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54 Cards in this Set

  • Front
  • Back
congenital heart disease
abnormal development of structures of the heart or major vessels of the heart
congenital heart disease (maternal exposure to agents)
- alcohol
- lithium
- coumadin
- retinoic acid
- sex hormones
congenital heart disease (maternal condition or infection)
- viral infections
- diabetes
- systemic lupus erythematosus
- PKU phenylketonuria
genetic/syndrome factors related to congenital heart disease
- trisomy 21
- turner syndrome
- digeorge syndrome
- heterotaxy syndrome (organs reversed)
- VACTERL syndrome
congestive heart failure
heart is unable to pump sufficient cardiac output to meet metabolic demands of the body
causes of CHF
- congenital heart disease (excess pressure/volume load)
- non structural myocardial factors
- dysrhythmias
CHF CM (low cardiac output)
- tachcardia
- decreased peripheral perfusion (blue feet)
- cardiomegaly
- oliguria
CHF CM (pulmonary congestion)
- tachypnea
- retraction, nasal flaring
- cough
CHF CM (systemic venous congestion)
- hepatomegaly
- edema (coccyx, genital, face. adults lower limbs)
- pleural/percardial effusions
CHF s/s
- **irritability**
- diaphoresis
- poor feeding (too tired to eat)
- failure to thrive
- low urine output
CHF management
- myocardial efficiency: digoxin, afterload reducers
- minimize volume overload: diuretics
- decrease cardiac workload
- adequate nutrition: increased calories d/t increased HR
CHF provide adequate nutrition
- small frequent feedings
- position during feeding
- high caloric feeding
- gavage feeding
- gastronomy tube
CHF parent support/teaching
- manage high stress level
- feeding education/support
- medication administration
- when to call the provider (when seeing s/s)
cyanosis
blue color of skin and mucuous membranes d/t increased concentration of reduced hemoglobin. 80-85% O2 stat
causes of cyanosis
- intracardiac mixing
- decreased pulmonary blood flow
cyanosis CM
- blue color at lips, nailbeds, mucous membranes
- tachypnea
- increased crying
- long term: polycythemia
cyanosis management
- **O2 not usually effective
- child will usually limit self
- interventional cardiac cath
- surgery
work up for congenital heart disease
- H & P
- chest x ray
- ECG
- echocardiogram
- cardiac cath
pre cath procedure
- vital signs, pulse ox
- mark pedal pulses
- labs: coagulopathies, CBC, electrolytes
post cath procedure
- assess peripheral pulses
- direct pressure on site if bleeding
- keep leg still to prevent further bleeding
patent ductus arteriosis
opening that causes mixing of blood
how is patent ductus arteriosis corrected
coil occlusion: coil threaded up aorta and create thrombus to close the hole
atrial septal defect
blood flow from LA -> RA through atrial septum. >9mm large needs to be treated. if left untreated heart will be enlarged.
correction of atrial septal defect
atrial septal defect occlusion device
coarctation of the aorta
narrowing/stenosis of the aorta
classic finding of coarctation of the aorta
arm BP > leb BP by 10+mmHG
correction of coarctation of the aorta
w/surgery but can cause scarring which can require additional surgery
pulmonary stenosis
hypertrophy of RV which can cause CHF.
correction of pulmonary stenosis
balloon valvuloplasty
post cath problems
- dysrhythmias
- thrombosis
- vessel dissection
- bleeding at the site
post cath nursing
- assess recover from anesthesia
- vital signs, pulse oximeter
- assess cath site
- assess perfusion distal to cath site
- keep leg still
ventricular septal defect
> 9 mm must be repaired, btw 2 ventricals
tetralogy of fallot
- pulmonic stenosis (primary defect)
- overriding aorta
- ventricular septal defect
- rt. ventricular hypertrophy
tet spell
pulmonary congestion. increased pressure in the lungs, more breathless. pull legs to chest. and meds to reduce pressure on the heart
transposition of great arteries
normal O2 stat < 70. complete absence of tricuspid valve. requires corrective surgery
tricuspid atresia
lack of tricuspid, surgery upon being born to create opening
discharge teaching
- review of defect and repair
- when to call the practitioner
- medications
- activity limits
- diet
- wound care
- FU appointment
bacterial endocarditis
infection of endocardium and intracardiac valves.
which children are at highest risk of bacterial endocarditis
CHD (valvular abnormalities, prosthetic valves, high pressure shunts)
bacterial endocarditis (pathophysiology)
- organisms grow on area of endocardium that has been exposed to abnormal blood streaming, jet of blood or turbulance
- thickened endocardium prediposes growth of pathogens
- vegetation, fibrin, thrombi form on valves
- risk of emboli to CNS, lung, spleen, kidney
bacterial endocarditis (portals of entry)
- dental work (antibiotics before dental work)
- cath with UT
- cardiac surgery
- indwelling lines
bacterial endocarditis CM
- insidious onset
- unexplained low grade, intermittent fever
- malaise, headaches, myalgias, arthralgias, diaphoresis, weight loss
- new or changed murmer
- splenomegaly
bacterial endocarditis Dx
- positive blood culture
- prolonged PR
- cardiomegaly
- anemia
- increased ESR, WBC
- hematuria
- vegetations seen on ECG
bacterial endocarditis Tx
- high dose IV antibiotics
- periodic blood cultures to check response to Tx
bacterial endocarditis prognosis
- 80% success if treated early
- death from CHF, MI, emboli, cardiac perforation
- high risk pts (infants, resistant organisms, fungal infection)
bacterial endocarditis nursing
- oral health and care
- teach need for specifics of subacute bacterial endocarditis prophylaxis
- high index of suspicion for children with CHD
- teach symptoms of subacute bacterial endocarditis
kawasaki disease
- acute systemic vasculitis
- males > females
- self limiting
kawasaki disease etiology
- unclear
- infectious agent
- late winter, early spring outbreaks
kawasaki disease pathophysiology
- inflammation of small and medium size vessels
- pancarditis (inflammation of heart)
- decreased left ventricular function
- aneurysms
- deadly
kawasaki disease Dx
5 of the following including fever
- fever > 5 days
- bilateral conjunctival injection w/o exudate
- strawberry tongue
- extremity edema, erythema and desquamation
- rash
- cervical lymphadenopathy
kawasaki disease s/s
- unresponsive high fever
- fussy and inconsolable
- temporary arthritis
kawasaki disease lab
- increased WBC
- increased sedimentation rate
- increased LFT
- thrombocytosis
- baseline and serial echocardiograms
kawasaki disease management
- high dose IVIG
- aspirin
- warfarin if giant aneurysms (> 8mm)
kawaksaki disease nursing
- control fever
- monitor for cardiac decompensation
- quiet environment
- soft foods
- symptomatic relief (cool cloth, meticulous oral care)