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85 Cards in this Set

  • Front
  • Back
ASTHMA Card 1
- characterized by airway narrowing r/t branchospasm, mucous membrane edema, & increased production of thick sputum
- inpredictable and variable. attack lasts mins to hrs, & then may be assymptomatic
1) extrensic:allergic rxn to specific allergan (pollen, spores, milk)
2) Intrinsic: no identifiable factor (maybe virus, exercise, emotion)
3) Mixed: MOST COMMON
ASTHMA Card 2
Following exposure to causative agent, 3 things hapen causing airway obstruction:
1. Bronchospasm due to constriction of bronchial smooth muscle
2. Histamine release due to allergic rxn (increased mucous production)
3. Histamine can also cause dialation of capillaries in bronchial tree (causing mucosal edema)
ASTHMA Card 3
Major difficulty is on expiration b/c it's difficult to force air out through constricted swollen bronchioles. aka Hyperinflation
Pt. will exhibit wheezing, prolonged expirations
ASTHMA Card 4
May sit upright, use accessory muscles, chest tightness, cough may be productive or nonproductive.
tx: open airway
Status asthmatics: sustained asthma attack, resistent to treatment, stat situation
CHRONIC BRONCHITIS Card 1
Primary cause: SMOKING, hypertrophy of mucus secreting glands, increase in goblet size & #, decreased ciliary activity, & bronch edema w/ narrowing airways
S/X: chronic cough w/ sputum production, frequent infections
EMPHYSEMA Card 1
Lung disorder: characterized by abnormal enlargement of air spaces (alveoli) distal to terminal bronchioles, alveolar walls & capillary network are destroyed, airways are narrowed, & decreased lung elasticity.
EMPHYSEMA Card 2
Patho: destruction of elastin & collagen structure in the lungs, bronchioles collapse on expiration, air trapped in lungs, & overinflation.
As alveoli destroyed, merge into blebs in pleaural space.
Decreased surface area available for diffusion of O2 & CO2.
Bulla: if belbs b/c really big
EMPHYSEMA Card 3
Manifestations: early dyspnea, chest b/c barrel shaped due to lung over inflation, minimal cough, & no cyanosis.
Tx: Can't undo damage, but relieve airway obstruction
CHRONIC OBSTRUCTIVE PULMONARY DISEASE
(COPD)
COPD pts often have combination of bronchitic & emphysema.
Often develop heart problems b/c heart has to pump hard to deliever enough oxygenated blood. (R sided heart failure family)
Atelectasis
mucus plugs/obtructs small airway

precurser to pnemonia
PNEUMONIAS Card 1
"infection in the lungs", bacterial, fungal, or viral
Predisposing factors: decreased cough reflex, tube in respiratory track, smoking, immobility, U.R. infections, chronic disease, & immunosuppresed
PNEUMONIAS Card 2
Organisms enter lungs by:
1. inhalation
2. aspiration
3. hematoygenous (via blood, uncommon)
Classified by CAUSITIVE AGENT:
1. lobar (entire lobe consolidated)
2. bronchopnemonia
3. interstitial (in alveolar walls)
BACTERIAL PNEUMONIA
90% Community Aquired
Sx: chills, fever, chest pain, rusty sputum, & ~ 12 hrs later cough
X-ray: diagnostic, consolidation of lung/lungs
Lab: elevated WBC count, neutrofils dominate, increased O2 levels, sputum sample
Tx: antibiotics
Bateremia
bacteria moves into blood stream
VIRAL PNEUMONIA
COMMON CAUSE IN CHILDREN.
Sx: vague: headache, fever, dry cough
Tx: none. no antibiotics
(antibiotics preventative for secondary infection)
HYPOSTATIC PNEUMONIA
RARE, result of no movement.
Fungal Infections
Diagnose: w/ skin tests & sputum cultures
Tx: 6-8 wks
ASPIRATION PNEUMONIA
Pt aspirates orophayngeal drainage, food/water from mouth or GI contents
TUBERCULOSIS Card 1
High Risk in: immuno suppressed, lower income, close living situations, elderly, & alcoholics
AFB: myobacterium is gram + acid fast bacilli
Mode of transmission: droplets
Sx: fatigue, weight loss, late afternoon fever, night sweats
TUBERCULOSIS Card 2
Skin Testing: does not tell us active vs. exposure
redness:
10 or more mm = +
5-9 mm = "doubtful" repeat test
0-4 mm = negative
TUBERCULOSIS Card 3
Sputum Culture: takes 2-4 wks, definitive mechanisms for diagnosis
Sputum Smear: heavy growth needed, not as accurate
X-ray: lesion in the lungs would appear
Tx: combination therapy, 2 or more drugs, meds taken for active dz until 6 months after sputum culture converts to -
CANCER IN THE LARYNX
- common in smokers, men, & air polution exposure
Sx: cough, clears throat freq., enlarged lymph nodes
Dx: laryngoscopy, biopsy of growths
Tx: depends on extent of cancer (remove, radiate, chemo)
CANCER IN THE LUNGS Card 1
Causitive Factors
1. SMOKING- 85%
2. increased risk in some occupations
3. diet
4. familial
5. passive smoke
CANCER IN THE LUNGS Card 2
Patho: classified by cell type
Sx: cough, increased sputum, hemoptysis, dyspnea, aching in chest, anorexia, fatigue, & wt loss
Dx: x-ray, CAT scan
CANCER IN THE LUNGS Card 3
Tx: SURGERY tx of choice combined with radiation & chemo
BLUNT TRAUMA
MOST COMMON FX OF RIBS

little damage externally, but internal damage

commonly caused by car accidents, fall, crushing injures
PENETRATING TRAUMA
sharp foreign object penetrates chest wall

(knife, bullet, "running with scissors"
CHEST TRAUMA
2 kinds: blunt & penetrating
Sx: pain on inspiration, splinting, pneumonia
Tx: prevent pain so will breathe normally
FLAIL CHEST
fx of multiple & adjacent ribs

injured area moves inward on inspiration & bulges out on expiration

Tx: O2 & stabilization
PNEUMOTHORAX
Define: accumulation of air in the pleural space which may collapse lung
Sx: DOE, increased resp., increased pulse, deminished breathe sounds
Tx: goal is to remove air from pleural space & reestablish - pressure so that lung can expand
CLOSED PNEUMOTHORAX
1. BLUNT TRUMA PUNCTURES LUNG, RUPTURE IN VISCERAL PLEURA
2. ALVEOLAR OR BRONCHIAL DAMAGE CAUSES LEAKAGE OF AIR INTO PLEURAL SPACE
COMMON CAUSE: EMPHYSEMA, OVER GROWN ALVEOLI BURST
OPEN PNEUMOTHORAX
EXTERNAL OPENING THRU THE CHEST WALL (KNIFE, SURGERY, GUN)
TENSION PNEUMOTHORAX
PROGRESSION OF TRAPPED AIR
Sx: quick, deviated thorax
CHEST TUBES
THORACTOMY TUBES, may be inserted in surg., ER, bedside between parietal & visceral space
PULMONARY DISEASE
USUALLY FROM DVT IN LEG,
Classic Sx: sudden dyspnea, tachynea, tachycardia, restlessness
Dx: lung scan, pulmonary angiogram
Tx: O2, heparin, cardiac monitoring
Preventative: low does heparin or coumadin
PULMONARY EDEMA
ACCUMULATION OF FLUID IN THE ALVEOLI CAUSED BY AN INCREASE IN THE HYDROSTATIC PRESSURE IN THE PULMONARY CIRCULATION OR A DECREASE IN THE ONCOTIC PRESSURE OF THE PULMONARY CIRCULATION
Tx: reduce hydrostatic pressure
PLEURAL EFFUSION
FLUID ACCUMULATED IN PLEURAL CAVITY/LUNGS
Tx: w/ draw fluid w/ needle
Lower GI Tract Anatomy

SMALL INTESTINE
Function: responsible for COMPLETING DIGESTION & ABSORBTION of sugars, fatty acids, amino acids, H2O, electrolytes, & vitamins
20-25 ft long
3 segments: DUODENUM, JEJUNUM, ILEUM
Lower GI Tract Anatomy

LARGE INTESTINE = COLON
Funtion: ABSORBTION OF H2O & ELECTRLYTES, final digestion of protiens, & production of Vitamin K
5-6 ft long
BEGINS AT THE CECUM W/ APPENDIX, ASCENDING COLON, TRANSVERSE COLON, DESCENDING COLON, SIGMOID, RECTUM, ANUS
DIAGNOSTIC STUDIES FOR INTESTINAL DISEASES
1. proctoscopy, sigmoidoscopy, colonoscopy: direct visualization of bowel & means to obtain biopsy material
2. stool samples
3. barium swallow (upper GI series) & ba enema (lower GI series) x-rays taken after barium ingested
DIARRHEA
SYMPTOM, NOT A DISEASE
frequent watery stools
Cause: contaminated food & H2O, viral infection, meds, malabsorbtion syndromes, & stress
Leads to loss of fluids, electrolytes, & nutrients
Tx: commonly support pt. w/ IV fluid
CONSTIPATION
Occasional constipation caused by decreased fiber or fluid intake & lack of activity.
Other causes: disease of the colon & rectum, meds, spinal cord injury, & habitual laxative use
Sx: fullness, distention, nausea, & flatus
Impaction: dried stool in bowel
Tx: treat cause if possible (diet, stool softners, exercise)
"ACUTE ABDOMEN"
TEMPORARY DIAGNOSIS UPON ADMITTING
used for pts. w/ Sx suggesting an abdominal condition
Causes: appendicitis, bowel obstruction, gall bladder inflammation, cancerous condtion, ulcers
APPENDICITIS Card 1
1. obsruction of lumne of the appendix w/ feces
2. leads to inflammation & necrosis
3. perforation of the appendix
4. results in leakage of the infection into the peritoneal area
APPENDICITIS Card 2
Sx: EARLY- vague, nausea, slightly elevated temp., abd pain, NVD LATER- pain localizes at Mc Burney's pt, rebound tenderness
Tx: appendectomy
PERITONITIS
Inflammation of peritoneum from BACTERIA or CHEMICAL
Sx: abd pain, board-like rigidity of the abd, elevated temp, WBC, pulse, & rate, decreased B/P
Dx: (abd paracentesis)w/draw fluid from peritonial cavity w/ needle/syringe
Tx: identify & treat cause
GASTROENTERITIS
NVD, fever, elevated WBC
usually VIRAL in origin- can be caused by bacteria or protozoa
Tx: NPO till NV cease, food and electrolyte replacement
IRRITABLE BOWEL SYNDROME (IBS)

"SPASTIC COLON"
Chronic, non-infectious disorder, associated w/ stress
alternate b/t diarrhea & constipation
common in women
Tx: stress management, high fiber diet, meds
INFLAMMATORY BOWEL DISEASE

(CHROHN'S DISEASE,
ULCERATIVE COLITIS)
Define: recurrent inflammatory diseases of the intestines: chronis & unpredictable
ETIOLOGY UNKNOWN, but possibly related to infection, autoimmunity, & famialial tendency
ULCERATIVE COLITIS Card 1
Define: inflammation & ulceration of colon & rectum
MOVES IN A CONTINUAL PATTERN (rectum, up the sigmoid, up the colon)
NO INFLAMMATION IN SMALL INTESTINE
INCREASED RISK OF COLON CANCER
ULCERATIVE COLITIS Card 2
Manifestations: bloody diarrhea, cramps, pain, fever, wt loss, perforation of the bowel
Dx: colonoscopy reveals areas of scarring, barium enema
Tx: REMOVE ENTIRE COLON
CHROHN'S DISEASE Card 1

"REGIONAL ENTERITIS"
CAN AFFECT ANY PART OF THE BOWEL
Patho: NOT CONTINUOUS "SKIP LESIONS", AFFECTED AREAS ARE SEPERATED BY AREAS OF NORMAL TISSUE
"COBBLESTONE" APPEARANCE
FISTULAS ARE COMMON
CHROHN'S DISEASE Card 2
Sx: similar to ulcerative colitis w/ mild to severe diarrhea
Complications: scarring, obstructions, fistula formation
Dx: barium enema "string sign"
Tx: remove colon, doesn't cure
BOWEL OBSTRUCTIONS
GI contents cannot pass thru the bowel
INTESTINAL POLYPS
Define: little growths, most are found in the sigmoid colon & rectum
Tx: polyps are removed ("polypectomy") & biopsy performed to determine if malignat or benign
CANCER OF THE COLON & RECTUM Card 1
20% of all CA deaths in US
Patho: usually it is an adenocarcinoma, the cancer spread thru the wall of the bowel into the lymph & blood vessels & metabolizes via the blood & lymph, usually to the liver
CANCER OF THE COLON & RECTUM Card 2
Sx: R side of colon- pt may present w/ ANEMIA, weakness, fatigue, & occult bleeding.
Left side of colon (MORE COMMON)- changes in bowel habits, narrow stools, & rectal bleeding
Dx: digital rectal exam, sigmoidoscopy & colonoscopy
DIVERTICULOSIS & DIVERTICULITIS
Define: outpouching in the intestinal wall, may be related to chronic constipation
Sx: NO SYMPTOMS USUALLY
Tx: high fiber diets & metamucil
In acute situation, pt needs to be NPO or on clear liquids
HERNIA Card 1
Define: protusion of part of intestine thru an abnormal opening or weakened area in abd wall
Sx: bulging occurs in the area when the pt stands or strains
Tx: surgery called" herniorrhaphy, loop of the bowel is placed back where it belongs
HERNIA CLASSIFICATIONS
REDUCIBLE: protrusion of part of intestine goes back into place when pt lies down or manually presses on it
IRREDUCIBLE: the protrusion can't be replaced or reduced
STRANGULATED: loop of intestine b/c trapped in the herniated area which reduces both intestinal flow & blood supply to the bowel
INGUINAL, FEMORAL, & UMBILICAL HERNIAS
INGUINAL- MEN
FEMORAL- WOMEN
UMBILICAL- BABIES
HEMMORHOIDS
Define: varicosities in veins of anus & rectum
Prediposing factors: pregnancy, chronic constipation, straining to defecate. heavy lifting
Tx: hemorrhoidal oint, stool softners, sitz baths for comfort & clensing
Hemorrhoidectomy- surgery where hemorrhoids are ligated & removed
LIVER ANATOMY
ESSENTIAL FOR LIFE
LARGEST BODY ORGAN
receives blood from HEPATIC ARTERY & PORTAL VEIN
1st organ exposed to substances (meds, food)
Kupffer Cells
Found in the Liver
macrophages present to engulf bacteria & foreign substances
LIVER FUNCTIONS
8 GENERAL
1.FORMATION & EXCRETION OF BILE
2.METABOLISM OF FOODSTUFFS DELIEVERED BY THE PORTAL VEIN
3. PROTEIN METABOLISM
4. FAT METABOLISM
5. STORAGE
6. STEROIDS
7.DETOXIFICATION
8. "FLOOD & FILTER"
Liver Function: Metabolism of foodstuffs delievered by the portal vien after absorbtion from intestines.
A. GLYCOGENESIS
B. GLYCOGENOLYSIS
C. GLUCONEOGENESIS
A. glucose converted to glycogen & stored
B. glycogen broken back down to glucose as needed
C. synthesis of glucose from amino acids & fats in absence of glucose
Liver Function:
Protein Metabolism
1. manufacture of plasma proteins such as albumin
2. clotting factors, prothrombin, fibrinogen
3. urea formation- NH3 is formed from breakdown of amino acids. ammonia is synthesized into urea & excreted by kidneys & intestines
Liver Function: Fat Metabolism
Fat metabolism of cholesteral & tri-glycerides into fatty acids w/ ketones as end-product
Liver Function: Storage
Storage of fat soluable vitamins and minerals
Live Function: Steroids
liver inactivates & excretes aldosterone, estrogen, progesterone, testosterone, & glucocorticoids
Liver Function: Detoxification
drugs & other potentially harmful substances are reduced to inactive substances which can then be excreted by the kidneys
Liver Function: "Flood & Filter"
flood- blood backed up from the vena cava & liver becomes congested
filter- kupffer macrophage cells remove bacteria & debris
GALL BLADDER
STORES & CONCENTRATES BILE TILL NEEDED
HORMONE: CCK, released when food enters the duodenum, bile assists w/ digestion of fats, bile salts reabsorbed & returned to liver for re-use
PANCREAS
Endocrine & Exocrine
Exocrine- production of digestive enzymes: amylase, lipase, & peptidase
Enzymes empty into sm intestine thru CBD
Diagnostic Liver Tests
1. serum bilirubin
2. urine bilirubin
3. SGPT - elevated in liver dz.
4. Serum proteins - decreased in liver dz.
5. serum amylase & lypase - increased in obstruction or inflammation of pancreas
JAUNDICE aka icteris
elevated bilirubin
EXCESS PRODUCTION OF BILIRUBIN- due to increased destruction of RBCs
OBSTRUCTING JAUNDICE- bilirubin gets conjugated but the bile can't flow from liver
HEPATOCELLULAR JAUNDICE- problem w/ liver cells, causes a decreased uptake of bilirubin
Disease of the Liver:
HEPATITIS
inflammation of the liver
may be viral or toxic
Hep A thru Hep E
HEPATITIS A Card 1
SPREAD THRU FECAL - ORAL ROUTE
2 WKS B4 symp. occur
High incidence in: poor hygiene, food-bourne outbreaks, restaurant workers, food handlers, & day care centers
MAINLY SEEN IN CHILDREN
HEPATITIS A Card 2
Onset: abrupt onset of symp., course of 1-2 months
INCUBATION PERIOD: 28 - 30 days
Dx: based on symp. & blood test
Prevention: HANDWASHING!
HEPATITIS B
Spread thru: blood, body fluids (sex, IV drug use)
Incubation Period: 60-90 days
Onset: insidious (don't feel well)
Detection: blood work
Prevention: vaccine Hep B
HEPATITIS - ALL
Patho: inflammation of liver w/ areas of necrosis
Systemic effects: enlarged lymph nodes, jaundice, pancreatitis
During acute phase, increase in liver enzymes
Bilirubin- urine may turn dark B4 jaundice obvious on skin
Sx: asymptomatic to fatal
Dx: based on symtoms, exposure history, elevated bilirubin may persist for 6 months
Tx: preventative care
Gall Bladder Dz. Card 1
cholecystitis
acute or chronic inflammation of the GB due to bile statis secondary to a stone (cholelithiasis)
Incidence: 4x more common in women, fam. hx, obese, diabetes, sedentary, elevated cholesterol levels
Gall Bladder Dz Card 2
sx: onset of RUQ pain, radiates to R shoulder due to colicky spasms, fever, clay colored stool, brown urine
Dx: ultrasouns
Tx: NG tube, meds, antibiotics
Surgical: cholectstectomy
Traditional- upper abd incision, T-tube
Laproscopic- 24 hrs, no acute inflammation
PANCREATITIS Card 1
Define: inflammation of the pancreas, ass. w/ GB or liver dz., trauma
ESP. COMMON IN ALCOHOLICS
Sx: sudden pain in epigastric area, radiates to back, NV, abd rigidity, decr. BS, fever, elev. WBC elev. serum amylase
PANCREATITIS Card 2
tx: eliminate causative factor, rest pancreas to decrease enzyme production by keeping NPO, NG tube to suction
Pain relief w/ demerol, NOT morphine