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19 Cards in this Set
- Front
- Back
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T/F
nicotine is a long acting drug |
F
it is very short acting that is why it is so addictive |
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1/2 life of nicotine
a) 30 min b) 1 hr c) 2 hr d) 15 min |
30 min
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untrue statement about nicotine
a) it is a stimulant b) it is a depressant c) inhibits reuptake of catecholamine d) stimulates production of norepinephrine e) stimulates production of epinephrine |
inhibits catecholamines = cocaine
nicotine releases catecholamines -> cardiac arrhythmia |
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_____is a negative inotropic agent
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nicotine - slows contractile forces of the HT -> congestive HT failure
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Why should caution be used if a smoker taking meds suddenly decides to quite smoking
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nicotine increases metabolism of many drugs there a person may be needing higher doses when smoking
suddenly quiting could cause OD |
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true statement about nicotine
a) inhibits platelet function/clotting b) Increase CO retention c) increase tone of GI d) decrease motor activity of GI |
increase CO, increase tone = true
causes platelet clotting/thrombi increase motor activity of GI->diarrhea, |
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list the major chemical groups of marijuana
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cannabinoids- alter perception of time, distance, speed
alkaloids- increase HR, BO, serum glucose |
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T/F
adverse effects of marijuana are reversible after quitting use |
True-
2mo-2yrs req to rid THC from body tissue (will test + for THC with weight loss even after cessation of long term use) |
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acts by inhibiting catecholamine re-uptake into sympathetic nerve terminals
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cocaine
symp absorbtion cause descending CNS stim |
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False statement about cocaine
a) can be absorbed thru mucus membranes b) vasoconstrictive action limits absorption c) crack is lethal d) decreased absorption with inflammation |
increased absorption with inflammation
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adverse affect of Cocaine
a) precipitate thrombi b) NT depletion c) Korsakoff psychosis d) fomication |
NT depletion
Fomication (bugs) also, hi BP, V fib, convulsions thrombi- nicotine korsakoff- ethanol |
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Muscle relaxers typically work by affecting which NT
a) dopamine b) epinephrine c) acetylcholine d) none |
acetylcholine
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the actions of muscle relaxers take place in
a) pre synaptic terminals b) nueromuscular junction c) cerebral cortex d) synaptic cleft |
neuromuscular junction (NMJ)
cause depol or prevent repol |
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drug(s) - short acting neuromuscular blocking agent that depolarizes the NMJ to prevent muscle contraction
a) Succinylcholine b) Tubocurarain (tubarine) c) Vecurinium (Norcuan) d) Pacuronium (pavulon) |
succinylcholine
Non-depolarizing agents: Tubocurarain= long lasting vecuronium, pancuronium= derivatives of tubocurarain |
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Non depolarizing agent(s) that block nerve impulse transmission causing muscle relaxation/paralysis
a) Succinylcholine b) Tubocurarain (tubarine) c) Vecurinium (Norcuan) d) Pacuronium (pavulon) |
Tubocurarain (tubarine)
Vecurinium (Norcuan) Pacuronium (pavulon) |
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describe how non-polarizing muscle relaxing agents work
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compete w ACh at cholinergic receptor sites on skeletal muscle endplates and block nerve transmission
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local anesthetic
a) cocaine b) Nitrous Oxide c) Halothane d) Tubocurarine (tubarine) |
cocaine
also: (all end in ___aine) benzocaine, dibucaine, lidocaine, procaine NO2, halothane- general anesthetics tubocurarine (tubarine)- non depol muscle relaxant |
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General anesthetic
a) Halothane b) enflurane c) forane d) nitrous Oxide |
all
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short acting muscle relaxers often used to set up ventilation devices
a) pancuronium (pavulon) b) benzocaine c) vecuronium (norcuon) d) succinylcholine |
pancuronium (pavulon)
vecuronium (norcuon) tuburcurarine (tubarine) is a long lasting |
