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60 Cards in this Set
- Front
- Back
classic symptom-cranping in the lower extramities
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Arterial intermittent claudication
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determined by the amount of docimented exercise that the pt can tolerate before pain actually occurs
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progression of arterial occlusion
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occurrs with exercise, relieved by rest
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ischemic pain
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persistant in forefoot when at rest..does not go away with rest,,when there is severe occlusion and ish.
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rest pain
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cool aple extremities, pallor with elevation, rubor with dependant positioning, cyanosis, shiny skin with brittle nails and hair loss
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arterial insufficiancy
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altered tissue perfusion
risk for inpaired skin integrety pain-due to insufficient o2 |
Nursing Dx=atrial insuff
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aching cramping pain
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venous
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diminished or absent pulses
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arterial
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pigmentation in gaitor area, skin thickened and tough, may be reddish blue, associated with dermatitis
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venous
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pulses present, may be diffacult to palpate through edema
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venous
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granulation tissue--beefy red to yellow in chronic long term ulcer
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venous
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pale to black and dry gangrene
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arterial
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circular ulcer
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art
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deep ulcers, often involving joint space
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art
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superficial ulcers
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venous
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warm enviornment, aviod cold, avoind hot, avoind restrictive clothing, don't cross legs, keep dependent, no smoking
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Nursing interventions..promoting tissue perfusion
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daily skin assess, gentle cleaning, moisturize, properly fitting shoes, protect feet-no barefoot, lose weaight, proper nutrition
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maintaining skin integrety
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increacing exercise tolerance-exercise develops collateral circu. slow progressive exercise 30-45 min.bid ROM
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reducing pain
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handheld doppler when pulsus are not palpable,
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doppler flow studies
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compares systolic BP of arm to BP of ankle, usually ratio of 1:0, if less then arterial insufficiancy is presesnt
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ankle-brachial index-ABI
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ultrasonic waves produce waveform with peaks and valleys, flattened waveform=obstruction
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duplex ultrasound
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treadmill 5 min or untill apin is disabling, ankle BP monitores
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exercise testing
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determine location and extent of disease, assess for contrast media uses contrast--allergies
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angiography
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maintain circulation through repair, VA and pulses frequently, color and temp, I+o, CVP, LOC, fluid imbalances, elevate legs, pressure dressings
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Post op care after vascular procedures
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smoking, HTN, diabetes, viruses, hyperlipidemia
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endothelial injury...causes of artherosclerosis
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platelets and monocytes aggragate at site, fatty streaks and fibrous plaques form, rupture causing thrombus
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artherosclerosis
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legs most oftern affected, severity depends on extent of obstructive lesions, confined to segments of aorta below renal arteries to popliteal art.
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arterial occlusive disease
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position below heart, exercise, postural exercise, sit in chair with feet flat on floor
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improve arterial circulation
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warmth with caution, stop smoking, stress managment, no cross legs, constrictive clothing, vasodialators, adrenergic blockers
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promoting vasodialation
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med that reduces blood viscosity
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Trental
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ASA, Persantine, Ticlid, Plavix
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anti-platelets
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vasodialators, adenergic blocking agents, CCB, trental, anti-platelet, meds for DM, HTN
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Meds for arterial occlusive disease
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ADA-low fat, low cholesterol, because of artherosclorosis
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diet for AOD
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chronic tissue ish, gangrene, necrosis, last resort when med. intervention fails
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amputation
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tissue is dry, cold, and black
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dry gangrene
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after trauma with infection..very bad odor
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moist gangrene
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obstructive vascular disorder cause by recurring inflammation in arteries and veins
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Buerger's Disease (Thromboangiitis obliterans)
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localized, episodic, vasoconstriction of disorder of small arteries in hands and feet that cause temp and color changes
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Raynaud's Disease
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Thrombosis, embolisim, trauma, occurs suddenly nad w/o warning
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Acute arterial Occlusive Disease
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autoimmune vasculitis, inflammatory response, white cells infiltrate, fibrosis occurs w/healing causing occlusion
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Buergers
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rheumatic heart disease, artificial heart valves, MI, AFib, vascualr surgeryinvasive arterial procedures, trauma or compression of artery
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risk factors for acute arterial occlusive disease
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necrotic lesions at tips of fingers and toes, see areas that are inflammed then they become necrotic
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Buergers
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winter, lupus, RA, trauma or obstruction, cooupational trauma--typistis, pianists, cold, stress, caffine, tobaco
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Risk factors--Raynauds
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pain, pallor, pulselessness, parasthesia, poikilnthermia, paralysis
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6 p's of NV assess. Acute Occ. art. disease
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bilateral, instep claudication-main symptom! intense rubor, cyanosis,rest pain, diminished or absent pulses
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S/S Buergers
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stop smoking, CCB or antiplatelet, sympathectomy to eliminate vasospas. amputation.BKA vs. toe
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Buergers
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symmetrical and bilateral, usually tip, not thumb, vasospas. in fingers, pallor early, then reddness as blood returns..very painful
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Raynauds
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emergency embolectomy, balloon angioplasty, artherectomy stents, bypass
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trestment of Acute occlusion
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circumferential dialation, relatively uniform in shape
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fusiform
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localized outpouching on one side
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saccular
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tear in the intima layer with accumulation b.t the intima and the media layer
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dissecting
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elderly, female, immobility, increaced viscosity, intimal damage, oral contraceptives
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rick factors for DVT
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pain or tenderness, edema, reddness, fever, +Homans
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s/sDVT
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promanant, abmormally dialated veins
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varacose
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pregnancy, obsiety, prolonged standing, chronic diseases
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varacose veins..rick fac.
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inadequate venous return over a long period of time, chronic pooling of blood leads to hyperpigment and edema over ankles
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chronic venous insufficinecy
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used for small, localized vari., agent in injected into the vein (NS)
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sclerotherapy
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dyspnea, hypotension, tachy card./tacypnea, hypoxemia, hemoptysis, chest pain
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S/S pulm embolus
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increaced venous tension, and valve imcompatence leads to venous stasis, poor venous return, edema and ulceration
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venus ulcer path
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irregular margins, copious serous exudate, occurrs over the medial or lateral malleolus(ankle bone)
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Venous ulcer
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