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39 Cards in this Set
- Front
- Back
ECG boxes
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1 small box = 0.04 seconds; 1 large box - 0.2 seconds; 5 large boxes = 1 second
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p wave
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small deflection = spread of depol from SA node over atria. electrical representation of atrial systole
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PR segment
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brief baseline period btw P wave and QRS complex; results from delay at AV node. Little separation of charge because structures are very close together. .12-.25 seconds
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PR of greater than .2 seconds indicates
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1st degree AV block
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Q wave
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small neg. deflection due to spread of depol in IV septum; from bundle branches to apical end of septum. L to R.
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R wave
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large positive deflection due to spread of depolarization through ventricular apex;
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S wave
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small neg deflection; represents later stages of ventricular depol, spreading through L vent wall
- in avf, II, III |
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QRS complex
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spread of depolarization through ventricles; signals onset of ventricular systole. Should be less than .15 seconds
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ST segment
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period at baseline btw QRS and T wave. Time when most ventricular muscle is depolarized; no electrical signal b/c all cells are depolarized and contracting. no separation of charge
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T wave
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small, broad deflection due to repolarization of ventricular muscle.
+ in leads I, II - in leads AVR |
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repolarization direction
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from epicardium to endocardium; reverse of depol
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QT interval
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from start of QRS to end of T wave; duration of ventricular electrical systole
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Magnitude of electrical ECG signal
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determined by # of myocytes depolarized and degree of depolarization
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direction of electrical ECG signal
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location of depolarized cells and direction of depolarization spread
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If vector is in same direction as axis
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positive deflection
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if vector is oblique to axis
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only parallel component is recorded
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if vector is perpendicular to axis
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no deflection recorded
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if vector is in opposite direction as axis
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negative deflection recorded
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HR and QT inerval
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QT strongly impacted by HR. Slower HR means longer QT.
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Mean electrical axis
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direction of R wave; provides into on spread of depol through ventricles and about the position of the heart in the chest.
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Normal MEA
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QRS is + or zero in I, II or AVF and is neg in avr
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RAD value
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QRS is neg in I, pos in AVR; R vent hypertrophy
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LAD value
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QRS is neg in III, AVF, II; L ventricular hypertrophy
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sinus bradycardia
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all complexes evenly spaced, but less than 60 bpm. could be due to increased vagal tone, beta adrenergic blockage, excellent physical conditioning. treat w/ pacemaker if necessary
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sinus tachycardia
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all complexes evenly spaced, more than 100 bpm. could be due to decreased vagal tone, excessive adrenergic tone, electrolyte abnormality, CHF, amphetamines, caffeine
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sinus bradycardia and tachycardia based in
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SA node
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ectopic pacemaker
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rogue part of heart that starts to depolarize from rest during phase 4. could be due to ischemia, electrolyte imbalance, increased catecholamines, etc.
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premature atrial contractions
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arise from early sinus beat or ectpic pacemaker in atria. may see some abnormal p waves but most everything else normal. treat w/ beta blocker or Na or Ca channel blocker
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atrial bigeminy
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every other beat premature
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premature ventricular contractions
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arise from ectopic pacemaker in ventricle; abnormal QRS that is prolonged and has no visible p wave before it.
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bidirectional block
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prevents re-entry, not so problematic
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unidirectional block
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blocks anterograde signal, but retrograde signal can penetrate through, re-enter loop and activate normal path. leads to regenerative loop of excitation, independent pacemaker circuit. Tachycardia.
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3 things needed for re-entry loop
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obstacle to propagation, unidirectional block of impulse propagation, region of slow conductance
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supraventricular re-entry arrhythmias
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re-entry circuit around AV node becomes pacemaker; undetectable or inverted P wave.
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atrial flutter
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re-entry circuit in atria; rhythmic, high-freq oscillations instead of normal P wave. atrial rate above 200bpm; partial block in AV node may reduce ventricular rate.
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atrial fibrillation
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chaotic excitation of atria with coarse or fine oscillations; no detectable p waves. vent rate is variable and set by AV node or ventricular pacemaker
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1st degree AV block
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prolonged but uniform PR interval. minor defect.
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2nd degree AV block
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worsening of AV conduction; PR interval increases with time until beat is skipped. single p wave not followed by QRS. 2:1 block: every other p wave is followed by QRS.
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third degree av block
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complete block at AV node; ventricular rhythm is independent of atrial and is slow. QRS independent of P waves. treat w/ pacemaker.
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