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49 Cards in this Set
- Front
- Back
Saccadic eye movement allows for? relative eye speed versus others?
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rapid change of eye position. fastest in terms of speed, eye movement.
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Classifications of saccades
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1. volitional saccades, voluntary saccades.
2. reflexive saccades 3. Spontaneous saccades 4. Quick phase of nystagmus 5. rapid eye movement sleep |
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Overview of saccades generation
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Cortex - command to make saccade
Brainstem - generates pulse and steps signals EOMS - make the actual movement |
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Main sequence is the ____ portion of the data/curve
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linear portion
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velocity amplitude relationship
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saccadic speed must be faster with longer duration
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Duration amplitude relationship
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larger saccades require more time.
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Static hypometria
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step size error, static undershoot, corrected by a corrective saccade, occurs more in periphery.
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Static hypermetria
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step size error, signal is too large, static overshoot. occurs more often in central.
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glassadic hypometria
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error in pulse signal, glassadic undershoot, corrected by glissade
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glassadic hypermetria
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error in pulse signal, glassadic overshoot, corrected by glissade.
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Combined dysmetria
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pulse and step problem
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If step size error then corrected by a?
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corrective saccade
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If pulse size error then corrected by a?
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Glassadie
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Sampled data model
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Once programmed saccade, saccade cannot be modified
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Continuous sampling model
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saccade can be modified up to 70 msec before saccade occurs.
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Saccadic omission
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intervention to prevent blurring of vision during a saccade
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Saccadic suppression
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reduction in visual sensitivity during saccadic eye movement.
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Saccadic masking
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reduction in ability to detect blur during saccadic eye movements.
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Higher visual levels of saccades include?
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frontal cortex, parietal cortex, thalamus, superior colliculus and basal ganglia.
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Premotor neurons generate?
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pulse and step.
From burst neurons 2 classes excitatory burst neurons, generate pulse signal. inhibitory burst neurons, inhibit antagonist EOMs. |
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excitatory burst neurons for horizontal saccades are located in the?
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PPRF, paramedian pontine reticular formation
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inhibitory burst neurons for horizontal saccades are located in the?
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rostral medulla
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Burst neurons for vertical and torsional saccades are in the
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riMLF,
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Tonic neurons for horizontal saccades
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carry the step signal, and are the neural integrates which integrate burst signals to step signals. Nucleus prepositus hypoglossi (NPH) and medial vestibular nucleus (MVN).
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Tonic neurons for vertical or torsional saccades
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INC or interstitial nucleus of cajal, integrates burst signal from excitatory burst neurons in the riMLF to step signals.
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Pause neurons
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discharge in the absence of saccades but stop discharging before and during saccades. pause to allow saccades to occur. Also stop firing during blinks. located in the nucleus raphe interpositus.
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Cerebellum plays a role in saccades?
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control of saccadic accuracy.
Dorsal vermis and fastigial nucleus - saccadic amplitude. flocculus calibrates the saccadic pulse-step match. |
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Pursuits are generated to? And are evolved from?
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follow or track a moving target. evolved from OKN.
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Pursuit latency
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100-150 msec
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OKN latency
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70 msec
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VOR latency
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16 msec
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Saccadic latency
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200 msec
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Ramp target stimulus of pursuits?
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100 msec latency, as eye begins to move there is a pursuit that lags behind and is corrected by a catch up saccade. The pursuit then become accurate.
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Step-ramp target stimulus of pursuits?
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150 msec latency, as eye begins to move there is a pursuit that becomes increasingly accurate as the target and eye movments coincide.
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Initial stage of pursuits are considered?
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open loop, first 100 msec of pursuits, does not recieve feedback to modify its performance, this is the intrinsic property of the pursuit system.
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2nd stage of pursuits are considered?
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closed loop, remainder of pursuits, able to receive feedback and modify its performance.
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Pursuit gain, up to 40 deg/sec
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nice and smooth
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pursuit gain, 40-100 deg/sec
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saccadic intrusion, target moving too fast or in unpredictable path.
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Pursuit gain, above 100 deg/sec
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jerky saccadic pursuit
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Neurophysiology of pursuits
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retina -> LGN ->(motion info) Middle temporal, Medial superior temporal, and posterior parietal -> Dorsolateral pontine nuclei, accesory optic system, nucleus of optic tract, cerebellum, flocculus and dorsal vermis synthesis pursuit signal -> neural integrators -> final pathways.
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Lesions on horizontal pursuits, Primary visual cortex
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Contralateral field defect, no vision or pursuit occurs
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Lesions on horizontal pursuits, Middle temporal
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contralateral field, does not see or percieve motion well.
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lesions on horizontal pursuits, medial superior temporal
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impairs pursuits for targets moving toward size of lesion, ipsilateral.
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lesions on horizontal pursuits, posterior parietal
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contralateral inattention
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lesions on horizontal pursuits, frontal eye frield
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ipsilateral defect
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lesions on horizontal pursuits, dorsolateral pontine nucleus
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ipsilateral defect
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lesions on horizontal pursuits, cerebellum
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ipsilateral defect.
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Center for horizontal conjugate eye movements is?
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abducens nucleus. recieves information from PPRF (excitatory burst neurons), NPH, MVN (step signal) and sends information via MLF to contralateral ocularmotor nucleus.
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ultimate cerebellum ensure that?
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all classes of eye movement are calibrated.
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