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27 Cards in this Set

  • Front
  • Back
open and closed comedones
inflammatory papules and nodules
psychosocial & cutaneous scarring
acne vulgaris
causes:
sebum under adrogenic hormone influence
occlusion of pilosebaceous unit by keratinocytes
effects of Propionibacterium acnes
inherited predisposition
acne vulgaris
unplug

live in sebum-kill
oxidized melanin in dead keratinocytes
blackhead
pink edematous papules and plaques. Move around. IgE. Pruritic
Urticaria-hives
itch
antihistamine
round, erythematous flat-top papule. Target lesions. Palms and soles. Bullae possibly in center. Drugs, so usually short-term.
erythema multiforme

large blister
Stevens Johnson Syndrome
mucous membrane involvement. can die. (EM).
recurrent cause of EM
herpes simplex
keratinocyte necrosis and dermal inflammation
EM
epidermal hyperplasia
epidermal and dermal inflammation
dermatitis/psoriasis
papulosquamous diseases
weeping, oozing and blistering
OR
lichenification (thickening)
dermatitis (exema)
common in children and can lead to asthma
atopic dermatitis
can have accompanying RA etc
affects 1-2% of population
salmon colored plaques w/ thick white scale on top.
scalp, elbows, knees, sacrum
pitting and lifting up of nail plate
psoriasis
skin lesions appearing on lines of trauma. Psoriasis.
Koebner phenomenon
coalescent tiny vesicles
vermilion border of upper lip and genitalia
3-7 day incubation period.
persists in 1+ dorsal root ganglia
HSV
80% labial infections
20% labial infections
Opposite for genital
HSV 1
HSV 2
1' oral HSV is a disease of what age?
Genital
childhood
early adulthood
itraepidermal vesicle caused by degeneration of epidermal cells resulting in acantholysis w/ viral inclusion bodies and multinucleated keratinocyte giant cells.
HSV AND Herpes zoster
a breakdown of a cell layer in the epidermis (as in pemphigus).
acantholysis-hsv pathology
after primary varicella infection, virus lives in sensory ganglia.
10-20% of population
1-4 day prodrome of fever, malaise, localized pain or paresthesia
grouped vesicles on an erythematous base develop, usually along a single dermatome stopping at the midline. vesicles become pustular, then crust, and generally resolve in 10-21 days.
Herpes zoster
often on top of cold sore
staph aureus
in children around nose and mouth
blisters->rupture->crusted erosion
autoinoculate
impetigo
dermatophyte infections
tinea- cutaneous & superficial fungi and yeasts
scaling, inflamed patches
scalp, feet, groin
yeast &/or hyphae in the stratum corneum
cutaneous superficial fungi and yeasts
(candida & tinea vesicolor)
erythematous
satellite pustules
groin
candida
pityrosporum yeast
hypo or hyperpigmented scaly macules on the trunk
young people
dark spots---from normal flora
tinea vesicolor
intensely itchy, very contagious dermatitis saused by the mite saroptes scabiei. Burrows, the track of the mite thru the stratum corneum are present and if scraped may yield a mite for diagnosis.
Scabies
area of ischemic necrosis caused by occlusion of either ARTERIAL SUPPLY or venous drainage in particular tissue.
INFARCTION
marantic
nonbacterial