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30 Cards in this Set

  • Front
  • Back
Patent Ductus Arteriosus (PDA)
Failure of the fetal ductus arteriosus to close within the first weeks of life. This makes a reversal in blood flow due to increased aortic pressure.

At risk for endocarditis and pulmonary vascular obstruction disease.
PDA Clinical Signs
May be asymptomatic or show signs of CHF.

These children may become diaphoretic while eating and they may tire while eating.
Murmur
A sound that is produced by vibrations within the heart chambers or in the major arteries from the back and forth flow of blood.

rated on a scale of 1-6 with grade 1 and 2 barely audible.
Atrial Septal Defect (ASD)
An abnormal opening between the atria, allowing blood from the higher pressure left atrium to flow into the lower pressure right-ASD1, ASD2, Sinus venous defect. More common in females.
Heart sounds
S1-beginning systole, loudest at apex and best heard over the mitrial and tricuspid areas. Closure of the AV valves.
S2- Loudest at the base. Closure of the semilunar valves, best heard over the pulmonic and aortic areas.
S3-Normal in some children and young adults. Best heard over mitrial area.
S4-not usually good to hear.
Coarctation of the Aorta
Narrowing of the aorta usually distal to the origin of the left subclavian artery.Most common site is at the ductus. Aortic narrowing increases resistance to aortic flow. There will be increase left ventricular pressure and work load.

Coartation common w/ VSD
Coartation manifestation
Difference in extremity BP’s and pulses-uppers will be hypertensive with bounding pulses while the lower extremity. Pulses may be weak and hypotensive. Decrease systemic perfusion. Always document where pulses were taken on a child.
Hemodynamics
Older children will have increase blood flow to upper body and decrease blood flow to the lower body. Increase Afterload to left ventricle. The renal arteries will receive decreasing flow. Rennin will then be released, causing hypertension in the ascending aorta.

Associated with neonates with a closed PDA
VSD
Most common heart defect
Ventricular Septal Defect (VSD)
Most common CHD.Abnormal opening between the right and left ventricles. 20%-60% are thought to close spontaneously during the first year of life.
Tetralogy of Fallot (TET)
Most common cyanotic heart defect.10% of CHD.
Requires 4 defects: Pulmonic stenosis, Overriding aorta, ventricular septal defect (VSD), right ventricular hypertrophy.
TET Manifestation
have episodes of cyanosis or hypoxia
use calm approach, O2, morphine, place in knee-chest position. Possible neurological complications as well as dehydration ( may cause strokes). may have cyanosis, poor weight gain, and short statue.
Transposition of the great vessels
Pulmonary artery leaves the left ventricle and Aorta exits from the right ventricle. Results in no communication between the systemic and pulmonary circulation.
Tricuspid Atresia
Failure of the tricuspid valve to develop.
Cyanosis in newborn period, tachycardia and dyspnea.
Older children may have signs of chronic hypoxemia and clubbing. Newborns may be given continuous infusion of prostaglandin E until surgical intervention arranged.
Heart Failure
Occurs most often with children with a CHD.
Cardiomyopathy
Myocardium cannot contract properly:
3 types: restrictive, dilated, hypertrophy.
Kawasaki Disease
Acute systemic vasculitis most commonly seen in infants and young children. Usually seen in winter and spring.
Kawasaki manifestation
Fever>102.2 x 5 days
Strawberry tongue (cracked and red), Desquamation of perineal area, fingers, toes

Treat with aspirin - prevent clot formation from swelling vessels.
Hypertension
Primary-rare, but increases with age
Secondary-related to underlying medical problem (usually renal)
Prolonged Q-T
Usually seen in adolescents.
Complaints of fatigue, syncope, inability to keep up with peers.
Rest, pacemakers.
Decrease physical activity.
Growth hormone deficiency (GHD)/ Hypopituitary
Inadequate production of secretion of growth hormone which results in poor growth and short stature.
Normal weight and length at birth. By age 1, ht<5% for age and sex. Micropenis and undescended testicles
Hyperpituitarism
Treat cause-removal of tumor, radiation therapy or oral bromocriptine
Diabetes Insipidus
Inability to concentrate urine due to deficiency of vasopressin or ADH.

Synthetic vasopressin (DDAVP) administered intranasally, SQ, IM or po-amount dependent on age and how administered. Intranasal given through a small flexible premarked tube twice a day or metered spray twice a day.
DI diagnostic
Water Deprivation-usually a 7 hour procedure where the child is deprived of water. In a child without DI, you would expect decrease urine output with high urine spec. gravity and no change in the Na level. A child with DI will continue to have large amount of dilute urine with low spec. gravity and increase Na level.
SIADH-Syndrome of Inappropriate Antidiuretic Hormone
Excessive production or release of ADH or vasopressin.

Fluid retention, weight gain
Hypotonicity
Precocious Puberty
Puberty before 8 for girls and before 9 for boys
Congenital Hypothyroidism
Present from birth since the thyroid gland does not produce enough thyroid hormones to meet metabolic needs of the infant.
Untreated leads to mental retardation.
Con. Hypothyroidism manifestations
Prolong jaundice, lethargy, constipation
Feeding problems, cold to touch, skin mottling, bradycardia
Umbilical hernia, hypotonia/ slow reflexes
Large tongue, large fontanel, distended abdomen
hyperthyroidism
Goiter, dry, thick skin and course hair, hair loss.
Tiredness/fatigue, cold intolerance, constipation
Weight gain, decreased growth, irregular menses
Edema of face, eyes, and hands
Hyperthyroidism Graves’ Disease
Excessive thyroid hormone by enlarged thyroid gland
Most in 6-15 year olds. Goiter, weight loss, increased appetite, diarrhea
Nervousness, increased perspiration, tremors, overheated/fatigue during physical exercise
Heat intolerance, increased heart rate