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15 Cards in this Set

  • Front
  • Back
course of the ureter
anterior to external iliac, posterior to the uterine artery (in women)
degraded tetracycline
associated with fanconi syndrome (proximal tubule reabsorption dysfunction that causes tubular dysfunction)
antihypertensive of choice in diabetes
ACE inhibitors
location of JGA
afferent arteriole
paradoxical aciduria
acidic urine in the presence of metabolic alkalosis - primarily due to hypokalemia in the presence of volume contraction
medication contraindicated in patients with renal artery stenosis
ACE inhibitors
amphetamine excretion in urine
amphetamine is a weak base so you want to acidify the urine to put it in its ionized form, use ammonium chloride
urachal cyst or sinus
remnant of the allantois that continues to drain urine from the bladder into the abdominal wall; allaNtois gives rise to the mediaN umbilical ligament
mechanism of furosamide induced calcium excretion
normally K+, Na+, 2Cl- in; Na+ out and K+ in via basolateral Na/K ATPase; K+ out via BL and apical K+ channels; Na+/K+ transports three Na out and 2 K in, results in net cation out --> makes inside of cell negative relative to the tubular lumen; inhibiting this transporter therefore ablates this positive tubular charge gradient and decreases driving force of Ca back into the cells
ace inhibitors with K-sparing diuretics
dangerous levels of hyperkalemia secondary to the inhibition of aldosterone
effect of ANII on renal arterioles
at low concentrations preferentially contricts efferents, at high concentrations (as in hemorrhage) constricts both afferents and efferents and decreases GFR
bence jones proteins
kappa light chains
Arginine vasopressin
ADH (another name)
digoxin toxicity
severe bradycardia in a patient with complete heart block, increase in furoemide would increase K+ secretion in nephron, causing hypokalemia, which potentiates the effects of digoxin
interaction between furosemide and digoxin
furosemide --> hypokalemia which potentiates the effects of digoxin