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66 Cards in this Set

  • Front
  • Back
Why is enteral feeding advocated over TPN for a multi trauma victim?
because it maintains bowel function and prevents translocation of bacteria
If a patient was admitted with an acute GI bleed what would be one of the first actions the nurse would want to take?
start a large bore IV before they became hypovolemic
Acute GI bleed causes a decrease in...
blood volume which can decrease the venous return to the heart thus decreasing CO
What are some causes of acute GI bleed...
peptic ulcer disease(most common)
-hypersecretion of gastric acid with impaired GI mucus secretion
-PUD can often be caused by helicobactor pylori (infection in the mucosa)

gastroesophageal varices
-if blood cannot flow easily to the liver because of obstruction; it is diverted to collateral channels
-esophagus--esophageal varices
-veins in the proximal stomach (gastric varices)
-rectal vault (hemmorrhoids)
What is the portal vein? Hepatic artery?
portal vein is how GI tract gets blood/nutrients to the liver (hepatic artery supplies oxygen)
What does the liver do?
every nutrient we consume passes through the liver
metabolizes carbs (synthesizes glucose), protein, lipids (produce cholesterol)
bile formation (which clears bilirubin from the from the blood-which results from dead blood cells-increase in bilirubin means jaundice)
(above pancreas and small intestine it is right upper abdomen)
How fast can PUD occur
6-8 hrs especially with stress in the hospital
Mallory Weiss syndrome
nonperforating tear that occurs in the gastric mucosa near the gastroesophageal junction (esoph and stomach)
-result of ETOH abuse and inflammatory conditions
-occurs most often due to the pressure changes in the stomach when vomiting
Hemorrhagic gastritis and it's cause
"stress ulcers"
gastric lesions that do not penetrate the muscularis mucosa
caused by NSAID use, ETOH abuse and severe stress (increased acid production)
What are some NSAIDS and what can they cause?
ibuprofen (motrin, advil) aspirin (bayer) aleve

they can cause hemorrhagic gastritis
What are signs and symptoms of an acute GI bleed?
hematemisis-throwing up blood
melena (tarry black sticky) /maroon colored stool
nausea
epigastric pain
abdominal distention
bowel sounds increased or decreased
orthostatic hypotension, tachy, decreased pulses
cold clammy skin
How do you diagnose acute GI bleed?
labs: H+H, CBC, PTT h. pylori

endoscopic procedure: scope
What are important steps to manage GI bleed
-insert two large bore IVs and IVFs
-administer blood products
-insert NG (decompress abdomen)
-position patient in left lateral decubitus position to decrease aspiration associated with hematemesis
What are surgical therapies for PUD/ gastritis
biliroth I and II
removal of lower portion of stomach (pylorus) with end to end anastomosis of the remaining stomach with the duodenum
I-anastomosis is directly to the duodenum
II-gastric stump joins to the jejunum
endoscopic therapy for variceal UGI bleed
sodium morrhuate is injected into the vessels which makes them shrink

rubberband ligation/hemoclips/
What is a temporary fix of variceal UGI bleed
sengstaken-blakemore tube (which emergently decreases blood flow through the varix to control bleeding)
What pharm therapy could be done for variceal UGI bleed
vasopression (vasoconstricts gastric inflow and portal pressure)

somatostatin (inhibits gastric blood flow)
What surgical option is available for varices?
TIPS (transjugular inrahepatic portosystemic shunt)

artificial channel in the liver from the portal vein to the hepatic vein
Diverticulitus
small pouches that develop in the large intestine secondary to aging
(occurs in 1/2 americans from60-80)
fecal material gets trapped in pouches leading to inflammation
(lack of fiber plays a role)
S&S of Diverticullitus
localized pain in the left lower quadrant
fever
chills
nausea and vomiting
cramping
constipation
Diagnostics of diverticulitus
CBC-check if WBC is high for a rupture
abdominal xray or CT
Management of diverticulitus
rehydration with IVFs
NPO status
NG placed for persistent vomiting
medications
-anticholingergics-reduce colonic spasms (cogentin, atrovent, atropine, robinul, spiriva, wellbutrin)
-opiates-methadone, morphine, oxy
-antibiotics
Crohns
chronic inflammation of the digestive tract that can involve one or more areas of any portion of the GI tract from mouth to anus
most common sites of inflammation in crohns
ilium, colon, and perianal
is crohns curable
no
causes of crohns
-genetic
-environmental-smoking, (constricts blood flow)hormonal contraception, NSAID use
S&S of Crohns
pain-at first right sided cramping that feels like appendicitis but later crampy abdomen pain associated with different foods

meals avoided to reduce pain-leads to weight loss/malnutrition

N/V watery stool steatorrhea (fatty)
fever( inflam process)
Diagnosing Crohns
labs CBC-albumin and BMP
colonoscopy and biopsy
Treatment of Crohns
antibiotics
anti inflammatory (sulfasalzine)
corticosteroids (hydrocortizone, decadron, prednisone)-act on immune system preventing inflammation but can also decrease WBC
immunosuppressive drugs
bowel restrictions
What can occur with increased doses of steroids
moonface/swollen
ulcerative colitis
type of IBD that affects large intestine
cause of ulcerative colitis
unknown
S&S of ulcerative colitis
sensation of rectal urgency
crampy abd. pain and tenderness
bloody watery diarrhea
(up to 30 stools/day)
weight loss vomiting diarrhea
surgical treatment of ulcerative colitis
coelctemy of lg intestine with ileostomy
bowel infarction
restricted blood flow to bowel
causes: abdominal distention, alpha stimulation sympathomimetic amines (epi and nor epi)
hypovolemia, decreased CO
bowel infarction can lead to...
bowel necrosis and then gangrene of the bowel
bowel obstructions causes
mechanical obstructions (ileum is most common site), adhesions from scar tissue, strangulated hernias (abd. muscle weakened or stressed-opening where bowel is caught and loses blood flow)
functional obstruction-paralytic ileus (no peristalsis) abd. surgeries
How would you position a pt with bowel obs/infarc
HOB elevated to relieve pressure from distended abdomen
goal of gastric bypass
decrease caloric intake
two catagories of bypass
restrictive-less invasive banding the upper portion of the stomach creating a smaller stomach pouch
(1-3 oz)

malabsorptive techniques
permanent include actually bypassing segment of sm. intestine
size of gastric pouch is reduced and anastomosed to a distal segment of the sm. bowel
after GI surgeries what precautions should be taken with med administration
no sustained release meds or diabetes meds because they are not taking in enough food and fluids so there will be decreased insulin resistance
What is hepatitis
injury to the liver characterized by the presence of inflammatory cells in the tissues of the organ
what causes hepatitis
viral ABCDE
autoimmune
drug related
hepatitis A
fecal oral (most common) usually via contaminated drinking water
caused by hep A virus (HAV)
hep B
DNA virus is present in all body secretions
exposure to blood/body fluids containing blood

(needles, childbirth, sexual contact, blood transfusions)
hep C
blood to blood
hep D
single stranded RNA virus
requires presence of hep B to establish infection

parenterally
what form of hep requires another form to be transmitted
hep D requires hep B
hep E
similar to A
fecal oral
autoimmune hep
idiopathic genetic predisposition of acute liver infection
drug related hep
acetaminophen, allopurinol, amoxicillin, anti TB meds

form of drug allergy in which the immune response is directed at the liver cells
causes necrosis
S&S of HEP
usually asymptomatic
possibly flu like symp.
diagnosis of hep
labs: liver panel, CBC, bilirubin levels
Treatment of hep
meds, transplant
gamma globulin...hep?
increases available antibodies
most common cause of cirrhosis of the liver
ETOH abuse

ETOH is directly toxic to liver cells, causing them to die
liver cells are replaced by fibrous tissue causing liver to progressively fail
S&S of Hepatic failure
jaundice
pruritis
ascites (fluid collection in the peritoneum)
hepatic encephalopathy
electrolyte imbalances (hypo-)
hepatic encephalopathy
worsening of brain function because the liver is unable to remove toxins from blood
ammonia is neurotoxic
muscle tremors slurred speech and mental status changes
hepatic failure diagnostics
labs
bilirubin
PT/PTT
ammonia
liver panel ALT AST
abd ultrasound (CT, MRI)
liver angiography and biopsy
HOB for hepatic failure pt.
45 degrees
Pancreatitis
inflammation of the pancreas resulting from premature activation of exocrine enzymes
causes of pancreatitus
Gallstones
Ethanol
Trauma

Steroids
Mumps
Autoimmune causes
Scorpion venom
Hyperlipidemias
Ercp (dye)
Drugs (tylenol, lasix, cocaine)
what diet form pancreatitis
NPO rest (TPN usually without lipids due to the high triglycerides anyways)
S&S of appendicitis
RLQ pain
rebound tenderness
anorexia
N&V
Cholecystitis
RUQ pain caused by gallstones (inflammation of gallbladder)
Managing cholecystitis
IVF, abx.