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22 Cards in this Set

  • Front
  • Back
ID
HASHIMOTO'S THYROIDITIS:
Thyoid is diffusely enlarged with an in-tact capsule and on gross exam, a cut surface that is grey-tan and nodular. Much of the thyroid is infiltrated by mononuclear inflammation containing plasma cells, small lymphocytes and well developed lymphoid germinal centers. Increased inter-follicular CT.
ID
HASHIMOTO'S THYROIDITIS:
Much of the thyroid is infiltrated by mononuclear inflammation containing plasma cells, small lymphocytes and well developed lymphoid germinal centers. Increased interfollicular CT.
ID
HASHIMOTO'S THYROIDITIS:
Much of the thyroid is infiltrated by mononuclear inflammation containing plasma cells, small lymphocytes and well developed lymphoid germinal centers. Increased inter-follicular CT.
ID
HASHIMOTO'S THYROIDITIS:
Infiltrate of lymphocytes, plasma cells and cell-developed germinal centers. Thyroid follicles that remain are small and atrophic. Some have abundant eosinophilic cytoplasm in follicular epithelial cells (Hurthle cells).
ID
HASHIMOTO'S THYROIDITIS:
Infiltrate of lymphocytes, plasma cells and cell-developed germinal centers. Thyroid follicles that remain are small and atrophic. Some have abundant eosinophilic cytoplasm in follicular epithelial cells (Hurthle cells).
ID
LEUKOCLASTIC VASCULITIS:
Dermis from slide where surface epithelia has been removed. Surrounding the capillaries is a perivascular infiltrate of neutrophils. Swollen gross appearance would be due to blood vessel dilation.
ID
LEUKOCLASTIC VASCULITIS:
Perivascular infltration of neutrophils. Some neutrophils in the walls of vessels are undergoing cellular fragmentation (leukocytoCLAST). Fibrinoid necrosis of vessel media may be present (not shown).
ID
LEUKOCLASTIC VASCULITIS:
Perivascular infltration of neutrophils. Some neutrophils in the walls of vessels are undergoing cellular fragmentation (leukocytoCLAST). Fibrinoid necrosis of vessel media may be present (not shown).
ID
LEUKOCLASTIC VASCULITIS:
Dermis from slide where surface epithelia has been removed. Surrounding the capillaries is a perivascular infiltrate of neutrophils. Swollen gross appearance would be due to blood vessel dilation.
ID
LEUKOCLASTIC VASCULITIS:
Dermis from slide where surface epithelia has been removed. Surrounding the capillaries is a perivascular infiltrate of neutrophils. Swollen gross appearance would be due to blood vessel dilation.
ID
LEUKOCLASTIC VASCULITIS:
Perivascular infltration of neutrophils. Some neutrophils in the walls of vessels are undergoing cellular fragmentation (leukocytoCLAST). Fibrinoid necrosis of vessel media may be present (not shown).
ID
LEUKOCLASTIC VASCULITIS:
Dermis from slide where surface epithelia has been removed. Surrounding the capillaries is a perivascular infiltrate of neutrophils. Swollen gross appearance would be due to blood vessel dilation.
ID
LEUKOCLASTIC VASCULITIS:
Dermis from slide where surface epithelia has been removed. Surrounding the capillaries is a perivascular infiltrate of neutrophils. Swollen gross appearance would be due to blood vessel dilation.
ID
SLE GLOMERULONEPHRITIS:
Note the increased number of glomerular cells + thickened capillary membranes (wire loops). Some of the parietal glomerular epithelia form crescent-shaped foci.

Immunoflourescene would show immune complex deposition in subendothelial and subepithelial areas, with antibodies to DNA.
ID
SLE GLOMERULONEPHRITIS:
SLE causing immune complex deposition in the kidney leads to MEMBRANOPROLIFORATIVE GLOMERULONEPHRITIS: 1) proliforation of epithelia in glomeruli and Bowman's capsule (proliforative glomerulonephritis) 2) thickening of glomerular capillary walls (membranous glomerulonephritis).

This biopsy also has a focal interstitial inflammation surounding some renal tubules (lymphocytes and plasma cells).
ID
SLE GLOMERULONEPHRITIS:
Note the increased number of glomerular cells + thickened capillary membranes (wire loops). Some of the parietal glomerular epithelia form crescent-shaped foci.

Immunoflourescene would show immune complex deposition in subendothelial and subepithelial areas, with antibodies to DNA.
ID
SLE GLOMERULONEPHRITIS

Focal interstitial inflammatory infiltrate surrounding some renal tubules? Lymphocytes and plasma cells.
ID
SLE GLOMERULONEPHRITIS:
Focal interstitial inflammatory infiltrate surrounding some renal tubules, composed of lumphocytes and plasma cells.
ID
SLE GLOMERULONEPHRITIS:
Note the increased number of glomerular cells + thickened capillary membranes (wire loops). Some of the parietal glomerular epithelia form crescent-shaped foci.

Immunoflourescene would show immune complex deposition in subendothelial and subepithelial areas, with antibodies to DNA.
ID
SLE GLOMERULONEPHRITIS:
SLE causing immune complex deposition in the kidney leads to MEMBRANOPROLIFORATIVE GLOMERULONEPHRITIS: 1) proliforation of epithelia in glomeruli and Bowman's capsule (proliforative glomerulonephritis) 2) thickening of glomerular capillary walls (membranous glomerulonephritis).

This biopsy also has a focal interstitial inflammation surounding some renal tubules (lymphocytes and plasma cells).
ID
SLE GLOMERULONEPHRITIS

proliferative glomerulonephritis - A proliferative response of the glomerular endothelial cells as well as the parietal epithelium lining Bowman's capsule.

membranous glomerulonephritis - A thickening of the glomerular capillary walls.

membranousproliferative - When both major components are present.

Wire loops - Thickened walls of capillaries

Crescent shaped foci - parietal epithelium proliferation
ID
SLE GLOMERULONEPHRITIS:
SLE causing immune complex deposition in the kidney leads to MEMBRANOPROLIFORATIVE GLOMERULONEPHRITIS: 1) proliforation of epithelia in glomeruli and Bowman's capsule (proliforative glomerulonephritis) 2) thickening of glomerular capillary walls (membranous glomerulonephritis).

This biopsy also has a focal interstitial inflammation surounding some renal tubules (lymphocytes and plasma cells).