• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/61

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

61 Cards in this Set

  • Front
  • Back

What is pain?

it is whatever the experiencing person says it is; existing whenever the person experiencing says it does/

Alternative definition

is it unpleasant sensory and emotional experience associated with actual or potential tissue damage or described in terms of such damage.

define suffering

is the state of severe distress associated with events that threaten the intactness of the person

Why does pain exist?

Protective mechanism, or warning and cardinal sign of inflammation

why is untreated or under treated pain a bad thing?

reactions to pain are tissue breakdown, increased metabolic rate, impaired immune function and negative emotions, prevents self care activities, hurts to cough, walk, etc.

Different types of pain

mild or severe pain, chronic or acute, intermittent or intratable, burning, dull, or sharp


poorly or precisely localized or reffered

Categorized into 3 main types

acute, chronic nonmalignant , cancer

Referred pain?

Pain felt at a site other than the injured or diseased for example in pain in coronary artery insufficiency that may be felt in left shoulder, arm, or jaw

Acute pain

intense <6 months warns individual of actual or potential tissue damage. Autonomic response that originates in SNS, Flood boy with epinephrine "fight or flight response"

Acute pain cont.

causes injury, medical conditions (pneumonia, angina) surgical procedure. described as aching or throbbing. Pt's may be agitated, restless, splint the area. Have increased heart rate, bp, resp rate

Chronic pain

-pain >6 months -can be continuous or intermittent and may be as intense as acute


-does not serve as a warning sign of tissue damage; may be due to damage that has already

Chronic pain cont.

causes: arthritis, back problems


described as dull, constant, shooting, tingling,burning


don't usually see increases in pulse, BP,resp

Synergistic relationship

Action of two or more substances or organs to achieve an effect of which each is individually incapable. examples: fatigue, sleep disturbance, and depression can change a person's perception of pain

Intractable pain

pain that cannot be relieved such as from severe arthritis or cancer

superficial pain

also called cutaneous pain


originates in the skin or tissue just below the skin


an injection



Visceral pain

Deep pain originating in bones and muscles or organs, usually dull or diffuse


an ulcer, angina, organ pain after surgical manipulation, fracture

radiating pain

pain perceived at the source and extending to nearby tissue

phantom pain

pain felt in an amputated body part


nerves transmit messages of pain even though limb (body part) gone

Non pharmacological techniques

pain caused by stimulation of nerve endings

Gate control theory

pain impulses can be regulated or even blocked by gating mechanisms located along the CNS.




Pain and other sensations of skin and muscle travel the same pathways through the large nerves in spinal cord.



When the dorsal horn gates are closed the pain is blocked.

Applied to nursing (gate theory)

small diameter nerve fibers transmitting pain can be closed by stimulating large diameter nerve fibers thru vibration and massage




-pressure of a backrub, heat of a warm compress, or the cold from ice applications, massage




high sensory input close pain impulses to brainstem to distraction will help relieve pain (visitors, activities)

Applied to nursing cont.

Gating mechanisms can be altered by thoughts, feelings, and memories

cerebral cortex and thalamus open pain gate when impulses originate from an increase in anxiety.


Endorphins

-Naturally occurring opiate like substances within the body


-stress and pain activate endorphins


-analgesia results when certain endorphins attach to opioid receptor sites in the brain and prevent the release of neurotransmitters, thereby inhibiting the transmission of pain impulses.



endorphin cont.

People who have less pain than others from a similar injury have higher endorphins levels.




Pain relief measures, such a TENS, acupuncture, placebos decrease pain by increasing endorphins.

Nociceptive pain

body's normal or physiological reaction to noxious stimuli

Neuropathic pain

pain associated with injury to PNS or CNS and is non-physiologic- not localized, may involve other area along pathway




-dm, gullain barre sydrome, cancer, HIV, shingles




analgesics and opioids do not relieve pain

4 phases of nociceptive

Transduction: tissue damage causes release of substances that stimulate nociceptors and start sensation of pain




Transmission: movement of pain to the spinal cord




Perception: pain impulses reach the brain and pain is recognized




Modulation: neurons in brain send signals back down the spinal cord by release of neurotransmitters

Tx of the 4 phases

NSAIDS block production of substances that trigger the nociceptors in the transduction phase


opioids interfere with transduction phase

Tx of neuropathic pain

adjuvant meds: NSAIDs, tricyclic anti-depressants, anticonvulsants, corticosteriods

Non invasive pain relief techniques

1. TENS- transcutaneous electrical nerve stimulation- provides a continuous, mild electrical current to the skin via electrodes causing release of endorphins which blocks pain impulses to the brain.




2. Distraction relaxation- pt think of things other than pain


- music, reading, mediation, visiting, controlled breathing, watching TV, guided imagery, hypnosis, homeopathy, biofeedback

Invasive approaches

Nerve blocks


epideral analgesics


neurosurgical procedures


acupuncture

ceiling effect

doses beyond which there is no improvement in analgesic effect and there may be an increase in adverse side effects

physical dependence

physiological phenomenon that most people experience after a few weeks of continuous opioid use so should wean off.




may experience withdrawal syndrome- sweating, tearing, runny nose, restlessness, irritability, tremors, dilated pupils, sleeplessness, nausea, vomiting, and diarrhea)

Tolerance

takes larger dose to provide same level of pain relief

Addiction

continued craving for opioid and the need to use the opioid for effects other than pain relief

Pseudoaddiction

syndrome in which behaviors similar to addiction appear as a result of inadequate pain control or fear of inadequate pain control

Analgesics

meds that relieve pain.


3 main classes


opioids


non-opioids (non-narcotic)


adjuvants

Non-opioids

generally 1st class of drugs used for pain tx


have ceiling effect to analgesia


do not produce tolerance or physical dependence


most have antipyretic


works at site of injury or peripherally (not CNS)

Opioids

Classified by ability to bind to opioid receptor in brain and spinal cord and other areas




similar to morphine added to nonopioids for pain not controlled




No ceiling effect if used alone. Increases in dose can result in respiratory distress

Classification of opioids

full agonist


-complete response at receptor site


partial agonist


-partial response at receptor site


mixed agonist


-activates one type of receptor while blocking another


antagonist


affinity to cell receptor by binding to it thus preventing it to respond

Adverse effects of opioids

sedation, constipation, nausea/vomiting, itching, respiratory depression*, constricted pupils*


*not as common but alert nurse to possible overdose



Controlled or time-release meds

never crush or chew


for prolonged, continuous pain


oxycontin, MS contin


have immediate release med for breakthrough pain (pain occurs between controlled doses.)



Morphine

Opioid of choice to treat to severe pain


affordable convenient


standard to which other analgesics compared



Hydromorphone

aka dilaudid- mod to severe pain


shorter acting than morphine and faster acting



Meperdine (demerol)

for short term use or allergic to other opioids


breaks down into metabolites (normeperidine) which can cause adverse effects (dysphoria,irrable mood, tremors, seizures)


Avoid if over age of 65 , impaired renal function, recieving MAOI antidepressants

Fentanyl (Sublimaze, Durgesic)

Parental, intraspinal,transdermal patch


short acting and must give more frequntly


patch is q 3 days



methdone (dolophine)

potent, longer duration than morphine


long half-life and accumulates with continued dosing


very effective orally at similar dosing to IV


used for pain, drug detoxification

Opioid antagonist

Naloxone/ Narcan


-pure opioid antagnist


Tx of OD, respiratory distress



Agonist-antagonist



Common ones include butorphanol, stadol, nalbuphine, nubain( for itching and nausea given in small doses will treat side effects but prevent analgesia

Adjuvants

May potentiate effects of opioids or nonopioids, provide analgesia, counteract unwanted effects of other analgesics

Steroids

Tx acute and chronic cancer related pain


Decrease inflammation and compression


standard tx of spinal cord compression

Benzodiazepines

Tx anxiety or muscle spasms


may cause sedation


midazolam(versed) or diazepam (valium)



Tricyclic Anti-depressants

Tx pain related to neuropathy and other painful nerve-related conditions


must be taken for days to weeks to be fully effective


elevates mood and improves ability to sleep


amitrityline, imipramine,desipramine, doxepin

Anticonvulsants

Relieves sharp or cutting pain caused by peripheral nerve syndromes


must be taken regularly before full benefit


carbamazepine (tegretol), gabapentin,neurontin

Stimulants

Used to counteract sedating effects of opioids


methylphenidate hydrochloride (ritalin) caffine-containing medications

Level 1

Mild pain


pt able to sleep, perform ADLs, and work


non-opioid analgesics (prn or OTC)



Level 2

Mild or moderate pain


may not be able to sleep, trouble working, stayed focused


add opioid

Level 3

Moderate to severe pain


affecting quality of a pt's life, may not be able to perform ADLs


Stronger opioid or increase



PCA

patient controlled analgesia


IV,allows pt to control at set intervals


cannot overdose


morphine,fentanyl, demerol

Epidural

deliver anesthesia or strong opiates to major nerves at base of spinal cords


placed by anesthesiologist


meds given thru catheter


fentanyl, morphine

Myth: A person who is laughing and talking is not in pain

Fact: A person in pain is likely to use laughing and talking as a distraction. May be easily distracted when they have vistors and may ask for pain meds as soon as their family leaves