• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/31

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

31 Cards in this Set

  • Front
  • Back

-spirometry


-what do you measure it with, and how does it work

-pulmonary function test to determine the amount and rate of inspired and expired air


-electronic spirometers- act as air flow transducers

spirogram

lung volumes and capacities

-tidal volume (TD)


-inspiratory reserve volume (IRV)


-expiratory reserve volume (ERV)


-residual volume (RV)



-the amount of air inspired and expired during each cycle


-amount of air you can force in after normally breathing in - THE MAX


-opposite to IRV


-amount of air left in your lungs after forced exhalation that can't be accessed, cant use spirometer on it





-capacity measures mean...


-vital capacity (VC)


-inspiratory capacity


-functional residual capacity (FRC)


-total lung capacity (TLC)



-capacities of two or more lung volumes


-max amount of air that can be exhaled after a max inhalation (IRV, TV, ERV)


- max amount of air that can be forcibly inhaled (TV, IRV)


- amount of air left in the lungs after a normal exhalation (RV, ERV)


- amount of air in the lungs after max inspiration (TV, IRV, FRC -or- RV, VC)

minute (total) ventilation


alveolar ventilation

-total air moved into respiratory system per minute


-into alveoli

FEV1


FVC



-forced expiratory volume- amount of air you can breathe out in 1 second


-amount of air you can force out after max inspiration as fast as possible

3 main patterns in a spirometery test

normal, obstructive, restrictive

obstructive pattern


FEV/FVC


name the diseases

-shortness of breath due to difficulty in exhaling a normal amount of air. Breathing is slower and a large amount of air remains in the lung


FEV more reduced than FVC. FEC/FVC<0.7


-bronchial asthma, cystic fibrosis, obstructive


pulmonary disease

restrictive pattern


FEV/FVC



-can't fill lungs with enough air, lungs are


restricted from fully expanding (usually due to stiff lungs, nerve damage or weak muscles)


-FEV and FVC reduced and VC. REV/FVC normal





helium dilution method

helium insoluble in blood


after several breaths, helium equilibrates


C2 measures after expiration



static properties of the lung

mechanical properties when no air is flowing


PIP, PTP


static compliance of the lung


surface pressure of the lung

dynamic properties of the lung

mechanical properties of the lung when air is flowing in and out and when the lung volume changes


PAV


dynamic compliance of the lung


airway and tissue resistance

-ventilation


-bulk flow


-Boyle's law

-exchange of air between atmosphere and alveoli


-air moves from high to low pressure


-at room temp, pressure and volume are


inversely proportional

parietal pleura


visceral pleura

parietal: cover thoracic wall and superior diaphragm


visceral: cover the lungs

2 resistive forces

inertia of respiratory system


friction

3 airflow patterns

-laminar flow (small radius, distal from terminal bronchioles


-transitional airflow (bronchial tree)


-turbulent flow (highest resistance, highest radius, highest velocities, big airways)



laminar flow and poiseuilles law

resistance proportional to the viscosity and length of tube, but inverse to the radius



air moves in and out of lungs due to variations in which 3 pressures

P(IP)


P(ALV)-dynamic


P(TP)- static

PTP responsible for what

keeping the alveoli open (pressure gradient across alveolar wall). Always want it to be above 0. Does not cause airflow, but affects lung volume. P(AV) will cause air flow

steps in inspiration

-diaphragm and inspiratory intercostals contract


-thorax expand


-P(IP) more sub atmospheric


-P(TP) gets higher


-lungs expand


-P(ALV) are sub atmospheric


-air moves in

expiration

exact opposite

in diseases, small airways play a larger role in determining airflow resistance than large ones (usually other way around). why (3)

get blocked by


-smooth muscle contraction


-edema


-mucous

lung compliance

change in lung volume / transpulmonary pressure


(static or dynamic)

when is static lung compliance measures

FRC (end of breath)

dynamic compliance

P(TP) always changing


reflects lung stiffness and resistance to airflow


always less or equal to static lung compliance

hysteresis

difference between inflation and deflation


compliance paths.


It exists because a greater pressure difference is required toopen a previously closed (or


narrowed) airway than to keep anopen airway from closing.

what happens with age

lower elastics and collagen= high compliance

emphysema

lower elasticity= higher compliance and alveolar wall destruction


time to fill and empty lungs is increased


dont need as much PTP

pulmonary fibrosis

collagen deposition=higher compliance


need more PTP

lung compliance determined by (2)

-elastic components (collagen, elastin)


-surface tension at the air-water interface at the alveoli (negative relationship) makes up 2/3 of the compliance

surface tension

a measure of the attracting forces acting to pull aliquid’s surface molecules together at an air-liquid interface.