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10 Cards in this Set

  • Front
  • Back
b adrenergic recep. blking drgs: rduce amt of nitroglyc, usd by pts, rduce inc. cardiac wk and oxy consum w/strss
both
ca cnl blking drugs like nifedipine: a. increas abililty of myocardium to utilize oxygen b. inhib coronary art spsm
b. inhibits coronary artery spasm
F abt nitroglycerine: alleviate acute at of angina pec; rdcs symp.act 2 hrt; rducs preld so dec. wk/oxy; prvnt angin w/ ex
F: reduces sympathetic nerve activity to heart
not beneficial effect of ACE inhib? rdced preload; rdcd afterld; inc intracell calc conc in cardiac myo; inc sod excr
iF: nc intracell calcium concentration in cardiac myocytes
in pts with ht failure, aldosterone receptor anatagonist would likely have what effect of digitalis glycosid
inhibit digitalis glycoside action
Digitalis glycosides: a. have wide margin safety; b. increase intracellular free Ca concentrati cardiac mycyts
b. increase intracellurlar free Ca concentration in cardiac myocytes
centlly acting HTN drgs; clonidine, have sev. unplsnt side eff; rdce symp outflw, almost a last resort
all of the above
thiazide dirutetics rduce bld press in htn pts by: stim production of renin by kid; reduce peripheral resis
reduce peripheral resistance
ACE inhibitors: blk AT1 recep; inc angtensII, rduce renin conc, reduce angiotensin II lvls, none of the above
ACE inhibitors reduce angiotensin II levels
beta 1 adrenergic receptr antago: directly dilate ven. bld vess; inhib ACE; reduce CO; cause prfnd diruesis
beta 1 adrenergic receptor antagonists reduce cardiac output