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79 Cards in this Set

  • Front
  • Back
Right Sided HF symptoms are seen where
Peripherially
What is Cardiac Output?
volume of blood the heart expels per minute
Dialated Cardiomyopathy
*Impaired pumping action of the heart
*Most cases
*Causes include ETOH abuse and chemo
Pts. look like HF pts
S/S OF CARDIAC TAMPONADE
BECKS TRIAD

1. Hypotension
2. JVD w/ clear lungs
3. Paradoxical Pulses
Extra complications of Pericarditis
Pericardial Effusion & Cardiac tamponade
NURSING CARE FOR PERICARDITIS
Assess pain - MI or Pericarditis?
Auscultate for friction rub
Position
NSAIDS
Assess for pulsus paradox & other S/S of cardiac tamponade
Central Venous Pressure (CVP)
CVP measures preload and is used for the RIGHT side of the heart.
NORMAL VALUE: 2-8 mmHg

ELOW 2: dehydration, blood loss
ABOVE 8: Fluid Overload
clients with angina should seek medical attention if chest pain lasts longer than
20 min
do not give NTG to patient who presents with angina whose SBP is <
90
meds for angina
-Heparin
-aspirin
-beta blockers
-ca+ channel blockers
-nitrates (NTG)
during anginal pain episode, EKG shows
-ST depression

-inverted T
1 risk for patients with aortic aneurysm
hypovemia --> shock

-monitor VS
-I&Os
-Lab results
-S/S of shock
drug therapy for abdominal aortic aneurysm
- Oxycodone
-B-blockers (metoprolol, propanolol)
-anti-hypertensives (minipress, Nitropress, NTG)
/S of abdominal aortic aneurysm
-abdominal mass to the left of midline
-abdominal pulsations
-bruits over the site of aneurysm
-diminished femoral pulses
-Lower back/abd pain
-SBP in legs lower than SBP in arms
dissecting aneurysm
aneurysm vessel wall ruptures and a blood clot is retained in an outpouching of tissue
Pulse Deficit
Radial pulse rate falls behind apical rate
What dx are usually associated with S4 sounds?
HTN, CAD, aortic stenosis, cardiomyopathy
Prinzmetal’s (variant) Angina
a variant form that is characterized by chest pain during rest and by an elevated ST segment during pain and that is typically caused by an obstructive lesion in the coronary artery
Unstable Angina
pain characterized by sudden changes (as an increase in the severity or length of anginal attacks or a decrease in the exertion required to precipitate an attack) especially when symptoms were previously stable
What are the actions of Adenosine and therapeutic uses?
Decreases automaticity, conduction velocity at the AV node.
Increases the refractory period at the AV node
Used in treatment of paroxysmal supraventricular tachycardia
Given as rapid IV bolus ONLY, followed by saline flush
Continuous telemetry monitoring
What are the adverse effects of lidocaine (Xylocaine)?
(Xylocaine)? Most common:
CV system (dysrhythmias and hypotension)
CNS (dizziness/lightheadedness, fatigue, and drowsiness)
Excessive levels can produce confusion and seizures. Monitor ECG continuously and switch to another dysrhythmic as soon as stable.
Atropine SO4
anticholinergic & Antiarrythmic
decreases HR, BP, systemic vascular resistance.
Vasopressin
smooth muscle contraction of all areas of the vascular bed. can increase BP in hypotensive pt
Lidocaine HCl
decreases ventricular excitability

Antiarrhythmic
Dobutamine HCL
ardiac stimulant, increases co, by improving stoke volume c min increase in hr, bp
Dopamine HCL
Vasopressure,cardiac stimulant corrects hemodynamic imbalances-bradycardia
condition in which fluid in the pericardial cavity creates enough pressure to cause cardiac compression
tamponade
What are the symptoms of dilated cardiomyopathy?
fatigue
weakness
palpitations
What can cause CHF?
coronary artery disease
past history of MI
untreated hypertension
valvular disease
cardiomyopathy
infection
heart defect
What are the symptoms of mitral valve prolapse?
many cases are asymptomatic

can cause:
palpitations
tachycardia
light-headedness
fatigue
dyspnea
inflammation of the endocardium, especially the cardiac valves, caused by bacteria, viruses, fungi, parasites, etc. that get into the bloodstream
nfective endocarditis
What are the signs and symptoms of shock?
tachycardia
very low BP
cold and mild skin
decreased capillary refill
low urine output
metabolic acidosis
What are the CM for cardiogenic shock?
impaired mentation
systemic and pulmonary edema
low cardiac output
dusky skin color
low BP
oliguria
ileus (intestinal obstruction)
dyspnea
What is the cause of hypovolemic shock?
loss of blood, plasma, or interstitial fluid in large amounts
What are the seven serum markers of myocardial damage?
Troponin (T and I)
- Creatine kinase (CK)-MB
- Myoglobin
- Serum lipids (Cholesterol, Triglycerides, HDLs, LDLs, HDL /LDL ratio)
- Homocysteine
- C-reactive protein (hsCRP)
- Blood coagulation tests (PT,PTT,INR)
Troponin (T and I)
- Creatine kinase (CK)-MB
- Myoglobin
- Serum lipids (Cholesterol, Triglycerides, HDLs, LDLs, HDL /LDL ratio)
- Homocysteine
- C-reactive protein (hsCRP)
- Blood coagulation tests (PT,PTT,INR)
serum markers of an mi
EKG changes:
What does ST depression indicate?
Ischemia
EKG changes:
What does ST segment elevation indicate?
Injury
lidocaine
antiarrhtyhmic
Conversion of PSVT to sinus rhythm
Contraindications

Second- or third-degree AV block, or sick-sinus syndrome. Atrial flutter. Atrial fibrillation. Ventricular tachycardia. Hypersensitivity to adenosine

Adverse Reactions

Facial flushing Lightheadedness Paresthesia Headache Diaphoresis Palpitations Chest pain Hypotension Nausea Metallic taste Shortness of brea
amiodarone
antiarrhymic
atropine
antarrhymic
Contraindications

Tachycardia Hypersensitivity Unstable cardiovascular status in acute hemorrhage and myocardial ischemia Narrow-angle glaucoma

Adverse Reactions

Tachycardia Paradoxical bradycardia when pushed slowly or when used at doses less than 0.5 mg Palpitations Dysrhythmias Headache Dizziness Anticholinergic effects (dry mouth or nose, photophobia, blurred vision, urine retention) Nausea and vomiting

Flushed, hot, dry skin Allergic reactions



Hemodynamically significant bradycardia Asystole give 4 this
procanimide
Indications

Suppressing PVCs refractory to lidocaine

Suppressing ventricular tachycardia (with a pulse) refractory to lidocaine

Suppressing ventricular fibrillation refractory to lidocaine when bretylium tosylate is not readily available

PSVTs with wide-complex tachycardia of unknown origin (drug of choice when associated with WPW)

Contraindications

Second- and third-degree AV block

Digitalis toxicity Torsades de pointes

Adverse Reactions Hypotension Bradycardia

Reflex tachycardia AV block

Widened QRS Prolonged PR or QT interval

PVCs Ventricular tachycardia, ventricular fibrillation, asystole

CNS depression Confusion Seizure
digoxin (Lanoxin)
cardiotonic - inotropic
Increases force of contraction, used in CHF
adenosine (Adenocard)
antiarrhythmic
atropine
antiarrhythmics - anticholinergics
Vasopressin considerations
Only given one time. May cause cardiac ischemia and angina. May replace first or second dose of Epi. Not recommended for responsive patients with coronary artery disease.
agnesium sulfate (electrolyte)

Indicated for?
Cardiac arrest if torsades or Hypomagnesemia
Lidocaine (antiarrhythmic)
V-Fib/Pulseless V-Tach, Stable V-Tach
1-1.5 mg/kg
Dopamine (catecholamine
Symptomatic Bradycardia, Hypotension
Atropine (parasympathetic blocker)

Indicated for?
radycardia
0.5 mg q 3-5 min as needed

PEA, Asystole
What is acute coronary syndrome?
Ischemic coronary events that may result in a blood supply and demand mismatch.
Stable and unstable angina pectoris.
Myocardial infarction.
Characteristics of stable angina?
INTERMITTENT chest pain (usually with exercise or exertion)
Episodes CONSISTENT in duration and intensity (usually only last a few minutes).
CONTROLLABE with medications (ie nitroglycerine).
Can develop into unstable angina.
Diagnostic studies for an MI:
Cardiac enzymes?
CK-MB elevated (>5ng/mL)

Troponin I (>0.1 ng/mL)

Myoglobin

Myoglobin levels peak earlier, but Troponin is most frequently used to diagnose
EKG changes:
What does ST depression indicate?
Ischemia
EKG changes:
What does ST segment elevation indicate?
Injury
EKG changes:
STEMI v NSTEMI:
STEMI characteristics?
ST elevation MI

Tends to be more extensive

COMPLETE coronary artery occlusion
Medical management of MI:
How do you--
reduce afterload?
nitroprusside for short term

ACE inhibitors and ARBs for longer term
Medical management of MI:
How do you--
decrease preload
nitrates, morphine, diuretics
Heart Valves:

Name the two atrioventricular.
mi tri
Mitral valve regurgitation:
Clinical Manifestations:
Chronic:
List 5 possible manifestations.
Asymptomatic/no Sx

2. Weakness, general malaise

3. Dyspnea (orthopnea/pnd)

4. PERIPHERAL edema (advance Sx)

5. S3 and murmur at apex
Mitral Valve Stenosis:
Clinical manifestations:
Name 4
1. Dyspnea (hemoptysis)

2. Palpitations (A fib)

3. Diastolic murmur (apex)

4. Predisposed to EMBOLI
CVP (Central Venous Pressure)
What does it measure?
The pressure of blood within the right atrium.
Used to approximate the right ventricular end diastolic pressure (RVEDP
CVP:
Normal values?
High indicates?
Low indicates?
Normal 2-6 mmHg
High=overhydration, increased venous return or right sided cardiac failure.
Low=hypovolemia or decreased venous return
Mean Arterial Pressure(MAP):
What is it?
Normal range?
Reflects changes in the relationship between cardiac output (CO) and systemic vascular resistance (SVR) and reflects the arterial pressure in the vessels perfusing the organs.
70-100 mmHg
Platelets
140,000-340,000/mm3
PT
10-12
INR
2-3 for patients on coumadin therapy
no P waves on the ECG
svt
cardiac tamponade
medical emergency condition where liquid accumulates in the pericardium in a relatively short time. The elevated pericardial pressure prevents proper filling of heart cavities. Instead of reducing the filling of both ventricles equally, the septum of the heart will bend into either the left or right ventricle. The end result is low stroke volume, shock and often death
True/False a pacemaker is senstivie to magnetic fields
True, should not be exposed to these fields for more than a few seconds, avoid welding or chain saws, no gasoline powered tools, carry medical alert especially in airports inorder to be hand searched.
Arteriosclerosis
hardening that affects the small arterioles
Teaching on Beta blockers
symptoms may worsen and may take a few weeks before improvement in cardiac status, meds are titrated slowly
signs of hypokalemia
weak pulse, faint heart sounds, hypotension, muscle flabbiness, diminished deep tendon reflexes and generalized weakness
signs and symptoms of hyponatremia
pprehension, weakness fatigue, malaise, muscle cramps and twitching and a rapid thready pulse
dobutamine (Dobutrex) action
ncrease cardiac contractility.
A vasopressor is
a drug used to constrict the arteries and increase blood pressure. These agents include dobutamine, dopamine, epinephrine, isoproterenol, norepinephrine, phenylephrine and ephedrine. They are most commonly used in intensive-care and post-operative hospital settings for patients with critical hypotension (low blood pressure)
Right sided heart failure
pedal edema
ascites
hepatic congestio
Drug classes of
verapamil (Calan)
Calcium channel-blocker
- Antianginal agent
- Antiarrhythmic
- Antihypertensive
Inhibits the movement of calcium ions across the membranes of cardiac and arterial muscle cells
Desired actions of
verapamil (Calan)
1. decreased cardiac workload
2. decrease oxygen demand
3. decreased cardiac energy consumption
4. increased delivery of oxygen to myocardial cells
SVT
too!