Use LEFT and RIGHT arrow keys to navigate between flashcards;
Use UP and DOWN arrow keys to flip the card;
H to show hint;
A reads text to speech;
207 Cards in this Set
- Front
- Back
(T/F) Operculated Retinal holes have a higher chance of RD. Why?
|
F. No more traction b/c retina is torn clear away.
|
|
What test should you do to see if operculated retinal holes are truly fully detached?
|
Scleral depression
|
|
(T/F) Pigment is a sign of stability for retinal holes.
|
T
|
|
(T/F) Operculated retinal hole pieces tend to shrink over time. Therefore, you can tell if it is old by comparing the size of the free floating piece to the hole.
|
|
|
Operculated retinal hole management: New hole with photopsia?
|
RTC 6 weeks
|
|
Operculated retinal hole management: New hole with no symptoms?
|
RTC 6 months
|
|
Operculated retinal hole management: Multiple holes
|
RTC 6 months
|
|
Operculated retinal hole management: Cuff edema < 2 DD with no pigmentation?
|
RTC 3 months
|
|
Operculated retinal hole management: Hole with pigment?
|
RTC 6 months
|
|
Operculated retinal hole management: Cuff edema greater than 2 DD in size?
|
Refer to retinal specialist.
|
|
___% of linear retinal tears develop into retinal breaks. ___% develop into RDs.
|
10-15%. 30%
|
|
Risk factors for linear retinal tears. (4)
|
High myopes, trauma history, aphakia, premature birth (ROP)
|
|
What does a linear retinal tear look like?
|
Horseshoe with red center and grey tissue around edge.
|
|
Where do linear retinal tears normally occur?
|
Posterior border of vitreal base
|
|
What is Shaefer's Sign in the context of linear retinal tears?
|
Pigmentation enters the vitreous.
|
|
If there is so much hemorrhaging that you can't see the retina what do you do?
|
Get a retinal B scan.
|
|
What is the treatment for a linear retinal tear?
|
Laser photocoagulation
|
|
Linear retinal tear management: When you find one?
|
Refer to retinal specialist.
|
|
Retinal dialysis management: When you find one?
|
Refer to retinal specialist.
|
|
Retinal dialysis management: When a person has a recent trauma, what is the dilation schedule?
|
Do at exam. Then repeat in 3-6 months b/c they they develop slowly.
|
|
(T/F) Retinal dialysis is often asymptomatic
|
T - 60%
|
|
Name the typical retinal dialysis pt.
|
Younger. Males under 40 years that had trauma.
|
|
Why are retinal dialysis tears so slow to develop? How long does it take?
|
They are often inferior. Within 140 days.
|
|
Why might IOP go up or down during retinal dialysis or detachment??
|
Up because pigment released into vitreous. Down because vitreous has another place to leave.
|
|
Giant Retinal Tear management: When you find one?
|
Refer to retinal specialist.
|
|
What causes Commotio Retinae?
|
Blunt trauma.
|
|
What do you see with Commotio Retinae?
|
At first, looks normal. Hours later it becomes swollen (opaque white, edema). Possibly hemorrhagic (black and blue).
|
|
What might happen as the swelling of Commotio Retina subsides?
|
Pt may develop holes or tears.
|
|
What is Berlin's Edema?
|
Retinal swelling that can lead to retinal dialysis because the mucopolysaccharide bonds of the retina are weakened. Associated with Commotio Retinae
|
|
What is a typical pt complaint with Commotio Retinae?
|
Blur due to retinal swelling (and black eye!)
|
|
Why do RDs happen most often after age 60?
|
PVD has vitreous pulling on retina.
|
|
If you have RD in one eye, how likely are you to get it in the other eye?
|
10-30%
|
|
(T/F) RD symptoms are always symptomatic.
|
F - can be asymptomatic because they are variable
|
|
What are the classic symptoms of RD?
|
Flashes and floaters
|
|
What is the old was to treat RD? The new way?
|
Cryopoxy. Laser photocoagulation
|
|
What 2 things do you see in the retina when a pt has RD?
|
Dissapearance of choroidal layer (Can't see ampules and other details.) Edema that makes the retina opaque.
|
|
What technology is very usefule in outlining retinal detachments, especially when there is hemorrhaging in the vitreous?
|
B Scan
|
|
Retinal detachment management: New one
|
Immediate referral
|
|
Retinal detachment management: Old one newly detected
|
Consultation within a few days
|
|
(T/F) Flashes and floaters stop right after the retina has detached.
|
F - They can continue for up to 6 months afterwards
|
|
What is snail track degeneration?
|
Thinning of parts of the retina
|
|
Name 2 reasons why snail track degeneration pts have a higher risk of RD?
|
Liquefied vitreous gets under the retina and lifts it up. Vitreoretinal traction.
|
|
(T/F) Active PVD increases your chances for a retinal tear.
|
T b/c the vitreous is peeling away and taking the retina with it.
|
|
What does an atrophic retinal hole look like?
|
Red dot. Possibly greyish edema around edges.
|
|
How does an atrophic retinal hole lead to RDs?
|
Fluid enters along the edges and lifts the retina up.
|
|
(T/F) Linear Retinal tears have a higher risk of RDs.
|
T
|
|
(T/F) Giant retinal tears do not have a higer risk of RDs
|
F
|
|
What is degenerative retinoschisis?
|
Splitting of the retina.
|
|
Degenerative retinoschisis management: You see it for the first time.
|
Refer to retinal specialist
|
|
Degenerative retinoschisis management: Watching old one w/ no flashes and floaters
|
RTC 6 months
|
|
Degenerative retinoschisis management: Watching old one w/ flashes and floaters
|
Refer to retinal specialist
|
|
Degenerative retinoschisis management: Watching old one w/ vitreoretinal traction.
|
Refer to retinal specialist
|
|
What is lattice degeneration?
|
Thinning of the retina with vitreoretinal traction on the edges with liquefied vitreous in that area
|
|
What is a typical lattice degeneration pt?
|
Young (6-10% of normal population)
|
|
(T/F) Lattice degeneration is typically bilateral
|
T
|
|
(T/F) Lattice degeneration can lead to holes or tears. Why?
|
T - Because it's thin
|
|
What is the typical size of a lattice degeneration?
|
1-4 DD in length. .5-2 DD in width.
|
|
(T/F) RPE hyperplasia with lattice degeneration is good
|
T - Pigmentation is a sign of stability
|
|
What does Lattice degeneration look like?
|
Fishboning or small crisscrossed lines.
|
|
What 3 other things might you see with lattice degeneration?
|
White without pressure. Lacunae of vitreous over the region (liquefied fluid). Choroidal retinal atrophy. Retinal tears.
|
|
Lattice degeneration management: No symptoms
|
RTC 1 year
|
|
Lattice degeneration management: Has flashes/floaters but scleral depression does not show a hole/tear.
|
RTC 6 months
|
|
Lattice degeneration management: Flashes/floaters with holes/tears
|
Retinal consultation
|
|
Lattice degeneration management: Lattice with horseshoe tear
|
Retinal consultation
|
|
Lattice degeneration management: Asymptomatic but scleral depression shows linear retinal tears
|
Retinal consultation
|
|
Management: snail track degeneration with no symptoms, no holes/tears
|
RTC 1 year
|
|
Management: snail track degeneration with flashes/floaters and scleral depression shows inferio holes.
|
RTC 6 months
|
|
(T/F) Inferior retinal holes are worse than superior ones.
|
F
|
|
Management: superior snail track degeneration, flashes/floaters, no holes/tears
|
Retinal consultation
|
|
Management: Big retinal detachment managed for 6 months. Lattice degeneration superiorly in other eye with no holes/tears. Flashes/floaters.
|
Retinal consultation
|
|
(T/F) Snail track degeneration looks like a slimy snail track and is managed similarly to lattice degeneration.
|
T
|
|
What should be done if an atrophic retinal hole has whitish edema around it?
|
Refer for laser photocoagulation because may lead to detachment
|
|
___% of atrophic retinal holes develop into RD
|
7%
|
|
(T/F) A hole at the border of the vitreous base has less risk than a hole inside of it.
|
F
|
|
Management: atrophic retinal hole, asymptomatic, no traction, no edema
|
RTC 1 year
|
|
Management: Retinal tear with pigmentation
|
Retinal consultation
|
|
Management: Many tiny retinal holes located closely together
|
Retinal consultation b/c can coalesce into a RD
|
|
(T/F) After PVD, there is an increased risk of RD
|
F - Because vitreous has already pulled away so no traction
|
|
At what age has PVD completed pulling away so there is little traction?
|
60-70
|
|
Name 5 risk factors for RD
|
Younger patients (b/c vitreous attached), family history, high myopes, history of RD in other eye, any breaks in retina
|
|
How do you fix an RD?
|
Laser photocoagulation
|
|
Management: Isolated retinal holes, no symptoms, small edema cuff (<1DD)
|
Monitor 3-6 months or retinal consultation
|
|
Management: Isolated holes, flashes and floaters
|
Retinal consultation
|
|
Management: Isolated holes, asymptomatic, no edema cuff
|
RTC 1 year
|
|
Management: Isolated holes with flashes/floaters
|
Retinal consultation
|
|
Management: Isolated holes with flap tear
|
Retinal consultation
|
|
Management: Isolated holes with edema cuff > 1 DD
|
Retinal consultation
|
|
Management: Isolated holes > 1 DD in size
|
Retinal consultation
|
|
Management: Isolated holes with pigment
|
RTC 1 year
|
|
Management: Isolated superior holes with pigment, flashes/floaters
|
Retinal consult
|
|
How does arteriosclerotic retinopathy mess up the eye?
|
HTN -> arteriole walls harden -> A/V crossing changes (pinch) -> Turbulence -> Thrombosis builds and impedes blood flow.
|
|
What is the most common occlusive disease?
|
BRVO
|
|
(T/F) BRVO typically does not have flame hemorrhages
|
F - Has mutiple flame hemorrhages
|
|
How is hollenhorst plaque, fibrinoplatelet and calcific plaque different materials?
|
Cholesterol, platelet/cholesterol, cacified heart valve piece
|
|
Carotic occlusive disease will cause and (artery/vein) occlusion
|
Artery
|
|
(T/F) Arteriosclerotic retinopathy is always indicative of the severity of hypertension
|
F. But might!
|
|
How are the 4 grades of arteriosclerotic retinopathy different?
|
Broadened light reflex w/ simple vein concealment. Deflection of veins at A/V crossings. Copper wire. Silver wire. (All cumulative)
|
|
How are Salus', Gunn's and Bonnet's sign different?
|
A deflection of vein at A/V crossing. 90 degree deflection. Venous banking (dilation of vessels distal to crossing)
|
|
What is 'humping'?
|
When an arteriole is under a vein
|
|
Management: Pt has arteriosclerotic retinopathy
|
Dilate yearly
|
|
A pt complains of sudden vision loss because plaques are blocking things in the eye. What can we do?
|
Reduce the IOP so blood vessels get bigger and clogger moves further out to less damaging area
|
|
(T/F) The likelihood of an artery occlusion from a hollenhorst plaque is much lower than a fibrinoplatelet or calcific plaque.
|
T
|
|
(Hollenhorst, fibrinoplateley, calcific) plaques have a higher risk for artery occlusion. Why?
|
Because the platelets cause clotting/stickiness and they stay longer.
|
|
What does a fibrinoplatelet plaque look like?
|
Elongated shape
|
|
What does a hollenhorst plaque look like?
|
Shiny dot
|
|
How does the retina change if a fibrinoplatelet plaque reduces blood flow?
|
Whitish and edematous
|
|
Name 2 things that predisposes a pt to calcific plaques
|
Rheumatic fever, bacterial endocarditis
|
|
What kind of occlusion do calcific plaques most cause?
|
CRAO
|
|
What do calcific plaques look like?
|
Chalky white
|
|
Management: artery occlusions in general
|
Systemic disease workup. (Especially vascular)
|
|
If you see several flame hemorrhages in one area, it is probably a…
|
BRVO
|
|
What age and gender does CRVO's most affect?
|
60's. Males.
|
|
Name 6 things that can cause a CRVO
|
HTN, diabetes, hyperviscosity syndrome diseases (leukemia, sickle cell, diabetes), glaucoma, hyperlipidemia, head injuries
|
|
If a thrombosis builds up in a venule, how does it cause a CRVO?
|
It goes to the lamina cribosa.
|
|
What are the symptoms of a CRAO?
|
Painless vision loss over 1-2 days.
|
|
(T/F) Vision cannot improve with a CRAO
|
F
|
|
What are the 2 signs of hypoxia in the retina?
|
Nerve fiber layer infarcts with Cotton wool spots. Neo.
|
|
How does CRVO lead to glaucoma?
|
Neovascularization of iris leads to neovascular glaucoma
|
|
What are the 6 ophthalmic signs of a CRAO?
|
Blood and thunder of retina. Swollen ONH with no venous pulse, although ONH may pulse. Hypoxia signs. Retinal hemorrhaging. Vitreous hemorrhaging. Venous engorgement.
|
|
How does venous engorgement happen with CRAO?
|
Blood backs up so venules engorge and things seep out causing edema.
|
|
The (hemorrhagic/venous stasis) is the worse one.
|
hemorrhagic
|
|
With ____ days, a CRAO patient will run the risk for glaucoma to occur.
|
90-100 days
|
|
What happens with venous stasis?
|
Circulation is reduced
|
|
What is the VA range for venous stasis?
|
20/20 - 20/200
|
|
(T/F) Venous stasis pts are almost always diagnosed for diabetes or HTN
|
F
|
|
How does the retina look with venous stasis?
|
Can still see most of the retina and ONH. Venous engorgement. Cotton wool spots. Hemorrhaging (dot blot and flame)
|
|
(T/F) Venous stasis often leads to rubeosis and glaucoma
|
F
|
|
(T/F) Venous stasis can show dot blot hemorrhages and lots of flame hemorrhages
|
T
|
|
(T/F) Hemorrhagic retinopathy (ischemic) pts often have poorly controlled diabetes and HTN
|
T
|
|
(T/F) Hemorrhagic retinopathy can show lots of hemorrhaging, cotton wool spots NFL infarcts and rubeosis.
|
T
|
|
What helps reabsorb the fast growing tissue from neo?
|
Anti-Vegf medication like Avastin.
|
|
Why does neo often occur in the iris?
|
It moves anteriorly from the retina to the iris.
|
|
(T/F) With hemorrhagic CRVO, we can often see most of the retina.
|
F
|
|
What sign do we see when there is neo starting in the iris?
|
little red spots.
|
|
(HTN/Diabetes) is most often the cause of a hemorrhagic CRVO
|
HTN
|
|
What is the standard bloodwork tha should be done for a retinal vascular occlusive disease?
|
CBC.
|
|
What 3 things do retinal specialists do when there is a retinal vascular occlusive disease?
|
Pan-retinal laser photocoagulation. Anti-VEGF medication. Triamcinoline (kenalog)
|
|
How does triamcinolone help with retinal vascular occlusive disease?
|
Decreases inflammation, swelling and vascular permeability. Therefore less leaking/bleeding.
|
|
(T/F) Avastin should be used even before there is neo.
|
F
|
|
How does laser work help with hypoxia?
|
Kills tissue to reduce oxygen demand
|
|
(T/F) With lots of hemorrhaging for retinal vein occlusive disease, it is good to give blood thinners
|
T b/c it can break up the thrombosis.
|
|
How often should we follow up for retinal vein occlusive disease?
|
3-4 weeks or every 6 months.
|
|
What are the 2 risks of steroid use?
|
Increased IOP and cataracts
|
|
What is the normal name for "altitudinal" or "dual branch" occlusion?
|
Hemicentral Retinal Vein Occlusion
|
|
Where does a hemicentral retinal vein occlusion occur?
|
Just past the lamina where the vessels first branches
|
|
How does a large CD ratio cause hemorrhaging?
|
The venule is kinked because the turn is too tight, causing hemorrhaging
|
|
Hemicentral retinal vein occlusion can create what 4 things?
|
Macular edema, disc edema, hypoxia of the retina, neo of the retina/iris
|
|
Management: Hemicentral retinal vein occlusion
|
Retinal consult to get FANG to determine spots to treat with laser.
|
|
What causes BRVO to be symptomatic?
|
If it's on the retina.
|
|
Management: BRVO not threatening the macula.
|
Control systemic disease and RTC in 1 month.
|
|
What blood pressures could indicate a BRVO?
|
140+ or 80+
|
|
How does a retinal hemorrhave move to the macula?
|
It is superior so gravity causes the fluid to go down to the macula
|
|
Management: BRVO with neo
|
Refer to retinal specialist.
|
|
What might a retinal specialist do for a BRVO with neo?
|
Laser photocoagulation or anti-VEGF
|
|
Management: BRVO threatening macula.
|
Retinal consult.
|
|
What might a retinal specialist do for a BRVO threatening the macula?
|
Triancinolone injection or laser photocoagulation.
|
|
(T/F) CRAO is often bilateral.
|
F
|
|
What are the #1 and #2 causes of CRAO?
|
calcific plaques. Fibrinoplatelet plaques
|
|
What are 4 causes of a CRAO?
|
Giant cell arteritis, migraines, contraceptives, man made emboli (talc from drugs)???
|
|
What is the symptom of CRAO?
|
Acute, painless monocular vision loss.
|
|
What are the ophthalmic signs of a CRAO?
|
Edema and hypoxia. Distended veins. Cherry Red spot on macula.
|
|
(T/F) A CRAO can cause an APD
|
T
|
|
What is a sign of an old CRAO?
|
Optic atrophy where retina thins and is replaced by glial tissue. Vessels look very thin.
|
|
Neo often occurs with (CRAO/CRVO)
|
CRVO
|
|
Why does neo not occur too much with CRAO?
|
Because it happens so fast.
|
|
Why might peripheral vision be terrible but VAs be good for a CRAO?
|
Cilioretinal artery can feed macula.
|
|
What do we do for a CRAO?
|
Lower IOP within 15-90 minutes.
|
|
(T/F) Beta blockers and lumigan are good for lowering IOP for a CRAO.
|
F. Too slow.
|
|
What do retinal specialists do to reduce IOP in a CRAO?
|
Paracentesis (draw out aqueous with needle)
|
|
(T/F) The point of digital massage is to make pressure go up. How?
|
F. Go down. Causes high pressure that pushes aqueous out. The pressure drops.
|
|
How does breathing in a brown paper bag help for CRAO?
|
It increases CO2 levels, which dilates blood vessels.
|
|
What does a CRAO ophthalmically look like?
|
Milky white edematous retina with cherry red spot.
|
|
What treatment do we do for a CRAO?
|
Get the IOP down (paracentesis needle), brown paper bag to dilate vessels, IV acetazolamide. Get vascular workup.
|
|
(T/F) Topical glaucoma drugs are good for reducing IOP for CRAO
|
F. Too slow
|
|
What 3 things mimic CRAO?
|
Giant cell arteritis, high blood pressure, temporal arteritis.
|
|
How do we r/o temporal arteritis when we have a CRAO suspect?
|
ESR (erythrocyte sedimentation rate) workup.
|
|
What race gets sickle cell retinopathy?
|
Blacks
|
|
How is sickle cell and diabetic retinopathy similar?
|
They both mishape RBS. Sickling. Dumbells.
|
|
How does sickle cell retinopathy lead to RD?
|
hypoxic -> neo -> traction -> RD
|
|
What are sunbursts of melanin made of?
|
Melanin and hemosiderin
|
|
______ lesions are often found in sickle cell retinopathy.
|
Salmon patch lesions/hemorrhages.
|
|
(T/F) Fibrovascular proliferation can occur in sickle cell retinopathy.
|
T
|
|
What are angiod streaks?
|
Breaks in bruch's membrane.
|
|
(T/F) Angiod streaks are found in sickle cell retinopathy.
|
T
|
|
I see a see fan of neo. What disease does the pt have?
|
Sickle cell retinopathy.
|
|
Why does neo lead to a traction retinal detachment?
|
Root is in retinal. Tops are in vitreous. Vitreous moves, but retina doesn't. Rip!
|
|
(T/F) Rubeosis neovascularization is very common in sickle cell retinopathy.
|
F. It can happen but not common
|
|
What therapies do we do for a sickle cell retinopathy pt?
|
Fluorescein angiogram, pan-retinal photocoagulation, anti-VEGF injection
|
|
How does retinopathy of prematurity occur?
|
Premature infant put in O2 rich environment. Eyes get used to it. When pulled out, the hypoxic signal makes neo grow.
|
|
When does the nasal and temporal retinal vessels reach the ora serrata?
|
8 months. After birth.
|
|
A dragged disc appearance is caused by what?
|
Retinopathy of prematurity.
|
|
How are ROP patients treated?
|
Cryoplasty or laser photocoagulation
|
|
If an adult is seeing 20/20 but had ROP, what is the management?
|
Yearly DFE
|
|
When the peripheral retinal forms little cystic space within the sensory retina, what can you get?
|
Retinoschisis
|
|
(T/F) Retinoschisis tends to grow in size.
|
F
|
|
Retinoschisis tens to be located ____ _____.
|
Inferior temporal
|
|
Retinal detachments tend to be locate _____ ____
|
superior temporal
|
|
(T/F) Retinoschisis pts tend to be asymptomatic.
|
T.
|
|
(T/F) Retinoschisis can lead to a scotoma.
|
T
|
|
What kind of scotoma does an RD give? Retinoschisis?
|
Relative. Absolute.
|
|
Why does retinoschisis give an absolute scotoma?
|
Because the retina isn't there any more!
|
|
What does cystoid degeneration look like?
|
Translucent lesion
|
|
(T/F) Retinoschisis makes the retina look moth eaten like beaten metal.
|
T
|
|
(T/F) Flat retinoschisis is more common than bullous retinoschisis.
|
F
|
|
____ retinoschisis looks like a transparent ballooning that looks elevated. Why?
|
Bullous retinoschisis. Because hyaluronic acid from the vitreous filles in the area.
|
|
___% of retinoschisis patients have a retinal hole or tear
|
32%
|
|
What does flat retinoschisis look like? But how does it look different?
|
White without pressure. No little whitish spots or snowflake specks.
|