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146 Cards in this Set

  • Front
  • Back

fluorescein eye stain

indication diagnosis of corneal ulcers, corneal lacerations and cuts, foreign body puncture wounds and test of nasolacrimal duct patency


green dye can come out the animals nose

schirmer tear test

indication is to measure the rate of tear production


normal tear production between 15mm-20mm/min

tonometery/tonometer

indications to measurement of intraocular pressure/IOP


diagnosis of glaucoma-high reading


anterior uveitis-low reading


normal in dogs is 15-25 mm Hg


normal in cats is 15-30 mm Hg

telephone triage

name and phone number!


ask questions to determine whats a serious emergency


always offer exam

dystocia

difficult labor

in hospital triage

3 groups


brief history, quick PE, and target discussion with owners


TPR, MM color, hydration status and CRT

initial triage exam

systemic evaluation of essential organ systems


cardiovascular, respiratory, neurologic

tachypnea

increased respiratory rate

bradypnea

decreased respiratory rate

inspiratory dyspnea

low, slow inspirations with short exhalations

expiratory dyspnea

increased abdominal effort on expiration

labored breating

prolonged and deep

orthopnea

patient is air hungry


neck extended


crouching


distress or aggression


elbows abducted

apneustic pattern

deep inhalation with an abnormally long pause before exhalation

cheyne-stokes breathing

pattern of alternating tachypnea and bradypnea occurring when carbon dioxide regulation of respiration is interrupted

kussmauls breathing

slow deep regular respiratory compensation for metabolic acidosis

paradoxical chest excursion

segment of thoracic wall moves in teh opposite direction in relation to the rest of the chest wall during respiration

decreased mentation

shock state

tachycardia

abnormally fast heart rate


dogs



bradycardia

inappropriately slow heart rate


cats

prolonged CRT

fluid resucitation

shortened CRT

hyperdynamic stage of sepsis

obtunded mentation

reacts to stimuli but at slower pace than normal

dull mentation

not bright or eager, still interactive with environment

stuporous mentation

completely disconnected only respond to noxious stimuli

comatose mentation

completely disconnected, doesn't react to any stimuli

mydriatic pupils

unresponsive big pupils


fixed and dilated



anisocoria

one big pupil and one little pupil


acute cerebral injury

TBI

traumatic brain injury

decerebrate posture

disconnect between forebrain and brain stem


extreme rigidity of all 4 legs


may have arching of back and neck


comatose or stuporous mentation



schiff sherrington posture

normal mentation


ambulatory when picked up and placed on feet


t3-t4 spinal cord lesion

decerebellate posture

severe injury to cerebellum


rigid forelimbs and flexed hindlimbs


normal mentation

tympany

pinging



fluid wave during abdominal palpation

hemorrhaging

signs of severe pain

arched back


praying posture



dehydration

decrease in water component of blood


tacky or dry mm


lack of skin turgor


mental status


body weight





hypovolemia

loss of blood volume


hypovolemic shock


tachycardia


weak pulses


hypotension


prolonged CRT
emergency

treating hypovolemia

catheterization


IV fluids


blood transfusion


identification of source-emergency surgery


intensive monitoring and nursing



treating dehydration

fluid replacement over several hours


slower if patient is hyperatremic- can cause cerebral edema


monitoring

initial diagnostics

packed cell volume


total protein


blood glucose


blood gas analysis


blood pressure


pulse oximetry


ECG


fast ultrasound scan


electrolytes


colloid osmotic pressure

pulse oximetry

saturation of oxygen in blood


anything below 92 should be put on oxygen therapy


oxygen level should always be 100

electrolytes

major role in the maintenance of extracellular compartmental water balance and cell function


commonly measured are potassium, sodium, chloride, magnesium and ionzied calcium

colloid osmotic pressure/COP

used to guide guide therapy


normal is 20-25 mm Hg


keep greater than 18 mm Hg

basic first aid for wounds

clipped


cleaned


flushed with sterile saline


bandages applied

emergency care station

easily accessible


clean and well stocked


equipment organized and labeled


oxygen source


suction apparatus


crash cart


functional clippers



crash cart

emergency medications


needles


syringes


laryngoscope


endotracheal tubes


ambu bag


instrument packs


red rubber catheters


large bore intravenous catheter

shock

altered blood flow or impaired delivery of oxygen to tissues


early stages are; depressed or anxious, tachycardic and tachypneic, normal, decreased or increased pulse, hyperglycemia

hypovolemic shock

most common in dogs and cats


decreased circulating blood volume


weak pulse, delayed CRT, pale mm and altered mentation


treatment- IV crystalloids, colloids or hypertonic solutions


blood transfusion

distributive shock

maldistribution of blood flow from inappropriate vasodilation and from pooling of blood in capillaries


anaphylaxis, heatstroke and envenomation


treatment- fluid therapy and vasopressors


weak or bounding pulse and pink mm

obstructive shock

venous return to the heart impaired


GDV


treatment-surgery or pericardiocentesis

cardiogenic shock

impaired cardiac output secondary to problems with the heart


cardiomyopathy, valvular heart disease and arrhythmias


weak pulses, hypotension and pale mm


treatment- antiarrhytmics, inotropes, vasopressores and pacemakers

septic shock

severe infectious insult


pneumonia, parvovirus, gastric or intestinal perforation or infected bite wound


sequeal to severe tissue damage-heatstroke or pancreatitis

complications of shock

systemic inflammatory response syndrome/SIRS


disseminated intravascular coagulation/DIC


multiple organ dysfunction syndrome/MODS

systemic inflammatory response syndrome

sepsis just a source of active infection

disseminated intravascular coagulation



thrombosis and bleeding


often fatal


throw blood clots


clotting cascade

multiple organ dysfunction syndrome

permanent organ failure and death

reperfusion injury

cells become starved for oxygen after shock-elevated levels of lactate and by products causing tissue damage


blood flow and oxygen are restored in tissue lactate and free radical molecules can be released into circulation


risks-SIRS, complications, organ failure

lactate concentration

hyerlactatemia is an increased lactate concentration


lactic acidosis results and decrease in systemic blood pH


Type A- inadequate oxygen delivery or increased oxygen demand


type B- inadequate utilization of oxygen

tracheostomy tube placement

blocked airway or distress


cant get endotracheal tube in


allows breathing until upper airway is cleared

capnography

measures carbon dioxide

flow by oxygen

flow rates need to be 100-150 ml/kg/mi



mask oxygen

with diaphragm on or off


tight mask can stress patient


without diapragm reduces oxygen blast



oxygen hood/cage

some have benefit to control temp and humidity


ensure cage is safe and patient cant get trapped


whole body or head and neck can be involved

nasal cannula

prongs


works well for brachycephalic breeds


can irritate and cause patient to sneeze


benefit is they are sold with etCO2 capabilities

naso-oxygen catheter

most invasive


best for long term oxygen


placed into nasal cavity, nasopharynx or trachea


complication-gastric distention or epitaxis


contraindicated in patients with known coagulation disorders

transtracheal oxygen catheter

administer oxygen directly into trachea


same catheters used for nasal oxygen


may cause patients to cough


prolonged use should include humidified

hyperbaric oxygen therapy

use increased atmospheric pressure to allow more dissolved oxygen in teh blood


hand off type therapy-pressurized chamber cant be simply opened, must be depressurization first


more indicated for wounds, severe burns and infections


helps healing process

oxygen toxicity

damage to pulmonary epithelium


pulmonary edema


fibrosis


can cause damage after 6 hours and signs not observable until later



pulse oximetry

normal saturation- 95-100%


doesnt measure partial pressure of oxygen


below 95 need oxygen therapy

capnography

measures carbon dioxide levels


inserted into breathing circuit of intubated patient


end tidal carbon dioxide reflect amount of carbon dioxide present in expired air at the the end of exhalation


increased-hypoventilation


decreased-hyperventilation or decreased cardiac output

arterial blood sample

assess pulmonary function


measured on pH and blood gas analyzer


reveals ability to ventilate and oxygenate



arterial blood gas monitoring

SaO2 arterial hemoglobin saturation


PaO2 partial pressure of oxygen-oxygenation


PaCO2 partial pressure of carbon dioxide-ventilation status



hypoventilation

increased PaCO2


occur with airway obstruction, lung disease, brain disease, sedation/anesthesia, toxicities


can cause academia/decreased blood pH

hyperventilation

decreased PaCO2


can be caused by hypoxemia, metabolic acidosis, stress, pain and anxiety

arterial blood samples

arteriopuncture


dorsal metatarsal artery


femoral artery

urinary output

normal urine production is 1-2ml/kg/hr


decreases when perfusion decreases or when MAP is less than 60 mm Hg

central venous pressure/CVP


measures the hearts ability to pump fluids returned to it


an estimate of the relationship of blood volume to blood volume capacity


used when heart failure is suspected

do not use jugular catheter when

a patient has a coagulation problem

do not use back legs to catheterize when

cats have a thromoembilism

winged need IV catheter

for short term use, pet has limited movement


blood collection


administration of nonirritating medications


easy to puncture vessel wall



multilumen IV catheter

2-3 lumens in one catheter


allow simultaneous infusions at one catheter site



through the needle catheter

placed in jugular vessels or saphenous


8-12 length and 8-20 gauge


needle guard protects need from sticking animal and shearing catheter



over the needle catheter

most common


placed in peripheral vessels


10-24 gauge, 3/4 to 5 1/2 length


catheter is fitted outside or over a steel needle


needlepoint extends beyond the catheter for entry into the vein

general supplies for peripheral catheters

catheter


syringe with heparinized saline flush


injection cap


tape


bandage material


clippers


antiseptic scrub and solutions

catheter maintenance

inspect every 24-48 hours


check every 2 hours if receiving fluid therapy


flushed with heparinized saline every 6-8 hours to prevent clots


dont leave in longer than 72 hours/3 days



phlebiitis

vein inflammation

cardiopulmonary arrest

cessation of spontaneous respirations and lack of perfusion heart rhythm

CPR

cardiopulmonary resuscitation

CPCR

cardiopulmonary cerebral resuscitation


importance of restoring blood flow to the brain as well as the heart and lungs

reasons for CPR

obstructed airway


shock


poisoning


prolonged seizure


coma


head injury


electric shock


sudden cessation of heart activity and breathing



ABC

airway


breathing


circulation



CAB

circulation


airway


breathing



closed chest compressions

cardiac pump


place animal in right right lateral recumbency


technician's hands encircle the ventral chest


perform compressions directly over the heart


use whole hands


120-130 compressions per minute]


large dog-must let the wall recoil back to normal positioning before next compression, 100 compressions per minute


never stop compressions even with other interventions being provided

closed chest compressions with abdominal compressions

used to enhance venous return to the heart during CPCR


increased abdominal pressure can facilitate blood flow towards the heart


abdomen is compressed during the recoil phase of the chest compression


complications-organ contusion, hemoabdomen

open chested CPCR

incision in the left 5th intercostal space, freeing heart from attachments


directly massages heart from apex to base


can occlude descending aorta promoting preferential delivery of blood to the brain and heart


decision must be decided after 1-2 cycles of resuscitation to maximize chances of success


indications- pleural/pericardial effusion, pneumothorax, diaphragmatic hernia, flail chest, penetrating chest trauma, giant breeds

airway during CPR

insert endotracheal tube in lateral recumbency


regardless of size 8-12 respirations per minute


governors vessel 26/G26 acupuncture helpful in stimulating respirations-25 gauge needle and twirl to stimulate receptors in brainstem

one cycle of CPCR

compressions and ventilations administered for 2-3 minutes at a time before the patient is reassessed

3 main arrest rhythms

asystole/flatline


pulseless electrical activity/PEA


ventricular fibrilation

asystole

complete cessation of all mechanical and electrical activity of heart


epinephrine, vasopressin, atropine

pulseless electrical activity

electrical system of heart is functioning but no mechanical heartbeat occurs in response to electrical stimulation


epinephrine, vasopressin, naloxone

ventricular fibrillation

disorganized contractile activity of the heart


proceeded by ventricular tachycardia


defribrillation treatment

defibrillator

never use alcohol for risk of fire


initial dose is 2-4 j/kg



NAVEL

drugs used in the endotracheal tube


naloxone


atropine


vasopressin


epinephrine


lidocaine

cardiovascular system after post arrest

effects of local ischemia and reperfusion injury of the heart muscle


can develop arrhythmias and systemic hypotension



respiratory system after post arrest

at risk for pulmonary edema, atelectasis, pulmonary thromboembolism, acute respiratory distress syndrome, pulmonary contusions hemorrhage, or rib fractures

gastrointestinal tract post arrest

microscopic breakdown of GI mucosal barrier


at risk for sepsis to bacterial translocation


GI hemorrhaging

kidneys post arrest

acute kidney injury, hypotension and hypoxia, monitor urine output and electrolytes

central nervous system post arrest

mechanisms lost to keep supply of oxygen, hypoxic brain injury, altered mental state and nerve function, blindness


evaluate neurologic function hourly

post arrest

physical examination every 30-60 minutes


monitor blood glucose concentration- at risk of hyperglycemia from stress and epinephrine exposure


hypoglycemia due to sepsis or reperfusion

lidocaine

antiarrhythmic medication


treat rapid or unstable ventricular tachycardia


given as bolus



mannitol

osmostic diuretic remains in intravascular space, draws water from interstitial space between cells


causes shift of fluid from tissue into blood stream


decreases cerebral edema


caution with patients in renal failure

dopamine

synthetic catecholamine


peripheral vasoconstriction improves blood pressure



dobutamine

synthetic catecholamine


increases cardiac output by enhancing cardiac contractility

vasopressin

used in hypotensive patients to improve blood pressure



furosemide

loop diuretic


increases urine output and causes volume contraction


help resolve cardiogenic pulmonary edema or fluid overload


do not use with hypovolemic patients

fluid therapy for critically ill

used to restore and or maintain body water in animals that are unable to keep up with their daily losses through eating and drinking

routes of fluid therapy

subcutaneous-slowest


intravenous-faster and larger volumes


intraosseous


PO- per os- by mouth-slowest

healthy animal loses how many mg/ml of fluid per day

50mg/ml

signs of dehydration

sunken eye


scruffy, dull coat


lethargy

fluid administration calculation after determining perceived percentage of deydration

volume to deliver/ml= %dehydration(decimal) X body weight in kg X 1000


divide by 24 to determine volume replaced over the first 24 hours

maintenance fluid requirements

50 ml/kg/day-compensates for measurable and immeasurable losses


weight in kg X 50=maintenance


combine initial fluid rate with maintenance for fluid plan

isotonic crystalloids

most common


osmolality equal to blood


hypovolemic shock


electrolyte concentration similar to extracellular fluid


lactated ringers, normal saline

hyertonic crystalloids

osmolality greater than blood


7.5% saline


saline causes fluids shifts from the intracellular space to the extracellular space resulting in improved venous return and cardiac output


causes vasodilation and reduced swelling


must be given slowly

colloids

high molecular weight solutions that dont cross capillary membrane readily


better blood volume expanders


natural-plasma/syntethic colloids


not indicated for hypovolemia shock

hypotonic fluids

osmolality less than blood


contain too much free water and distribute excessively to the intracellular compartment


5% dextrose in water


never use for resuscitation



whole blood or packed RBC fluid

treating hemorrhagic shock

hypertonic solution

cells shrink

isotonic solution

cells are normal



hypotonic solution

cells swell

hydration assessment

PCV


urine output


urine specific gravity


changes in body weight


clinical signs of dehydration



how do you know if a patient isnt tolerating fluid rate

patients hock will show loss of detail or edema


thoracic auscultation reveals moist lung sounds


cardiac auscultation reveal irregular rhythms



complications of fluid therapy

volume overload


electrolyte/acid base distrubance

volume overload in fluid therapy

peripheral edema


cough


increased rr


serous nasal discharge


restlessness


vomiting, diarrhea


pulmonary edema


pleural effusion

fluid additives

potassium chloride/KCl-hyokalemia- increase blood sugar


dextrose- hypoglycemia


sodium bicarbonate- metabolic acidosis, severe hyperkalemia, chronic metabolic acidosis

micro drip set

used for pediatrics



macro drip sets

60 gtt/ml set-60 gtt/min=1 cc


10 gtt/ml set-10 gtts/min=1 cc


15 gtts/ml set-15 gts/min=1 cc



determining GGT/drops

determine volume based on maintenance and fluid deficit


volume X drip set(10,15,60) / minutes= ggt/min


60 / ggt/min= ggt/sec

5% dehyrdation

mm tacky or dry


Hx of fluid loss



7% dehydration

dry mm


prolonged skin tent


increased HR


pulse pressure/BP normal


Hx of fluid loss somewhat prolonged

10% dehydrdation

Hx of fluid loss somewhat prolonged


dry mm


prolonged skin tent


increased heart rate


weak to thread pulse pressure


hypotension





12% dehydration

Hx of marked fluid loss


dry mm, prolonged skin tent


increased/decreased HR


hypotension


pulse weak or absent


decreased body temp, cool extremities


diminished mentation


sunken eye

sympathomimetics

drugs support myocardial contractility and BP


used when patient is unresponsive to vigorous fluid therapy


domamine and dobutamine



mechanical ventilation

central line catheter and central venous pressure measurement


arterial catheter


continuous direct BP monitoring, pulse ox, temp etCO2


indwelling urinary catheter

complications of mechanical ventilation

pressure ulcers


peripheral edema


edematous tongue


pneumonia

central venous pressure/CVP

refers to the blood pressure in central veins before it enters the right atrium


0-5 cm H2O is normal in dog


used to assess volume status and response to fluid therapy

normal values for arterial blood pressure

systoic- 80-140


diastolic- 50-80 mm Hg