Use LEFT and RIGHT arrow keys to navigate between flashcards;
Use UP and DOWN arrow keys to flip the card;
H to show hint;
A reads text to speech;
58 Cards in this Set
- Front
- Back
In the cervical spine, which spinal segments are most often injured? |
C1, C2, C5, C7 |
|
In the thoracolumbar area, which spinal segments are most often injured? |
T12 through L2 |
|
Why does injury occur more frequently at some levels than others? |
Movement(rotation) is greatest at these segments and leads to instability |
|
What is the American Spinal Injury Association |
Created the international standards for neurological classification of spinal cord injury to standardize the way in which severity of injury is determined |
|
Neurological level (ASIA) |
the most caudal level of the spinal cord with normal motor and sensory function on both the left and right sides of the body |
|
Motor level (ASIA) |
The most caudal segment of the spinal cord with normal motor function bilaterally |
|
Sensory Level (ASIA) |
The most caudal segment of the spinal cord with normal sensory function bilaterally |
|
How is the sensory level (ASIA) determined? |
Sensitivity to light touch and pinprick test |
|
What are the types of spinal cord injury? |
Traumatic Non-traumatic Complete Injury Incomplete Injury |
|
What is the number one cause of traumatic spinal cord injury? |
MVA-40% Falls-27% ***Most frequent cause of spinal cord injury |
|
In the cervical region, what is the most common type of injury cause? |
Flexion and rotation forces **Rear-end MVA produce flexion and rotation |
|
A pure hyperflexion force causes what type of fracture? |
Anterior compression fracture due to stretching of the posterior longitudinal ligaments |
|
Cervical hyperflexion usually results in what type of spinal cord injury? |
Severs the anterior artery and results in incomplete anterior cord syndrome **head on collision |
|
What is GM-1 Ganglioside |
Limits the amount of inflammation, decreases effects of ischemia, and enhances blood flow |
|
Once a pt. is injured, what should you do? |
Immobilize the patient to stabilize the spine to prevent further damagge |
|
What does recovery depend on? |
1. the extent of pathologic changes 2. The prevention of further trauma 3. Prevention of secondary medical complications |
|
What are some examples of non-traumatic injury? |
Arteriovenous malformation thrombosis/embolus Hemmorrhage Vertebral subbluxations Abscess of the spinal cord neurological diseases infections |
|
Complete Injury |
No sensory or motor function in the lowest sacral segments (s4-s5) |
|
How are sensory and motor function determined at s4-s5 |
by anal sensation and voluntary external anal sphincter contraction |
|
Incomplete injury |
having partial motor and/or sensory function below the neurological level including sensory and/or motor function at s4-s5 |
|
What must be present for an injury to be classified as incomplete? |
Perianal sensation must be present |
|
Tetraplegia (quadriplegia) |
Complete paralysis of all four extremities and trunk Injuries to cervical spine |
|
Paraplegia |
Complete paralysis of all or part of the trunk and both lower extremities Results from lesions in the thoracic or lumbar spine |
|
Cauda equina injuries |
Injuries at L1 or below |
|
Is it better to have an incomplete or complete lesion? |
Incomplete-recovery or motor function **may be able to walk |
|
Brown-sequard syndrome |
Hemisection of the spinal cord caused by penetration wounds Features on the same side as lesion |
|
Clinical features of brown-sequard syndrome |
Ipsilateral-paralysis, sensory, and motor function loss Contralateral-loss of sense of pain and temperature |
|
Anterior Cord Syndrome |
Flexion injuries of the cervical region-damage to anterior spinal cord |
|
Clinical features of anterior cord syndrome |
Loss of motor function and sense of pain and temperature bilaterally below the lesion -proprioception, light touch, and vibratory sense are preserved |
|
Central Cord Syndrome |
**Most common SCI syndrome results from stenosis, compression that is the consequence of hyperextension in the cervical region |
|
Clinical characteristics of central cord syndrome |
More neurological involvement of UE than LE Sensory impairment< motor impairment Preservation of sexual, bowel and bladder |
|
Cauda Equina Injuries |
Direct trauma from a fracture-dislocation below L1 -Usually incomplete -Peripheral nerve injuries |
|
Clinical characteristics of cauda equina injuries |
Flaccidity, areflexia, loss of bowel and bladder. Lower extremity paralysis and paresis |
|
Root escape |
Damage to the nerve root within the vertebral foramen can lead to a peripheral nerve injury |
|
Dorsal Column/Posterior Cord |
Incomplete injury that results from damage to posterior spinal artery Loss of sensory functions **rare |
|
Spinal Shock |
Following an SCI Period of flaccidity, areflexia, loss of bowel and bladder function, and loss of autonomic functions |
|
How long does spinal shock last? |
24 to 48 hours |
|
Autonomic Dysreflexia |
Autonomic reflex that can be life threatening |
|
Where does autonomic dysreflexia occur? |
Above T6 |
|
Autonomic dysreflexia is more commin in what what type and stage of recovery? |
Chronic stage Complete injury |
|
What happens during autonomic dysreflexia? |
Acute onset of autonomic activity from noxious stimuli below the level of lesion. Afferent input reach the lower spinal cord and initiate a mass reflex response resulting in Hypertension |
|
What are the results of hypertension from Autonomic Dysreflexia? |
seizures renal failure retinal hemorrhage cardiac arrest stroke subarachnoid hemorrhage |
|
What is the most common cause of AD ? |
Bladder and bowel distension/irritation |
|
Symptoms of AD |
Profuse sweating Bradycardia INcreased spasticity Constricted pupils Piloerection Blurred vision Headache
|
|
Intervention for AD |
Identify the source of the noxious stimuli Decrease pt's blood pressure Loosen tight clothing
|
|
Spastic hypertonia |
65% of SCI Pasticity, muscule spasms, abnormally high muscle tone, hyperactive stretch reflexes, clonus |
|
Spastic hypertonic occurs in what spinal level |
cervical-level |
|
What is the cause of spastic hypertonia |
Result of altered input at the spinal segmental level, imbalance between excitation and inhibition |
|
What cardiovascular impairments occur post SCI |
bradycardia and dilation of the peripheral vasculature below the level of the lesion |
|
During SCI is sympathetic or parasympathetic communication lost? |
sympathetic |
|
Symptoms of orthostatic hypotension |
Blurred vision, ringing in the ears, fainting, light-headedness |
|
Orthostatic hypotension occurs in what spinal level? |
About t6 |
|
Is temperature control impaired post SCI? |
Yes, the hypothalamus can no longer control cutaneous blood flow or level of sweating |
|
What is tthe leading cause of death in people with high cervical injuries? |
Pulmonary impairments |
|
Injuries below what level will have normal respiratory function |
below T10 |
|
Paradoxical breathing pattern |
Paralysis or paresis of the scalenes and intercostal muscles results in altered breathing pattern |
|
Postural hypotension |
SCI often develop low blood pressure due to lack of efficient skeletal muscle pump |
|
BP below what numbers will result in cardiac arrest? |
70/40mmhg |