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107 Cards in this Set

  • Front
  • Back
Gypsum:Fixation casts to articulators
w/p=63cc/100g
Quick Set Plaster
Gypsum: Diagnosis/Planning
w/p=45 to 55 cc/100g
Plaster Study Models
Gypsum: Fabricate Appliances
w/p=30cc/100g
Stone Casts
Gypsum: Crown Fabrication
w/p=22cc/100g
Improved Stone Dies
If more water is used than recommended. Mixes have _______ setting time, are _________, poor _________ (too fluid), and ________ hard, so it ________more easily
LONGER
WEAKER
STACKING
LESS
ABRADES
Accelerators
2% KSO4
Slurry Water
Longer Mix/Vibration
Retarders
Blood
Salivia
Alginate
Less mixing/vibration
6 to 8 minutes
initial set/loss of gloss
15 to 30minute
final set
30 to 60minutes
separate from impression
Mixing Principles
-Sift powder into water bowl with vibrator on high
-Mix without fluffling until homogeneous on Vibrator
-Flex bowl vigourously and repeatedly while pressing bowl hard on vibrator on high setting
sources of macroscopic porosity
-air trapped in btw powder particles
-air mixed in while homogenizing
-air trapped while pouring the mix in the mold
Gypsum Powder
Gypsum Cast
Gypsum Rxn
Forming Gypsum powder into Gypsum Cast is what type of chemical reaction?
Exothermic-releases heat
3 things that cause Alginate's poor dimensional stability
-water evaporation
-imbibition
-syneresis
explain Water evaporation in relating to poor dimensional stability
evaporation from gel causes 25+% shrinkage
explain imbibition in relating to poor dimensional stability
water absorption with water soaking
explain syneresis in relating to poor dimensional stability
shrinkage with continued reaction in humidor. AT 100% HUMIDITY
When do you pour your cast after taking the Alginate impression?
ASAP-as soon as possible
sets alginate too fast
hot water
slow set and flows too much
cold water-also problem for sensitive teeth.
a type whose physical condition is changed by a chemical action that is not reversible. It is an impression material that is elastic when set.
irreversible hydrocolloid
a type whose physical condition is changed by temperature. The material is made fluid by heat and becomes an elastic solid on cooling. Can change from a gel to solid set with the addition or removal of heat
reversible hydrocolloid
particles less than 0.5um
colloid
Steps in making successful alginate impressions
1. fluff powder and scoop parallel to container
2. level scoop, NO packing
3. 1 scoop/water ring
4. use room temperature water
5. wet powder slowly
6. spread in thin layers with flat spatula, to eliminate air bubbles by "strooping" back and forth til cmppth and creamy
7. load tray back to front, must be full
8. seat pt upright, wipe alginate on tray with wet glove finger
9. rotate corner of tray into mouth, center tray on midline of arch
10. pull lips over the tray before seating. seat posterior first. (3-5mm material tray btw teeth and soft tissue)
11. wit 2 minutes
12. break air lock
13. rinse in water for 30s
14. check impression for voids
15.spray with disinfectant and wrap in damp towl and placed in plastic bag
16. clean metal tray/ pour casts
the condylar housing is on the upper member of the articulator and the condylar ball is on the lower articulator
arcon articulator
Same relationship found in the human skull
arcon articulator
the condylar housing is on the lower member of the articulator and the condylar ball is on the upper member of the articulator
non arcon articulator
opposite of what is found in the human skull
non arcon articulator
caliper-like instrument which is used to relate the maxillary cast to the condyle elements of the articulator just like the maxilla is related to the condyles and fossa of your patient
facebow
facebow utilizes an estimate of the location of the transverse horizontal or hinge axis
arbitary/ average value
facebow has adjustible caliper ends which are used to locate the transverse horizontal axis of the mandible
kinematic
precise enough for most diagnosis and treatment
arbitrary facebow and fixed conylar width articulator
mechanical instrument that represents the tempromandibular joints and jaws, to which maxillary and mandibular casts are attached to stimulate some or all mandibular movements
articulator
Masseter
Superficial head
O: Anterior 2/3 of lower border
of the zygomatic arch
I: On the angle and ramus of the
mandible

Deep head
O: Medial surface ( posterior) of
the zygomatic arch
I : On ramus and part of the coroniod
process

Action: elevation (as in closing of the mouth) and protraction of mandible
temporalis
O: Lateral surface of the skull;
temporal fossa and temporal fascia

I: Coronoid process of mandible and
border of ramus

A:Elevation and retraction of mandible
Occipitalis
Origin: Superior nuchal line of the occipital bone and mastoid process of the temporal bone

Insertion: Galea aponeurosis

Actions: Moves the scalp back
frontalis and occipitalis joined at _______ by fascial connective tissue sheath and aponeurosis (tendon) sheath
vertex
Sternocleidomastoid
Origin: Manubrium sterni and medial portion of the clavicle

Insertion:Mastoid process of the temporal bone, superior nuchal line

Actions: Acting alone, tilts head to its own side and rotates it so the face is turned towards the opposite side.
Acting together, flexes the neck, raises the sternum and assists in forced inspiration.
digastric
Origin:
anterior belly - digastric fossa (mandible);
posterior belly - mastoid process of temporal bone

Insertion: Intermediate tendon (hyoid bone)

Actions: Opens the jaw when the masseter and the temporalis are relaxed.
Medial Pterygoid
Origin:
deep head: medial side of lateral pterygoid plate behind the upper teeth
superficial head: pyramidal process of palatine bone and maxillary tuberosity

Insertion: medial angle of the mandible

Actions: elevates mandible, closes jaw, helps lateral pterygoids in moving the jaw from side to side
Lateral Pterygoid Muscle
Origin: Great wing of sphenoid and pterygoid plate

Insertion: Condyloid process of the mandible

Actions: depresses mandible, protrude mandible, side to side movement of mandible
Occurs when there is contact
between the maxillary and
mandibular posterior teeth
on the same side of the arches
as the direction in which the
mandible has moved
working interference
An occlusal contact between
maxillary and mandibular teeth
on the side of the arches opposite
the direction in which the mandible
has moved in a lateral excursion
nonworking interference
The proximity of the teeth to the
muscles and the oblique vector
of the forces make contacts
between opposing posterior
teeth during protrusion
protrusive interference
Pattern of Disclusion

A pattern of mutually-protected
articulation in which the
vertical and horizontal overlap of the
canine teeth disengage the posterior
teeth in excursive movements of
the mandible
anterior/canine guidance
Pattern of dis-occulsion

Multiple mandibular teeth
contact the maxillary teeth
during lateral movement
on the working side

Occlusal forces distributed
over a group of teeth
group function
It is characteristic where the
incisal edges of the maxillary
incisors extend below the
incisal edges of the
mandibular incisors when
the posterior teeth are
fully interdigitated.
overbite
vertical dimension
overbite
It is the characteristic where the incisal edges of the maxillary teeth
extend labially to the incisal edges of the mandibular incisors when
the posterior teeth are inter digitated
overjet
horizontal dimension
overjet
When the upper canine
occludes in the
embrasure between the
lower canine and the
lower first premolar
Class 1
The maxillary first
molar mesiobuccal
cusp tip occludes in
the lower first molar
buccal developmental
groove
Class 1
When the upper canine occludes
anterior to the embrasure between the lower canine
and the lower first premolar
Class 2
When the maxillary first molar
mesiobuccal cusp tip occludes
anterior to the buccal
developmental groove
on the mandibular first molar
Class 2
When the upper canine
occludes posterior to
the embrasure between lower canine and the first
premolar
Class 3
When the upper first molar mesial cusp
tip occludes distal to
the buccal develop-mental groove
of the lower first
molar
Class 3
diagnostic dilemma
PATIENTS WITH PRIMARY MYOFASCIAL PAIN PROBLEMS CAN DEVELOP SECONDARY TEMPOROMANDIBULAR JOINT PATHOLOGY

PATIENTS WITH PRIMARY TMJ PATHOLOGY CAN DEVELOP SECONDARY MYOFASCIAL PAIN PROBLEMS
joint pain is
constant and localized
muscle pain is
intermittent and poorly localized
muscle pain increases when biting on the
painful side
joint pain increases when biting on the
opposite side
congenital anomalies of the TMJ are characterized by
agenesis of the condyle
IS A CHRONIC LIMITATION OF MOVEMENT DUE TO JOINT CONSOLIDATION
ankylosis
IS LIMITATION OF MOVEMENT DUE TO MUSCLE INFLAMMATION OR SPASM
trismus
Patients with disc displacement have a history of
prior clicking
Patients with disc adhesion have a sudden onset of
locking without a prior history of any TMJ problems
TREATMENT OF THE MPD PATIENT Phase I (2-4 Wks)
INITIAL EXPLANATION OF THE PROBLEM
HOME THERAPY
MEDICATIONS FOR PAIN AND ANXIETY
MEDICATION FOR SLEEP
POSSIBLE USE OF A BITE APPLIANCE
TREATMENT OF THE MPD PATIENT Phase II (4Wks)
RECONSIDER THE DIAGNOSIS
CHECK FOR COMPLIANCE
CONTINUE HOME THERAPY AND MEDICATIONS
CONSIDER USING A BITE APPLIANCE
TREATMENT OF THE MPD PATIENT Phase III (4-6 Wks)
CONTINUE HOME THERAPY AND MEDICATIONS
RE-EVALUATE THE BITE APPLIANCE
INITIATE PHYSICAL THERAPY
TREATMENT OF THE MPD PATIENT Phase IV
RE-EVALUATE DIAGNOSIS AND COMPLIANCE
DETERMINE THE NEED FOR CONSULTATION
PSYCHOLOGICAL COUNSELING
REFERRAL TO A PAIN CENTER
oral devices that have been fitted over the occlusal surfaces of teeth for the treatment of different masticatory disorders
oral orthotic
occlusal/bite splint
occlusal/bite guard
night guard
deprogrammer
modes of action
use of appliances will induce muscle relaxation, and usually makes it possible to record centric relation clinically by jaw manipulation to produce stable records

some beneficial effects of the occlusal splint may be psychologic and based on congnitive awareness,

allows for optimal positioning of the condyles and discourages bruxism through elimination of the habitual dysfunctional patterns of the teeth
How do they work?
Stabilizing Occlusal Splints Indications
1. patients with tmj or muscle disorders such as bruxism
2. Diagnosis and treatment of trauma from occlusion 1
3. Establishment of the optimal condylar position in centric relation prior to definitive occlusal therapy
4. Stabilization of mobile teeth by mainting them in stable position
5. Holding maxillary teeth in the desired position following orthodontic therapy or loss of opposing teeth
6. temporary disocclision of teeth for orthodontic or other purpose
7. differential diagnosis in patients with signs and symptoms imitating TMJ or muscle
8. treatment of headaches caused by neurousclar tension
Full Coverage: Advantages
1. worn continuously w/o tooth movement
2. good for bruxism, even if uncontrolled
3. can be worn on maxillary of mandibular teeth
Full Coverage: Disadvantages
1. longer adjustment period
2. patients with jaw pain and manipulation during adjustment difficult
3. commercial lab fee
occlusal scheme full coverage appliance
stabilizing splint with stops for all opposing teeth and controlled cuspid rise
Partial Coverage: Advantages
Good diagnostic device
Easy adjustment and more acceptable to patients with jaw pain
Lack of bulk makes it easier to wear
Partial Coverage: Disadvantages
Should not be worn for extended periods of time
Less acceptable by patient because palatal coverage is necessary
Functional Activities: Force of contact
17,200 lb-sec/day
Functional Activities: Direction of Force to teeth
Vertical-tolerated
Functional Activities: Mandibular position
C.O.-stable
Functional Activities: Protective relfexes influence
present
Functional Activities: Pathology
unlikely
Para-Functional Activities: Force of Contact
57,600 lb-sec/day
Para-Functional Activities: Direction of force to teeth
horizontal-not tolerated
Para-Functional Activities: Mandibular position
Eccentric-unstable
Para-Functional Activities: Protective reflexes influence
absent
Para-Functional Activities: Pathology
highly likely
involuntary activity of the jaw musclature characterised by parafunctional activities such as jae clencing, tooth gnashing, and grinding
bruxism
Awake Bruxism
Unrelated to sleep bruxism
•Ususally occurs subconsciouly without cognitive awareness, however, it is under voluntary control
•Protective reflexes intact
•Often occurs during periods of concentration , exertion, stress, anxiety of grinding
•May accompany other types of parafunctional oral habits such as cheek biting, tongue posturing, nail biting, lip biting, jaw bracing or posturing
•20% are aware of at least occasional daytime clenching
Sleep Bruxism
•(International Classifacation of Sleep Disorders)
ICSD -2 (2005) : Sleep bruxism was re-classified as a sleep-related movement disorder (relatively simple, usually stereotyped movements that disturb sleep and are less complex that other parasomnias)

•An oral activity characterized by grinding or clenching of the teeth during sleep
•Usually associated with microarousals
• Often seen in lighter stages of sleep
• Change in sympathetic-parasympathetic activity
• Tonic muscle contractions = clenching
• Phasic, repetitive muscle contractions = grinding
•Not under voluntary control
•Protective reflexes decreased
Sleep Bruxism Etiology
The most accepted theory of SB is that it is a movement disorder involving a cascade of physiological events characterized by autonomic-cardiac activities as related to sleep arousal.
NREM Bruxism
Stage 1: Transitional lighter sleep
•Stage 2: Deeper sleep
•Stage 3: Slow wave activity
•Stage 4: Slow wave activity
•Stages 3 and 4 are referred to as slow-wave sleep or delta sleep
•Parasympathetic nervous system predominates. Hormonal changes to promote anabolism. Protein synthesis and tissue repair. Restorative period
REM Bruxism
•“Dream” stage
•Sympathetic nervous system predominates
•– Increased oxygen consumption
•– Increased BP, pulse, respirations, CO
•Increased chance of plaque rupture due to surges in pressure and changes in coronary artery tone
Bruxism: Diagnosis Based on History
Self Awareness
•Report of witnessed sounds of tooth grinding or tapping
•Jaw discomfort, fatigue or stiffness in the morning
•Morning headache
•Generalized sensitivity of the teeth to cold
Bruxism: Diagnosis Based on Clinical Findings
•Tooth wear (100% of bruxers; 40% of nonbruxers)
•Masseter muscle hypertrophy
•Mandibular angle hypertrophy on panoramic radiograph
•Masticatory muscle tenderness to palpation
•TMJ tenderness to palpation
•Maxillary exostosis and mandibular tori
•Other evidence of parafunctional jaw activity (tongue scalloping/buccal mucosal lesions)
2 types of Sleep Bruxism
1.Idiopathic: without clear cause
2.Secondary Bruxism: Associated with an identifiable cause; may also occur while awake
2 types of Secondary Bruxism
a)Drug induced (alcohol, caffeine, cigarettes, SSRI, cocaine, ecstacy)
b)Medical disorders (cerebral palsy, Parkinson’s disease, mental retardation, autism)
Evidence of Bruxism
1. excessive tooth wear
2. fractured tooth structure or restoration
3. tooth sensitivity
4. tooth mobility
5. pulp necrosis
6. abfractions
7. alveolar exostosis and tori
8. periodontal bone loss
9. masticatory muscle hypertrophy
10. muscle pain and headache
11. traumatic ulcer
12. linea alba and tongue indentation
Management of Bruxism
Identify the cause and work toward reducing factors

behavioral modifications
occlusal appliances
pharmacology
Behavioral Modifications
1.Avoidance of known triggers (smoking, caffeine, SSRIs)
2.Stress management (hypnosis, biofeedback)
3.Sleep Hygiene education
4.Change sleep position
Occlusal Splints-management
Comprehensive reviews by Dao et al and Kato et al conclude that splints are useful adjuncts in the management of sleep bruxism, but are NOT DEFINITIVE treatment
PHARMACOTHERAPY
The short term use of pharmacotherapy aimed at reducing sleep onset, increasing sleep continuity and total sleep time has been helpful in managing sleep disturbances, including bruxism.

1) muscle relaxants ( Methocarbamol; Robaxin)
2)Benzodiazepines ( Valium)
3)low dose of tricyclic antidepressants