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86 Cards in this Set

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Cerebral Palsy
A static (not changing) encephalopathy (brain injury that is non-progressive disorder of posture and movement); etiologies are varaible- brain injury, trauma during birthing process; often associated with epilepsy, speech problems, vision compromise, and cognitive dysfunction; most will have neuromuscular challenges
Most common speech disorder individuals with CP experience
Dysarthria
Prevalence of CP
2-4/1000; 7-10,000 new babies born each year in the U.S.
Physiologic CP
look at what's going on with the individual e.g. they have spastic CP; use elements of physiology to label what their deficit(s) is
Athetoid CP
Physiological characteristic: constant uncontrolled motion of limbs, head and eyes; appear intoxicated, can walk but unsteady; speech tends to be slurred; imprecision with speech
Ataxic CP
physiological characteristic: poor sense of balance, often causing falls and tumbles; imprecise articulation
Rigid-Spastic CP
physiological characterisic: rigidity means tight muscles that resist efforts to make them move;
Spastic means tense, contracted muscles (most commone type of CP)
Atonic CP
associated with marked hypotonia, brisk reflexes, and severe cognitive delays
Mixed CP
spastic and athetoid
Monoplegic CP
single limb; topographic classification
Paraplegic CP
lower limbs; topographic classification
Hemiplegic CP
one side of the body; topographic classification
triplegic CP
three limbs; topographic classification
quadraplegic CP
four limbs; topographic classification
Diplegic CP
symmetrical parts; topographic classification
Etiologic CP- prenatal
(70%)- something going on during prenatal development such as infection, anoxia, toxicity, vascular, Rh disease, genetic, congenital malformation of brain
Etiologic CP- natal
(5-10%)- anoxia, traumatic delivery, metabolic
Etiologic CP- post natal
trauma, infection, toxic, e.g. infant who is dropped = trauma; allergic reaction = toxic
Clinical symptoms and complications of CP
Spacticity, weakness, increase reflexes, clonus(repetitive movement), seizures, articulation and swallowing difficulties (these are typically treated by SLP) visual compromise, deformation, hip dislocation, kyphoscoliosis, constipation, urninary tract infection
Management of CP
Speech
OT
PT
Adaptive equipment (AA)
Neurologic
Surgical (Rhizotomy- nerves section- cut the nerves that are causing the muscle to be hypercontracted)
Pharmaceutical (e.g. baclofen pumps, botoxin spacticity reduction)
Rrehabilitation challenges for those with CP
Mobility
Communication
Learning
Self Care
Self Direction
Independent Living
Economic Sufficiency
*Help individual to grow to be as independent as possible
Speech
just one modality of communication (others = gestures, facial expression), most natural, efficient and culturally acceptable form of communication, typically the long-term goal of individuals with impairments, not always achievable
Communication
Functions to control an individual's enviroment (to obtain or reject something), functions to regulate social interactions, functions to receive and convey information and other ideas, can be as simple as changing body tension
Assistive technology
Any item, piece of equipment, or system that is commonly used to increase, maintain, or improve functional capabilities of individuals with disabilities; used to enhance the effectiveness of one's communication
Augmentative communication
used by individuals who have some speech but are unintelligible or have limited abilities to use speech; facilitating their natural ability to communicate; e.g. person talks very softly so they use an amplifier to enhance communication
Alternative Communication
Used when an individual has no speech and must rely on another method to make their communicative intents (ideas, desires and needs) known; individual has unintelligible speech so substitute something that replaces natural speech; use gestures, signs, pointing device- create an alternate source of speech
Purpose of AAC
increase/improve an individual's ability to achieve basic communication functions in the environments and activities in which they participate or are expected to participate- gives someone the ability to have their needs met and share information
AAC systems
integrated group of components, including the strategies, aids, techniques, and symbols used to enhance communication; we all use a system to communicate;e.g. texting is part of our communicative system and it is an alternate communication device; e.g. answering maching allows us to communicate with someone even when they are not there
Aided Communication
the use of devices (computer), tools (pen), objects (pictures), individuals etc. to communicate;
-external materials and/or devices are necessary, generally less efficient
unaided communication
use of one's own self (speech, gestures, body language) to communicate; no external material or device is necessary; generally more efficient; e,g, sign language
low-tech aided communication
pictures/objects
schedules
menus
pencil & paper
communication board
communication books
high-tech aided communication
recorders
amplifiers
synthetic speech
comnputer based communication
core vocabulary
used to meet basic communication needs in ALL environments
Fringe vocabulary
specific to certain environments; e.g. menun = eating, calendar = morning circle time, tv guide = leisure time,
closed set of vocabulary
Static- paper board--> items dont change
AND
Dynamic- computerized device, pictures can easily change
Generative vocabulary
picture/gesture sequencing
spelling
talking
e.g."hi im rick"- combining utterances and spell words
visual representation system
real objects
miniature objects
photographs
realistic drawings
line drawings
symbols
written words
type of access/selection for AAC devices
Direct selection: method of choice, most efficient- quickest e.g. key boards or pointing to pictures
Indirect Selection: less efficient, more likely the choice with increased physical movement impairments
e.g. single switch, multi switch, scanning, coding
Goal of AAC systems
facilitate communication by increasing opportunities and reducing barriers
increasing opportunities
-way to facilitate communication
- shape communicative environment
-peer involvement
-patience
-reinforcement
reducing barriers
-way to facilitate communication
-policy
-environment
-resource
-communication
AAC considerations
-multi-modal system
-access rate- efficiency
-adaptability- must grow with the user
>pre-literate
>literate
>non-literate
-support and acceptance
Caaruso & Strand Model
adult model
sensorimotor planning
sensorimotor programming
neuromuscular execution
Sensorimotor planning
initial process in the transformation from the cognitive/linguistic processes to speech movements- specifying motor goals
frontal lobe's role in sensorimotor planning
specify the direct and indirect system motor plans
parietal lobe's role in sensorineural planning
contribute to planning based on expected performance and aspects of internal (proprioceptive) feedback
motor planning
goal-orientated spatial and temporal specifications of speech movements (respiration, phonation, resonance, ariculation)
-described in PLACE and MANNER of production
-coordination, sequencing of sounds and coarticulation
sensorimotor programming
a set of muscle commands structured prior to onset of a movement & executed ina sequence while influenced by feedback
-accounts for major movement determinations of respiratory, phonatory, resonatory, and articulatory position and timing
Force function
element of sensorimotor programming
level of muscle contraction to complete target
timing relations
element of sensorimotor programming
temporal characteristics of within each element of the target
interstructural/component relations
element of sensorimotor programming
positioning and timing across elements
status of the position dynamics
element of sensorimotor programming
feedback to ensure movement adheres to the plan to reach the intended target
Sensorimotor programming: what do the programs specify?
programs specify muscle tone, movement direction, force, range, rate, as well as mechanical stiffness of joints
why is sensorimotor program flexible?
to account for dynamic nature of phonological targets (e.g. coarticulation)
it prevents over- and under-shoot
speech motor equivalence
the contributions of various structures (e.g. articulators) can vary considerably across multiple productions of a segment, yet the end product is perceptually consistent
Neurosubstrates associated with programming
basal ganglia control circuit
cerebellar control circuit
fronto-limbic system
SMA
Cortex
Sensorimotor execution
realization of the dynamix sensorimotor plan and programming
-even after speech has begun, system is flexible to make "on-line" changes due to the integrated rocess of all aspects of the sensorimotor system (planning, programming, execution)
Speech Movement
production of acoustic signal (based on voltional recruitment of respiratory, phonatory, articulatory, musculature) in corrdinated and rapid fashions
very complex postures and dynamic characteristics (movements) are highly skilled volitional neuromuscular events
functional trade-off between speech and accuracy (varies based on personal and situational factors)
Netsell's model
respiration, phonation, resonance, and articulation all lead to/ have an impact on prosody; dysarthria can affect any/all of these subsystems
Motor speech disorders
speech disorders resulting from neurologic impairments affecting the motor planning, programming, neuromuscular control or execution of speech; disorder of speech with a sensorimotor difficulty
Dysarthria
neurologic speech disorder resulting from abnormalities in the strength, speed, range, steadiness, tone,or accuracey of movements required for control of the respiratory, phonatory, resonatory, articulatory and prosodic aspects of speech production; neurologic in origin; disorder of movement or control
what causes the pathophysiologica disturbances responsible for dysarthria?
central or peripheral nervous system abnormalities and most often reflect weaknesses; spacticity; incoordination; involuntary movements, excessive or reduced muscle tone
Apraxia of speech
neurologic speech disorder reflecting an impaired capacity to plan or program sensorimotor commands necessary for directing movements that result in phonetically and prosodically normal speech;
occurs in the absence of physiologic disturbances associated with the dysarthrias and in teh absence of disturbance in the compent of language
Childhood Apraxia of Speech (CAS)- symptoms
difficulty initiating speech movements, difficulty achieving and maintaining articulatory configurations, groping for articulatory posture,halting, effotful sequencing of phonemes, inabilitiy to imitate speech sounds in absence of structural or functional abnormalities of articulators, vowel distortion,timing errors,
why does CAS have more widespread linguistic effects than adult apraxia?
language is still developing in children
Different terms for CAS
developmental articulatory dyspraxia, verbal dyspraxia, developmental verbal apraxia, developmental apraxia of speech
Dysarthria
execution challenges
speech distortions
consistent errors
little or no groping for sounds
all speech is affected
little difference in errors based on types of speech (automatic vs. volitional)
Apraxia
speech substituions
inconsistent errors
groping for articulatory sound/target
islands of fluency
error differences based on types of speech (automatic is fluent, volitional is not)
Types of dysarthria
flaccid
spastic
ataxic
unilateral upper motor neuron
hyperkinetic
hypokinetic
mixed
Flaccid Dysarthria symptoms
hypotonia, breathy voice, hypernasal
Flaccid Dysarthria: lesions
Lower motor neurons (LMN) located in cranial and spinal nerves
V. trigeminal nerve: jaw movement
VII. Facial nerve: lower half of face
IX. glossopharyngeal nerve: velum
VII. hypoglossal: tongue
clinical Dx of flaccid dysarthria
myasthenia gravis
Spastic dysarthria: symptoms
stiff, rigid muscle tone, strained, strangled speech
Spastic dysarthria: lesions
most commonly caused by stroke affecting the brainstem; must have bilateral damage
clinical Dx of Spastic Dysarthria
spasmodic dysphonia
Ataxic Dysarthria: symptoms
poor muscle coordination; gives impression of intoxication; weak muscle tone; unsteady in their gate; speech also sounds like they are intoxicated
Ataxic Dysarthria: lesions
lesions/damage to cerebellum
Unilateral Upper Motor Neuron Dysarthria (UUMN): symptoms
speech difficulties are minimal usually and often only occur during early stage following brain injury
Unilateral Upper Motor Neuron Dysarthria:lesions:
caused by damage to upper motor neurons on ONE side
usually caused by single stroke affecting middle cerebral artery
can co-occur with Broca's aphasia or RHS, depending on hemisphere of damage
Hyperkinetic Dysarthria: symptoms
too much movement of muscles; tremors (rhythmic muscle contraction and relaxation), tics (sudden repetitive nonrhythmic), involuntary movement can be quick or slow-->
*dystonia- slow movements
*Chorea- quick movements
Hyperkinetic Dysarthria: causes
damage to the extrapyramidal system; involves basil ganglia
Hypokinetic dysarthria: symptoms
muscles become rigid and stiff; too little muscle movement
hypokinetic dysarthria: cause
lesions to extrapyramidal tract; most common cause is Parkinson's disease
Prosody
Naturalness of speech
supersegmental aspects of speech
>rate/duration
>loudness/intensity
>pitch/frequency
linguistic stress
social communication/pragmatics
interpretation of prosody is done by right hemisphere
Mixed dysarthria:
combination of dysarthrias; site of lesion not so precise
treatment of dysarthria
articulation: intelligibility drills, placement cues
resonance: prosthesis, medical intervention
prosody: contrastive stress drills
phonation: hypoadduction or hyperadduction exercies
respiration: controlled respiration/phonation tasks; chunking