Use LEFT and RIGHT arrow keys to navigate between flashcards;
Use UP and DOWN arrow keys to flip the card;
H to show hint;
A reads text to speech;
54 Cards in this Set
- Front
- Back
congenital heart disease
|
abnormal development of structures of the heart or major vessels of the heart
|
|
congenital heart disease (maternal exposure to agents)
|
- alcohol
- lithium - coumadin - retinoic acid - sex hormones |
|
congenital heart disease (maternal condition or infection)
|
- viral infections
- diabetes - systemic lupus erythematosus - PKU phenylketonuria |
|
genetic/syndrome factors related to congenital heart disease
|
- trisomy 21
- turner syndrome - digeorge syndrome - heterotaxy syndrome (organs reversed) - VACTERL syndrome |
|
congestive heart failure
|
heart is unable to pump sufficient cardiac output to meet metabolic demands of the body
|
|
causes of CHF
|
- congenital heart disease (excess pressure/volume load)
- non structural myocardial factors - dysrhythmias |
|
CHF CM (low cardiac output)
|
- tachcardia
- decreased peripheral perfusion (blue feet) - cardiomegaly - oliguria |
|
CHF CM (pulmonary congestion)
|
- tachypnea
- retraction, nasal flaring - cough |
|
CHF CM (systemic venous congestion)
|
- hepatomegaly
- edema (coccyx, genital, face. adults lower limbs) - pleural/percardial effusions |
|
CHF s/s
|
- **irritability**
- diaphoresis - poor feeding (too tired to eat) - failure to thrive - low urine output |
|
CHF management
|
- myocardial efficiency: digoxin, afterload reducers
- minimize volume overload: diuretics - decrease cardiac workload - adequate nutrition: increased calories d/t increased HR |
|
CHF provide adequate nutrition
|
- small frequent feedings
- position during feeding - high caloric feeding - gavage feeding - gastronomy tube |
|
CHF parent support/teaching
|
- manage high stress level
- feeding education/support - medication administration - when to call the provider (when seeing s/s) |
|
cyanosis
|
blue color of skin and mucuous membranes d/t increased concentration of reduced hemoglobin. 80-85% O2 stat
|
|
causes of cyanosis
|
- intracardiac mixing
- decreased pulmonary blood flow |
|
cyanosis CM
|
- blue color at lips, nailbeds, mucous membranes
- tachypnea - increased crying - long term: polycythemia |
|
cyanosis management
|
- **O2 not usually effective
- child will usually limit self - interventional cardiac cath - surgery |
|
work up for congenital heart disease
|
- H & P
- chest x ray - ECG - echocardiogram - cardiac cath |
|
pre cath procedure
|
- vital signs, pulse ox
- mark pedal pulses - labs: coagulopathies, CBC, electrolytes |
|
post cath procedure
|
- assess peripheral pulses
- direct pressure on site if bleeding - keep leg still to prevent further bleeding |
|
patent ductus arteriosis
|
opening that causes mixing of blood
|
|
how is patent ductus arteriosis corrected
|
coil occlusion: coil threaded up aorta and create thrombus to close the hole
|
|
atrial septal defect
|
blood flow from LA -> RA through atrial septum. >9mm large needs to be treated. if left untreated heart will be enlarged.
|
|
correction of atrial septal defect
|
atrial septal defect occlusion device
|
|
coarctation of the aorta
|
narrowing/stenosis of the aorta
|
|
classic finding of coarctation of the aorta
|
arm BP > leb BP by 10+mmHG
|
|
correction of coarctation of the aorta
|
w/surgery but can cause scarring which can require additional surgery
|
|
pulmonary stenosis
|
hypertrophy of RV which can cause CHF.
|
|
correction of pulmonary stenosis
|
balloon valvuloplasty
|
|
post cath problems
|
- dysrhythmias
- thrombosis - vessel dissection - bleeding at the site |
|
post cath nursing
|
- assess recover from anesthesia
- vital signs, pulse oximeter - assess cath site - assess perfusion distal to cath site - keep leg still |
|
ventricular septal defect
|
> 9 mm must be repaired, btw 2 ventricals
|
|
tetralogy of fallot
|
- pulmonic stenosis (primary defect)
- overriding aorta - ventricular septal defect - rt. ventricular hypertrophy |
|
tet spell
|
pulmonary congestion. increased pressure in the lungs, more breathless. pull legs to chest. and meds to reduce pressure on the heart
|
|
transposition of great arteries
|
normal O2 stat < 70. complete absence of tricuspid valve. requires corrective surgery
|
|
tricuspid atresia
|
lack of tricuspid, surgery upon being born to create opening
|
|
discharge teaching
|
- review of defect and repair
- when to call the practitioner - medications - activity limits - diet - wound care - FU appointment |
|
bacterial endocarditis
|
infection of endocardium and intracardiac valves.
|
|
which children are at highest risk of bacterial endocarditis
|
CHD (valvular abnormalities, prosthetic valves, high pressure shunts)
|
|
bacterial endocarditis (pathophysiology)
|
- organisms grow on area of endocardium that has been exposed to abnormal blood streaming, jet of blood or turbulance
- thickened endocardium prediposes growth of pathogens - vegetation, fibrin, thrombi form on valves - risk of emboli to CNS, lung, spleen, kidney |
|
bacterial endocarditis (portals of entry)
|
- dental work (antibiotics before dental work)
- cath with UT - cardiac surgery - indwelling lines |
|
bacterial endocarditis CM
|
- insidious onset
- unexplained low grade, intermittent fever - malaise, headaches, myalgias, arthralgias, diaphoresis, weight loss - new or changed murmer - splenomegaly |
|
bacterial endocarditis Dx
|
- positive blood culture
- prolonged PR - cardiomegaly - anemia - increased ESR, WBC - hematuria - vegetations seen on ECG |
|
bacterial endocarditis Tx
|
- high dose IV antibiotics
- periodic blood cultures to check response to Tx |
|
bacterial endocarditis prognosis
|
- 80% success if treated early
- death from CHF, MI, emboli, cardiac perforation - high risk pts (infants, resistant organisms, fungal infection) |
|
bacterial endocarditis nursing
|
- oral health and care
- teach need for specifics of subacute bacterial endocarditis prophylaxis - high index of suspicion for children with CHD - teach symptoms of subacute bacterial endocarditis |
|
kawasaki disease
|
- acute systemic vasculitis
- males > females - self limiting |
|
kawasaki disease etiology
|
- unclear
- infectious agent - late winter, early spring outbreaks |
|
kawasaki disease pathophysiology
|
- inflammation of small and medium size vessels
- pancarditis (inflammation of heart) - decreased left ventricular function - aneurysms - deadly |
|
kawasaki disease Dx
|
5 of the following including fever
- fever > 5 days - bilateral conjunctival injection w/o exudate - strawberry tongue - extremity edema, erythema and desquamation - rash - cervical lymphadenopathy |
|
kawasaki disease s/s
|
- unresponsive high fever
- fussy and inconsolable - temporary arthritis |
|
kawasaki disease lab
|
- increased WBC
- increased sedimentation rate - increased LFT - thrombocytosis - baseline and serial echocardiograms |
|
kawasaki disease management
|
- high dose IVIG
- aspirin - warfarin if giant aneurysms (> 8mm) |
|
kawaksaki disease nursing
|
- control fever
- monitor for cardiac decompensation - quiet environment - soft foods - symptomatic relief (cool cloth, meticulous oral care) |