOOD BEING CIRCULATED
CAUSES OF VHD
CONGENITAL OR RHEUMATIC FEVER
MED MGMT FOR VHD
RESTRICT ACTIVITY, LOW SODIUM DIET, DIURETICS, DIGOXIN, MONITOR I & O
2 SURGERIES FOR VHD
MITRAL COMMISSURUTOMY, VALVE REPLACEMENT (PIG), MAIN VALVE AORTIC SEMILUNAR VALVE