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46 Cards in this Set

  • Front
  • Back
0-3 month
Heart rate
Mean 140
Awake 85-205
Sleep 80-160
3 mon-2 years
Heart rate
Mean 130
Awake 100-190
Sleep 75-160
2-10 years
Heart rate
Mean 80
Awake 60-140
Sleep 60-90
> 10 years
Heart rate
Mean 75
Awake 60-100
Sleep 50-90
Tachyarrhythmias
Nonspecific signs
Heart palpitations
Light-headedness
syncope
Congestive heart failure signs
in an infant
Poor feeding
irritability
rapid breathing
hepatomegaly
edema
crackles lungs
Tachyarrhythmias
Hemodynamically unstable
respiratory distres/failure
signs of shock with or without hypotension
Altered mental status
Sudden collapse with rapid, weak pulse
Narrow Complex Tach
Sinus tach (ST)
Supraventricular tach (SVT)
Atrial flutter
Wide Complex Tach
Ventricular tach (VT)
Supraventricular tach (SVT) with aberrant intraventricular conduction
Sinus Tach
Varies with;
respirations
positional changes
relaxation
fluid administration
Sinus Tach
Causes
exercise
pain
anxiety
tissue hypoxia
hypovolemic shock
fever
metabolic stress
injury
toxins/poisons/drugs
anemia
Sinus Tach
Less common causes
cardiac tamponade
tension pneumothorax
thromboembolism
Sinus Tach
EKG Characteristics
Infants- Usually <220
Children- Usually <180
Variable with activity and stress
P waves present/normal
PR constant/normal
R-R Variable
QRS narrow
Signs of SVT
Infants
irritability
poor feeding
rapid breathing
unusual sleepiness
vomiting
pale, mottled, gray or cyanotic skin
Signs of SVT
Children
Palpitations
shortness of breath
chest pain or discomfort
dizziness
light-headedness
fainting
AIRWAY
SVT effects
Usually patent unless LOC is significantly impaired
BREATHING
SVT effects
Tachypnea
Increased work of breathing
Crackles (or wheezing in infants) if CHF develops
Grunting if CHF develops
CIRCULATION
SVT effects
Tachycardia beyond ST range. fixed rate/ abrupt onset
Delayed cap refill
Weak peripheral pulses
Cool extremities
Diaphoretic, pale, mottled, gray, cyanotic skin
Hypotension
Jugular venous distention if CHF develops (diff. to see in young children)
DISABILITY
SVT effects
Altered mental status (LOC)
Sleepiness or lethargy
Irritability
EXPOSURE
SVT effects
Defer eval of temp until ABC's supported
SVT
EKG Characteristics
Usually >220 infants
Usually >180 Children
No beat to beat variability
P; absent/ or abnormal
PR; undeterminable
R-R; Constant
QRS; Narrow/ wide uncommon
Atrial Flutter
Clasically a "sawtooth" pattern on EKG
can be >300
More common
newborn infants with normal hearts
Kids with congenital heart disease, especially following heart surg
V. Tach
Uncommon among children; Electrolyte disturbances
Drug toxicity (tricyclic antidepressants, cocaine, methamphet)
underlying heart disease
long QT syndrome
myocarditis/cardiomyopathy
V. Tach
EKG Characteristics
>120/regular
QRS; wide
P; usually not identifiable
Torsades de pointes
Wide polymorphic v.tach
Hypomagnesemia
hypokalemia
Drug toxicity (quinidine, procainamide, diisopyramide, encainide, flecainide, sotalol, amiodarone
Torsades de Pointes
Can quickly deteriorate to V. fib
CIRCULATION
Questions
PULSES + OR -
No- Cardiac arrest algorithm
Yes- Tachycardia algorithms
PERFUSION
Questions
Poor or Adequate
Poor- Tachycardia with pulse/ poor perfusion algorithms
Adequate- Tachycardia with pulse/ poor perfusion algorithms (Consider cardiology consult)
RHYTHM
Questions
Narrow or Wide
Narrow- Differential of ST vrs SVT
Wide- Differential of SVT vrs VT, treat as presumed VT unless known aberrant history
Atrial Flutter
Clasically a "sawtooth" pattern on EKG
can be >300
More common
newborn infants with normal hearts
Kids with congenital heart disease, especially following heart surg
V. tach
Uncommon in children
Wide complex
>200
V. Tach in children
Causes
Electrolyte disturbances (hyperkalemia, hypocalcemia, hypomagnesemia)
Drug Toxicity (Tricyclic antidepressants, cocaine, methamphetamines)
Heart disease
long QT syndrome
V. tach
EKG characteristics
>120
QRS; Wide
P; Often not identifieable
V. Tach
Assume wide complex rhythm is VT unless known aberrant conduction
Torsades de pointes
causes
Long QT syndrome
Hypomagnesemia
Hypokalemia
Antiarrhythmic Drug toxicity (quinidine, procainamide, sotalol, amiodarone)
Other drug toxicity (tricyclic antidepressants, calcium channel blockers, phenothiazines)
Circulation
Questions
pulse + or -
Absent- cardiac arrest algorithm
Present- Tachycardia algorithm
Perfusion
Questions
Poor- Tachycardia with a pulse/ poor perfusion algorithm
Adequate- Tachycardia with a pulse/ poor perfusion algorithm (consider cardiac consult)
Rhythm
Questions
Narrow- Differentiate ST vrs SVT
Wide- Differentiate ST vrs VT
Tachycardia Management
ABC's and O2
Monitor/ defibrillator and pulse ox
IV
12 lead EKG
Labs (Potassium, glucose, ionized calcium, magnesium, blood gas ph)
LOC
Anticipate meds
identify and treat reversible causes
Vagal maneuvers
Infant- ice to face 15-20 sec
child- blow through narrow straw
older child- carotid sinus massage
Vagal maneuvers
ABC
12 lead before and after if possible- dont delay treatment
If stable- may attempt twice if first is unsuccessful
If unstable- Only attempt while pharmacological and electrical conversions are being set up
Cardioversion
Establish iv and sedation
If unstable, do not delay treatment for iv access
Potential problems with
synchronized cardioversion
Synch button must be activated for synchronization to occur, and monitors usually return to default settings
R wave may not be identified by machine, and other lead may need to be used
Staff may be unfamiliar with operation
Synchronized cardioversion
Initial dose; 0.5-1 J/kg
Subsequent dose; 2 J/kg
When Synchronized Cardioversion is used
Hemodynamically unstable (poor perfusion, low BP, or heart failure)
Tachyarrhythmias (SVT, atrial flutter , VT) with pulses
Tachycardia medications
Adenosine
Amiodarone
Procainamide
Lidocaine
Magnesium sulfate