Use LEFT and RIGHT arrow keys to navigate between flashcards;
Use UP and DOWN arrow keys to flip the card;
H to show hint;
A reads text to speech;
115 Cards in this Set
- Front
- Back
Differential Dx: Corneal Abrasion
|
Ocular/adnexal foreign bodies
Corneal Ulcer Corneal laceration |
|
Differential Dx: Blepharitis
|
Chalazion
Hordeolum Rosacea Blepharitis |
|
Differential Dx and Symptoms: Viral Eyelid Disease
|
Diff: Impetigo
vesicular lesions with an erythematous base Fluorescein dye and cobalt blue light |
|
Differential Dx: Eyelid Ptosis
|
Congenital v. traumatic v. neurogenic ptosis, Horner Syndrome
|
|
Differential Dx: Ophthalmia Neonatorum
|
May be bacterial, or viral or due to tx w/silver nitrate prophylaxis at birth, Chlamydia - most common,
Gonorrhea - threatens vision HSV - possibly systemic |
|
Buphthalmos
|
Abnormal enlargement of eye
Seen in Congenital Glaucoma; buphthalmos is glaucoma until proven otherwise |
|
Nasolacrimal Duct Obstruction Treatment, Outcomes, and Risks
|
Massage to remove debris (debatable),
Topical antibiotics, Surgical tx - probing, effectiveness decreases after 1 year of age 90% spontaneously resolve in 1 year Monitor for possible glaucoma If light sensitivity and blepharospasm present - urgent referral to ophthalmologist (Risks of Superinfection, Dacryocystitis, preseptal cellulitis, orbital cellulitis) |
|
Retinopathy of Prematurity Tx
|
Follow up every 1-2 wks
Surgical tx necessary within 72 hours to reduce bad visual outcome if disease progressed. However, often cases don’t require tx and ROP doesn’t progress to retinal detachment. |
|
Amblyopia - 3 types
|
kinds: 1. Strabismic 2. Refractive 3. Deprivation (causes worst visual loss)
Sequalae (leads to)- permanent blurry vision, vision loss, |
|
Retinoblastoma
|
the most common primary intraocular malignancy of childhood
-usually presents before age 3 |
|
Periorbital Cellulitis
|
often present with erythematous and edematous eyelids, pain, and mild fever. The vision, eye movements, and eye itself are normal (unlike orbital cellulitis)
Perform H test to R/o orbital cellulitis IV Abx |
|
Orbital cellulitis
|
Decreased vision, restricted eye movements, and an afferent papillary defect suggest orbital cellulitis.
Orbital cellulitis presents with signs of periorbital disease as well as proptosis (a protruding eye), restricted eye movement, and pain with eye movement. Fever is usually hig |
|
Orbital Tumors
|
Both benign and malignant orbital lesions occur in children.
-The most common benign tumor is capillary hemangioma -The most common primary malignant tumor of the orbit is rhabdomyosarcoma. |
|
Symptoms of Orbital Tumors
|
proptosis,
orbital ecchymosis (raccoon eyes), displacement of the globe |
|
Tx of Orbital Tumors
|
- Treatment is indicated if the lesion is large enough to cause amblyopia
-Induced astigmatism or amblyopia (or both) are treated with glasses and patching, respectively. -Treatment of orbital dermoids is by excision -Treatment of metastatic disease requires management by an oncologist |
|
Nystagmus
|
Oscillation of eyes
|
|
Recurrent Aphthous
|
small ulcers (3-10mm) on inner aspect of lips or tongue, rarely on tonsils or palate. No fever or cervical adenopathy. Painful and lasts 1-2 weeks. May recur several times throughout life. PFAPA = episodes last 5 days without other URI symptoms
Recurrent = canker sores |
|
Herpes Simplex Gingivostomatitis
|
10+ ulcers, fevers, cervical adenopathy, and inflammation
<3 y/o, lasts 7-10 days If caught early, tx w/Acyclovir Usually pain --> dehydration --> hospitalization required |
|
tx of Acute Stomatitis
|
coat leasons with corticosteroid ointment (triamcinolone). Reduce pain by bland diet (no acidic, salty food or juice) or acetaminophen/ibuprofen. PFAPA = cimetidine treatoment or tonsillectomy.
Refer to otolaryngologist - if PFAPA suspected |
|
Acute Stomatitis: Herpes Simpex
|
initially: 10+ small ulcers of buccal mucosa, anterior tonsillar pillars, inner lips, tongue, gingival. Posterior pharynx usually spared. Associated with fever, tender cervical adenopathy, generalized oral inflammation preceding the ulcers
|
|
Acute Stomatitis: Thrush
|
white curdlike plaques, predominantly on buccal mucosa (can’t wash away
|
|
Hx of Thrush
|
affects infants or older children in debilitated state. Use of broad-spectrum abx, systemic/inhaled corticosteroids, HIV + (erythematous candidiasis, producing red patches on palate and dorsum of tongue usually assoc with abx use, corticosteroids and HIV)
|
|
Acute stomatitis: traumatic ulcers
|
trauma due to biting by molars (mechanical), thermal trauma (hot food), chemical trauma (contact with aspirin). Leukemia or cyclic neutropenia
|
|
Pharyngitis: acute viral and tonsillitis (can’t often distinguish causative viral agent):
|
all: sore throat, fever (90% of sore throats and fever due to viral infection). Some have ulcers (see PhysEx)
Types: 1. infectious mono - palpable speen, increased IgM's, >5 y/o, axillary adenopathy, dx with EBV serology 2. Herpangina - 3mm ulcers surr. by halos, Coxsackie A grp. viruses, self-limiting 3. Hand Foot Mouth Disease - enteroviruses, rarely causes Encephalitis, ulcers in mouth, vesicles/papules on palms, soles, fingers, and buttocks 4. Pharyngoconjunctival Fever - Adenovirus - epidemic in kids, exudative tonsillitis, conjunctivitis, lymphadenopathy, fever, Tx is symptomatic. |
|
Patho of Pharyngitis
|
all: sore throat, fever (90% of sore throats and fever due to viral infection). Some have ulcers (see PhysEx)
|
|
Phys Exam of Pharyngitis
|
M: exudative tonsillitis, cervical adenitis, fever, palpable spleen or axillary adenopathy. H: ulcers (3mm) surrounded by halo on anterior tonsillar pillars, soft palate and uvula. HFMD: ulcers anywhere on mouth; vesicles, pustules or papules on palms, soles, interdigital areas and buttocks. PCF: exudative tonsillitis, conjunctivitis, lymphadenopathy, fever
|
|
Tx of Pharyngitis
|
all tx is symptomatic IM: no amoxicillin bc produces rash.
HFMD - can lead to encephalitis |
|
Pharyngitis: bacterial
|
Pharyngitis: bacterial
|
|
Recurrent Rhinitis Allergic
|
intranasal corticosteroids, oral and intranasal antihistamines, leukotriene antagonists, decongestants, nasal saline rinses
|
|
Rhinsosinusitis: Recurrent/Chronic
|
ABRS clears bur recurs with each or most URI = recurrent. Child does not clear infection in expected time, but acquires no acute complications = chronic. Allergies, anatomic variations (causing obstruction, eg septum deviation) and disorders in host immunity. Gastroesophageal reflux could contribute to chronic ABRS
If allergic polyps found in child<10 --> cystic fibrosis workup |
|
Acute Bacterial Rhinosinusitis
|
Bacterial infection of paranasal sinuses; almost always preceded by viral URI
gradual or sudden onset of symptoms that last less than 30 days: nasal drainage, nasal congestion, facial pressure or pain, postnasal drainage, hyposmia or anosmia, fever, cough, fatigue, maxillary dental pain, ear pressure or fullness |
|
Acute Viral Rhinitis
|
Common Cold
clear or mucoid rhinorrea, nasal congestion, sore throat, possible fever. Symptoms resolves within 7-10 days. Nose, throat and TM may appear red and inflamed. Nasal secrections more purulent after day 2 (color change should not be assumed to indicate bacterial rhinosinusitus) |
|
Acute trauma to middle ear - Tx
|
1. Mainly watchful waiting.
2. Antibiotics are not necessary unless there are visual signs of infection. 3. If accompanied with vertigo, refer to otolaryngologist and should be attended to more urgently. Refer ! |
|
Mastoiditis
|
1. postauricular pain
2. ear protrusion with outwardly displaced pinna. 3. fever present 4. Mastoid is swollen and red. 5. AOM is almost always present (acute otitis media). Can have brain abscess, persistent headaches, recurring fever. |
|
Mastoiditis Presentation
|
If acute: diffuse inflammatory clouding of mastoid cells occurs in an uncomplicated AOM.
Postauricular pain and swelling (ddx external otitis) Body destruction and resorption of mastoid air cells occurs in severe AOM cases. |
|
Mastoiditis
|
If acute: diffuse inflammatory clouding of mastoid cells occurs in an uncomplicated AOM.
Body destruction and resorption of mastoid air cells occurs in severe AOM cases. |
|
Mastoiditis Complications
|
Meningitis is a complication of acute mastoiditis. Meningitis Sx are high fever, stiff neck, severe headache...diagnosis found by lumbar puncture.
Can progress to sigmoid sinus thrombosis, epidural abscess, or intaparenchymal brain absess |
|
Mastoiditis Complications
|
Meningitis is a complication of acute mastoiditis. Meningitis Sx are high fever, stiff neck, severe headache...diagnosis found by lumbar puncture.
Can progress to sigmoid sinus thrombosis, epidural abscess, or intaparenchymal brain absess |
|
Snoring, mouth breathing and upper airway obstruction
|
Nightly snoring or mouth breathing. Unrefreshed sleep, behavioral problems, daytime fatigue.
- Enlarged (4+) tonsils Recurrent tonsillitis tonsillectomy or polysomnography |
|
Dacryostenosis
|
Obstructed nasolacrimal duct
tears build up on eye Usually since birth Recurrent conjunctivitis Tx: duct massage, moist heat, eyelid cleaning, topical ABX prn if persistent, ophthalmology referral, tube placement to open up adhesed duct |
|
Blepharitis Peds Tx
|
• management
o antibiotics o gentle cleaning with gentle soap o conservative approach Monitor for corneal changes associated with dry eye |
|
Chalazion Tx
|
conservative management
• warm compress few times day • time to go away • significant red/pus – topical antibiotics prn (NOT oral) • 2 months doesn’t go away – opthalmologist can remove them |
|
Ophthalmia Neonatorum
|
o Newborn conjunctivitis
• 1st month of life • contracted at birth from mother (vaginally) o Chlamydia (CT) – most common • Prophylaxis is NOT effective need oral antibiotics • culture from eye d/c and nasopharynx • 1-2 wks post-natal presentation subtle • 50% acquisition rate if mom infected (vaginal birth) • long term effects - scarring blindness |
|
Tx - Ophthalmia Neonatorum
|
• management
screen pregnant patients before delivery • regular prenatal care? o vaginal swabs screening for infection – less likely to have undiagnosed infection oral macrolide antibiotics -azithromcyin, e-mucin • has to be ORAL antibiotics • would progress over time if untreated |
|
Gonorrheal Conjunctivitis
|
• More rare
Prophylaxis is usually effective • Typical age 1-7 days (EARLIER – more acute/severe) • corneal perforation and scarring • dissmeinated gonococcal dz • management - hospitalize, IV antibiotics |
|
HSV Keratoconjunctivitis
|
• Rare
• onset 3 days - 3 weeks • corneal lesions, scarring, recurrent outbreaks • management – topical or oral antivirals, no steroids (may make it worse) |
|
Pt presents with: Pain/Foreign Body Sensation
|
i. Foreign body
ii. Corneal abrasion iii. Laceration iv. Acute infections of globe/ocular adnexa v. Iritis vi. Angle-closure glaucoma vii. Trichiasis viii. Contact Lens Misuse |
|
Pt presents with: Photophobia
|
1. Corneal abrasions
2. Foreign bodies 3. Iritis 4. Squinting of one eye in bright light → intermittent exotropia |
|
Pt presents with: no ocular pathology
|
a. Migraines
b. Meningitis c. Retrobulbar optic neuritis |
|
Pt presents with: leukocoria
|
1. Retinoblastoma
2. Retinopathy of prematurity 3. pupillary membrane 4. cataract 5. vitreous opacities 6. retinal detachment 7. Toxocara infection 8. Retinal Dysplasia |
|
Peds Ophthalmic Exam
|
i. Hx of poor vision/misalignment of eyes
ii. Visual acuity outside expected level iii. Abnormal red reflex (Infants) 1. Cataracts 2. Intraocular tumors iv. Abnorm pupil reactivity v. → refer to ophtalmologist vi. prompt tx vii. birth – 3 y/o 1. hx, vision assessment, inspection of lids and eyes, pupil exam, ocular motility, and red reflex check viii. 3 y/o + 1. everything PLUS visual acuity testing ix. Visual acuity 1. The MOST important test of visual fx |
|
Pathogens in Preseptal Infections
|
S. aureus and S. pyogens
|
|
Retinoblastoma
|
• The most common primary intraocular malignancy of childhood
o Age at presentation o DO: Red Reflex Check – white pupil, reflex absent • Etiology o Retinal cell mutation o Hereditary or sporadic • Sx o Single or multiple tumors o Unilateral or bilateral o leukocoria, strabismus, glaucoma, blindness, proptosis, red eye, pus in anterior chamber |
|
AOM Pathogens
|
• S pneumo – PCN drug resistance common
Most invasive pathogen May be decreasing • H influenzae – on the rise, but less invasive • M. catarrhalis • S. pyogenes |
|
Prevnar
|
pneumococcal vaccine (Prevnar) – to prevent certain strains of strep pneumo in ear infections in kids
|
|
1st line med for AOM
|
– HD Amox (not if “allergic)
• (high dose - HD – 90 mg per kg per day) • HD because S pneumo is becoming more resistance and the y need to bypass it |
|
2nd line med for AOM
|
HD Augmentin ES-600mg (can’t use another formula – causes severe diarrhea)
• clinical failure of 1st line tx OR • recurrence within 4 wks of prior antibiotic • broadens the coverage to cover more of the pathogens than Amox alone |
|
3rd line med for AOM
|
Rocephin IM (2 or 3 doses (based on severity), 48 hours apart) OR ENT referral (will culture fluid)
• clinical failure of 2nd line tx • broad spectrum cephalosporin – more broad coverage |
|
AOM tx - if allergic to PCN or AMOX
|
• Omnicef, Ceftin, Or Zantin (rash rxn to PCN/AMOX)
• Bactrim or Zithromax (severe rxn) – don’t give any cephalosporins to these pts --- don’t cover the pathogens as well. o May give Rocephin IM 1st Line w/compelling reason and no h/o SEVERE allergies (1 injection) • Uncommon • VERY expensive, but great drug, does well w/infections • Compelling – going out of country, can’t do 10 days of tx |
|
Neonatal Risk Factors for SN Hearing Loss
|
-NICU admission > 48 hrs
-Craniofacial anomalies -In utero infections (from mom) -Severe neonatal jaundice Other risk factors: -Meningitis -Head trauma -Medications -Loud Noises -Hypoxia |
|
Signs of SN Hearing Loss
|
-No startle, reflex, language delay, in attention, psychosocial problems
-Consider reassessment for high risk patients Parents would usually complain of child not being able to hear. After 6 months should be able to babble. |
|
Newborn Hearing Screening
|
-Universal screening at birth
-Otoacoustic emission test (OAE) (simple and quick) - If OAE fails, auditory brain stems response (ABR) (Wears headphones..Use of electrodes to measure the nerve response from cranial nerve VIII )..may need to sedate the child if moving too much. If ABR fails, referral goes to Audiologist -Early detection critical (very important) -Tx: hering aids, cochlear implants, sign language, lip reading, gestures -Refer: ENT opthamology, geneticist |
|
Mastoiditis
|
-Infection of the periosteum of the mastoid bone
-More common under age of two -Fever, severe pain behind the ear, redness behind the ear, displacement of the ear (pinna) -From acute otitis media most of the time -Pathogens: (same one) haemo, step neumo, etc -Complications: meningitis, abscess in the brain -Treatment: STAT (immediate) consult with ENT -Direct admit for myringotomy, culture, IV antibiotics |
|
ACUTE VIRAL RHINITIS (URI/ COMMON COLD)
|
Incidence and Frequency
-SX: -Runny nose, congestion, fever, sore throat, cough, seezing -Purulent/ thick nasal mucus after 2 days -Cough and runny nose last longest -Management if < 2 y/o—Usually treatment is not recommended if kids have issues with upper respiratory problems. Also, lot of young babies coming in ER with overdose of meds because parents giving combination of meds -Saline nose drops with bulb suctioning -1/4 tsp salt in 1 c water -Humidifier/ vaporizer -Tylenol or ibuprofen -(Benadryl may be okay if > 12 months of age) -Nighttime secretions -Management for ages 2-6yrs: -Use of cough, suppressants, decongestants, antihistamines, expectorants or any combination thereof are still under investigations… -OTC meds are not recommended in kids < 4 y/o -Management for children > 6 y/o |
|
Influenza
|
“worse than the common cold”
-URI sx + significant amount of; -fever, myalgias, headache, chills, fatigue -Sometimes AGE (abdominal…) sx in younger kids -Most common complications: -secondary bacterial infections -Croup -Wheezing -High risk children: those with immunodeficiency, like kids with diabetes, with lungs, with hearts etc -Prevention: -Seasonal flu shot -H1N1 flu shot -Relenza ((antiviral med) |
|
RHINOSINUSITIS:
|
Inflammation in the sinuses or pathways
Diagnosis: if you have URI for ten days and not getting any better. It is Rhinosinusitis IF URI is also 5 to 7 days and conditions getting worse then it is also RHINOSINUSITIS -Pathology is often secondary post URI or AR -Mucosal injury/ swelling -Increased mucus -Obstruction -Decreased ciliary action Sinus development= -Maxillary and Ethmoid infection Younger children Rarely infected before 1 yr old Just sinusitis usually doesn’t occur with the child less than 1 yr old but rhoinosinutis does Frontal sinuses -- Form by approx 7 – 8yrs -- Rarely infected before age 10 yrs |
|
Rhinosinusitis
|
Major pathogens:
-the usual -Complications -Cellutitis, bone infection or intracranial complications Rhinosinusitis-symptoms Common: Nasal congestion, purulent nasal d/c, cough Other: Post nasal drip, bad breath, anosmia, toothache, earache, fever, facial pain o Signs: Often minimal in younger children X ray -Best for children > 6 y/o CT scans -For severe disease or surgical candidates |
|
Mgmt/Tx of Rhinosinusitis
|
X ray
-Best for children > 6 y/o CT scans -For severe disease or surgical candidates Management (Figure 17-8, pg 456) First line therapy –Amoxicillin HD (not in day care, mild moderate sx, no recent antibiotics) First line therapy- Augmentil HD Secondary options: same as Acute otitis Media (omnicef, septin…if hx of allergic reactic to Zithromax, Biacyn (if severe allergic reaction) or Bactrim (doesn’t cover H influenza) -Consider nasal corticosteroids or decongestants -Consider pain management -Rx antibiotics for at least 10 days. -Look for improvement with 48-72 hrs |
|
Allergic Facies
|
Elongated Relaxed facial, dry eyes, exaggerated skin lines, usually dark circles or red circles under the eye,
o Allergic Rhinitis Exam: -Pale and boggy turbinates -red turbinates -Cobblestoning o Symtoms: sneexing, runny nose, itchy eyes, itchy throat Daytime sleepiness common |
|
Allergic P/E
|
-Pale and boggy turbinates
-red turbinates -Cobblestoning o Symptoms: sneezing, runny nose, itchy eyes, itchy throat Daytime sleepiness common o Complications: -OM -Sinusitis -Obstructive sleep apnea -Misalignment of the jaw |
|
Allergic Tx
|
-Non sedating antihistamines
-Claritin -Zyrtec -Nasal corticosteroids -Nasonex -Flonase -Singulair (leukotreiene antagonist) |
|
Epistaxis
|
Common causes in children-
-trauma -dry air/ mucosa -Nasal sprays -Most common site: front of the nasal septum -Exam findings- Raw nasal mucosa, crusting, blood Usually benign, work-up if: Fam hx of bleeding disorder (like hemophilia) H/O easy bleeding Bleeding > 30 mins Abnormal Hgb Onset < 2yrs of age Initial labs -CBC, PT, PPTT -TX- pressure, keep mucosa moist |
|
ORAL CANDIDIASIS (Thrush)
|
White curd like plaques
Mild mouth discomfort Adherent (may come off with vigorous rubbing) Usually mild sources Can occur after use of antibiotics, cause of inhaled corticosteroids like treating asthma, doesn’t come off usually |
|
Thrush Tx
|
-Nystatin suspension X 1 week
-Consider Fluconazole (Diflucan) oral suspension -Wash bottles, pacifiers -Apply to breast if breastfeeding |
|
HERPES GINGIVOSTOMATITIS
|
SVI
-Generalized- can be on the tongue, around the mouth, looks like bunch of canker sores -More common < 3 yrs of age Source: somebody shedding the virus HSVI o Symptoms/ signs include: Lots of PAN, bleeding gums, decreases appetite, drooling, fever, ulcerations, cervial Lymphadenopathy (LAD) HSV can also affect other place |
|
HERPES GINGIVOSTOMATITIS
Tx |
-Motrin or Tylenol
-Acyclovir Susp- 200 mg/5 cc -No Corticosteroids -Magic Mouthwash-various combos -Benadryl, Maaloxm Viscous Lidocaine 2% (20 cc each) -Education -Prevent dehydration, decrease infectivity |
|
Apthous Ulcers
|
Anterior
Pain Self limited Recurrent Unknown cause |
|
Apthous Ulcers Tx
|
-Betamethasone ointment BID
-Bland foods -Mortin or Tylenol -Warm H20 gargles |
|
Herpangina
|
Coxsackie virus
Summer/ fall Ulcers, runny nose, low fever Anterior pillars, soft palate, uvula Recurrent self-limited |
|
Herpangina Tx
|
-Motrin or Tylenol
-Magic Mouthwash Pharyngitis (Viral) -90% are viral origin -Signs/ Symptoms -Lab-rapid strep or throat c/s + treat it … (-) then culture it |
|
Strep Throat
|
-GAS/ SABHS/ Strep pyogenes
-Signs and Symptoms -Acute onset of fever and sore throatBeef red throat, exudate, petechiae (small red dots on soft palate) cervical LAD |
|
Tx of Strep Throat
|
LABS: order rapid strep screening/ culture sensitivity
TxL oral PCN TID o Alternatives- IM PCN, Amox, Cepthalosporin Zithromax, E mycin -NO Bactrim (doesn’t prevent rheumatic fever and doesn’t deal with strep..) -Can give PCN injection (once) o Complications -Scarlet Fever (benign…rash on stomach and tongue)…so always check the throat carefully with non specific rashes -Rheumatic Fever (can occur in skin, cardiac problems, joint problems---need to be careful..This is due to toxins of strep) -Acute glomerulonepthritis (can affect kidneys) -Abscess |
|
Complications of Strep Throat
|
-Scarlet Fever (benign…rash on stomach and tongue)…so always check the throat carefully with non specific rashes
-Rheumatic Fever (can occur in skin, cardiac problems, joint problems---need to be careful..This is due to toxins of strep) -Acute glomerulonepthritis (can affect kidneys) -Abscess |
|
Peritonsillar cellulitis or abscess
|
-Source: same as step throat.
Sx: -Severe sore throat-often unilateral -Fever -Dysphagia -Hot Potato voice (gargles type of sound) Exam: Uvula and soft palate LAD Muffled voice Tx: |
|
Peritonsillar cellulitis or abscess Tx
|
Tx:
Admit for IV antibiotics, possible I & D |
|
Management of bacterial conjunctivitis
|
o topical
• Erythromycin, ocuflox, polymyxin-bacitracin, sulfacetamide (topical) • Ocuflox – flouroquinolone Only use topical flouroquinolones on eye or ears (not IV or oral w/kids) • Some may burn – sulfacetamide |
|
Viral Conjunctivitis Tx
|
o Very contagious, usually self limited
• Secondary bacterial infections DO occur • Avoid corticosteroids, esp if it pertains to Herpes |
|
Allergic Conjunctivitis Tx
|
o Acular (anti-inflam eye drop), Patanol (anti-histamine eye drop), Naphcon-A (anti-histamine and vasoconstrictor)
|
|
Pink Eye Education and Tx
|
o Infectivity, no improvement, return to school
• Viral – contagious at least a week • Bacterial – after on abx for a couple of days can go back to school, redness will be better by then, not contagious • General – if no improvement after being on abx for a couple days o May be resistant, misdiagnosis, follow up |
|
Periorbital/Preseptal Cellulitis
|
• Orbital septum separates eyelid from orbit
• Preseptal infection - of eyelid only and some surrounding skin around lid (eyelid infection) o Sx • LID edema, LID redness, LID pain, mild fever o Source • Eyelid abrasions, insect bites, or nearby infections o Common Pathogens • Staph aureus, Strep pyogenes o Tx: Oral Abx w/ close follow up o IV or IM abx if severe |
|
Orbital Cellulitis
|
• Infection in the orbit of eye, surrounding the eye
• Sx: o Same as periorbital PLUS: • Restricted eye movement (do H Test) • Proptosis • Usually a HIGH fever, may appear toxic • Source o Contiguous sinus infection o May be a secondary preseptal infection • Pathogens o Strep pneumo, H Influenzae, M Catt, Staph aureus o Also consistent w/otitis media/HEENT disorders o When treating – make sure rx will cover all of these |
|
Orbital Cellulitis Tx
|
: IV ABX, surgical drainage
o Watch for orbital compression of optic nerve → vision loss • CT Scan – check for pus in orbit to differentiate o Difficult to tell apart by the visual appearance |
|
Corneal Abrasion
|
• Sx
o Infants – usually scratched cornea w/ their own hand o Older children – hx of eye trauma • Exam o Check visual acuity first o Evert the upper lid, o Check lower lid well o Checking for foreign body o Stain the eye to see uptake of dye in the abrasion • Management o Watchful waiting o Time o DAILY follow ups to observe improvement/watch for infections • Prognosis o Great, unless significant scratch/scarring |
|
Retinopathy of Prematurity
|
• Common risk factors
o First check to see if they are under the care of an ophthalmologist already o Retina is not completely vascularized/ not fully mature o Vessels predisposed to hemorrhage o If retina not getting a full blood supply → it will detach |
|
Retinoblastoma
|
• The most common primary intraocular malignancy of childhood
o Age at presentation o DO: Red Reflex Check – white pupil, reflex absent • Etiology o Retinal cell mutation o Hereditary or sporadic • Sx o Single or multiple tumors o Unilateral or bilateral o leukocoria, strabismus, glaucoma, blindness, proptosis, red eye, pus in anterior chamber • Management o Detect and refer • Prognosis o Depends on characteristics of the specific tumor |
|
Leukocoria
|
cataract is more diffuse, hazy cornea, retinoblastoma is intraocular, so it’s not hazy, it’s a sharp margin of a white pupil in the light
|
|
The most appropriate test for assessing vision in a newborn infant:
|
looking for blepharospastic response to bright light
|
|
Infant Ears
|
50% of time won’t see eardrum well
• Flat eardrum in infants • Angulate speculum down |
|
AOM Risk Factors
|
o Flat eustachian tube
o Age – bottle feeding • Young – haven’t been exposed to many viruses/coughs/less immune than older • Colonization – predisposed to bacterial infections, more likely to have bacterial infection • URI’s • Daycare – pathogen exposure • Impaired immune system • Smoking – inflammation to middle ear, blockage of ear, higher risk for middle ear infection • Bottle feeding – fluids don’t drain well in recumbent position |
|
AOM Key Features
|
o Key Features
• Fever, ear pain, inflammation, effusion • Other Recent URI, decreased sleep, increasing crying, ear tugging, decreased appetite, vomiting Decreased TM mobility, decreased light reflex • Special Considerations Fever, (fever can cause a red eardrum – may not be AOM),crying (can cause red eardrum), cerumen (can obstruct view of TM – may need to dig out wax to see) Decreased incidence with increasing age |
|
AOM Pathogens
|
• S pneumo – PCN drug resistance common
Most invasive pathogen May be decreasing • H influenzae – on the rise, but less invasive • M. catarrhalis • S. pyogenes |
|
AOM Vaccine
|
– pneumococcal vaccine (Prevnar) – to prevent certain strains of strep pneumo in ear infections in kids
o Now with the vaccine, the other strains are on the rise, and the strep pneumo decreasing |
|
Recurrent Aphthous Stomatitis
|
Small (3-10 mm) ulcers; lips/tongue
No fever/LAD Painful - last 1-2 wks Tx: CTSD paste: Triamcinolone/Betamethasone Bid Bland Diet Acetaminopen/Motrin Assoc. w/Behcet disease/Med. fever adn PFAPA |
|
Thrush
|
C. albicans
Patho: vertically passed at birth; ABX/ICS use predisposition; immunocompromised Tx: Nystatin, supsension x1 wk, apply 4x/day, stop when gone (3-4 days) Systemic antifungal if no response Replace infected pacifiers, and tx mother (breast feeding) |
|
Acute stomatitis:
Herpes Simplex Gingivostomatitis |
HSV1
Sx: Painful/bleeding gums/decreased appetite/HIGH fever (7-10 days) Ulcerations (>10) Cervical LAD, Halitosis; diffuse mouth infxn preceding ulcer appearance MC <3 y/o -Herpetic Whitlow - spread to fingers --> can then spread to eyes Tx: Pain mgmt (if >6mos/o) Magic Mouthwash: Combo: Benadry, Maalox, +/- viscous lido (20 cc each) -older swish + spit -younger - wipe mouth w/Qtip -Acyclovir - if tx early Comp: dehydration; herpetic laryngotracheitis |
|
Infectious Mononucleosis (EBV)
|
Gray exudative tonsillitis
Cervical Adenitis Splenomegaly Fever usually >5 y/o R/o strep: strep test first, then Mono spot (2-3 days) (+) supports; (-) doesn't R/o Mono --> false (-) if < 5 y/o EBV Ab Titer (IgM) - Elevated IgM capsid = DEFINITIVE CBC = abnormal shape Blood Smear > 10% atypical lymphocytes --> falsey (-) if <5 y/o NO AMOX --> Rash Tx: Symptomatic |
|
Herpangina
|
Ulcers 3mm w/halos: anterior pillars, soft palate, uvula
Benign Glossitis (geographic tongue) Coxsackie A Virus - summer/fall Enteroviral PCR available, but usually unnecessary --> Self-limited Symptomatic Tx (Possible several episodes) |
|
Pharyngoconjunctival Fever
|
Adenovirus
Often = epidemic; URI Sxs: Exudative tonsillitis Conjunctivitis Lymphadenopathy Fever Tx = symptomatic |
|
Hand Foot Mouth Disease
|
Several Enteroviruses cause it
Enterovirus 71: can cause encephalitis Ulcers: anywhere in mouth Vesicles; pustules; papules: palms, soles, interdigital; buttocks Younger - distal extremities; face |
|
Acute Bacterial Pharyngitis
Dx |
Dx: Sore Throat +
@ least one of the following -Cervical LAD (tender or >2 cm) -Tonsillar exudates -Fever >38.3C (+) culture for group A B-hemolytic Strep Do a rapid Antigen test, but confirm (-) rapid w/a Culture 10% of kids w/sore throat+fever = group A Strep (sudden onset, beefy-red uvula, sore throat/odynophagia, palatal petechiae |
|
Acute Bacterial Pharyngitis
Tx |
1) PCN V, tid, PO, 10d course
--> beware of increasing PCN resistance and ABX failure after max dose for 10d. --> fail to respond --> AUG or Azith Alt: IM PCN, AMOX, Erythromycin/Azith, Ceph (NO bactrim --> doesn't prevent Rheumatic Fever/doesn't cover Strep) Comp: Scarlet Fever, acute glomerulonephritis, Rheumatic Fever (nodular rash/movement d/o's/Chronic Heart Dz/Hematuria) Consider anti-streptolysin-o titers if elevated & pt wasn't tx'ed |
|
Peritonsilar Cellulitis or Abscess
|
M/C is B-hemolytic Streptococcus
-tonsillar infxn that penetrates tonsillar capsule and spreads to surrounding tissue - Unilateral sore throat (severe!)/dysphagia/trismus/drooling -Hot Potatoe voice/ gargling/gutterall -High Fever -Tonsils bulge medially -Soft palate swelling -LAD; drooling PE: Observe uvula/soft palate --> locate abscess Difficult to differentiate cellulitis from abscess -Admit 12-24 hrs w/IV ABX - aggressive tx prevents suppuration - PCN or Clindamycin -DC on oral ABX if fever cleared > 24 hrs and improved dysphagia OR closely followed by ENT **HIGH suspicion for abscess if failure to respond to ABX in 12-24 hrs |
|
Retropharyngeal Abscess
|
Retropharyngeal Nodes: drain adenoids, nasopharynx, paranasal sinuses
--> become infected --> pyogenic adenitis --> Untx'ed = Abscess formation M/C B-hemolytic Strep or S. aureus Suspect in infants w/: fever, RESPIRATORY SXS, Neck HYPEREXTENSION, Dysphagia, Drooling, Dyspnea, Gurgling, prominent unilateral swelling of posterior pharyngeal wall --> stops at midline --> confirms Dx Difficult to differentiate abscess from Cervical Adenitis --> Immediate Hospitalization + IV ABX (PCN or Clinda) 12-24 hrs of ABX helps ddx based on tx response If abscess (+): surg. drainage |
|
Ludwig Angina
|
Rapidly progressive
Cellulitis of submandibular space - M/C d/t dental DZ or lacerations/injuries Group A Strep -Fever -Tenderness w/swelling to floor of mouth -Enlarged tongue - tender/red Upwardly displaced tongue - dysphagia/drooling --TX: ICU; IV Clindamycin; monitor airway closely for obstruction -->Refer to otolaryngologist for drainage procedure |
|
Acute Cervical Adenitis
|
M/C B-hemolytic Strep (also S. aureus, viral)
Local ENT infxns that involve a reginal node --> develop abscess -Typically a unilateral, solitary, anterior Cervical Node -Isolated, tender LAD w/fluctuance -Fever LABS: CBC w/diff Rapid A group Strep test Monospot - if multiple nodes PPD - Anti-TB Mycobacterium -TX: ABX and aspiration culture/I&D if fluctuance R/o neoplasm Also Cat Scratch DZ - papule @ scratch site Cervical Lymphadenitis - NonTB Myco - unilateral, w/several matted nodes (+ PPD) |