• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/115

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

115 Cards in this Set

  • Front
  • Back
Differential Dx: Corneal Abrasion
Ocular/adnexal foreign bodies
Corneal Ulcer
Corneal laceration
Differential Dx: Blepharitis
Chalazion

Hordeolum

Rosacea Blepharitis
Differential Dx and Symptoms: Viral Eyelid Disease
Diff: Impetigo

vesicular lesions with an erythematous base

Fluorescein dye and cobalt blue light
Differential Dx: Eyelid Ptosis
Congenital v. traumatic v. neurogenic ptosis, Horner Syndrome
Differential Dx: Ophthalmia Neonatorum
May be bacterial, or viral or due to tx w/silver nitrate prophylaxis at birth, Chlamydia - most common,

Gonorrhea - threatens vision
HSV - possibly systemic
Buphthalmos
Abnormal enlargement of eye

Seen in Congenital Glaucoma; buphthalmos is glaucoma until proven otherwise
Nasolacrimal Duct Obstruction Treatment, Outcomes, and Risks
Massage to remove debris (debatable),

Topical antibiotics,
Surgical tx - probing, effectiveness decreases after 1 year of age

90% spontaneously resolve in 1 year

Monitor for possible glaucoma

If light sensitivity and blepharospasm present - urgent referral to ophthalmologist

(Risks of Superinfection,
Dacryocystitis, preseptal cellulitis,
orbital cellulitis)
Retinopathy of Prematurity Tx
Follow up every 1-2 wks
Surgical tx necessary within 72 hours to reduce bad visual outcome if disease progressed. However, often cases don’t require tx and ROP doesn’t progress to retinal detachment.
Amblyopia - 3 types
kinds: 1. Strabismic 2. Refractive 3. Deprivation (causes worst visual loss)
Sequalae (leads to)- permanent blurry vision, vision loss,
Retinoblastoma
the most common primary intraocular malignancy of childhood
-usually presents before age 3
Periorbital Cellulitis
often present with erythematous and edematous eyelids, pain, and mild fever. The vision, eye movements, and eye itself are normal (unlike orbital cellulitis)
Perform H test to R/o orbital cellulitis
IV Abx
Orbital cellulitis
Decreased vision, restricted eye movements, and an afferent papillary defect suggest orbital cellulitis.
Orbital cellulitis presents with signs of periorbital disease as well as proptosis (a protruding eye), restricted eye movement, and pain with eye movement. Fever is usually hig
Orbital Tumors
Both benign and malignant orbital lesions occur in children.
-The most common benign tumor is capillary hemangioma
-The most common primary malignant tumor of the orbit is rhabdomyosarcoma.
Symptoms of Orbital Tumors
proptosis,
orbital ecchymosis (raccoon eyes), displacement of the globe
Tx of Orbital Tumors
- Treatment is indicated if the lesion is large enough to cause amblyopia
-Induced astigmatism or amblyopia (or both) are treated with glasses and patching, respectively. -Treatment of orbital dermoids is by excision
-Treatment of metastatic disease requires management by an oncologist
Nystagmus
Oscillation of eyes
Recurrent Aphthous
small ulcers (3-10mm) on inner aspect of lips or tongue, rarely on tonsils or palate. No fever or cervical adenopathy. Painful and lasts 1-2 weeks. May recur several times throughout life. PFAPA = episodes last 5 days without other URI symptoms

Recurrent = canker sores
Herpes Simplex Gingivostomatitis
10+ ulcers, fevers, cervical adenopathy, and inflammation

<3 y/o, lasts 7-10 days
If caught early, tx w/Acyclovir
Usually pain --> dehydration --> hospitalization required
tx of Acute Stomatitis
coat leasons with corticosteroid ointment (triamcinolone). Reduce pain by bland diet (no acidic, salty food or juice) or acetaminophen/ibuprofen. PFAPA = cimetidine treatoment or tonsillectomy.

Refer to otolaryngologist - if PFAPA suspected
Acute Stomatitis: Herpes Simpex
initially: 10+ small ulcers of buccal mucosa, anterior tonsillar pillars, inner lips, tongue, gingival. Posterior pharynx usually spared. Associated with fever, tender cervical adenopathy, generalized oral inflammation preceding the ulcers
Acute Stomatitis: Thrush
white curdlike plaques, predominantly on buccal mucosa (can’t wash away
Hx of Thrush
affects infants or older children in debilitated state. Use of broad-spectrum abx, systemic/inhaled corticosteroids, HIV + (erythematous candidiasis, producing red patches on palate and dorsum of tongue usually assoc with abx use, corticosteroids and HIV)
Acute stomatitis: traumatic ulcers
trauma due to biting by molars (mechanical), thermal trauma (hot food), chemical trauma (contact with aspirin). Leukemia or cyclic neutropenia
Pharyngitis: acute viral and tonsillitis (can’t often distinguish causative viral agent):
all: sore throat, fever (90% of sore throats and fever due to viral infection). Some have ulcers (see PhysEx)

Types:
1. infectious mono - palpable speen, increased IgM's, >5 y/o, axillary adenopathy, dx with EBV serology

2. Herpangina - 3mm ulcers surr. by halos, Coxsackie A grp. viruses, self-limiting

3. Hand Foot Mouth Disease - enteroviruses, rarely causes Encephalitis, ulcers in mouth, vesicles/papules on palms, soles, fingers, and buttocks

4. Pharyngoconjunctival Fever - Adenovirus - epidemic in kids, exudative tonsillitis, conjunctivitis, lymphadenopathy, fever, Tx is symptomatic.
Patho of Pharyngitis
all: sore throat, fever (90% of sore throats and fever due to viral infection). Some have ulcers (see PhysEx)
Phys Exam of Pharyngitis
M: exudative tonsillitis, cervical adenitis, fever, palpable spleen or axillary adenopathy. H: ulcers (3mm) surrounded by halo on anterior tonsillar pillars, soft palate and uvula. HFMD: ulcers anywhere on mouth; vesicles, pustules or papules on palms, soles, interdigital areas and buttocks. PCF: exudative tonsillitis, conjunctivitis, lymphadenopathy, fever
Tx of Pharyngitis
all tx is symptomatic IM: no amoxicillin bc produces rash.
HFMD - can lead to encephalitis
Pharyngitis: bacterial
Pharyngitis: bacterial
Recurrent Rhinitis Allergic
intranasal corticosteroids, oral and intranasal antihistamines, leukotriene antagonists, decongestants, nasal saline rinses
Rhinsosinusitis: Recurrent/Chronic
ABRS clears bur recurs with each or most URI = recurrent. Child does not clear infection in expected time, but acquires no acute complications = chronic. Allergies, anatomic variations (causing obstruction, eg septum deviation) and disorders in host immunity. Gastroesophageal reflux could contribute to chronic ABRS

If allergic polyps found in child<10 --> cystic fibrosis workup
Acute Bacterial Rhinosinusitis
Bacterial infection of paranasal sinuses; almost always preceded by viral URI

gradual or sudden onset of symptoms that last less than 30 days: nasal drainage, nasal congestion, facial pressure or pain, postnasal drainage, hyposmia or anosmia, fever, cough, fatigue, maxillary dental pain, ear pressure or fullness
Acute Viral Rhinitis
Common Cold

clear or mucoid rhinorrea, nasal congestion, sore throat, possible fever. Symptoms resolves within 7-10 days. Nose, throat and TM may appear red and inflamed. Nasal secrections more purulent after day 2 (color change should not be assumed to indicate bacterial rhinosinusitus)
Acute trauma to middle ear - Tx
1. Mainly watchful waiting.
2. Antibiotics are not necessary unless there are visual signs of infection.
3. If accompanied with vertigo, refer to otolaryngologist and should be attended to more urgently.

Refer !
Mastoiditis
1. postauricular pain
2. ear protrusion with outwardly displaced pinna.
3. fever present
4. Mastoid is swollen and red.
5. AOM is almost always present (acute otitis media).

Can have brain abscess, persistent headaches, recurring fever.
Mastoiditis Presentation
If acute: diffuse inflammatory clouding of mastoid cells occurs in an uncomplicated AOM.
Postauricular pain and swelling (ddx external otitis)
Body destruction and resorption of mastoid air cells occurs in severe AOM cases.
Mastoiditis
If acute: diffuse inflammatory clouding of mastoid cells occurs in an uncomplicated AOM.
Body destruction and resorption of mastoid air cells occurs in severe AOM cases.
Mastoiditis Complications
Meningitis is a complication of acute mastoiditis. Meningitis Sx are high fever, stiff neck, severe headache...diagnosis found by lumbar puncture.
Can progress to sigmoid sinus thrombosis, epidural abscess, or intaparenchymal brain absess
Mastoiditis Complications
Meningitis is a complication of acute mastoiditis. Meningitis Sx are high fever, stiff neck, severe headache...diagnosis found by lumbar puncture.
Can progress to sigmoid sinus thrombosis, epidural abscess, or intaparenchymal brain absess
Snoring, mouth breathing and upper airway obstruction
Nightly snoring or mouth breathing. Unrefreshed sleep, behavioral problems, daytime fatigue.

- Enlarged (4+) tonsils
Recurrent tonsillitis

tonsillectomy or polysomnography
Dacryostenosis
Obstructed nasolacrimal duct
tears build up on eye
Usually since birth
Recurrent conjunctivitis
Tx: duct massage, moist heat, eyelid cleaning, topical ABX prn if persistent, ophthalmology referral, tube placement to open up adhesed duct
Blepharitis Peds Tx
• management
o antibiotics
o gentle cleaning with gentle soap
o conservative approach

Monitor for corneal changes associated with dry eye
Chalazion Tx
 conservative management
• warm compress few times day
• time to go away
• significant red/pus – topical antibiotics prn (NOT oral)
• 2 months doesn’t go away – opthalmologist can remove them
Ophthalmia Neonatorum
o Newborn conjunctivitis
• 1st month of life
• contracted at birth from mother (vaginally)
o Chlamydia (CT) – most common
• Prophylaxis is NOT effective
 need oral antibiotics
• culture from eye d/c and nasopharynx
• 1-2 wks post-natal presentation
 subtle
• 50% acquisition rate if mom infected (vaginal birth)
• long term effects - scarring blindness
Tx - Ophthalmia Neonatorum
• management
 screen pregnant patients before delivery
• regular prenatal care?
o vaginal swabs screening for infection – less likely to have undiagnosed infection
 oral macrolide antibiotics -azithromcyin, e-mucin
• has to be ORAL antibiotics
• would progress over time if untreated
Gonorrheal Conjunctivitis
• More rare
 Prophylaxis is usually effective
• Typical age 1-7 days (EARLIER – more acute/severe)
• corneal perforation and scarring
• dissmeinated gonococcal dz
• management - hospitalize, IV antibiotics
HSV Keratoconjunctivitis
• Rare
• onset 3 days - 3 weeks
• corneal lesions, scarring, recurrent outbreaks
• management – topical or oral antivirals, no steroids (may make it worse)
Pt presents with: Pain/Foreign Body Sensation
i. Foreign body
ii. Corneal abrasion
iii. Laceration
iv. Acute infections of globe/ocular adnexa
v. Iritis
vi. Angle-closure glaucoma
vii. Trichiasis
viii. Contact Lens Misuse
Pt presents with: Photophobia
1. Corneal abrasions
2. Foreign bodies
3. Iritis
4. Squinting of one eye in bright light → intermittent exotropia
Pt presents with: no ocular pathology
a. Migraines
b. Meningitis
c. Retrobulbar optic neuritis
Pt presents with: leukocoria
1. Retinoblastoma
2. Retinopathy of prematurity
3. pupillary membrane
4. cataract
5. vitreous opacities
6. retinal detachment
7. Toxocara infection
8. Retinal Dysplasia
Peds Ophthalmic Exam
i. Hx of poor vision/misalignment of eyes
ii. Visual acuity outside expected level
iii. Abnormal red reflex (Infants)
1. Cataracts
2. Intraocular tumors
iv. Abnorm pupil reactivity
v. → refer to ophtalmologist
vi. prompt tx
vii. birth – 3 y/o
1. hx, vision assessment, inspection of lids and eyes, pupil exam, ocular motility, and red reflex check
viii. 3 y/o +
1. everything PLUS visual acuity testing
ix. Visual acuity
1. The MOST important test of visual fx
Pathogens in Preseptal Infections
S. aureus and S. pyogens
Retinoblastoma
• The most common primary intraocular malignancy of childhood
o Age at presentation
o DO: Red Reflex Check – white pupil, reflex absent
• Etiology
o Retinal cell mutation
o Hereditary or sporadic
• Sx
o Single or multiple tumors
o Unilateral or bilateral
o leukocoria, strabismus, glaucoma, blindness, proptosis, red eye, pus in anterior chamber
AOM Pathogens
• S pneumo – PCN drug resistance common
 Most invasive pathogen
 May be decreasing
• H influenzae – on the rise, but less invasive
• M. catarrhalis
• S. pyogenes
Prevnar
pneumococcal vaccine (Prevnar) – to prevent certain strains of strep pneumo in ear infections in kids
1st line med for AOM
– HD Amox (not if “allergic)
• (high dose - HD – 90 mg per kg per day)
• HD because S pneumo is becoming more resistance and the y need to bypass it
2nd line med for AOM
HD Augmentin ES-600mg (can’t use another formula – causes severe diarrhea)
• clinical failure of 1st line tx OR
• recurrence within 4 wks of prior antibiotic
• broadens the coverage to cover more of the pathogens than Amox alone
3rd line med for AOM
Rocephin IM (2 or 3 doses (based on severity), 48 hours apart) OR ENT referral (will culture fluid)
• clinical failure of 2nd line tx
• broad spectrum cephalosporin – more broad coverage
AOM tx - if allergic to PCN or AMOX
• Omnicef, Ceftin, Or Zantin (rash rxn to PCN/AMOX)
• Bactrim or Zithromax (severe rxn) – don’t give any cephalosporins to these pts --- don’t cover the pathogens as well.
o May give Rocephin IM 1st Line w/compelling reason and no h/o SEVERE allergies (1 injection)
• Uncommon
• VERY expensive, but great drug, does well w/infections
• Compelling – going out of country, can’t do 10 days of tx
Neonatal Risk Factors for SN Hearing Loss
-NICU admission > 48 hrs
-Craniofacial anomalies
-In utero infections (from mom)
-Severe neonatal jaundice
Other risk factors:
-Meningitis
-Head trauma
-Medications
-Loud Noises
-Hypoxia
Signs of SN Hearing Loss
-No startle, reflex, language delay, in attention, psychosocial problems
-Consider reassessment for high risk patients
Parents would usually complain of child not being able to hear. After 6 months should be able to babble.
Newborn Hearing Screening
-Universal screening at birth
-Otoacoustic emission test (OAE) (simple and quick)
- If OAE fails, auditory brain stems response (ABR)
(Wears headphones..Use of electrodes to measure the nerve response from cranial nerve VIII )..may need to sedate the child if moving too much.
If ABR fails, referral goes to Audiologist
-Early detection critical (very important)
-Tx: hering aids, cochlear implants, sign language, lip reading, gestures
-Refer: ENT opthamology, geneticist
Mastoiditis
-Infection of the periosteum of the mastoid bone
-More common under age of two
-Fever, severe pain behind the ear, redness behind the ear, displacement of the ear (pinna)
-From acute otitis media most of the time
-Pathogens: (same one) haemo, step neumo, etc
-Complications: meningitis, abscess in the brain
-Treatment: STAT (immediate) consult with ENT
-Direct admit for myringotomy, culture, IV antibiotics
ACUTE VIRAL RHINITIS (URI/ COMMON COLD)
Incidence and Frequency
-SX:
-Runny nose, congestion, fever, sore throat, cough, seezing
-Purulent/ thick nasal mucus after 2 days
-Cough and runny nose last longest


-Management if < 2 y/o—Usually treatment is not recommended if kids have issues with upper respiratory problems. Also, lot of young babies coming in ER with overdose of meds because parents giving combination of meds
-Saline nose drops with bulb suctioning
-1/4 tsp salt in 1 c water
-Humidifier/ vaporizer
-Tylenol or ibuprofen
-(Benadryl may be okay if > 12 months of age)
-Nighttime secretions
-Management for ages 2-6yrs:
-Use of cough, suppressants, decongestants, antihistamines, expectorants or any combination thereof are still under investigations…
-OTC meds are not recommended in kids < 4 y/o
-Management for children > 6 y/o
Influenza
“worse than the common cold”
-URI sx + significant amount of;
-fever, myalgias, headache, chills, fatigue
-Sometimes AGE (abdominal…) sx in younger kids
-Most common complications:
-secondary bacterial infections
-Croup
-Wheezing
-High risk children: those with immunodeficiency, like kids with diabetes, with lungs, with hearts etc
-Prevention:
-Seasonal flu shot
-H1N1 flu shot
-Relenza ((antiviral med)
RHINOSINUSITIS:
Inflammation in the sinuses or pathways
Diagnosis: if you have URI for ten days and not getting any better. It is Rhinosinusitis IF URI is also 5 to 7 days and conditions getting worse then it is also RHINOSINUSITIS
-Pathology is often secondary post URI or AR
-Mucosal injury/ swelling
-Increased mucus
-Obstruction
-Decreased ciliary action
Sinus development=
-Maxillary and Ethmoid infection
Younger children
Rarely infected before 1 yr old
Just sinusitis usually doesn’t occur with the child less than 1 yr old but rhoinosinutis does
Frontal sinuses
-- Form by approx 7 – 8yrs
-- Rarely infected before age 10 yrs
Rhinosinusitis
Major pathogens:
-the usual
-Complications
-Cellutitis, bone infection or intracranial complications

Rhinosinusitis-symptoms
Common:
Nasal congestion, purulent nasal d/c, cough
Other:
Post nasal drip, bad breath, anosmia, toothache, earache, fever, facial pain
o Signs:
Often minimal in younger children
X ray
-Best for children > 6 y/o
CT scans
-For severe disease or surgical candidates
Mgmt/Tx of Rhinosinusitis
X ray
-Best for children > 6 y/o
CT scans
-For severe disease or surgical candidates
Management (Figure 17-8, pg 456)
First line therapy –Amoxicillin HD (not in day care, mild moderate sx, no recent antibiotics)
First line therapy- Augmentil HD
Secondary options: same as Acute otitis Media (omnicef, septin…if hx of allergic reactic to
Zithromax, Biacyn (if severe allergic reaction) or Bactrim (doesn’t cover H influenza)
-Consider nasal corticosteroids or decongestants
-Consider pain management
-Rx antibiotics for at least 10 days.
-Look for improvement with 48-72 hrs
Allergic Facies
Elongated Relaxed facial, dry eyes, exaggerated skin lines, usually dark circles or red circles under the eye,
o Allergic Rhinitis
Exam:
-Pale and boggy turbinates
-red turbinates
-Cobblestoning
o Symtoms: sneexing, runny nose, itchy eyes, itchy throat
Daytime sleepiness common
Allergic P/E
-Pale and boggy turbinates
-red turbinates
-Cobblestoning
o Symptoms: sneezing, runny nose, itchy eyes, itchy throat
Daytime sleepiness common
o Complications:
-OM
-Sinusitis
-Obstructive sleep apnea
-Misalignment of the jaw
Allergic Tx
-Non sedating antihistamines
-Claritin
-Zyrtec
-Nasal corticosteroids
-Nasonex
-Flonase
-Singulair (leukotreiene antagonist)
Epistaxis
Common causes in children-
-trauma
-dry air/ mucosa
-Nasal sprays
-Most common site: front of the nasal septum
-Exam findings-
Raw nasal mucosa, crusting, blood
Usually benign, work-up if:
Fam hx of bleeding disorder (like hemophilia)
H/O easy bleeding
Bleeding > 30 mins
Abnormal Hgb
Onset < 2yrs of age
Initial labs
-CBC, PT, PPTT
-TX- pressure, keep mucosa moist
ORAL CANDIDIASIS (Thrush)
White curd like plaques
Mild mouth discomfort
Adherent (may come off with vigorous rubbing)
Usually mild sources
Can occur after use of antibiotics, cause of inhaled corticosteroids like treating asthma, doesn’t come off usually
Thrush Tx
-Nystatin suspension X 1 week
-Consider Fluconazole (Diflucan) oral suspension
-Wash bottles, pacifiers
-Apply to breast if breastfeeding
HERPES GINGIVOSTOMATITIS
SVI
-Generalized- can be on the tongue, around the mouth, looks like bunch of canker sores
-More common < 3 yrs of age
Source: somebody shedding the virus HSVI
o Symptoms/ signs include:
Lots of PAN, bleeding gums, decreases appetite, drooling, fever, ulcerations, cervial Lymphadenopathy (LAD)

HSV can also affect other place
HERPES GINGIVOSTOMATITIS
Tx
-Motrin or Tylenol
-Acyclovir Susp- 200 mg/5 cc
-No Corticosteroids
-Magic Mouthwash-various combos
-Benadryl, Maaloxm Viscous Lidocaine 2% (20 cc each)
-Education
-Prevent dehydration, decrease infectivity
Apthous Ulcers
Anterior
Pain
Self limited
Recurrent
Unknown cause
Apthous Ulcers Tx
-Betamethasone ointment BID
-Bland foods
-Mortin or Tylenol
-Warm H20 gargles
Herpangina
Coxsackie virus
Summer/ fall
Ulcers, runny nose, low fever
Anterior pillars, soft palate, uvula Recurrent self-limited
Herpangina Tx
-Motrin or Tylenol
-Magic Mouthwash
Pharyngitis (Viral)
-90% are viral origin
-Signs/ Symptoms
-Lab-rapid strep or throat c/s
+ treat it … (-) then culture it
Strep Throat
-GAS/ SABHS/ Strep pyogenes
-Signs and Symptoms
-Acute onset of fever and sore throatBeef red throat, exudate, petechiae (small red dots on soft palate) cervical LAD
Tx of Strep Throat
LABS: order rapid strep screening/ culture sensitivity
TxL oral PCN TID
o Alternatives-
IM PCN, Amox, Cepthalosporin
Zithromax, E mycin
-NO Bactrim (doesn’t prevent rheumatic fever and doesn’t deal with strep..)
-Can give PCN injection (once)
o Complications
-Scarlet Fever (benign…rash on stomach and tongue)…so always check the throat carefully with non specific rashes
-Rheumatic Fever (can occur in skin, cardiac problems, joint problems---need to be careful..This is due to toxins of strep)
-Acute glomerulonepthritis (can affect kidneys)
-Abscess
Complications of Strep Throat
-Scarlet Fever (benign…rash on stomach and tongue)…so always check the throat carefully with non specific rashes
-Rheumatic Fever (can occur in skin, cardiac problems, joint problems---need to be careful..This is due to toxins of strep)
-Acute glomerulonepthritis (can affect kidneys)
-Abscess
Peritonsillar cellulitis or abscess
-Source: same as step throat.
Sx:
-Severe sore throat-often unilateral
-Fever
-Dysphagia
-Hot Potato voice (gargles type of sound)
Exam:
Uvula and soft palate
LAD
Muffled voice
Tx:
Peritonsillar cellulitis or abscess Tx
Tx:
Admit for IV antibiotics, possible I & D
Management of bacterial conjunctivitis
o topical
• Erythromycin, ocuflox, polymyxin-bacitracin, sulfacetamide (topical)
• Ocuflox – flouroquinolone
 Only use topical flouroquinolones on eye or ears (not IV or oral w/kids)
• Some may burn – sulfacetamide
Viral Conjunctivitis Tx
o Very contagious, usually self limited
• Secondary bacterial infections DO occur
• Avoid corticosteroids, esp if it pertains to Herpes
Allergic Conjunctivitis Tx
o Acular (anti-inflam eye drop), Patanol (anti-histamine eye drop), Naphcon-A (anti-histamine and vasoconstrictor)
Pink Eye Education and Tx
o Infectivity, no improvement, return to school
• Viral – contagious at least a week
• Bacterial – after on abx for a couple of days can go back to school, redness will be better by then, not contagious
• General – if no improvement after being on abx for a couple days
o May be resistant, misdiagnosis, follow up
Periorbital/Preseptal Cellulitis
• Orbital septum separates eyelid from orbit
• Preseptal infection - of eyelid only and some surrounding skin around lid (eyelid infection)
o Sx
• LID edema, LID redness, LID pain, mild fever
o Source
• Eyelid abrasions, insect bites, or nearby infections
o Common Pathogens
• Staph aureus, Strep pyogenes
o Tx: Oral Abx w/ close follow up
o IV or IM abx if severe
Orbital Cellulitis
• Infection in the orbit of eye, surrounding the eye
• Sx:
o Same as periorbital PLUS:
• Restricted eye movement (do H Test)
• Proptosis
• Usually a HIGH fever, may appear toxic
• Source
o Contiguous sinus infection
o May be a secondary preseptal infection
• Pathogens
o Strep pneumo, H Influenzae, M Catt, Staph aureus
o Also consistent w/otitis media/HEENT disorders
o When treating – make sure rx will cover all of these
Orbital Cellulitis Tx
: IV ABX, surgical drainage
o Watch for orbital compression of optic nerve → vision loss

• CT Scan – check for pus in orbit to differentiate
o Difficult to tell apart by the visual appearance
Corneal Abrasion
• Sx
o Infants – usually scratched cornea w/ their own hand
o Older children – hx of eye trauma
• Exam
o Check visual acuity first
o Evert the upper lid,
o Check lower lid well
o Checking for foreign body
o Stain the eye to see uptake of dye in the abrasion
• Management
o Watchful waiting
o Time
o DAILY follow ups to observe improvement/watch for infections
• Prognosis
o Great, unless significant scratch/scarring
Retinopathy of Prematurity
• Common risk factors
o First check to see if they are under the care of an ophthalmologist already
o Retina is not completely vascularized/ not fully mature
o Vessels predisposed to hemorrhage
o If retina not getting a full blood supply → it will detach
Retinoblastoma
• The most common primary intraocular malignancy of childhood
o Age at presentation
o DO: Red Reflex Check – white pupil, reflex absent
• Etiology
o Retinal cell mutation
o Hereditary or sporadic
• Sx
o Single or multiple tumors
o Unilateral or bilateral
o leukocoria, strabismus, glaucoma, blindness, proptosis, red eye, pus in anterior chamber
• Management
o Detect and refer
• Prognosis
o Depends on characteristics of the specific tumor
Leukocoria
cataract is more diffuse, hazy cornea, retinoblastoma is intraocular, so it’s not hazy, it’s a sharp margin of a white pupil in the light
The most appropriate test for assessing vision in a newborn infant:
looking for blepharospastic response to bright light
Infant Ears
50% of time won’t see eardrum well
• Flat eardrum in infants
• Angulate speculum down
AOM Risk Factors
o Flat eustachian tube
o Age – bottle feeding
• Young – haven’t been exposed to many viruses/coughs/less immune than older
• Colonization – predisposed to bacterial infections, more likely to have bacterial infection
• URI’s
• Daycare – pathogen exposure
• Impaired immune system
• Smoking – inflammation to middle ear, blockage of ear, higher risk for middle ear infection
• Bottle feeding – fluids don’t drain well in recumbent position
AOM Key Features
o Key Features
• Fever, ear pain, inflammation, effusion
• Other
 Recent URI, decreased sleep, increasing crying, ear tugging, decreased appetite, vomiting
 Decreased TM mobility, decreased light reflex
• Special Considerations
 Fever, (fever can cause a red eardrum – may not be AOM),crying (can cause red eardrum), cerumen (can obstruct view of TM – may need to dig out wax to see)
 Decreased incidence with increasing age
AOM Pathogens
• S pneumo – PCN drug resistance common
 Most invasive pathogen
 May be decreasing
• H influenzae – on the rise, but less invasive
• M. catarrhalis
• S. pyogenes
AOM Vaccine
– pneumococcal vaccine (Prevnar) – to prevent certain strains of strep pneumo in ear infections in kids
o Now with the vaccine, the other strains are on the rise, and the strep pneumo decreasing
Recurrent Aphthous Stomatitis
Small (3-10 mm) ulcers; lips/tongue
No fever/LAD
Painful - last 1-2 wks
Tx: CTSD paste: Triamcinolone/Betamethasone Bid
Bland Diet
Acetaminopen/Motrin
Assoc. w/Behcet disease/Med. fever adn PFAPA
Thrush
C. albicans
Patho: vertically passed at birth; ABX/ICS use predisposition; immunocompromised

Tx: Nystatin, supsension x1 wk, apply 4x/day, stop when gone (3-4 days)
Systemic antifungal if no response

Replace infected pacifiers, and tx mother (breast feeding)
Acute stomatitis:
Herpes Simplex Gingivostomatitis
HSV1
Sx: Painful/bleeding gums/decreased appetite/HIGH fever (7-10 days)
Ulcerations (>10)
Cervical LAD, Halitosis;
diffuse mouth infxn preceding ulcer appearance
MC <3 y/o
-Herpetic Whitlow - spread to fingers --> can then spread to eyes
Tx: Pain mgmt (if >6mos/o)
Magic Mouthwash: Combo: Benadry, Maalox, +/- viscous lido (20 cc each)
-older swish + spit
-younger - wipe mouth w/Qtip
-Acyclovir - if tx early
Comp: dehydration; herpetic laryngotracheitis
Infectious Mononucleosis (EBV)
Gray exudative tonsillitis
Cervical Adenitis
Splenomegaly
Fever
usually >5 y/o
R/o strep: strep test first, then Mono spot (2-3 days)
(+) supports; (-) doesn't R/o Mono --> false (-) if < 5 y/o
EBV Ab Titer (IgM) - Elevated IgM capsid = DEFINITIVE

CBC = abnormal shape
Blood Smear > 10% atypical lymphocytes --> falsey (-) if <5 y/o

NO AMOX --> Rash
Tx: Symptomatic
Herpangina
Ulcers 3mm w/halos: anterior pillars, soft palate, uvula
Benign Glossitis (geographic tongue)
Coxsackie A Virus - summer/fall
Enteroviral PCR available, but usually unnecessary --> Self-limited
Symptomatic Tx

(Possible several episodes)
Pharyngoconjunctival Fever
Adenovirus
Often = epidemic; URI Sxs:
Exudative tonsillitis
Conjunctivitis
Lymphadenopathy
Fever
Tx = symptomatic
Hand Foot Mouth Disease
Several Enteroviruses cause it
Enterovirus 71: can cause encephalitis
Ulcers: anywhere in mouth
Vesicles; pustules; papules: palms, soles, interdigital; buttocks
Younger - distal extremities; face
Acute Bacterial Pharyngitis
Dx
Dx: Sore Throat +
@ least one of the following
-Cervical LAD (tender or >2 cm)
-Tonsillar exudates
-Fever >38.3C
(+) culture for group A B-hemolytic Strep

Do a rapid Antigen test, but confirm (-) rapid w/a Culture

10% of kids w/sore throat+fever = group A Strep
(sudden onset, beefy-red uvula, sore throat/odynophagia, palatal petechiae
Acute Bacterial Pharyngitis
Tx
1) PCN V, tid, PO, 10d course
--> beware of increasing PCN resistance and ABX failure after max dose for 10d.
--> fail to respond --> AUG or Azith

Alt: IM PCN, AMOX, Erythromycin/Azith, Ceph
(NO bactrim --> doesn't prevent Rheumatic Fever/doesn't cover Strep)

Comp: Scarlet Fever, acute glomerulonephritis, Rheumatic Fever (nodular rash/movement d/o's/Chronic Heart Dz/Hematuria)
Consider anti-streptolysin-o titers if elevated & pt wasn't tx'ed
Peritonsilar Cellulitis or Abscess
M/C is B-hemolytic Streptococcus
-tonsillar infxn that penetrates tonsillar capsule and spreads to surrounding tissue
- Unilateral sore throat (severe!)/dysphagia/trismus/drooling
-Hot Potatoe voice/ gargling/gutterall
-High Fever
-Tonsils bulge medially
-Soft palate swelling
-LAD; drooling
PE: Observe uvula/soft palate --> locate abscess
Difficult to differentiate cellulitis from abscess
-Admit 12-24 hrs w/IV ABX - aggressive tx prevents suppuration
- PCN or Clindamycin
-DC on oral ABX if fever cleared > 24 hrs and improved dysphagia
OR closely followed by ENT
**HIGH suspicion for abscess if failure to respond to ABX in 12-24 hrs
Retropharyngeal Abscess
Retropharyngeal Nodes: drain adenoids, nasopharynx, paranasal sinuses
--> become infected --> pyogenic adenitis --> Untx'ed = Abscess formation
M/C B-hemolytic Strep or S. aureus
Suspect in infants w/:
fever, RESPIRATORY SXS, Neck HYPEREXTENSION, Dysphagia, Drooling, Dyspnea, Gurgling, prominent unilateral swelling of posterior pharyngeal wall --> stops at midline --> confirms Dx
Difficult to differentiate abscess from Cervical Adenitis
--> Immediate Hospitalization + IV ABX (PCN or Clinda)
12-24 hrs of ABX helps ddx based on tx response
If abscess (+): surg. drainage
Ludwig Angina
Rapidly progressive
Cellulitis of submandibular space
- M/C d/t dental DZ or lacerations/injuries
Group A Strep
-Fever
-Tenderness w/swelling to floor of mouth
-Enlarged tongue - tender/red
Upwardly displaced tongue - dysphagia/drooling
--TX: ICU; IV Clindamycin; monitor airway closely for obstruction
-->Refer to otolaryngologist for drainage procedure
Acute Cervical Adenitis
M/C B-hemolytic Strep (also S. aureus, viral)
Local ENT infxns that involve a reginal node --> develop abscess
-Typically a unilateral, solitary, anterior Cervical Node
-Isolated, tender LAD w/fluctuance
-Fever
LABS:
CBC w/diff
Rapid A group Strep test
Monospot - if multiple nodes
PPD - Anti-TB Mycobacterium
-TX: ABX and aspiration culture/I&D if fluctuance
R/o neoplasm
Also Cat Scratch DZ - papule @ scratch site
Cervical Lymphadenitis - NonTB Myco - unilateral, w/several matted nodes (+ PPD)