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39 Cards in this Set

  • Front
  • Back
ECG boxes
1 small box = 0.04 seconds; 1 large box - 0.2 seconds; 5 large boxes = 1 second
p wave
small deflection = spread of depol from SA node over atria. electrical representation of atrial systole
PR segment
brief baseline period btw P wave and QRS complex; results from delay at AV node. Little separation of charge because structures are very close together. .12-.25 seconds
PR of greater than .2 seconds indicates
1st degree AV block
Q wave
small neg. deflection due to spread of depol in IV septum; from bundle branches to apical end of septum. L to R.
R wave
large positive deflection due to spread of depolarization through ventricular apex;
S wave
small neg deflection; represents later stages of ventricular depol, spreading through L vent wall
- in avf, II, III
QRS complex
spread of depolarization through ventricles; signals onset of ventricular systole. Should be less than .15 seconds
ST segment
period at baseline btw QRS and T wave. Time when most ventricular muscle is depolarized; no electrical signal b/c all cells are depolarized and contracting. no separation of charge
T wave
small, broad deflection due to repolarization of ventricular muscle.
+ in leads I, II
- in leads AVR
repolarization direction
from epicardium to endocardium; reverse of depol
QT interval
from start of QRS to end of T wave; duration of ventricular electrical systole
Magnitude of electrical ECG signal
determined by # of myocytes depolarized and degree of depolarization
direction of electrical ECG signal
location of depolarized cells and direction of depolarization spread
If vector is in same direction as axis
positive deflection
if vector is oblique to axis
only parallel component is recorded
if vector is perpendicular to axis
no deflection recorded
if vector is in opposite direction as axis
negative deflection recorded
HR and QT inerval
QT strongly impacted by HR. Slower HR means longer QT.
Mean electrical axis
direction of R wave; provides into on spread of depol through ventricles and about the position of the heart in the chest.
Normal MEA
QRS is + or zero in I, II or AVF and is neg in avr
RAD value
QRS is neg in I, pos in AVR; R vent hypertrophy
LAD value
QRS is neg in III, AVF, II; L ventricular hypertrophy
sinus bradycardia
all complexes evenly spaced, but less than 60 bpm. could be due to increased vagal tone, beta adrenergic blockage, excellent physical conditioning. treat w/ pacemaker if necessary
sinus tachycardia
all complexes evenly spaced, more than 100 bpm. could be due to decreased vagal tone, excessive adrenergic tone, electrolyte abnormality, CHF, amphetamines, caffeine
sinus bradycardia and tachycardia based in
SA node
ectopic pacemaker
rogue part of heart that starts to depolarize from rest during phase 4. could be due to ischemia, electrolyte imbalance, increased catecholamines, etc.
premature atrial contractions
arise from early sinus beat or ectpic pacemaker in atria. may see some abnormal p waves but most everything else normal. treat w/ beta blocker or Na or Ca channel blocker
atrial bigeminy
every other beat premature
premature ventricular contractions
arise from ectopic pacemaker in ventricle; abnormal QRS that is prolonged and has no visible p wave before it.
bidirectional block
prevents re-entry, not so problematic
unidirectional block
blocks anterograde signal, but retrograde signal can penetrate through, re-enter loop and activate normal path. leads to regenerative loop of excitation, independent pacemaker circuit. Tachycardia.
3 things needed for re-entry loop
obstacle to propagation, unidirectional block of impulse propagation, region of slow conductance
supraventricular re-entry arrhythmias
re-entry circuit around AV node becomes pacemaker; undetectable or inverted P wave.
atrial flutter
re-entry circuit in atria; rhythmic, high-freq oscillations instead of normal P wave. atrial rate above 200bpm; partial block in AV node may reduce ventricular rate.
atrial fibrillation
chaotic excitation of atria with coarse or fine oscillations; no detectable p waves. vent rate is variable and set by AV node or ventricular pacemaker
1st degree AV block
prolonged but uniform PR interval. minor defect.
2nd degree AV block
worsening of AV conduction; PR interval increases with time until beat is skipped. single p wave not followed by QRS. 2:1 block: every other p wave is followed by QRS.
third degree av block
complete block at AV node; ventricular rhythm is independent of atrial and is slow. QRS independent of P waves. treat w/ pacemaker.