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28 Cards in this Set

  • Front
  • Back
What structures make up the upper aerodigestive tract?
- oral cavity
- nasal cavity
- paranasal sinuses
- pharynx
- larynx
what is the cause of acute infectious rhinitis or coryza?
viruses specifically adenovirus, echovirus, and rhinovirus
what are the clinical manifestations of coryza?
- inflammation
- edema (congestion)
- rhinorrhea (nasal discharge)
what is the cause of chronic infectious rhinitis?
a bacterial superinfection on top of coryza. these bacteria are usually part of the normal flora
what is empyema? when could you possibly see it?
- it is an accumulation of pus

- you could possibly see it as a complication to chronic infectious rhinitis
what type of hypersensitivity is involved in allergic rhinitis?
type I hypersensitivity; exposure to an allergen causes IgE to be made by plasma cells. subsequent exposure causes IgE mediated eosinophil degranulation
what are nasal polyps?
these are permanent swellings of the nasal mucosa
name 2 types of nasal polyps?
- atopic or allergic polyps
- nonatopic or inflammatory polyps
what is the make up of a nasal polyp?
- respiratory epithelium surrounding edema and eosinophils
what is the most likely cause of allergic fungal rhinosinusitis?
aspergillus
T or F: allergic fungal rhinosinusitis is non-invasive proliferation of a fungus.
true; there is little or no invasion into the tissue
you look at a specimen under the microscope from a patient with an upper airway problems. you can see branching organisms, eosinophils, and Curschmann spirals. what is the most likely diagnosis?
allergic fungal rhinosinusitis

eosinophils + Curschmann spirals = allergic mucin, which is seen in allergic fungal rhinosinusitis
what are 2 predisposing factors for developing invasive fungal rhinosinusitis?
- diabetic ketoacidosis
- immunosuppression
what is the most common organism involved in invasive fungal rhinosinusitis?
mucormycosis
what is the triad for Wegener's granulomatosis?
ELK
- ENT
- Lungs
- Kidneys
what is angiocentric NK/T cell lymphoma?
an aggressive type of non-Hodgkin lymphoma that is associated with EBV
a patient is found to have necrotizing granulomas in the nasal passages, lungs, kidneys, and ear canals and vasculitis. what is the most likely diagnosis?
Wegeners granulomatosis
what is lethal midline granuloma (also called polymorphic reticulosis)?
this is a syndrome when the center of the face becomes necrotic
what are the causes of lethal midline granuloma?
- invasive fungal rhinosinusitis
- wegener's granulomatosis
- angiocentric NK/T cell lymphoma (most common)
what is the most common cause of pharyngitis/tonsilitis?
viruses; echovirus, adenovirus, rhinovirus, EBV
a patient comes in complaining of a sore throat. physical exam shows a purulent exudate on the tonsils. what is the most likely diagnosis? are there any major complication you may worry about?
- bacterial pharyngitis/tonsilits

- if it is beta hemolytic streptococci causing the infection it could develop into rheumatic fever and a type of glomerulonephritis
a patient comes in complaining of a sore throat. physical exam shows enlarged, injected tonsils without any exudate. what is the most likely diagnosis? are there any complications you should worry about?
- viral tonsilitis

- adenotonsilar hyperplasia which can lead to airway obstruction
who gets nasopharyngeal angiofibroma?
males between the ages of 10-25
where are the most common locations for nasopharyngeal angiofibromas?
posterior and lateral wall of the nasopharynx
what is the major complication associated with nasopharyngeal angiofibromas?
the blood vessels lack a smooth muscle wall so hemorrhage is a major complication. if the blood vessels rupture they bleed a lot and are hard to stop. this is an especially big concern during surgery to remove them
what is the cause of sinonasal papillomas?
HPV type 6 and 11
how would a olfactory neuroblastoma look under the microscope?
these are small, round, blue cell tumors
T or F: the most common cause of death from olfactory neuroblastoma is invasion into the brain.
true