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29 Cards in this Set
- Front
- Back
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What occurs within 1 hour of death? (2)
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pale foci on the liver
PM clotting (persists for 6-7 hrs) |
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What occurs within 1-6 hours of death? (3)
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rigor mortis (persists 1-2 days)
bile imbibition mucousal sloughing |
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What occurs 6-7 hours after death?
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clotted blood begins to liquify
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What occurs 16-17 hours after death?
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clotted blood is completely re-liquified
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What happens 16-24 hours after death?
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pseudomelanosis
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What is the enzyme deficiency in Type I Glycogenosis?
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G-6-P
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What is the enzyme deficiency in Type II Glycogenosis (Pompe's Dz)?
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glucosidase (lysosomal acid maltase)
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What is the enzyme deficiency in Type III Glycogenosis (Cori's Dz)?
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amylo-1,6-glucosidase
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What is the enzyme deficiency in Type IV Glycogenosis?
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glycogen brancher enzyme
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What is the enzyme deficiency in Type VII Glycogenosis?
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M type PFK
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What is the main organ involved in Type I Glycogenosis?
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liver
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What is the main organ involved in Type II Glycogenosis (Pompe)?
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CNS, muscle, liver
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What is the main organ involved in Type III Glycogenosis (Cori)?
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liver, heart, nerves, sk & sm muscles
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What is the main organ involved in Type IV Glycogenosis?
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skeletal and cardiac muscle
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What is the main organ involved in Type VII Glycogenosis?
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muscle wasting, hemolytic anemia
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What species have Type I Glycogenosis?
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toy dogs
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What species have Type II Glycogenosis (Pompe)?
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cattle, sheep, dogs, cats, turkeys
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What species have Type III Glycogenosis (Cori)?
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GSD
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What species have Type IV Glycogenosis?
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norwegian forest cats, quarter horses, arabians, painted horses
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What species have Type VII Glycogenosis?
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english springer spaniels
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What species/breeds have familial amyloidosis?
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siamese, abyssynians, shar peis
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Which hemoglobin pigment does not stain for iron in the prussian blue rxn?
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hematins (formalin and parasitic hematin)
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Causes of pathological active hyperemia?
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primary response in inflammatory rxn
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Causes of acute local passive hyperemia?
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organ misalignment
venous thrombosis or embolism |
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Causes of chronic local passive hyperemia?
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compression (tumors, abscesses, fibrosis)
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Effects of local passive hyperemia?
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increased venous P --> edema (main effect)
blood stasis, anoxia, degen, necrosis (chronic) |
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Causes of general passive hyperemia?
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cardiac failure
impeded venous return increased pulmonary resistance |
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Effects of general passive hyperemia? (2) Long term effects?
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reduced output & increased venous pressure
edema & tissue hypoxia long term effects in the lungs - edema w/ hemorrhage --> fibrosis w/ loss of fxn |
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Effect of ischemia? Most affected? Least?
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degenerative lesions leading to infarct of cells bc of hypoxia & fibrosis
brain & myocardium most affected liver least affected |
