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113 Cards in this Set

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Should you perform tenodesis vs tenotomy for long head of the biceps repair?
Literature review by Frost et al. Am J Sports Med 2009 - inconclusive, recommend RCT
Levels of Evidence:
1A: Meta-analysis of RCTs
1B: RCT
2A: Non-R Controlled Trial
2B: Experimental, non-controlled trial
3: Non-Experimental Data (Case, Correlation, Comparative)
4: Panel of Experts
---short form--

1A: Meta-analysis
1B: Randomized
2A: Controlled
2B: Trial
3: Data
4: Expert Opinion
Describe Giant Cell Tumors:
Age Range, Location (Body & Bone), Cellular Morhpology, Recurrence
25-40 yo

Around the Knee, Distal Radius, Sacrum, Hand Phalanges
Metaphysis or Epiphysis (Not Diaphysis)

Multinucleated, but Nuclei look exactly like surrounding stromal cells

Recurrence Rate Significantly Increased if there is soft ts extension (Williams et al J Hand Surg Am 2010)
EXT1 & EXT2 genes
Autosomal Dominant
Tumor Suppressor Genes of Multiple Hereditary Exostoses
Multiple Osteochondromas
EXT1 mutation is more severe than EXT2 (more exostoses, worse, >flatbone involv, higher malign conversn, mn: named 1 because they found it first because more severe presentation)
Important for chondrocyte proliferation suppression

common locs? malignant risk?
Risk for malignant transformation: <1% solitary, 5-10% with MHE
Steiber: slightly increased risk of sarcomatous transformation in exsostosis (J Am Acad Orthop Surg 2005)
Pannier: EXT genes encode "exostosins" glycosyltransferases important in he...
Risk for malignant transformation: <1% solitary, 5-10% with MHE
Steiber: slightly increased risk of sarcomatous transformation in exsostosis (J Am Acad Orthop Surg 2005)
Pannier: EXT genes encode "exostosins" glycosyltransferases important in heparin sulfate synthesis
Multiple Hereditary Exostosis
Multiple Osteochondromas

EXT1 & EXT2 Genes = Tumor suppressors vs chondrocyte prolif

Locs: common locations: around the knee, prox humerus, subungual (esp hallux)

Risks for malig: 5-10% with MHE
Steiber: slightly increased risk of sarcomatous transformation in exsostosis (J Am Acad Orthop Surg 2005)
Pannier: EXT genes encode "exostosins" glycosyltransferases important in heparin sulfate synthesis
Steiber: slightly increased risk of sarcomatous transformation in exsostosis (J Am Acad Orthop Surg 2005)
Pannier: EXT genes encode "exostosins" glycosyltransferases important in heparin sulfate synthesis
Diastrophic Dyplasia

gene, gene fnx

4 morph features
DTDST gene, sulfate transporter important in cartilage conversion to bone

cauliflower ear, diastrophic dwarfism, clubfoot, hitchhiker thumbs +/- cleft palate
DTDST gene, sulfate transporter important in cartilage conversion to bone

cauliflower ear, diastrophic dwarfism, clubfoot, hitchhiker thumbs +/- cleft palate
dwarfism characterized by cauliflower ear, clubfoot, hitchhiker thumbs

sro, gene, gene fnx
Diastrophic Dyplasia: DTDST gene, sulfate transporter important in cartilage conversion to bone,
Diastrophic Dyplasia: DTDST gene, sulfate transporter important in cartilage conversion to bone,
Apert's Syndrome

Gene, Morph Features
FGFr2

dysmorphic face

syndactyly
sro, gene, fnx
sro, gene, fnx
Multiple Hereditary Exostoses

Autosomal Dominant
Tumor Suppressor Genes of Chonrocytes

Multiple Osteochondromas
EXT1 mutation is more severe than EXT2

common locs? malignant risk?
Risk for malignant transformation: <1% solitary, 5-10% with MHE
Steiber: slightly increased risk of sarcomatous transformation in exsostosis (J Am Acad Orthop Surg 2005)
Pannier: EXT genes encode "exostosins" glycosyltransferases important in heparin sulfate synthesis
Sro, Gene, Gene Funx, Morph Feat
Sro, Gene, Gene Funx, Morph Feat
Disatrophic Dysplasia

DTDST gene

sulfate transporter important in cartilage conversion to bone,

cauliflower ear, diastrophic dwarfism, clubfoot, hitchhiker thumbs +/- cleft palate
Sro, Gene, Gene Funx, Morph Feat
Sro, Gene, Gene Funx, Morph Feat
Disatrophic Dysplasia

DTDST gene

sulfate transporter important in cartilage conversion to bone,

cauliflower ear, diastrophic dwarfism, clubfoot, hitchhiker thumbs +/- cleft palate
FGFr2 gene

Sro, features
Apert's Sro

Dysmorphic Face & Syndactyly
Sro, Gene, Features
Sro, Gene, Features
Apert's Sro
FGFr2

dysmorphic face & syndactyly
Champagne Glass Pelvis of Achondroplasia

FGFR3 gene
cleidocranial dysplasia
RUNX2/CBFA1 gene

failure of formation of midline structures and absent clavicles
RUNX2/CBFA1 gene mutation
cleidocranial dysmorphism

failure to develop of clavicles & midline strx
cleidocranial dysmorphism

RUNX2/CBFA1 gene mutation

failure to develop of clavicles & midline strx
Hill-Sachs Lesion
cortical depression in posterolateral humeral head 2/2 anterior dislocxn vs glenoid rim
cortical depression in posterolateral humeral head 2/2 anterior dislocxn vs glenoid rim
Hill-Sachs Lesion
Treatment for Open Wounds
Gustillo Grade 1 & 2: 1G Cephalosporin (Ancef/Cefazolin)
Gustillo Grade 3: 1G Ceph + Aminoglycoside
Farm Injuries or possible bowel involvment: add PCN vs Clostridia
Continue for 24 hours following debridement
Tetanus prophylaxis in open fractures:
Wound: Clean & Minor
>=3 doses of tetanus in past:
give Tetanus vaccine if last dose >10 years, else not, no IVIG
<3 or ? = Give vaccine
All other wounds
>=3 doses in past: give tetanus vaccine if last dose >5 ya, no IVIG
<3 or ? = Give vaccine + IVIG

Toxoid Doses?
Toxoid 0.5mL regardless of age
IG:
<5 yo receives 75U
5-10 = 125 U
>10 = 250U

recap:

so IVIG only if <3 or ? status with non-minor or non-clean wound
Tetanus vaccine if <3 or unknown or >=3 and >5 ya or if minor clean wound >10 ya
Tx: clavicular non-union
aSx: observation for neurovascular sx
Sx: ORIF. no K wires since distraction & migration common
Boyer JBJS 97: lag screw + autograft
Bradbury Acta Orthop Scand 96: plate fix + graft
Conversion of Hip Arthrodesis to THA

Indications, Age Limit, Outcome Predictors & Complixns
Good for relieving knee & back pain
no time limit
most important predictor of ambulatory outcome: glut med status
perform an EMG on
Complications: Nerve Palsey, HO, Need for revision
Joshi JBJS 2002
What velocity constitutes low velocity vs high velocity gunshots:
Low: 1k feet/sec (civilian)
High: 2k f/s (military/hunting)
Tx: low velocity gunshot wound, neurovascularly intact s fx
Treat it like a normal open ound: tetanus prophylaxis, local I&D, 2-3 days of oral abx
Indications for distal radius corrective osteotomy c bone graft & internal fixation
symptomatic >25-30 degrees of angulated malunion in active pts s degenerative changes
--or--
who wish to have deformity corrected
Fernandez JBJS 82
MCC: adult acquired flatfoot deformity
posterior tibial tendon insufficiency (PTTI)
Single heel rise test
normally there is inversion of foot on plantarflexion (seen in creases of heel)
no inversion = posterior tibial tendon dysfunction
Insertions of Posterior Tibial Tendon
anterior limb: navicular & 1st cuneiform
middle limb: inferiorly to 2nd & third cuneiforms, cuboid & metatarsals 2-5
posterior limb: sustinaculum tali anteriorly
Watershed area of posterior tibia
between navicular and medeal malleolus
PTT fnx in gait
allows locking of transverse tarsal joints, creates a rigid lever arm for toe off
PTTI classification
I: Tenosynovitis (Normal Anatomy, Radiographs, Physical Exm)
II: Flatfoot Deformity, Flexible Hindfoot (Arch Loss, Loss of Single-Leg Toe Raise, +/- Mild Sinus Tarsi Pain)
IIA: Normal Forefoot
IIB: Forefoot abduction ("too many toes," >40% talonavicular uncoverage)
III:Rigid Hindfoot Valgus (Severe Sinus Tarsi Pain, Subtalar Arthritis)
IV:Deltoid Lig Compromise (Ankle Pain, Talar Tilt in Ankle Mortise)
Posterior Tibial Tendon Insufficiency:
Arch Loss is indicative of grade...
II+
PTTI classification
I: Tenosynovitis (Normal Anatomy, Radiographs, Physical Exm)
II: Flatfoot Deformity, Flexible Hindfoot (Arch Loss, Loss of Single-Leg Toe Raise, +/- Mild Sinus Tarsi Pain)
IIA: Normal Forefoot
IIB: Forefoot abduction ("too many toes," >40% talonavicular uncoverage)
III:Rigid Hindfoot Valgus (Severe Sinus Tarsi Pain, Subtalar Arthritis)
IV:Deltoid Lig Compromise (Ankle Pain, Talar Tilt in Ankle Mortise)
Posterior Tibial Tendon Insufficiency:
Flatfoot Deformity is indicative of grade...
II+
PTTI classification
I: Tenosynovitis (Normal Anatomy, Radiographs, Physical Exm)
II: Flatfoot Deformity, Flexible Hindfoot (Arch Loss, Loss of Single-Leg Toe Raise, +/- Mild Sinus Tarsi Pain)
IIA: Normal Forefoot
IIB: Forefoot abduction ("too many toes," >40% talonavicular uncoverage)
III:Rigid Hindfoot Valgus (Severe Sinus Tarsi Pain, Subtalar Arthritis)
IV:Deltoid Lig Compromise (Ankle Pain, Talar Tilt in Ankle Mortise)
Posterior Tibial Tendon Insufficiency:
Forefoot Abduction is indicative of grade...
IIB+
PTTI classification
I: Tenosynovitis (Normal Anatomy, Radiographs, Physical Exm)
II: Flatfoot Deformity, Flexible Hindfoot (Arch Loss, Loss of Single-Leg Toe Raise, +/- Mild Sinus Tarsi Pain)
IIA: Normal Forefoot
IIB: Forefoot abduction ("too many toes," >40% talonavicular uncoverage)
III:Rigid Hindfoot Valgus (Severe Sinus Tarsi Pain, Subtalar Arthritis)
IV:Deltoid Lig Compromise (Ankle Pain, Talar Tilt in Ankle Mortise)
Posterior Tibial Tendon Insufficiency:
Rigit Hindfoot Valgus is indicative of grade...
III+
PTTI classification
I: Tenosynovitis (Normal Anatomy, Radiographs, Physical Exm)
II: Flatfoot Deformity, Flexible Hindfoot (Arch Loss, Loss of Single-Leg Toe Raise, +/- Mild Sinus Tarsi Pain)
IIA: Normal Forefoot
IIB: Forefoot abduction ("too many toes," >40% talonavicular uncoverage)
III:Rigid Hindfoot Valgus (Severe Sinus Tarsi Pain, Subtalar Arthritis)
IV:Deltoid Lig Compromise (Ankle Pain, Talar Tilt in Ankle Mortise)
Posterior Tibial Tendon Insufficiency:
Talar Tilt in Ankle Mortise is indicative of grade...
Deltoid Ligament Compromise: IV+
PTTI classification
I: Tenosynovitis (Normal Anatomy, Radiographs, Physical Exm)
II: Flatfoot Deformity, Flexible Hindfoot (Arch Loss, Loss of Single-Leg Toe Raise, +/- Mild Sinus Tarsi Pain)
IIA: Normal Forefoot
IIB: Forefoot abduction ("too many toes," >40% talonavicular uncoverage)
III:Rigid Hindfoot Valgus (Severe Sinus Tarsi Pain, Subtalar Arthritis)
IV:Deltoid Lig Compromise (Ankle Pain, Talar Tilt in Ankle Mortise)
Posterior Tibial Tendon Insufficiency:
Severe Sinus Tarsi Pain is indicative of grade...
III+
PTTI classification
I: Tenosynovitis (Normal Anatomy, Radiographs, Physical Exm)
II: Flatfoot Deformity, Flexible Hindfoot (Arch Loss, Loss of Single-Leg Toe Raise, +/- Mild Sinus Tarsi Pain)
IIA: Normal Forefoot
IIB: Forefoot abduction ("too many toes," >40% talonavicular uncoverage)
III:Rigid Hindfoot Valgus (Severe Sinus Tarsi Pain, Subtalar Arthritis)
IV:Deltoid Lig Compromise (Ankle Pain, Talar Tilt in Ankle Mortise)
Posterior Tibial Tendon Insufficiency:
Subtalar Arthritis is indicative of grade...
III+
PTTI classification
I: Tenosynovitis (Normal Anatomy, Radiographs, Physical Exm)
II: Flatfoot Deformity, Flexible Hindfoot (Arch Loss, Loss of Single-Leg Toe Raise, +/- Mild Sinus Tarsi Pain)
IIA: Normal Forefoot
IIB: Forefoot abduction ("too many toes," >40% talonavicular uncoverage)
III:Rigid Hindfoot Valgus (Severe Sinus Tarsi Pain, Subtalar Arthritis)
IV:Deltoid Lig Compromise (Ankle Pain, Talar Tilt in Ankle Mortise)
Posterior Tibial Tendon Insufficiency:
Arch Loss is indicative of grade...
II+
PTTI classification
I: Tenosynovitis (Normal Anatomy, Radiographs, Physical Exm)
II: Flatfoot Deformity, Flexible Hindfoot (Arch Loss, Loss of Single-Leg Toe Raise, +/- Mild Sinus Tarsi Pain)
IIA: Normal Forefoot
IIB: Forefoot abduction ("too many toes," >40% talonavicular uncoverage)
III:Rigid Hindfoot Valgus (Severe Sinus Tarsi Pain, Subtalar Arthritis)
IV:Deltoid Lig Compromise (Ankle Pain, Talar Tilt in Ankle Mortise)
Pes Planus may be 2/2 to what
1. Posterior Tibial Tendon Insufficiency
2. Midfoot Pathology - OA, chronic Lisfranc injury
3. Incompetance of Spring Ligament (talonavicular stabalizer)
Tx: Stage I Posterior Tibial Tendon Insufficiency
Stage I is PTT tenosynovitis with no structural/functional changes
Treat with immobilization in walking/casting boot for 3-4 mos followed by custom molded in-shoe orthoses
Open Chain vs Closed Chain Exercise:
Open Chain: Foot or Hand is moving (end of a lever is moving)
Closed chain: foot or hand is planted, body is moving
Isokinetic Exercise
same speed throughout
Most predictive factor of outcome in SCIWORA injuries:
Severity of initial neurologic injury
complete transexn/major hemorrhage: profoundly poor
minor hemorrhage: 40% will return to mild grade
edema: 75% will return to mild grade
Lisfranc ligament
medial cuneiform to base of 2nd metatarsal
eponychium
epi (upon) onychion (little claw)
Tx: Subungual Hematoma
<50% of nailbed invovled --> perforation & drainage of hematoma
>50% of nailbed involved --> nail bed repair
Tx: nail bed laceration
tetanus/Abx, most cases req nail removal c D&I + nailbed repair
Tx: Nail Avulsion:
minimal or no loss of germinal matrix: nail removal, bed repair +/- Fx fix'n
significant matrix loss: add split thickness graft & matrix transfer
What is the chemical name of dermabond
2-octyl-cyano-acrylate
Tx: PIN transection with failed primary repair
Transfer of FCR to ED, and Palmaris Longus to EPL
Ropars et al J Hand Surg Br 2006
x
x
x
ossification of the clavicle
-clavicle is the last bone to ossify, medial growth plate fuses in early 20's
Last bone to ossify
-clavicle is the last bone to ossify, medial growth plate fuses in early 20's
Os acromiale:
unfused secondary ossification center within acromion
[which starts off as 4 separate ossification centers]
3% of population have it, 60% who have it are bilateral
If Sx --> ORIF
Glenoid Tilt:
4 degrees upward
7 retro to 10 anteroversion
[nb: humeral head is 30 degrees retrotoverted]
Average diameter of humeral head:
43mm
Humeral Head Tilt:
30 degrees retroverted
130 degrees from shaft
Capsuloligamentous Strx of GH Joint:
Coracohumeral Ligament
Superior GH Lig
Middle GH Lig
Inferior GH Lig complex
Posterior Capsule
Biceps Tendon
Rotator Interval Boundaries & Contents
(Btw Supraspinatus and Subscapularis/coracoid base & transverse humeral ligament):
cocracohumeral Ligament
Superior GHL
Biceps Tendon
Glenohumeral Capsule
90 degrees of motion at the shoulder produces what clavicular rotation
40 degrees
what is the primary restraint of excessive AP translation of the sternoclavicular joint
posterior capsule
AC Joint (& Coracoid) Ligaments
Acoracoclavicular Ligments: Surround Joint Capsule, vs AP translation
Coracoacromial Ligaments: prevents inferior translation of coracoid
Coracoclavicular Ligaments:
Trapezoid (lateral)
Coronoid (medial)
Prevent Inferior Translation from Clavicle
Zanca View:
X ray beam directed 10* cephalad at 50% normal penetrance
best view of AC joint
Zanca View:
X ray beam directed 10* cephalad at 50% normal penetrance
Ratio of GH to Scapulothroacic motion (abduction)
2:1
Layers of the shoulder:
Outer: Deltoid, Cephalic Vein, Pect Major
2nd: Conjointed Tendon, Pect Minor, Clavipectoral Fascia
3rd: Subdeltoid Bursa, rotator cuff, GH capsule Tuberosities, LH of Biceps, Synovium, glenoid suprascapular neurovascular bundle
Inner: GH capsule, Glenoid synovium, long head of biceps
Changes of Dominant vs Non-Dominant Shoulder
Dominant: Anterior Capsule selectively Stretched, Posterior Capsule Tightened
--> "GIRD" Glenohumeral internal Rotation Deficit
Dominant hand more easily externally rotates, does not internally rotate as far
Standard Shoulder Athroscopy Portals
Posterior: 2 cm distal & medial to posterolateral border of acromion
Anterior: Just Anterior to AC joint
Lateral: 1-2 cm distal to lateral acromial edge
What is at risk with anterior shoulder dislocations
Axillary nerve because of its relatively fixed location
Bankart lesion:
anteroinferior labral tear
Osseus Bankart lesion
glenoid rim fx c fnx labral detachment
ie nondisplaced labral tear with intact medial scapular perisoteum
ALPSA:
anterior labroligamentous poseriosteal sleeve avulsion
ie medialized bankart lesion: medial anterioinferior labral tear displaced medially by intact medial scapular periosteum
reverse bankart lesion:
posteroinferior labral tear
Glenolabral articular disruption
labral tear extending into glenoid cartilage
GABL:
humeral avulsion of inferior glenohumeral ligament
Grading Load Shift of Shoulder
0: normal amt of translation
1: to but not over labrum
2: Over labrum but reducible
3: Over labrum & locking
TUBS Sro:
Traumatic Unilateral dislocations with a Bankart lesion
young pts with 90% recurrence if nonoperatively managed (surgical)
most important risk factor for recurrent instability: age at initial insult
AMBRI:
Atraumatic Multidriectional Bilateral shoulder dislocation/subluxation which responds to Rehabilitation & sometimes requries Inferior capsular shift or plication
Focus on closed kinetic chain exercises
closed kinetic chain exercises:
Extremities planted/fixed on a surface with motion against
ie pushups, pullups, squats
Tx: Glenoid deficiency greater than 25% of humeral head
Latarjet procedure ie coracoid transfer
Tx: failed repab for multidirectional instability
capsular shift
Tx: chronic shoulder dislocation with greater than 40% of articualr surface deficit:
allograft for young pts
prosthesis for old pts
Bankart procedure
reattachment of labrum (&IGHLC) to glenoid
a gold standard procedure
staple capsulorrhaphy:
capsular reattachment & tightening
complciations; staple migration, articular injury
Putti-Platt
subscapularis advancement capsular coverage
complx: decreased ER, DJD
Magnuson-Stack:
Subscapularis transfer to greater tuberosity
Complx: Nonanatomic, Recurrance
Bristow/Latarjet
Coracoid transfer to Inferior Glenoid
Complx: nonunion, migration, labral tears
Bone block osteotomy
(shoulder)
Anterior bone block
Complx: nonunion, migration, articular injury
(shoulder) Capsular Shift procedure:
Inferior capsule shifted superiorly "pants over vest"
Complx: overtightening;
"gold standard"for multidirectional instability
Rotator interval closure results
decreased external rotation in shoulder adduction and posteroinferiro translation
Thermal Capsular Shrinkage for Shoulder Instability
No longer recommended
Good short term outcomes, worse long term, and poor ts & chondral damage noted at revision
Dx: Fixed pOsteriro shoulder dislocation
lack of ER
Kim lesion:
incomplete & concealed avulsion of posteroinferior labrum
+Jerk, + Kim Tests
Epidemiology of Geriatric Rotator Cuff dz:
~1/3 over 60yo have full thickness tear
~2/3 over 70yo have full thickness tear
sic: if you have one tear, 50% chance its bilateral
Of those with aSx: 50% Sx w/in 3 yrs, 40% of these progressive
Social Factor with Poor Prognosis for Subacromial Decompression
Pts with workers comp claims
Rate limiting factor for recovery from rotator cuff suergy:
biologic healing of rotator cuff tendon to humerus: 8-12 weeks minimum
Classify Rotator Cuff Tear size:
Small: <1cm
Med: 1-3 cm
Large: 3-5 cm
Massive >5 cm or 2 tendons
Tx Timeline: Acute Rotator Cuff Tears
Repair Early
Tx: PASTA:
Partial articular supraspinatus tendon avulsion


debridement vs repair = controversial
Reverse Shoulder Prosthesis:
popular for rotator cuff arthropathy in elderly pts with low fnx demands
requires competaent deltoid, glood glenoid bone stock
more predictable fnx outcomes, but ?high rates of complxns (~40%)
Rotator Cuff Arthropathy:
Massive rotator cuff tear (ie >5cm or 2 tendons), combined with fixed superior migration of humeral head, and severe glenohumeral arthrosis (loss of congruence)
Subscap tears:
comma sign on arthroscopy is avulsed SGHL
Tx: surgical, if chronic --> pect transfer
Subcoracoid Impingement:
<7mm btw humerus and coracoid is abnormal
best evaluation: cross armed CT
Tx: resexn of lateral coracoid process
(open = reattach conjoined tendon, arthroscopic: never detach it)
Shoulder:
Internal impingement:
Bennet lesion: mineralization of posterior inferior glenoid
indicative of internal impignement from overhead throwing
associated with GIRD: tight posteroinferior capsule/lax anterior capsule: posterosuperior shift of humeral head & internal impingment
Tx: posterior/posteroinferior capsular stretching exercises ie sleeper stretch, & stretching pect minor
Operative: debride labrum, posterior capsular release
Dynamic Labral Shear Test:
Elbow Flexed to 90*
Forward Flex Shoulder Up to natural limit
Shift out until UE is in coronal plane
Adduct
Positive if pain or click on posterior joint line btw 120 and 90 degrees
86% sensitive, 100% specific for SLAP tear
Biceps Tendon Subluxation
Most Common Assoc: Subscap Tear
non-op: strengthening & corticosteroids
op: repair of subscapularis, strx, tenotonomy or tenodesis
Types of AC seprations:
Type I: sprain
II: AC only
III: AC & Coracoclavicular
IV: posterior displacement of clavicle through trapezius muscle (controverial for surg in older/inactive)
V: superior displacement of clavicle (surgical)
VI: inferior displacement of clavicle below conjoined tendon (surgical)
Structural Concerns for Ankle ports
Anterolateral portal: danger to dorsal intermediate cutaneous branch of superficial peroneal nerve
anteromeial: greater saphenous vein
anterior central: dorsalis pedis
posteromedial: posterior tibial artery
posterolateral: sural nerve
MSTS/Enneking System:
Arabic numerals for benign lesions.
1 = latent, 2 = active, 3 = aggressive
I: Low Grade
II: High Grade
III: Metastatic
A: Intracompartmental (within cortex)
B: Extracompartmental (beyond cortex)
AJCC Staging System:
I: Low Grade
II: High Grade
III: skip lesions or deep
IV: mets

For I & II: A if <8cm, B if >8cm
For IV: A if mets to lung, B if mets elsewhere
AJCC Staging System:
I: Low Grade
II: High Grade
III: skip lesions or deep
IV: mets

For I & II: A if <8cm, B if >8cm
For IV: A if mets to lung, B if mets elsewhere
MSTS/Enneking System

Arabic numerals for benign lesions.
1 = latent, 2 = active, 3 = aggressive
I: Low Grade
II: High Grade
III: Metastatic
A: Intracompartmental (within cortex)
B: Extracompartmental (beyond cortex)