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51 Cards in this Set

  • Front
  • Back
Intraspinous radicular pain
Neurofibroma, Ependymoma, Meningioma, Disc, Spinal stenosis, AVM of cord, Spinal AV fistula
Extraspinous radicular pain
piriformis, vascular disease, nerve root irritation, neoplasms, plexitis, polyneuropathy, trauma with neuropraxis, shingles
What is the major role in risk of acute LBP?
occupation
-nursing, garbage collection, warehouse, airlines
Non-Radicular pain
Traumatic, Chronic/sub acute, DJD, Referred pain, infection, neoplasm, rheumatologic
Alarm symptoms of LBP
age >50
prior cancer history
unexplained fevers, night sweats, weight loss
pain lasting more than one month often intractable and unrelieved with rest
no improvement following conservative therapy
Discogenic (radicular) pain
often acute,
constant, sharp, shooting
dermatomal radiation, unilateral
increased by cough, sneezing, sitting
improved with lying down
Risk factors for HNP
repetitive lifting activity
prolonged sitting
twisting/rotational movement
chronic cough
prior spinal injury or disc disease
tobacco use
exposure to prolonged vibratory forces
HNP characteristics
posterior lateral location
L4-L5, L5-S1
males more than females
pain worse with increasing intrathecal pressure
Cauda Equina syndrome
impingement of caudal equina
50% secondary to tumor
EMERGENT surgical decompression
Facet Trophism
asymmetry of facet joint which predispose patients to early degenerative joint disease and pain
Sacralization
deformity in which L5 fuses with sacrum, can predispose patient to pain and early disc herniation
Lumbarization
deformity in which S1 segment fuses to lumbar spine

less common
Spina Bifida Occulta
no herniation through defect
only physical sign is course hair over site
no neurological defects
Spina Bifida Meningocele
a herniation of meninges through defect
Spina Bifida Meningomyelocele
herniation of meninges and nerve roots through defect
associated with neurological deficits
Lumbosacral angle
angle formed by intersection of horizontal line with sacral base line
normal: 25-35 degress
increased: increased lumbosacral joint shear and LBP
Postural/ Tonic muscles respond to dysfunction how?
by becoming hypertonic, shortened, sometimes spastic
Dynamic/phasic muscles respond to dysfunction how?
by inhibition, hypotonicity and weakness
Motion of lumbar spine?
flexion and extension
Psoas muscle spasm (flexion contracture)
somatic dysfunction of L1 and L2
rotated to same side as spasm
Low back horizontal platforms
Thoraco-Abdominal Diaphragm
Pelvic Diaphragm
Meniscus of knee
plantar fascia of foot
Low back cables
erector spinae
Iliopsoas & QL
Quads & Hamstrings
TFL/ITB & hip adductors
gastroc/soleus & tibialis anterior
Lumbar spine xray, AP view
lumbar scoliosis
unlevel sacral base
functional short leg
Lumbar spine xray, Lateral view
loss of lumbar lordosis
DDD, facet syndrome
Lumbar spine xray, L-S Jctn spot view
Spondylolisthesis L5-S1
Straight leg test
raise leg 20-60 degrees
positive test reproduced radicular pain below knee
nerve root lesion
Test L1-L2 motor nerve root
psoas muscle,
flex hip up
Test L3 motor nerve root
quadriceps muscle
extend leg
L4 motor nerve root
Tibialis anterior muscle
dorsiflex foot
L5 motor nerve root
extensor hallucis longus
extend great toe
Si motor nerve root
gastrocnemius muscle
plantarflex foor
Grades of muscle strength
0=no tone, flaccid
1=tone w/o movement
2= movement without gravity, not against gravity
3= movement against gravity, not against resistance
4= slightly diminished strength
5= full strength against resistance
Patellar reflex
L4
Achilles reflex
S1
Grading deep tendon reflexes
0=no response
1=minimal response
2=mid-range normal response
3= slightly hyperactive response
4= hyperactive response with clonus
Patrick's Test
positive test reproduces pain in SI joint and anterior groin crease
FABERE
flexion, abduction, external rotation, extension (patricks test)
Thomas test
tight hip flexor, psoas
look for gap in politeal fossa (psoas)
Flexed lumbar segment-- still technique
flex to segment-- ADduct-- compress-- ABduct
Extended lumbar segment-- still technique
flex to segment-- ABduct -- compress-- ADduct leg
Treatment of ABL2
counterstrain by standing on ipsilateral side, flexing hip 90degrees, bringing knees together, rotating towards tender point--SB away
AL1
internal oblique mm
AL2
External oblique mm
AL3/4
Iliopsoas mm
AL5
Rectus abdominus mm
Charles Hazard D.O.
student of Dr. Still in 1897
teacher at American School of Osteopathy
Still similar to FPR
positioning and use of compression
Type 1/2 not needed for FPR (neutral)
not through barrier (Still is)
Force vector
point of compression to and through the dysfunctional tissue
Two types of FPR
directed at normalization of palpable abnormal tissue texture
modified to influence deeper muscles involved in joint mobility
FPR for dummies
put into neutral-- add compression--balance--hold-- return to neural
What is the order of treatment for still technique
lumbar spine and pelvic dysfunctions are treated BEFORE sacral dysfunction