• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/207

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

207 Cards in this Set

  • Front
  • Back
(T/F) Operculated Retinal holes have a higher chance of RD. Why?
F. No more traction b/c retina is torn clear away.
What test should you do to see if operculated retinal holes are truly fully detached?
Scleral depression
(T/F) Pigment is a sign of stability for retinal holes.
T
(T/F) Operculated retinal hole pieces tend to shrink over time. Therefore, you can tell if it is old by comparing the size of the free floating piece to the hole.
Operculated retinal hole management: New hole with photopsia?
RTC 6 weeks
Operculated retinal hole management: New hole with no symptoms?
RTC 6 months
Operculated retinal hole management: Multiple holes
RTC 6 months
Operculated retinal hole management: Cuff edema < 2 DD with no pigmentation?
RTC 3 months
Operculated retinal hole management: Hole with pigment?
RTC 6 months
Operculated retinal hole management: Cuff edema greater than 2 DD in size?
Refer to retinal specialist.
___% of linear retinal tears develop into retinal breaks. ___% develop into RDs.
10-15%. 30%
Risk factors for linear retinal tears. (4)
High myopes, trauma history, aphakia, premature birth (ROP)
What does a linear retinal tear look like?
Horseshoe with red center and grey tissue around edge.
Where do linear retinal tears normally occur?
Posterior border of vitreal base
What is Shaefer's Sign in the context of linear retinal tears?
Pigmentation enters the vitreous.
If there is so much hemorrhaging that you can't see the retina what do you do?
Get a retinal B scan.
What is the treatment for a linear retinal tear?
Laser photocoagulation
Linear retinal tear management: When you find one?
Refer to retinal specialist.
Retinal dialysis management: When you find one?
Refer to retinal specialist.
Retinal dialysis management: When a person has a recent trauma, what is the dilation schedule?
Do at exam. Then repeat in 3-6 months b/c they they develop slowly.
(T/F) Retinal dialysis is often asymptomatic
T - 60%
Name the typical retinal dialysis pt.
Younger. Males under 40 years that had trauma.
Why are retinal dialysis tears so slow to develop? How long does it take?
They are often inferior. Within 140 days.
Why might IOP go up or down during retinal dialysis or detachment??
Up because pigment released into vitreous. Down because vitreous has another place to leave.
Giant Retinal Tear management: When you find one?
Refer to retinal specialist.
What causes Commotio Retinae?
Blunt trauma.
What do you see with Commotio Retinae?
At first, looks normal. Hours later it becomes swollen (opaque white, edema). Possibly hemorrhagic (black and blue).
What might happen as the swelling of Commotio Retina subsides?
Pt may develop holes or tears.
What is Berlin's Edema?
Retinal swelling that can lead to retinal dialysis because the mucopolysaccharide bonds of the retina are weakened. Associated with Commotio Retinae
What is a typical pt complaint with Commotio Retinae?
Blur due to retinal swelling (and black eye!)
Why do RDs happen most often after age 60?
PVD has vitreous pulling on retina.
If you have RD in one eye, how likely are you to get it in the other eye?
10-30%
(T/F) RD symptoms are always symptomatic.
F - can be asymptomatic because they are variable
What are the classic symptoms of RD?
Flashes and floaters
What is the old was to treat RD? The new way?
Cryopoxy. Laser photocoagulation
What 2 things do you see in the retina when a pt has RD?
Dissapearance of choroidal layer (Can't see ampules and other details.) Edema that makes the retina opaque.
What technology is very usefule in outlining retinal detachments, especially when there is hemorrhaging in the vitreous?
B Scan
Retinal detachment management: New one
Immediate referral
Retinal detachment management: Old one newly detected
Consultation within a few days
(T/F) Flashes and floaters stop right after the retina has detached.
F - They can continue for up to 6 months afterwards
What is snail track degeneration?
Thinning of parts of the retina
Name 2 reasons why snail track degeneration pts have a higher risk of RD?
Liquefied vitreous gets under the retina and lifts it up. Vitreoretinal traction.
(T/F) Active PVD increases your chances for a retinal tear.
T b/c the vitreous is peeling away and taking the retina with it.
What does an atrophic retinal hole look like?
Red dot. Possibly greyish edema around edges.
How does an atrophic retinal hole lead to RDs?
Fluid enters along the edges and lifts the retina up.
(T/F) Linear Retinal tears have a higher risk of RDs.
T
(T/F) Giant retinal tears do not have a higer risk of RDs
F
What is degenerative retinoschisis?
Splitting of the retina.
Degenerative retinoschisis management: You see it for the first time.
Refer to retinal specialist
Degenerative retinoschisis management: Watching old one w/ no flashes and floaters
RTC 6 months
Degenerative retinoschisis management: Watching old one w/ flashes and floaters
Refer to retinal specialist
Degenerative retinoschisis management: Watching old one w/ vitreoretinal traction.
Refer to retinal specialist
What is lattice degeneration?
Thinning of the retina with vitreoretinal traction on the edges with liquefied vitreous in that area
What is a typical lattice degeneration pt?
Young (6-10% of normal population)
(T/F) Lattice degeneration is typically bilateral
T
(T/F) Lattice degeneration can lead to holes or tears. Why?
T - Because it's thin
What is the typical size of a lattice degeneration?
1-4 DD in length. .5-2 DD in width.
(T/F) RPE hyperplasia with lattice degeneration is good
T - Pigmentation is a sign of stability
What does Lattice degeneration look like?
Fishboning or small crisscrossed lines.
What 3 other things might you see with lattice degeneration?
White without pressure. Lacunae of vitreous over the region (liquefied fluid). Choroidal retinal atrophy. Retinal tears.
Lattice degeneration management: No symptoms
RTC 1 year
Lattice degeneration management: Has flashes/floaters but scleral depression does not show a hole/tear.
RTC 6 months
Lattice degeneration management: Flashes/floaters with holes/tears
Retinal consultation
Lattice degeneration management: Lattice with horseshoe tear
Retinal consultation
Lattice degeneration management: Asymptomatic but scleral depression shows linear retinal tears
Retinal consultation
Management: snail track degeneration with no symptoms, no holes/tears
RTC 1 year
Management: snail track degeneration with flashes/floaters and scleral depression shows inferio holes.
RTC 6 months
(T/F) Inferior retinal holes are worse than superior ones.
F
Management: superior snail track degeneration, flashes/floaters, no holes/tears
Retinal consultation
Management: Big retinal detachment managed for 6 months. Lattice degeneration superiorly in other eye with no holes/tears. Flashes/floaters.
Retinal consultation
(T/F) Snail track degeneration looks like a slimy snail track and is managed similarly to lattice degeneration.
T
What should be done if an atrophic retinal hole has whitish edema around it?
Refer for laser photocoagulation because may lead to detachment
___% of atrophic retinal holes develop into RD
7%
(T/F) A hole at the border of the vitreous base has less risk than a hole inside of it.
F
Management: atrophic retinal hole, asymptomatic, no traction, no edema
RTC 1 year
Management: Retinal tear with pigmentation
Retinal consultation
Management: Many tiny retinal holes located closely together
Retinal consultation b/c can coalesce into a RD
(T/F) After PVD, there is an increased risk of RD
F - Because vitreous has already pulled away so no traction
At what age has PVD completed pulling away so there is little traction?
60-70
Name 5 risk factors for RD
Younger patients (b/c vitreous attached), family history, high myopes, history of RD in other eye, any breaks in retina
How do you fix an RD?
Laser photocoagulation
Management: Isolated retinal holes, no symptoms, small edema cuff (<1DD)
Monitor 3-6 months or retinal consultation
Management: Isolated holes, flashes and floaters
Retinal consultation
Management: Isolated holes, asymptomatic, no edema cuff
RTC 1 year
Management: Isolated holes with flashes/floaters
Retinal consultation
Management: Isolated holes with flap tear
Retinal consultation
Management: Isolated holes with edema cuff > 1 DD
Retinal consultation
Management: Isolated holes > 1 DD in size
Retinal consultation
Management: Isolated holes with pigment
RTC 1 year
Management: Isolated superior holes with pigment, flashes/floaters
Retinal consult
How does arteriosclerotic retinopathy mess up the eye?
HTN -> arteriole walls harden -> A/V crossing changes (pinch) -> Turbulence -> Thrombosis builds and impedes blood flow.
What is the most common occlusive disease?
BRVO
(T/F) BRVO typically does not have flame hemorrhages
F - Has mutiple flame hemorrhages
How is hollenhorst plaque, fibrinoplatelet and calcific plaque different materials?
Cholesterol, platelet/cholesterol, cacified heart valve piece
Carotic occlusive disease will cause and (artery/vein) occlusion
Artery
(T/F) Arteriosclerotic retinopathy is always indicative of the severity of hypertension
F. But might!
How are the 4 grades of arteriosclerotic retinopathy different?
Broadened light reflex w/ simple vein concealment. Deflection of veins at A/V crossings. Copper wire. Silver wire. (All cumulative)
How are Salus', Gunn's and Bonnet's sign different?
A deflection of vein at A/V crossing. 90 degree deflection. Venous banking (dilation of vessels distal to crossing)
What is 'humping'?
When an arteriole is under a vein
Management: Pt has arteriosclerotic retinopathy
Dilate yearly
A pt complains of sudden vision loss because plaques are blocking things in the eye. What can we do?
Reduce the IOP so blood vessels get bigger and clogger moves further out to less damaging area
(T/F) The likelihood of an artery occlusion from a hollenhorst plaque is much lower than a fibrinoplatelet or calcific plaque.
T
(Hollenhorst, fibrinoplateley, calcific) plaques have a higher risk for artery occlusion. Why?
Because the platelets cause clotting/stickiness and they stay longer.
What does a fibrinoplatelet plaque look like?
Elongated shape
What does a hollenhorst plaque look like?
Shiny dot
How does the retina change if a fibrinoplatelet plaque reduces blood flow?
Whitish and edematous
Name 2 things that predisposes a pt to calcific plaques
Rheumatic fever, bacterial endocarditis
What kind of occlusion do calcific plaques most cause?
CRAO
What do calcific plaques look like?
Chalky white
Management: artery occlusions in general
Systemic disease workup. (Especially vascular)
If you see several flame hemorrhages in one area, it is probably a…
BRVO
What age and gender does CRVO's most affect?
60's. Males.
Name 6 things that can cause a CRVO
HTN, diabetes, hyperviscosity syndrome diseases (leukemia, sickle cell, diabetes), glaucoma, hyperlipidemia, head injuries
If a thrombosis builds up in a venule, how does it cause a CRVO?
It goes to the lamina cribosa.
What are the symptoms of a CRAO?
Painless vision loss over 1-2 days.
(T/F) Vision cannot improve with a CRAO
F
What are the 2 signs of hypoxia in the retina?
Nerve fiber layer infarcts with Cotton wool spots. Neo.
How does CRVO lead to glaucoma?
Neovascularization of iris leads to neovascular glaucoma
What are the 6 ophthalmic signs of a CRAO?
Blood and thunder of retina. Swollen ONH with no venous pulse, although ONH may pulse. Hypoxia signs. Retinal hemorrhaging. Vitreous hemorrhaging. Venous engorgement.
How does venous engorgement happen with CRAO?
Blood backs up so venules engorge and things seep out causing edema.
The (hemorrhagic/venous stasis) is the worse one.
hemorrhagic
With ____ days, a CRAO patient will run the risk for glaucoma to occur.
90-100 days
What happens with venous stasis?
Circulation is reduced
What is the VA range for venous stasis?
20/20 - 20/200
(T/F) Venous stasis pts are almost always diagnosed for diabetes or HTN
F
How does the retina look with venous stasis?
Can still see most of the retina and ONH. Venous engorgement. Cotton wool spots. Hemorrhaging (dot blot and flame)
(T/F) Venous stasis often leads to rubeosis and glaucoma
F
(T/F) Venous stasis can show dot blot hemorrhages and lots of flame hemorrhages
T
(T/F) Hemorrhagic retinopathy (ischemic) pts often have poorly controlled diabetes and HTN
T
(T/F) Hemorrhagic retinopathy can show lots of hemorrhaging, cotton wool spots NFL infarcts and rubeosis.
T
What helps reabsorb the fast growing tissue from neo?
Anti-Vegf medication like Avastin.
Why does neo often occur in the iris?
It moves anteriorly from the retina to the iris.
(T/F) With hemorrhagic CRVO, we can often see most of the retina.
F
What sign do we see when there is neo starting in the iris?
little red spots.
(HTN/Diabetes) is most often the cause of a hemorrhagic CRVO
HTN
What is the standard bloodwork tha should be done for a retinal vascular occlusive disease?
CBC.
What 3 things do retinal specialists do when there is a retinal vascular occlusive disease?
Pan-retinal laser photocoagulation. Anti-VEGF medication. Triamcinoline (kenalog)
How does triamcinolone help with retinal vascular occlusive disease?
Decreases inflammation, swelling and vascular permeability. Therefore less leaking/bleeding.
(T/F) Avastin should be used even before there is neo.
F
How does laser work help with hypoxia?
Kills tissue to reduce oxygen demand
(T/F) With lots of hemorrhaging for retinal vein occlusive disease, it is good to give blood thinners
T b/c it can break up the thrombosis.
How often should we follow up for retinal vein occlusive disease?
3-4 weeks or every 6 months.
What are the 2 risks of steroid use?
Increased IOP and cataracts
What is the normal name for "altitudinal" or "dual branch" occlusion?
Hemicentral Retinal Vein Occlusion
Where does a hemicentral retinal vein occlusion occur?
Just past the lamina where the vessels first branches
How does a large CD ratio cause hemorrhaging?
The venule is kinked because the turn is too tight, causing hemorrhaging
Hemicentral retinal vein occlusion can create what 4 things?
Macular edema, disc edema, hypoxia of the retina, neo of the retina/iris
Management: Hemicentral retinal vein occlusion
Retinal consult to get FANG to determine spots to treat with laser.
What causes BRVO to be symptomatic?
If it's on the retina.
Management: BRVO not threatening the macula.
Control systemic disease and RTC in 1 month.
What blood pressures could indicate a BRVO?
140+ or 80+
How does a retinal hemorrhave move to the macula?
It is superior so gravity causes the fluid to go down to the macula
Management: BRVO with neo
Refer to retinal specialist.
What might a retinal specialist do for a BRVO with neo?
Laser photocoagulation or anti-VEGF
Management: BRVO threatening macula.
Retinal consult.
What might a retinal specialist do for a BRVO threatening the macula?
Triancinolone injection or laser photocoagulation.
(T/F) CRAO is often bilateral.
F
What are the #1 and #2 causes of CRAO?
calcific plaques. Fibrinoplatelet plaques
What are 4 causes of a CRAO?
Giant cell arteritis, migraines, contraceptives, man made emboli (talc from drugs)???
What is the symptom of CRAO?
Acute, painless monocular vision loss.
What are the ophthalmic signs of a CRAO?
Edema and hypoxia. Distended veins. Cherry Red spot on macula.
(T/F) A CRAO can cause an APD
T
What is a sign of an old CRAO?
Optic atrophy where retina thins and is replaced by glial tissue. Vessels look very thin.
Neo often occurs with (CRAO/CRVO)
CRVO
Why does neo not occur too much with CRAO?
Because it happens so fast.
Why might peripheral vision be terrible but VAs be good for a CRAO?
Cilioretinal artery can feed macula.
What do we do for a CRAO?
Lower IOP within 15-90 minutes.
(T/F) Beta blockers and lumigan are good for lowering IOP for a CRAO.
F. Too slow.
What do retinal specialists do to reduce IOP in a CRAO?
Paracentesis (draw out aqueous with needle)
(T/F) The point of digital massage is to make pressure go up. How?
F. Go down. Causes high pressure that pushes aqueous out. The pressure drops.
How does breathing in a brown paper bag help for CRAO?
It increases CO2 levels, which dilates blood vessels.
What does a CRAO ophthalmically look like?
Milky white edematous retina with cherry red spot.
What treatment do we do for a CRAO?
Get the IOP down (paracentesis needle), brown paper bag to dilate vessels, IV acetazolamide. Get vascular workup.
(T/F) Topical glaucoma drugs are good for reducing IOP for CRAO
F. Too slow
What 3 things mimic CRAO?
Giant cell arteritis, high blood pressure, temporal arteritis.
How do we r/o temporal arteritis when we have a CRAO suspect?
ESR (erythrocyte sedimentation rate) workup.
What race gets sickle cell retinopathy?
Blacks
How is sickle cell and diabetic retinopathy similar?
They both mishape RBS. Sickling. Dumbells.
How does sickle cell retinopathy lead to RD?
hypoxic -> neo -> traction -> RD
What are sunbursts of melanin made of?
Melanin and hemosiderin
______ lesions are often found in sickle cell retinopathy.
Salmon patch lesions/hemorrhages.
(T/F) Fibrovascular proliferation can occur in sickle cell retinopathy.
T
What are angiod streaks?
Breaks in bruch's membrane.
(T/F) Angiod streaks are found in sickle cell retinopathy.
T
I see a see fan of neo. What disease does the pt have?
Sickle cell retinopathy.
Why does neo lead to a traction retinal detachment?
Root is in retinal. Tops are in vitreous. Vitreous moves, but retina doesn't. Rip!
(T/F) Rubeosis neovascularization is very common in sickle cell retinopathy.
F. It can happen but not common
What therapies do we do for a sickle cell retinopathy pt?
Fluorescein angiogram, pan-retinal photocoagulation, anti-VEGF injection
How does retinopathy of prematurity occur?
Premature infant put in O2 rich environment. Eyes get used to it. When pulled out, the hypoxic signal makes neo grow.
When does the nasal and temporal retinal vessels reach the ora serrata?
8 months. After birth.
A dragged disc appearance is caused by what?
Retinopathy of prematurity.
How are ROP patients treated?
Cryoplasty or laser photocoagulation
If an adult is seeing 20/20 but had ROP, what is the management?
Yearly DFE
When the peripheral retinal forms little cystic space within the sensory retina, what can you get?
Retinoschisis
(T/F) Retinoschisis tends to grow in size.
F
Retinoschisis tens to be located ____ _____.
Inferior temporal
Retinal detachments tend to be locate _____ ____
superior temporal
(T/F) Retinoschisis pts tend to be asymptomatic.
T.
(T/F) Retinoschisis can lead to a scotoma.
T
What kind of scotoma does an RD give? Retinoschisis?
Relative. Absolute.
Why does retinoschisis give an absolute scotoma?
Because the retina isn't there any more!
What does cystoid degeneration look like?
Translucent lesion
(T/F) Retinoschisis makes the retina look moth eaten like beaten metal.
T
(T/F) Flat retinoschisis is more common than bullous retinoschisis.
F
____ retinoschisis looks like a transparent ballooning that looks elevated. Why?
Bullous retinoschisis. Because hyaluronic acid from the vitreous filles in the area.
___% of retinoschisis patients have a retinal hole or tear
32%
What does flat retinoschisis look like? But how does it look different?
White without pressure. No little whitish spots or snowflake specks.