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18 Cards in this Set

  • Front
  • Back
DIABETIC KETOACDIOSIS (DKA)
ACUTE COMPLICATION OF DM

BEGINS W/ DEFICENCY OF INSULIN
MORE COMMON W/ TYPE 1
DUE TO UNDIAGNOISED DM OR DONT TAKE INSULIN
INCRS SERUM KETONE BODIES
HYPERGLYCEMIC
ACIDIC
FLUID LOSS=5-7 L
HYPEROSMOLAR HYPERGLYCEMIA NON KETOTIC SYNDROM (HHNK)
BS > 600
PLASMA OSMOLARITY >310
COMA OCCURS OFTEN IN TYPE 2, AND THOSE WITH IMPAIRED ABILITY TO RECOGNIZE THIRST
INCRS pH
FLUID LOSS OF 9L
DEHYDRATION MORE SEVERE!
DIABETIC HYPOGYLCEMIA
COMMON W/ TYPE 1 PTS

TAKE TOO MUCH INSULIN
SOMOGYI EFFECT
BLOOD GLUCOSE DROPS TO QUICKLY usually at noc when it goes unnoticed
AT FIRST HYPERGLYCEMIC SO TAKE EXTRA INSULIN, WHICH WILL THEN CAUSE HYPOGLYCEMIA
TX: DCRS INSULIN DOSE TO A LEVEL WHERE THE BLOOD GLUCOSE WILL NOT DROP SO LOW
Chronic complication in vasculatur of DM
DCRS DIAMETER OF VESSELS
RISK FOR ATHEROSCLEROSIS
MI
CVA
ATROPHY OF SKIN/HAIR, CLAUDICATION
NON HEALING ULCERS
NECROSIS
RBCs carry a HbA1c which measure BS for 120 days (lifespan of RBC)
chronic eye prob of DM
Retinopathy (blindness)
MICROANEURYSMS
HEMORRHAGES
INCRS RATE OF CATARACTS GLAUCOMA
hard exudates
cottonwool spots
chronic prob of kidneys in DM
nephropathy
RENAL ARTERY ATHEROSCLEROSIS
KIMMELSTIEL-WILSON DISEASE= THICKING OF GLOMERULOUS CAUSEING GLOMERULOSCLEROSIS CAUSEING DESTRICTION OF GLOMERULI
nodule necrosis
renal failure
azotemia
DIABETIC NEUROPATHY
SEXUAL IMPOTENCE
NEUROGENIC BLADDER=CANT VOID
PERPIPHERAL NEUROPATHY=NUMBNESS AND TINGLING INCRS H20
stocking-glove peripheral neuropathy
ulcers
DRUG=INSULIN
SE= HYPOGLYCEMIA

USED FOR TYPE 1
SULFONYLURES =DRUG
STIMULATE RELEASE OF ENDOGENOUS INSULIN FROM PANCREAS

SE=HYPOGLYCEMIA, DISULFIRAM LIKE EFFECTS
METFORMIN=DRUG
INHIBITS GLYCONEOGENESIS(making glucose from other things) AND STIMULATED GLYCOLYSIS(utilization of the glucose) TO DCRS SERUM GLUCOSE LEVELS

SE=LACTIC ACIDOSIS
GLITAZONES=DRUG
INCRS TARGET CELL RESPONSE TO INSULIN

SE=WEIGHT GAIN, HEPATOTOXIC
5 ALPHA-GLUCOSIDASE INHIBITORS
DM drug
INHIBITS INTESTINAL ENZYME
DELAYED HYDROLYSIS OF GLUCOSE AND DCRS ABSORPTION OF GLUCOSE

SE=GI DISTURBANCES
NKA Non-ketotic Hyperosmolar
hyperglycemia
hyperosmolarity
alkalosis
older pt w/ typeII
Causes:inability to sense thirst or obtain enought H20 infection, CVA, MI
incrs in plasma concentr. lose 9L
tx w/ saline and insulin
hypoglycemia
bs < 50-70
causes:
DM that take a med, dont eat exercise
alcoholism
carnitine deficiency-tissues depend on glucose for fuel not fat and other things
factitious hypogly-give self to much insulin so high serum insulin levels w/ low C peptide levels
sympotoms of hypoglycemia
sweating
trembling
anxiety
glucagon S&S- hunger, borborygmus, N&V, abd discomfort
tx of hypoglycemia
give 15-20 g of carb works w/in minutes
IV perfusion 50% dextrose
causes of HHNK
dcrs fluid intake
infection UTI pneuomnia
drugs
stressors
enviro chemicals