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40 Cards in this Set

  • Front
  • Back
lidocane hydrochloride

other name
Xylocaine
lidocane hydrochloride

Class
Antiarrythmeic
lidocane hydrochloride

mechanism of action
decreases automacity by slowing the rate oof phase 4 depolarization
lidocane hydrochloride

indicatons
alt. to amiodarone in cardiac arrest from v-fid /pulseless v-tach, stable monomorphic v-tach, stable polymorphic v-tach with normal baseline QT interval
lidocane hydrochloride

contraindications
hypersensitivity, 2nd and 3rd degree AV blocks in the absense of atrial pacemaker, stokes-adams syndrome, prophylactic use in AMI, wide-complex ventricular escape beats with brady
lidocane hydrochloride

adverse reactions
slurred speach, seizures (with high dose) altered mental status, confusion, lightheadedness, blurred vision, brady
lidocane hydrochloride

drug interactions
apnea induced succinlcholine may prolonged with high dose of lidocaine. Cardiac depression may occur in conjunction with IV phenytoin (dilantin). Procainamide may exasterbate CNS effects. metabilic clearance is decreased in patients with lever disease or in patients taking beta blockers
lidocane hydrochloride

how supplied
100mg in 5ml prefilled syrinfges and mpules(20ml)
1g and 2g additive syringes, 1 and 2 gram vials in 30ml solution
lidocane hydrochloride

ADULT
dosages and administration
cardiac arrest from v-fib or pulseless v-tach: initial 1-1.5mg/k IV/IO push. repeat .5 to .75 mg/k in 5 to 10 min.
-Max dose of 3mg.
-ET dose: 2 to 4mg/K
-stable v-tach, wide- complex tach of uncertain type. significant ectopy: .5 to .75mg/K and up to 1 to 1.5mg/K may be used. Rpt. .5 to .75 every 5 to 10 min
lidocane hydrochloride

duration of action
onset: 1 to 3 min
peak: 5 to 10 min
duration: 15min to 2 hours
morphine sulfate

other names
astramorph/PF and others
morphine sulfate

class
opioid analegesic (schedule 2 marcotic)
morphine sulfate

mechanism of action
alleviates pain through CNS action, suppresses fear and anxiety centers in the brain. depresses brain stem respiratory centers, increases peripheral venus capacitance and decreases venus return, decreases preload and afterload, which decreases myocardial oxygen demad
morphine sulfate

indication
Severe CHF, pulmonary edema, chest pain associated with AMI, analgesia for moderate to severe acute and chronic pain (use with caution)
morphine sulfate

contraindications
head injury, exacerbated COPD, depressed respiratory level, hypotension, undiagnosed abdominal pain, decreased level of consciousness, suspected hypovolemia, pts who have taken MAOIs in the past 14 days
morphine sulfate

adverse reactions
respiratory depression, hypotension, decreased LOC, nausea, emesis, brady, tachy, syncope, facial flushing, euphoria, bronchospasm, dry mouth
morphine sulfate

drug interactions
potentiates sedative effects of phenothiazines CNS depressant may potentiate effects of morphine. MAOIs may cause pardoxial excitation
morphine sulfate

how supplied
10mg in 1mL of solution, ampules and tubex syringes
morphine sulfate

ADULT
dosages and administration
initial dose: 2-4mg IV(over 1 ro 5 min) every 5 to 30min. rpt dose 2-8mg at 5 to 15min intervals
morphine sulfate

Duration of action
onset: immediate
peak: 20min
duration 2-7 hours
Furosemide

other names
Lasix
Furosemide

class
loop diuretic
Furosemide

mechanism of action
inhibits electrolyte reasorbtion and promotes excretion of NA, K, and Chloride
Furosemide

indications
CHF, pulmonary edema, HTN crisis
Furosemide

Contraindication
hypovolemia, anuria, hypotension (relative contraindication), hypersensitivity, hepatic coma
Furosemide

adverse reactions
may exerstabate hyovolenia, hypokalemia, ECG changes, dry mouth, hypochloremia, hyponateremia, hypergylcemia (due to hemoconcentration)
Furosemide

Drug interaction
Lithum toxicity may be potentiated by sodium depletion, digitalis toxicity may be potentiated by K+ depletion
Furosemide

How supplied
100mg/5mL, 20mg/2mL, 40mg/4mL vials
Furosemide

ADULT
dosage and administration
.5 to 1.0 mg/k injjected IV over 1 to 2 min. if no response double the dose to 2mg/K over 1 to 2 min
Furosemide

duration of action
onset: 5min
peak: 20 to 60 min
duration: 4 to 6 hours
Nitroglycerine

other names
nitrostat, tridil
Nitroglycerine

class
vasodilator
Nitroglycerine

mechanism of action
smooth muscle relaxant acting on vasuclar, bronchial, and intestinal smooth muscle; dilation of arterioles and veins in the periphery; reduces preload and afterload, decreases the work load of the heart and theyby myocardial oxygen demand
Nitroglycerine

indications
acute angina pectoris, ischemic chest pain, hypotension, CHf, pulmonary edema
Nitroglycerine

contraindications
hypotension, hypovolemia, intracranial bleeding or head injury, previous admin of viagra, revatio, levitra cialis or simular agenst with in 24 hrs.
Nitroglycerine

adverse reactions
headache, hypotension, syncope, reflex tachycardia, nausea, emesis, diaphoresis, muscle twitching
Nitroglycerine

drug interactions
additive effects with other vasodilators; incompatible with other IV drugs
Nitroglycerine

How supplied
tablets : .15mg; .3mg, .4mg, .6mg
NTG spray .4mg-.8mg under the tongue TG IV
Nitroglycerine

Duration of action
onset 1 to3 min
peak: 5 to 10 min
duration: 20 to 30 min :IF IV 1-10 min after discontinuation of infusion
Nitroglycerin

ADULT
dose and administration
tablets: .3 or .4mg SL rpt 3 to5min max 3 doses, NTG spray .4 SL 1 to 2 sprays. NTG IV - 10 to 20μg/min every 5min until desired effect.