Pathology Of The Gb

Front 1

Caroli's Disease

Back 1

It is a rare congenital disease of of the intrahepatic biliary tree that results from malformation of the ductal plates, the primordial cells that give rise to the intrahepatic bile ducts. It is associated with cystic real disease, most often renal tubular ectasia (medullary sponge kidney)

Front 2

Choledochal Cysts

Back 2

A hetergeneous group of congenital diseases that may manifest as focal or diffuse cystic dilation of the biliary tree. True cysts in RUQ with or without communication with biliary system Jaundice and possibly increased bilirubin.

Hint 2

Localized cystic dilation of the common bile duct, diverticulum from the common bile duct. invagination of the CBD into the duodenum. dilation of the entire CBD and the CHD

Front 3

Sludge

Back 3

thickened bile, which occurs from bile stasis. This may be seen in patients with prolonged fasting. Gravity dependent.

Hint 3

Low-level internal echoes layering in dependent part of 'GB, prominent GB size, changes with patient position

Front 4

Acute Cholecystitis

Back 4

Inflammation of the GB which is the most common cause of acute cholecystitis is cholelithiasis. Found more freq in females.

Hint 4

Increase Amylase, Ab LFTs, Dialtion and rounding of GB, + Murphey's sign, thick GB wall with irregular wall edema, stones,

Front 5

Chronic Cholecystitis

Back 5

The most COMMON form of gallbladder inflammation. Results of numerous attacks of acute cholecystitis.

Hint 5

Contraction of GB, stones, WES sign. Increased Amylase, ab LFT's RUQ pain.

Front 6

Acalculous cholecystitis

Back 6

Inflammation of the GB without the formation of stones. Most likely caused by decreased blood flow through the cystic artery.

Hint 6

GB wall is extremely thickened, greater than 4 to 5 mm. and echogenic sludge is seen. dilation of GB, + murphy's sign. Not caused by cholelithiasis.

Front 7

Emphysematous Choleystitis

Back 7

Gas-Forming bacteria in GB, Ab LFTs

Hint 7

Bright echo in area of Gb with ring down or comet tail artifact, may appear as WES.

Front 8

Gangrenous Cholecystits

Back 8

May lead to perforation of the GB. The gallbladder wall may be thickened.

Hint 8

No shadow, medium to coarse echogenic densities that fill GB lumen in absence of duct obstruction. does not layer.

Front 9

Cholelithiasis

Back 9

Is the MOST COMMON disease of the gallbladder. May be a single large stone or hundreds of tiny ones. Gallstones may contain cholesterol.

Hint 9

patient fall under the 5 f''s:
Fat, female, forty fertile, fair.
Dilated GB with thick wall, hyperechoic intraluminal echoes with posterior acoustic shadowing. WES sign, gravity dependent calcifications in GB.

Front 10

Adenoma of the GB

Back 10

Benign neoplasms of the GB with a premalignant potential much lower than colonic adenomas,. Solitary lesion.

Hint 10

No shadow
Occur as flat elevations located in the body of the GB. Does not change with patient position.

Front 11

Adenomyomatosis

Back 11

A hyperplastic change in the gallbladder wall. Papillomas may occur singly or in groups and may be scattered over a large part of the mucosal surface of the GB.

Hint 11

Benign tumors of the GB,
"comet tail" artifact.

Front 12

Choledocholithiasis

Back 12

Stones in the bile duct. The majority of the stones in the CBD migrated from the GB.

Hint 12

Increased direct bilirubin. Ab LFT's leukocytosis, increased Alk phos. Echogenic structure in extrahepatic duct. dialted biliary tree.

Front 13

Porcelain Gallbladder

Back 13

A rare occurence that is defined as calcium incrustation of the GB wall.

Hint 13

Bright echogenic echo is seen in the region of the GB with shadowing posterior.